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1 ry as analogues of spiroiminodihydantoin and guanidinohydantoin.
2 -deoxynucleoside-5'-triphosphate (dSpTP) and guanidinohydantoin-2'-deoxynucleoside-5'-triphosphate (d
3           The chemical oxidation resulted in guanidinohydantoin and spiroiminodihydantoin rearrangeme
4 yribonucleosides of spiroiminodihydantoin, 5-guanidinohydantoin, and oxazolone resulting from H2O as
5 nct substrate lesions, 5-hydroxycytosine and guanidinohydantoin, are more efficiently removed by the
6 oxidation of 2'-deoxyguanosine (dG) yields 5-guanidinohydantoin (dGh) as a product.
7 uanine (dOG), spiroiminodihydantoin (dSp), 5-guanidinohydantoin (dGh), oxazolone (dZ), 5-carboxamido-
8 ation products include potentially mutagenic guanidinohydantoin (Gh) and spiroiminodihydantoin (Sp) b
9 r ability to remove oxidized guanine lesions guanidinohydantoin (Gh) and spiroiminodihydantoin (Sp) f
10 droxy-7,9-dihydropurine-6,8-dione (5-OH-OG), guanidinohydantoin (Gh) and spiroiminodihydantoin (Sp) f
11 ptibility to further oxidation that produces guanidinohydantoin (Gh) and spiroiminodihydantoin (Sp) l
12 to secondary oxidation products, such as the guanidinohydantoin (Gh) and spiroiminodihydantoin (Sp) n
13 e lesions 8-oxo-7,8-dihydroguanine (8-oxoG), guanidinohydantoin (Gh) and spiroiminodihydantoin (Sp) r
14 to the formation of hydantoins, specifically guanidinohydantoin (Gh) and spiroiminodihydantoin (Sp),
15 ent work has identified two new DNA lesions, guanidinohydantoin (Gh) and spiroiminodihydantoin (Sp),
16        The four-electron oxidation products, guanidinohydantoin (Gh) and spiroiminodihydantoin (Sp),
17 ty of oxidants yields the hydantoin lesions, guanidinohydantoin (Gh) and spiroiminodihydantoin (Sp).
18 ro to form two secondary oxidation products, guanidinohydantoin (Gh) and spiroiminodihydantoin (Sp).
19 s further oxidation products, the hydantoins guanidinohydantoin (Gh) and spiroiminodihydantoin (Sp).
20             Of these, the hydantoin lesions, guanidinohydantoin (Gh) and the two diastereomers of spi
21 -2'-deoxyguanine (8-oxoG) oxidation products guanidinohydantoin (Gh) and the two stable stereoisomers
22  of synthesis, the oxidation products of OG, guanidinohydantoin (Gh) and/or its isomer iminoallantoin
23 in products spiroiminodihydantoin (Sp) and 5-guanidinohydantoin (Gh) in a sharply pH-dependent fashio
24 rther oxidation of 8-oxoG can produce either guanidinohydantoin (Gh) in duplex DNA or spiroiminodihyd
25 e genotoxic spiroiminodihydantoin (Sp) and 5-guanidinohydantoin (Gh) lesions.
26 xidized purines, spiroiminodihydantoin (Sp), guanidinohydantoin (Gh), 2,6-diamino-4-hydroxy-5-formami
27 f DNA lesions, such as 8-oxoguanine (8OG), 5-guanidinohydantoin (Gh), and spiroiminodihydantoin (Sp).
28 col (Tg), 8-oxo-7,8-dihydroguanine (8-oxoG), guanidinohydantoin (Gh), or spiroiminodihydantoin (Sp) c
29 ity of MutY on the 8-oxoG oxidation products guanidinohydantoin (Gh), two diastereomers of spiroimino
30 e, among them spiroiminodihydantoin (Sp) and guanidinohydantoin (Gh).
31 nome, whereas editing enhances repair of the guanidinohydantoin lesion by NEIL1.
32 te that an oxidized form of 8-oxoG, possibly guanidinohydantoin, may direct misreading and misinserti
33 the 8-oxoG site, resulting in formation of a guanidinohydantoin moiety as the major product.