ライフサイエンスコーパス: gut lumen

1 l output to the microbial environment of the gut lumen.

2 g in elevated levels of free stearate in the gut lumen.

3 as a free apical surface in contact with the gut lumen.

4 as microorganisms and metabolites within the gut lumen.

5 ies increase the levels of vitamin B6 in the gut lumen.

6 posed of myeloid cells extravasated into the gut lumen.

7 le TG2 when the protein is perfused into the gut lumen.

8 nd molecular oxygen from the tissue into the gut lumen.

9 xclusion and strain replacement in the mouse gut lumen.

10 companied by increased formate levels in the gut lumen.

11 substantially elevated lactate levels in the gut lumen.

12 go with nuclei of neighboring cells, and the gut lumen.

13 "blebbing" of the midgut epithelium into the gut lumen.

14 lization dependent on their proximity to the gut lumen.

15 reduced the growth of S. Typhimurium in the gut lumen.

16 estinal tract and aid in absorption from the gut lumen.

17 e non-pharmacological events elicited in the gut lumen.

18 resulting in high fucose availability in the gut lumen.

19 h M-cell-specific transcellular pores to the gut lumen.

20 ion of ovalbumin (OVA) to the blood from the gut lumen.

21 a and other pathogens that also exist in the gut lumen.

22 t markedly reduces their availability in the gut lumen.

23 nsitizing antigens when they reappear in the gut lumen.

24 etic microspheres were administered into the gut lumen.

25 rchenteron cavity actually gives rise to the gut lumen.

26 from the proteinase-rich environment of the gut lumen.

27 nteron lining does not become the definitive gut lumen.

28 migration into the intestinal mucosa and the gut lumen.

29 nd eubacterial-RNA within 14(th) weeks fetal gut lumen.

30 t cells release acetylcholine (ACh) into the gut lumen.

31 ated to facilitate egg extrusion through the gut lumen, a necessary step for parasite transmission.

32 that the worms harbour several taxa in their gut lumen absent from their diets and substrates.

33 be transported to internal tissues from the gut lumen across gut cells that lack SID-1.

34 revealed Helicobacter-like organisms in the gut lumen and attached to intestinal epithelial cells.

35 By facing the gut lumen and being directly connected with alphaSyn-con

36 decreased clearance of C. difficile from the gut lumen and diminished inflammation.

37 by mouse Paneth cells, is secreted into the gut lumen and has bactericidal activity against intestin

38 ial cells interact with both microbes in the gut lumen and host immune cells.

39 unction promotes microbial invasion from the gut lumen and induces the development of intestinal infl

40 unlike these it is secreted into the larval gut lumen and is an active beta1,3-glucanase.

41 sociated with increased endotoxin within the gut lumen and its associated portal circulation.

42 ransform into the procyclic stage within the gut lumen and later migrate to the ectoperitrophic space

43 s Listeria monocytogenes colonization of the gut lumen and prevents systemic dissemination.

44 ormed calcium phosphate nanoparticles in the gut lumen and show how this helps to shape intestinal im

45 , a key element at the interface between the gut lumen and the deeper intestinal layers.

46 biotic degradation and shuttling between the gut lumen and tissue-resident reservoirs can promote the

47 rs, symbiotic microorganisms residing in the gut lumen and/or hemocoelic tissues maintain complicated

48 (despite the immunological inertness of the gut lumen), and (iii) that there are very high benefits

49 VCBPs are secreted into the gut lumen, and direct binding to bacterial surfaces can

50 demonstrates that symbiotic bacteria in the gut lumen appear to function as partners both for symbio

51 in which TG2-gluten complexes formed in the gut lumen are taken up by TG2-specific B cells in GALT.

52 Our results highlight the gut lumen as a primary site of presystemic metabolism of

53 Upon feeding, chitinase is secreted into the gut lumen as an inactive pro-enzyme that is later activa

54 alyses pinpoint recipient migration into the gut lumen as rate-limiting for plasmid transfer dynamics

55 ular B.theta shows a longer lag phase in the gut lumen as well as a slightly slower net growth rate.

56 on by 1 week, beginning patch extrusion into gut lumen at 3 weeks, and total separation of patch with

57 uced in the intestine was transported to the gut lumen at a rate that exceeded transport to plasma by

58 al stimuli induces active DC sampling of the gut lumen at sites distant from organized lymphoid tissu

59 We found that, in the gut lumen, both the mycobiota and fungi present in the d

60 behavior, we developed optogenetics for the gut lumen by engineering a flexible fiberoptic.

61 that sorafenib-glucuronide excreted into the gut lumen can be cleaved by microbial enzymes to sorafen

62 lts in defective IgA transportation into the gut lumen, causing a dramatic increase in serum polymeri

63 e can trigger initial Salmonella Typhimurium gut-lumen colonization in mice, providing insight into t

64 unoglobulin A (IgA) across the mucosa to the gut lumen depends on the epithelial transport protein, p

65 uni establishes its niche and expands in the gut lumen during infection is poorly understood.

66 The gut lumen-dwelling tapeworm Hymenolepis diminuta, unlike

67 Various microbial products leaked from gut lumen exacerbate tissue inflammation and metabolic d

68 onstitutes a physical barrier separating the gut lumen from sterile internal tissues.

