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1 mply additive for the central and peripheral haemodynamics.
2 proBNP >= 125 ng/L) and controls with normal haemodynamics.
3 minantly female and had severely compromised haemodynamics.
4 t more symptomatic than patients with normal haemodynamics.
5 els in the microvasculature, affecting local haemodynamics.
6 used to identify key parameters representing haemodynamics.
7  to metabolism, cardiovascular structure and haemodynamics.
8 of tissue layers with optical information on haemodynamics.
9 te to sex differences in LV mechanics and LV haemodynamics.
10 relations were present for ADRB2 density and haemodynamics.
11 e brain lesions, sleep patterns and cerebral haemodynamics.
12 known of underlying changes in microvascular haemodynamics.
13 al input or of standard neural predictors of haemodynamics.
14 s on systemic microvascular permeability and haemodynamics.
15 ctroscopy (NIRS) device to record prefrontal haemodynamics.
16 may contribute to age-related impairments in haemodynamics.
17 actin and prepared for recording of ARNA and haemodynamics.
18 elling offers a valuable tool for simulating haemodynamics.
19  fundamentally alters central and peripheral haemodynamics.
20 hensive subject-specific analysis of central haemodynamics.
21 bling the non-invasive in vivo assessment of haemodynamics across various vascular territories of the
22 linical studies, apelin regulates glomerular haemodynamics and acts on the tubule to promote aquaresi
23 amics (CFD) can be used to simulate vascular haemodynamics and analyse potential treatment options.
24 oft robotic aortic sleeve that can mimic the haemodynamics and biomechanics of aortic stenosis.
25 ulation of tubule sodium reabsorption, renal haemodynamics and blood pressure.
26 erstanding of the interplay between abnormal haemodynamics and cardiovascular pathologies, promising
27 re (ICP) is associated with altered cerebral haemodynamics and cephalic pain.
28                              Skeletal muscle haemodynamics and circulating adenosine triphosphate (AT
29 ical leaflet thrombosis and subsequent valve haemodynamics and clinical outcomes on the basis of two
30 opportunity for further improvement in valve haemodynamics and clinical outcomes.
31 tionwide evaluation of the role of pulmonary haemodynamics and comorbidity in predicting survival amo
32 resistance artery function, improve exercise haemodynamics and enhance exercise performance compared
33 PHT) on resistance artery function, exercise haemodynamics and exercise performance relative to knee
34 s epoprostenol (prostacyclin, PGI2) improves haemodynamics and exercise tolerance, and prolongs survi
35  permit remote assessment of cardiopulmonary haemodynamics and facilitate early intervention that has
36  OA excitations, and of stimulus-evoked slow haemodynamics and fast calcium activity in the presence
37        However, they do not recapitulate the haemodynamics and flow patterns associated with the dise
38 me per hour with close monitoring of patient haemodynamics and fluid balance.
39 s suggest that EIAH has no effect on central haemodynamics and is likely to be a consequence of pulmo
40 rent understanding of the link between brain haemodynamics and local neuronal activity.
41  activity and whole-brain macroscopy of slow haemodynamics and metabolism.
42                                              Haemodynamics and muscle sympathetic nerve activity (MSN
43 ns in aortic regurgitation, favourable valve haemodynamics and myocardial remodelling, with associate
44 ce at 6 months of age, by measuring cerebral haemodynamics and neural activity to physiological senso
45 piratory and cardiovascular variables, renal haemodynamics and renal function were recorded while the
46 ventricular function and had no influence on haemodynamics and respiratory.
47 art failure have shown beneficial effects on haemodynamics and symptoms.
48       Catheterisation is necessary to assess haemodynamics and to evaluate vasoreactivity during acut
49 stent with a muscle pump effect on capillary haemodynamics), and (2) there would be a dynamic relatio
50  lamb1a mutants exhibit hallmarks of altered haemodynamics, and blocking cardiac contractility in lam
51  shock epidemiology, specific subphenotypes, haemodynamics, and cardiogenic shock severity staging ha
52 ures, regional (leg, arm, head) and systemic haemodynamics, and left ventricular (LV) volumes and fun
53 ined the influence of central and peripheral haemodynamics, and skeletal muscle factors linked to mec
54              Myocardial microvasculature and haemodynamics are indicative of potential microvascular
55 whether these age-associated changes in limb haemodynamics are mediated by tonically augmented sympat
56 es argues for the importance of intracardiac haemodynamics as a key epigenetic factor in embryonic ca
57 in the peripheral circulation jointly impact haemodynamics as MAC develops.
