ライフサイエンスコーパス: haematoma

1 tex by immunoblotting at 30 min and 4 h post-haematoma.

2 atoma and this persisted at 2 h and 4 h post-haematoma.

3 h following production of an acute subdural haematoma.

4 shaped and semi-erect ears may trigger aural haematoma.

5 carriage also showed increased odds of aural haematoma.

6 ble to induce the complete lysis of the 5 ml haematoma.

7 leading to the formation of an intracerebral haematoma.

8 procedure for drainage of a chronic subdural haematoma.

9 individuals, 5.4% of whom had a parenchymal haematoma.

10 e expense of adverse events such as subdural haematoma.

11 nd they all target reducing expansion of the haematoma.

12 from local vasculature or recruitment to the haematoma.

13 Trial of dexamethasone for chronic subdural haematoma.

14 ial conservative treatment in acute subdural haematoma.

15 patients with a symptomatic chronic subdural haematoma.

16 en for post-operative complications, such as haematoma.

17 egion which was compatible with intraorbital haematoma.

18 ny, are associated with presence of subdural haematoma.

19 n the areas identified during standard US as haematomas.

20 id collection in the operation site, two had haematoma, 10 had an abscess, and five had a pancreatic

21 pain, reduced lower limb motion, or visible haematoma; 11 patients had also anaemia (haemoglobin < 7

22 nderlying traumatic brain injury or subdural haematoma (4.4 [1.4-14.0]), a Glasgow Coma Scale score o

23 sults indicate that following acute subdural haematoma, a rapid cellular redistribution of apoE occur

24 g to treatment preference for acute subdural haematoma (acute surgical evacuation or initial conserva

25 enal injury by differentiating between mural haematoma and a duodenal perforation because the latter

26 On post operative day 6, an infected haematoma and an area of proximal skin necrosis were sur

27 standing that they enhance resorption of the haematoma and reduce neuroinflammation.

28 Mortality at 90 days and risk of parenchymal haematoma and symptomatic intracranial haemorrhage did n

29 examination, the average difference between haematoma and the renal cortex was 5 dB.

30 hroughout the cortical layers underlying the haematoma and this persisted at 2 h and 4 h post-haemato

31 Duodenal wall haematoma and traumatic duodenal perforation causing pne

32 rious adverse events related to kyphoplasty (haematoma and urinary tract infection); other serious ad

33 hom 1407 (31%) presented with acute subdural haematoma and were included in our study.

34 ssibility of a persistent idiopathic scrotal haematoma and/or haematoma secondary to a trauma of the

35 taxis); one receiving placebo (infusion site haematoma), and four during dabigatran etexilate pretrea

36 o study the natural history of such subdural haematomas; and to ascertain which obstetric factors, if

37 Subdural haematomas are thought to be uncommon in babies born at

38 A blood-fluid level within the haematoma, as revealed by computed tomography (CT), has

39 Iron deposition appeared on day 3 around the haematoma border but did not change synchronously with A

40 n the cortical levels of apoE at 30 min post-haematoma but, at 4 h post-haematoma, there was a signif

41 Intraorbital haematoma can be managed by conservative approach withou

42 The most severe form, parenchymal haematoma, can result in neurological deterioration, dis

43 Chronic subdural haematoma causes serious morbidity and mortality.

44 njury, increased blood alkaline phosphatase, haematoma, colitis, gastric obstruction, lung infection,

45 the levels of apoE in cortex underlying the haematoma compared to control levels.

46 s and 20 breeds showed reduced odds of aural haematoma compared with crossbred dogs.

47 difference in the echogenicity of perirenal haematomas compared to the routine examination in B pres

48 Aortic dissection and intramural haematoma comprise an aortopathy involving separation of

49 n reveals intracranial abnormalities such as haematomas, contusions and cerebral oedema.

50 Dogs diagnosed with aural haematoma during 2016 were identified from the VetCompas

51 were 2,249/905,554 dogs diagnosed with aural haematoma during 2016.

52 We experienced a case of intraorbital haematoma during a commercial flight.

53 Early surgery combined haematoma evacuation (within 24 h of randomisation) with

54 monstrate that O2L-001 provides new hope for haematoma evacuation and the treatment of patients with

55 ed tPA (OptPA), offers improved efficacy for haematoma evacuation as well as improved safety.

56 Open craniotomy haematoma evacuation has not been found to have any bene

57 in 27 countries, we compared early surgical haematoma evacuation within 12 h of randomisation plus m

58 utcome was 45% (95% CI 40% to 50%) and after haematoma evacuation, case fatality was 34% (95% CI 30%

59 e in patients treated conservatively or with haematoma evacuation.

60 treatment and 24% (95% CI 19% to 29%) after haematoma evacuation.

61 A total of 1171 patients (57%) underwent haematoma evacuation.

62 than 2 years (normal mental status, no scalp haematoma except frontal, no loss of consciousness or lo

63 on models, prior APT was not associated with haematoma expansion (OR, 0.97; 95% CI 0.60 to 1.57), maj

64 Haematoma expansion affects a fifth of patients within 2

65 eta-analysis to compare baseline ICH volume, haematoma expansion and clinical outcomes between NOAC-I

66 plex and encompasses mechanical mass effect, haematoma expansion and secondary injury.