69 e peritrophic matrix (PM) that separates the gut lumen from the epithelium.

70 ntestinal epithelial cells (IECs) lining the gut lumen functions as the site of nutrient absorption a

71 mpacted by presystemic metabolism within the gut lumen, gut wall, and liver and how this impact diffe

72 LPS administered enterally was found in the gut lumen in close proximity to the mucosa but was not d

73 omplex communities of bacteria reside in the gut lumen in direct contact with the ingested materials

74 gh concentrations of acetate and acidify the gut lumen in humans and mice, which, in combination, can

75 g presystemic metabolism, notably within the gut lumen, in human exposure and toxicokinetic modeling.

76 he intestine in vivo Uric acid levels in the gut lumen increased in response to exogenous DNA, and th

77 e demonstrate that S.Tm cells re-seeding the gut lumen initiate clonal expansion.

78 ansporter that would transport heme from the gut lumen into intestinal epithelial cells.

79 n the initial propagation of prions from the gut lumen into Peyer's patches.

80 ated molecular patterns translocate from the gut lumen into systemic circulation.

81 translocation of microbial products from the gut lumen into the bloodstream.

82 are required for S. marcescens to escape the gut lumen into the hemocoel, indicating that the flagell

83 ses move through the insect vector, from the gut lumen into the hemolymph or other tissues and finall

84 ic neural circuits respond to changes in the gut lumen is not well understood.

85 ity and low stability, metabolism within the gut lumen is the most remarkable of the three presystemi

86 2-ethylhexyl phthalate (DEHP), for which the gut lumen may even exceed the liver in importance of pre

87 n the gut; increased lipid hydrolysis in the gut lumen, normalization of peripheral unsaturated fatty

88 e dehydrogenase (RyGDH), which acidifies the gut lumen of fed mosquitoes, causing irreversible confor

89 Acid-base status in the hemolymph and gut lumen of insects is generally well regulated but var

90 irochete, Borrelia burgdorferi, inhabits the gut lumen of the tick vector.

91 in their hosts (i.e. as promastigotes in the gut lumen of their sandfly vectors and as amastigotes in

92 ally in acidic pH compartments either in the gut lumen or in vacuoles of the intestinal lining cells.

93 e changes in the bacterial population in the gut lumen or lamina propria that cause inflammation at t

94 an increase polyphenol concentrations in the gut lumen, primarily through the release of bound phenol

95 gs designed to induce autoproteolysis in the gut lumen, prior to toxin uptake, circumventing pathogen

96 e found that commensal bacteria in the mouse gut lumen produced a high concentration of the active re

97 pteran and dipteran insects that have acidic gut lumen, recombinant AtVSP2 significantly delayed deve

98 emic IgG antibodies neutralize toxins in the gut lumen remains unresolved, although it has been sugge

99 we were able to identify either systemic or gut lumen-restricted TGR5 agonists.

100 In the gut lumen, secretory IgA binds pathogens and toxins but

101 e the secretion of compensatory IgM into the gut lumen, sIgAd subjects displayed an altered gut micro

102 This is accompanied by an increase in the gut lumen size and a compromise in the intestine's abili

103 dding of LPS-containing enterocytes into the gut lumen, that has not been previously described.

104 lex life cycles and invasion of vertebrates' gut lumen, the hallmark features of these parasites, evo

105 yphimurium by promoting its outgrowth in the gut lumen through unknown mechanisms.

106 f how prions are initially conveyed from the gut lumen to establish infection on FDC.

107 tion alters the metabolic environment of the gut lumen to favor the outgrowth of the pathogen at the

108 rate mechanical and chemical inputs from the gut lumen to generate complex motor outputs.

109 processes between epithelial cells into the gut lumen to sample pathogens.

110 e monitored microbial translocation from the gut lumen to the bloodstream in the cohort with non-resp

111 ays mood-altering signals originating in the gut lumen to the brain.

112 translocation of the scrapie agent from the gut lumen to the GALT from which neuroinvasion subsequen

113 h TSE agents are initially conveyed from the gut lumen to the GALT is not known.

114 Here we show that fungi migrate from the gut lumen to the pancreas, and that this is implicated i

115 ing force responsible for diffusion from the gut lumen to the tissues of an organism.

116 assage on nutrients/pharmaceuticals from the gut lumen towards the epithelium, whilst preventing it f

117 Nutrient signals from the gut lumen trigger local intestinal reflexes in the enter

118 mutants thrive during initial growth in the gut lumen, where GlpT's capacity to import phosphate is

119 chemicals consumed orally passes through the gut lumen, where it modulates the composition of the gut

120 at undergo substantial metabolism within the gut lumen, where the metabolites have high permeability,

121 cretion of neutral sterols into bile and the gut lumen, whereas G1 transports cholesterol from macrop

122 reactive oxygen species are degraded in the gut lumen, which gives rise to molecular oxygen that sup

123 e of toxic and infectious molecules from the gut lumen while allowing selective paracellular absorpti

124 d the enteroblast/pre-enterocyte reaches the gut lumen with a fully formed brush border.

125 re mainly produced by micro-organisms in the gut lumen, with presumably important alterations in adva

126 motes absorption of monosaccharides from the gut lumen, with resulting induction of de novo hepatic l