58 onitoring, optimisation of volume status and haemodynamics, avoidance of nephrotoxic drugs and radioc
59 sms in T2D and hypertension by comparing the haemodynamics between healthy controls and subjects with
60 ndpoints included changes in cardiopulmonary haemodynamics, Borg dyspnoea index, WHO functional class
61                                      Central haemodynamics, brachial artery shear rate (SR) and blood
62 icular growth not only led to more realistic haemodynamics, but also impacted the magnitude of growth
63 icular growth not only led to more realistic haemodynamics, but also impacted the magnitude of growth
64 hibit similar sympathetic nerve activity and haemodynamics, but decreased pain perception, during a c
65           Here we show that placental oxygen haemodynamics can be non-invasively probed in real time
66                 Devices for monitoring blood haemodynamics can guide the perioperative management of
67 he regional (leg, arm and head) and systemic haemodynamics (cardiac output: Q ) during passive single
68 AH, improved exercise capacity and pulmonary haemodynamics compared with placebo but at the expense o
69 the decompression phase can lead to improved haemodynamics compared with standard CPR.
70                                        Local haemodynamics control arterial homeostasis and dysfuncti
71 isease depends on measurements of the global haemodynamics (criteria for heart function) and also the
72 teria for heart function) and also the local haemodynamics (detailed data on the dynamics of blood fl
73                                      Altered haemodynamics distal to the web cause flow stagnation an
74 is to analyse the characteristics of hepatic haemodynamics disturbances identified through perfusion
75 or future investigations of patient-specific haemodynamics during AF.
76  in the sympathetic neural control of muscle haemodynamics during exercise.
77 mic perfusion, we measured local and central haemodynamics during one-legged knee-extensor exercise (
78  rat spinotrapezius muscle (n = 5) capillary haemodynamics during recovery from 3 min of twitch muscl
79      Pharmacotherapies that alter intrarenal haemodynamics (eg, renin-angiotensin-aldosterone pathway
80 nimals were anaesthetized and catheter-based haemodynamics evaluated, followed by histological measur
81 ve approaches that quantify global and local haemodynamics for different CoA severities, innovative c
82 This 'neuroprosthetic baroreflex' controlled haemodynamics for extended periods of time in rodents, n
83          Our results therefore indicate that haemodynamics, generated by a Pitx2-induced morphologica
84           Conventional therapies that modify haemodynamics have lengthened the time to kidney failure
85 requency spectrum characteristics from these haemodynamics have never been exploited to test whether
86             Lambs on support maintain stable haemodynamics, have normal blood gas and oxygenation par
87            Biventricular geometry, function, haemodynamics, hypertrophy and fibrosis were compared be
88 tic sleeve recapitulates clinically relevant haemodynamics in a porcine model of aortic stenosis, as
89  for imaging myocardial microvasculature and haemodynamics in explanted pig hearts and in patients in
90 ltaneous recordings of neuronal activity and haemodynamics in motor and whisker barrel cortices.
91          In this study, we examined cortical haemodynamics in mouse preparations that modelled Alzhei
92 us, ENO can improve oxygenation and systemic haemodynamics in neonates, and seems to reduce rebound h
93 cular function and pulmonary and/or systemic haemodynamics in particular patients.
94 tan increases exercise capacity and improves haemodynamics in patients with pulmonary hypertension, s
95 are few studies on the evaluation of retinal haemodynamics in patients with type 1 diabetes mellitus
96 potential for continuous monitoring of blood haemodynamics in patients.
97 s work was to create a method for simulating haemodynamics in positive-displacement blood pumps, usin
98  substantially to the steady-state capillary haemodynamics in the contracting rat spinotrapezius musc
99                                      Femoral haemodynamics in the control limb were unaffected by phe
100                   Propranolol did not affect haemodynamics in the experimental or control limbs.
101 ltrafast volumetric photoacoustic imaging of haemodynamics in the human body at up to 1 kHz can be ac
102 ables the longitudinal volumetric imaging of haemodynamics in vasculature a few millimetres below the
103 phy (PAVT), allows for the quantification of haemodynamics in veins more than 5 mm deep, as we show f
104 possibilities for quantitative assessment of haemodynamics in vivo in real time, at the whole-body le
105 ternal (ICA) and common carotid artery (CCA) haemodynamics (indicative of CBF and extra-cranial blood
106 ional studies suggest that impaired cerebral haemodynamics is associated with symptomatic status in p
107 mprehensive understanding of MAC's impact on haemodynamics is lacking.
