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1 ds (DPPH, ORAC and erythrocyte resistance to haemolysis).
2 linked to microvascular red blood cell (RBC) haemolysis.
3 PNH and clinically significant extravascular haemolysis.
4 other for 1-14 days) would cause significant haemolysis.
5 PNH and clinically significant extravascular haemolysis.
6 o sustained reticulocytopenia, to near-fatal haemolysis.
7 ompromised, the RBCs are more susceptible to haemolysis.
8 rds hydrogen peroxide and on surface-induced haemolysis.
9 altering red blood cell rheology and causing haemolysis.
10 nd no evidence of hepatitis, cholestasis, or haemolysis.
11 c sucrose solutions at pH 6) supported their haemolysis.
12                     Cytochalasin B prevented haemolysis.
13 of individuals at risk of primaquine-induced haemolysis.
14 ver, abnormal liver function tests, and mild haemolysis.
15  EC necroptosis and complement-dependent RBC haemolysis.
16 site reactions (in 20 [26%] of 77 patients), haemolysis (15 [19%]), nasopharyngitis (12 [16%]), and d
17  flavonoid that potentiated copper-triggered haemolysis (155 +/- 81 % at twice the amount of Cu(2+)),
18 l mutant also restores full contact-mediated haemolysis activity to this bacterium.
19 lation of eight mutants that failed to cause haemolysis, all of which had transposon insertions in ge
20           To reduce the risk of drug-induced haemolysis, all patients should be tested for glucose-6-
21  significant improvements in haemoglobin and haemolysis among patients with pyruvate kinase deficienc
22 nillin or 5-hydroxymethyl, and urea) reduced haemolysis, an effect not due to increased oxygen affini
23                Hydroxyurea treatment reduces haemolysis and anaemia by increasing foetal haemoglobin,
24 ubarachnoid haemorrhage is often followed by haemolysis and concomitant oxidative stress, and is freq
25 hiphos on trout health through intravascular haemolysis and consequently from pathophysiologic proces
26  (PHA) test, bacteria-killing ability (BKA), haemolysis and haemagglutination assays.
27 nto the mechanism of streptolysin S-mediated haemolysis and have implications for the development of
28                      SMA is characterised by haemolysis and inadequate erythropoiesis, and is associa
29 ucture is a key step for membrane insertion, haemolysis and insecticidal activity.
30 SAT6 has recently been demonstrated to cause haemolysis and macrophage lysis.
31 rated 28 analogues of B1, showing comparable haemolysis and MICs against MRSP and P. aeruginosa.
32                There has been no significant haemolysis and no device-related complications.
33 12 patients reported serious adverse events; haemolysis and pyrexia were the most common (each occurr
34 oderate (2438 m) altitude increased rates of haemolysis and right ventricular systolic pressures in m
35 est inhibitory activity against heat-induced haemolysis and ROS formation in erythrocytes.
36 oposed direct link between contact-dependent haemolysis and Shigella entry, and demonstrate that IpaB
37 s accompanied by consumption of haptoglobin (haemolysis) and poor reticulocyte production.
38 ing to ineffective erythropoiesis, increased haemolysis, and deranged iron homoeostasis.
39 considerable antimicrobial activity, minimal haemolysis, and low cytotoxicity, we introduced the natu
40 of 41 through to week 16 due to breakthrough haemolysis, and ten [13%] of 77 due to severe treatment-
41 hildren showed evidence of treatment-related haemolysis, and the mean maximum decrease in haemoglobin
42  although the importance of chronic anaemia, haemolysis, and vasculopathy has been established.
43 utropenic typhlitis, pancreatitis, and acute haemolysis are very rare.