67 ains a medical emergency, with prevention of haematoma expansion as the key therapeutic target.

68 fect of intensive BP lowering on significant haematoma expansion at 24 hours warrants further investi

69 The primary outcome was haematoma expansion at 72 hours.

70 lowering and haemostatic treatment minimise haematoma expansion but have not led to improved functio

71 Haematoma expansion is a major cause of mortality in int

72 Good haemostatic efficacy, defined as haematoma expansion of <=35% or <=6 mL, was the primary

73 with ICH, prior APT was not associated with haematoma expansion or functional outcomes after ICH, re

74 and at 3 months, as well as ICH volumes and haematoma expansion rates in the two groups.

75 The rates of haematoma expansion were comparable in NOAC-ICH versus V

76 s were excellent haemostatic efficacy (<=20% haematoma expansion) and good functional outcome (modifi

77 five (19%) of 27 in the PCC group (none from haematoma expansion), the first of which occurred on day

78 ight (35%) of 23 in the FFP group (five from haematoma expansion, all occurring within 48 h after sym

79 rast CT features are promising predictors of haematoma expansion, but their potential contribution to

80 Our results confirm that ICH volume, haematoma expansion, mortality and functional outcome ap

81 activated factor VII (eptacog alfa) reduced haematoma expansion, mortality, and disability when give

82 were judged to be FFP related (four cases of haematoma expansion, one anaphylactic reaction, and one

83 Complications of ICH include haematoma expansion, perihaematomal oedema with increase

84 s are each associated with increased risk of haematoma expansion.

85 trials targeting patients at highest risk of haematoma expansion.

86 isation seemed to be associated with smaller haematoma expansion.

87 associated with a higher risk of parenchymal haematoma following recombinant tissue-plasminogen activ

88 ations associated with a risk of parenchymal haematoma following thrombolytic therapy in patients wit

89 d 18 years and older with a chronic subdural haematoma for burr-hole drainage were assessed for eligi

90 Therefore, in a patient with acute subdural haematoma for whom a neurosurgeon sees no clear superior

91 e, ventricular volume, volume of extra-axial haematomas) from CT scans were measured and correlated w

92 ere absolute (>6 mL) and proportional (>33%) haematoma growth at 24 hours.

93 =0.09) or absolute (0.84, 0.68-1.04; p=0.12) haematoma growth compared with guidelines treatment.

94 effects on functional recovery and relative haematoma growth decreased with increasing time from ons

95 hrombotic therapy is associated with greater haematoma growth, which may be reduced by early intensiv

96 justed OR 0.82, 95%CI 0.68 to 0.99; p=0.034) haematoma growth.

97 justed OR 0.82, 95%CI 0.68 to 0.99; p=0.034) haematoma growth.

98 nctional recovery, without a clear effect on haematoma growth.

99 All were rescanned at 4 weeks and haematomas had completely resolved.

100 s, including pneumothorax in 28 patients and haematoma in 3 cases.

101 ate the incidence and risk factors for aural haematoma in dogs under primary veterinary care in the U

102 of neurological impairment, and site of the haematoma in the brain.

103 ), evacuation of a supratentorial extradural haematoma in the medium HDI tier (189 [31%]) and high HD

104 vacuation of a supratentorial acute subdural haematoma in the very high HDI tier (155 [47%]).

105 troke that is defined by the occurrence of a haematoma in the wall of an intracranial artery.

106 s the echogenicity and the size of perirenal haematomas in patients after kidney transplant during ro

107 ed a more detailed assessment of the size of haematomas in the perirenal space that appeared during e

108 Following the haematoma induction, apoE immunoreactivity was dramatica

109 nt each with grade 3 postoperative wound and haematoma infection, grade 4 hypertensive encephalopathy

110 At 30 min post-haematoma, intense apoE staining was observed in cluster

111 of pearls appearance, concentric intramural haematoma, intimal flap (the most definite sign), and do

112 ng logistic regression, and (2) of increased haematoma+intraventricular haemorrhage volume (IVH) with

113 Intraorbital haematoma is a rare clinical entity which can be caused

114 d, acute surgery in traumatic acute subdural haematoma is based on low-grade evidence.

115 after burr-hole drainage of chronic subdural haematoma is safe and associated with reduced recurrence

116 he interpretation of the images of perirenal haematoma is their ability to change in time.

117 Intensive BP lowering reduced haematoma+/-IVH growth by 4.7/7.1 mL in patients on anti

118 ollow-up CT after 24 h, absolute increase in haematoma+/-IVH volume was larger (5.2/5.0 mL) in those

119 n of O2L-001 at 1 mg/ml into the core of the haematoma led to a 44% increase in thrombolysis compared

120 pital admission costs, which did not vary by haematoma location (p=0.90).

121 p with other ICH outcomes when stratified by haematoma location or study cohort.