108 les, in particular CBV, to regional cerebral haemodynamics is not clearly established in humans with
109      Although exercise did not alter fasting haemodynamics, it lowered the augmentation index (AIx75,
110 CO(2) -CR interactions restricted peripheral haemodynamics, likely by potentiating sympathetic vasoco
111 teraction facilitated central and peripheral haemodynamics, likely by potentiating sympathetic vasodi
112 ion, and an overview of important mediators (haemodynamics, matrix components, and soluble factors) i
113                                 Beat-to-beat haemodynamics (Modelflow), muscle sympathetic nerve acti
114 en generating high-fidelity patient-specific haemodynamics models.
115                              Despite adverse haemodynamics, no patient who died suddenly had undergon
116                                 Imaging deep haemodynamics non-invasively remains a quest.
117 tive and economic tool for investigating the haemodynamics of natural and artificial heart valves.
118 ular function combine to explain the complex haemodynamics of subjects affected by hypertension and/o
119 ificant alteration is observed in the normal haemodynamics of the aorta.
120                   Age and sex did not impact haemodynamics or AP discharge during PECO (range: P = 0.
121 -48 mL min(-1) with minimal changes to other haemodynamics or blood chemistry.
122 hout changes in gross LV structure, arterial haemodynamics or heart rate.
123 icantly affect baseline cardiac performance, haemodynamics or myocardial metabolism.
124         There were no significant changes in haemodynamics or renal biochemistry for either group.
125 on resting CBF, end-tidal CO(2) and systemic haemodynamics over a 2 h period post-ingestion in young
126  has the unique capacity of monitoring brain haemodynamics/oxygenation (measuring oxygenated and deox
127     To this aim, we examined etanet, central haemodynamics, peripheral circulation, and peripheral fa
128                                 Beat-by-beat haemodynamics (photoplethysmography) and MSNA (microneur
129                                              Haemodynamics play a key role in cardiovascular disease
130 e mitochondrial capacity, suggesting central haemodynamics plays a larger role in age-related decline
131 ach reflex) and hypo-additive for peripheral haemodynamics (responses during co-activation of the ref
132  interactions with restriction in peripheral haemodynamics, resulting from the EPR:CR interaction in
133                                 Through a 1D haemodynamics simulation, we study how the mechanical pr
134  was used to interrogate changes in vascular haemodynamics, structural response and hypoxia in C6 gli
135 -dimensional and reduced-order computational haemodynamics studies via a pipeline which involves: 1)
136                      Alterations in cerebral haemodynamics, such as arterial impulse propagation driv
137 re temperature and changes in cardiovascular haemodynamics, such as cardiac output and vascular shear
138 heart rate, and hypo-additive for peripheral haemodynamics, the interaction resulting from the EPR:CO
139 ing to investigate the relationships linking haemodynamics to vessel-wall pathobiology.
140 eeded to assess causes, pathophysiology, and haemodynamics, to determine prognosis and consider thera
141 nvironment that makes powerful computational haemodynamics tools accessible to a wide audience, inclu
142                       Techniques for imaging haemodynamics use ionizing radiation or contrast agents
143 nd (2) a mechanical subsystem that simulates haemodynamics using a reduced order model.
144 ns of left atrial (LA) myocardial motion and haemodynamics using patient-specific anatomies and fibro
145        Medial gastrocnemius microvasculature haemodynamics were assessed before, during and after sit
146 holamine levels and changes in fetal femoral haemodynamics were assessed following fetal glucocortico
147           Serum NT-proBNP, biochemistry, and haemodynamics were determined at baseline and at key tim
148 by-second by Doppler ultrasound, and central haemodynamics were measured by finger photoplethysmograp
149                                     MSNA and haemodynamics were measured supine and during 45 min 60
150                                     MSNA and haemodynamics were measured supine and during a graded u
151 Muscle sympathetic nerve activity (MSNA) and haemodynamics were measured supine, at 30 deg and 60 deg
152 ase in haematocrit and because ABP and renal haemodynamics were normalized: acute hypoxia in N rats p
153                Patients with mildly abnormal haemodynamics were not more symptomatic than patients wi
154 ive reflex effects on central and peripheral haemodynamics were quantified in healthy young women and
155           Baseline characteristics, MSNA and haemodynamics were similar between the groups.
156 terventional approaches can improve vascular haemodynamics, whereas established and emerging pharmaco
157 postductal arterial oxygenation and systemic haemodynamics, which were maintained during the off-drug
158 copy (2D-OIS) to measure changes in cerebral haemodynamics within the sensory cortex of the brain dur

 
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