ライフサイエンスコーパス: gut microflora

1 ic disease in individuals with a compromised gut microflora.

2 e of cleanliness of the mouse colony and the gut microflora.

3 is believed to be under the influence of the gut microflora.

4 ue between the immune system of the host and gut microflora.

5 th a broad spectrum of antibiotics to reduce gut microflora.

6 nal rabbits and thereby acquired a different gut microflora.

7 rvival, a function dependent on licensing by gut microflora.

8 enged by higher eukaryotes from the diet and gut microflora.

9 lasma isoflavone concentrations and modified gut microflora activities [beta-glucoside hydrolysis and

10 o be completely dependent on the presence of gut microflora and could be established by colonization

11 ed mice showed no significant decline in the gut microflora and developed EAE similar to untreated mi

12 Commensal gut microflora and dietary fiber protect against colonic

13 llus species are normal members of the human gut microflora and most are regarded as safe when admini

14 suggests that changes in the composition of gut microflora and/or deranged epithelial barrier functi

15 apoptosis of intestinal epithelium, changed gut microflora, and elevated ATP.

16 Aberrant immune response and changes in the gut microflora are the main causes of inflammatory bowel

17 t likely due to effects mediated through the gut microflora, bowel transit, and enhancement of gastro

18 We recently described that alteration of the gut microflora can affect a population of Foxp3(+)T(reg)

19 y (MIC = 19/38 muM), selectivity over normal gut microflora, CC(50)s > 606 muM against mammalian cell

20 y of environmental samples such as the human gut microflora, combined with the sustained exponential

21 ronment and with the presence of a different gut microflora compared to mice maintained in SPF facili

22 ns and participate in the development of the gut microflora for infants.

23 pectrum antibiotics-as well as transplanting gut microflora from a protective environment-attenuated

24 microbiome was observed, suggesting that the gut microflora has a direct impact on the drug metabolis

25 Introduction of gut microflora in GF mice rapidly initiated maturation o

26 an significantly affect the growth of select gut microflora in humans, which suggests the potential p

27 on is currently available on the role of the gut microflora in modulating isoflavone bioavailability

28 Additional investigation revealed commensal gut microflora in the mesenteric lymph nodes and elevate

29 to be modulated according to composition of gut microflora, including ingested probiotics.

30 Our results indicate that alteration of the gut microflora is associated with cognitive functioning

31 The human gut microflora is important in regulating host inflammat

32 Loss of immune tolerance to gut microflora is inextricably linked to chronic intesti

33 of drinking water affects the composition of gut microflora, leading to an altered autoimmune respons

34 d with amino acid, energy, purine, lipid and gut microflora metabolisms.

35 Concentrations of isoflavones and their gut microflora metabolites in the plasma, urine, and fec

36 Relationships between gut microflora, morphological and metabolic traits were

37 ved molecular techniques for analysis of the gut microflora, new manufacturing biotechnologies, and i

38 aning (postnatal day 21) on the behavior and gut microflora of 129S6/SvEv mice, a mouse strain common

39 d higher abundance of dominant genera in the gut microflora of group JD.

40 ollectively, these findings suggest that the gut microflora of the honey bee harbours bacterial membe

41 community-based mechanism for protecting the gut microflora, preserving its functional robustness dur

42 The normal gut microflora prevents C. difficile colonization; howev

43 hetaiotaomicron, a predominant member of the gut microflora, revealing a mechanism whereby intestinal

44 ditions, such as circulating glucose levels, gut microflora, time of year, and even diurnal rhythm, w

45 nization of the alimentary tract with normal gut microflora, which induced Ern2 expression.