122 Haematoma location was classified as lobar or non-lobar.

123 mentia and hospital care costs stratified by haematoma location.

124 functional outcomes after ICH, regardless of haematoma location.

125 Additionally, at 4 h post-haematoma marked apoE staining of discrete foci within t

126 Ex vivo haematoma models using human blood were used to demonstr

127 a [n=2], bone marrow failure [n=1], subdural haematoma [n=1], and intracranial haemorrhage [n=1]) and

128 e neocortex (13 cases, 37%) and intracranial haematoma (nine cases, 26%).

129 In clinical sized haematomas (obtained from 30 ml of human blood), a singl

130 Subdural haematomas occurred in two patients.

131 There were also fewer haematomas of any severity in the stenting group than in

132 1.03; p=0.04), the presence of intracerebral haematoma on initial CT scan (OR 2.0, 95% CI 1.2 to 3.5;

133 nal within the vessel wall due to intramural haematoma on T1 fat-saturated images.

134 he 1.5% twice daily group developed subdural haematoma; one patient in the 1.5% once daily group had

135 symptoms include gastrointestinal bleeding, haematomas or haemarthroses.

136 significant difference in the development of haematomas or seromas after surgery but the non-warmed g

137 polymorphism was associated with parenchymal haematoma (P = 0.01), and the overall association after

138 of the aorta: aortic dissection, intramural haematoma, penetrating aortic ulcer and other less commo

139 l hypotension, ischaemic stroke, retroclival haematoma, pituitary apoplexy, third ventricle colloid c

140 bital exploration and surgical evacuation of haematoma remains a second line intervention.

141 At the first month follow-up, intraorbital haematoma resolved without significant sequelae.

142 rsistent idiopathic scrotal haematoma and/or haematoma secondary to a trauma of the inguino-scrotal r

143 ), rates of wound complications (dehiscence, haematoma, seroma, bleeding, bruising), length of stay i

144 alysis, each copy of APOE epsilon2 increased haematoma size by a mean of 5.3 mL (95% CI 4.7-5.9; p=0.

145 tional outcome following ICH is dependent on haematoma size, with only patients with smaller haemorrh

146 ive bleeding from the ruptured aneurysm with haematoma spreading into the right retroperitoneum.

147 carriage within breeds and the risk of aural haematoma suggest that trauma along the line of cartilag

148 oE at 30 min post-haematoma but, at 4 h post-haematoma, there was a significant elevation (27%, P < 0

149 lasminogen activator; cases were parenchymal haematoma type 1 or 2 as defined by the European Coopera

150 20 (2%) versus 15 (2%) patients, parenchymal haematoma type 2 occurred in 37 (4%) versus 26 (3%) pati

151 parenchyma, who were suspected of perirenal haematoma, underwent a CE-US examination after intraveno

152 the thrombolytic agent into the core of the haematoma via a minimally invasive procedure.

153 y (OR, 0.89; 95% CI 0.47 to 1.85), admission haematoma volume (beta, -0.17; SE, 0.09; p=0.07) and shi

154 age was 61 years (IQR 51-68), and the median haematoma volume 57 mL (IQR 44-74).

155 Admission Glasgow Coma Scale, increasing haematoma volume and cortical involvement were associate

156 Baseline haematoma volume and gender appear to influence PHE grow

157 ssion to establish the effect of genotype on haematoma volume and logistic regression to assess the e

158 associate with severity of ICH, in terms of haematoma volume at presentation and subsequent outcome.

159 Ventricular volume and extra-axial haematoma volume did not correlate significantly with op

160 ive (>=33%) and absolute (>=6 mL) changes in haematoma volume from baseline to 24 h.

161 e relationship with PHE growth, but baseline haematoma volume had a direct correlation.

162 analyses, APT was associated with admission haematoma volume in lobar ICH (beta, 0.25; SE, 0.12; p=0

163 At baseline, median haematoma volume was 15.0 mL (IQR 7.9-29.2) with median

164 Secondary outcomes were admission haematoma volume, all-cause mortality, death or major di

165 ns of the HP CNV genotype and rs2000999 with haematoma volume, perihaematomal oedema (PHO) volume, fu

166 sion models adjusting for trial and baseline haematoma volume.

167 APT was associated with admission haematoma volumes in lobar ICH.

168 estimated one-year incidence risk for aural haematoma was 0.25% (95% confidence interval 0.24-0.26).

169 translational significance, a clinical sized haematoma was used to ensure catheter placement and to a

170 In six patients, the size of haematomas was comparable using both techniques, whereas

171 Haematomas were supratentorial and deep in most (170 [63

172 developed aortic dissections and intramural haematomas when stimulated with angiotensin II.

173 rction, cranial nerve palsy, and access site haematoma with endarterectomy.

174 e were observed in the cortex underlying the haematoma with minimal damage observed in shams.

175 t treatment for patients with acute subdural haematoma with similar characteristics differed dependin

176 disorders who presented with acute subdural haematoma within 24 h of traumatic brain injury.