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1 mutations of psaE and psaF caused defective haemagglutination.
2 inia pseudotuberculosis pH6 antigen mediates haemagglutination and adhesion to cultured mammalian cel
5 (x) ) and ovotransferrin concentrations, and haemagglutination and haemolysis titres) over two annual
6 oxide and ovotransferrin concentrations, and haemagglutination and haemolysis titres), body mass and
7 n of these surface structures, which mediate haemagglutination and have a demonstrated role in virule
9 binding by B. burgdorferi is associated with haemagglutination and we have identified a 26 kDa protei
12 significant inhibition of P fimbria-mediated haemagglutination assay of uropathogenic Escherichia col
15 antibodies that inhibited mannose-resistant haemagglutination by ETEC expressing CFA/I, CS4 and CS14
16 E. coli chi7122 conferred mannose-resistant haemagglutination (HA) and curli production to E. coli H
17 ed that the psaABC genes were sufficient for haemagglutination if they were expressed by a heterologo
18 s formed high levels of A/Cal-specific serum haemagglutination-inhibiting antibodies and were solidly
19 HD-MAP administration of 2.5 mug HA induced haemagglutination inhibition (HAI) and microneutralisati
23 ndividuals in Guangzhou, China, using 67,683 haemagglutination inhibition (HI) assay measurements aga
25 nza infection or vaccination and fit them to haemagglutination inhibition (HI) titres from 5 groups o
26 In the baseline serum samples from 2008, haemagglutination inhibition and microneutralisation ant
28 tal transfer of antibodies by measurement of haemagglutination inhibition and microneutralization tit
29 traditional antigenic characterization using haemagglutination inhibition and promote selection of HA
31 fficacy of high-titre anti-influenza plasma (haemagglutination inhibition antibody titre >=1:80) comp
32 t-boosted regression model trained on annual haemagglutination inhibition antibody titre inputs, we i
33 this influenza nanoparticle vaccine elicited haemagglutination inhibition antibody titres more than t
34 y and efficacy of anti-influenza plasma with haemagglutination inhibition antibody titres of 1:80 or
38 and compared the proportion of samples with haemagglutination inhibition titre 1:32 or more (deemed
40 difference in the proportion of samples with haemagglutination inhibition titre equal to or above 1:3
43 he placebo group had a four-fold increase in haemagglutination inhibition titres (group geometric mea
44 ted participants had a four-fold increase in haemagglutination inhibition titres (group geometric mea
45 hIVIG treatment produced a robust rise in haemagglutination inhibition titres against influenza A
50 r, and one person of eleven seroconverted by haemagglutination inhibition, microneutralisation, H5N3
51 ccine gave the highest seroconversion rates: haemagglutination inhibition, six of ten; microneutralis
52 unogenicity endpoints were seroconversion by haemagglutination-inhibition (HAI), defined as a four-ti
53 come was the proportion of participants with haemagglutination-inhibition (HI) geometric mean titre (
54 subsequent immunisation were evaluated using haemagglutination-inhibition and microneutralisation ass
57 ed influenza vaccine induces moderate-to-low haemagglutination-inhibition antibody responses in peopl
60 vaccinated with LAIV H5N2 had an increase in haemagglutination-inhibition titre of greater than four-
61 ants in the vaccine group had an increase in haemagglutination-inhibition titre of more than four-fol
65 by the organism which are biofilm formation, haemagglutination properties and capsule production.
66 strains exhibited a strong mannose-sensitive haemagglutination reaction with guinea pig erythrocytes,
68 ctive combined serology (positive T pallidum haemagglutination test and rapid plasmin reagin titre of