44       Significant improvements in markers of haemolysis, as assessed by the difference in adjusted me
45            A simple test represented by this haemolysis assay would be useful especially in less affl
46  due to clinically significant extravascular haemolysis can affect patients with paroxysmal nocturnal
47  may result in a ten-fold increase in sample haemolysis, compared to the recommended guideline proced
48    Both fetal erythropoietic suppression and haemolysis contribute to anaemia.
49              Long-term data indicate durable haemolysis control with iptacopan in paroxysmal nocturna
50 rated, samples were processed to obtain full haemolysis curves at precise times.
51  progressive changes in the profile of their haemolysis curves, as the curves migrated towards lower
52 showed no change in profile of the migrating haemolysis curves, suggesting that their PCl distributio
53 son, haptoglobin, NO(x) , ovotransferrin and haemolysis differed significantly between breeding and n
54 on (2 [6%]), and one (3%) each with anaemia, haemolysis, fatigue, and a neurological, metabolic, resp
55 on of patients with vivax malaria at risk of haemolysis following 8-aminoquinoline radical cure is su
56 ched the threshold of clinically significant haemolysis (fractional haematocrit reduction >25%) in G6
57 PNH and clinically significant extravascular haemolysis (haemoglobin <=9.5 g/dL; absolute reticulocyt
58 mmation; and systemic manifestations such as haemolysis, haemorrhage and rhabdomyolysis.
59                                              Haemolysis has features which suggest that it is linked
60 GS 8/9) was more effective against oxidative haemolysis (IC(50) 38 and 75 ug/mL).
61 nces (IC(50) = 23 +/- 2 ug/mL) and oxidative haemolysis (IC(50) at 60 min = 46.0 +/- 0.8 ug/mL).
62 bstances (IC(50) = 23 2 ug/mL) and oxidative haemolysis (IC(50) at 60 min = 46.0 0.8 ug/mL).
63 2438 m) altitude would have a higher rate of haemolysis, impaired cardiac function and reduced exerci
64 or dehydrating cells to attain resistance to haemolysis in a selected hypotonic medium.
65 re was no evidence of clinically significant haemolysis in any patient.
66 tioxidant power) and inhibition of oxidative haemolysis in erythrocytes.
67 nt of Plasmodium vivax malaria and can cause haemolysis in G6PD deficient subjects.
68 he potential to cause clinically significant haemolysis in G6PD heterozygous females who are reported
69 the relationship between primaquine dose and haemolysis in G6PDd.
70   The 8-aminoquinolines cause dose-dependent haemolysis in glucose-6-phosphate dehydrogenase deficien
71                       Either drug can induce haemolysis in individuals with glucose-6-phosphate dehyd
72 primaquine or tafenoquine) is complicated by haemolysis in individuals with glucose-6-phosphate dehyd
73        However, these drugs can cause severe haemolysis in individuals with glucose-6-phosphate dehyd
74 stfeeding mothers to avoid the potential for haemolysis in infants with G6PD deficiency.
75              However, primaquine can trigger haemolysis in patients with a deficiency in glucose-6-ph
76       However, concerns about dose-dependent haemolysis in people with glucose-6-phosphate dehydrogen
77 ial was designed to measure efficacy and not haemolysis in relation to G6PD genotype and that the het
78            Controversies include the role of haemolysis in sickle cell disease pathophysiology, optim
79  to week 72, changes in the concentration of haemolysis markers (absolute and percentage reticulocyte
80 etic deletion of Mlkl from ECs decreased RBC haemolysis, microvascular obstruction and reduced ischae
81 scending dose regimen was stopped because of haemolysis (n=1) and asymptomatic increases in transamin
82                                     At pH 6, haemolysis occurred even in oxygenated samples.
83 ss the 48-week trials, clinical breakthrough haemolysis occurred in seven (7%) of 96 iptacopan-treate
84 as significantly inhibited with little or no haemolysis occurring at 4 degrees C.
85 ls and loss of Dot/Icm T4SS-mediated contact haemolysis of erythrocytes.
86        Eculizumab inhibits the intravascular haemolysis of PNH, reduces transfusion requirements, sta
87                             Cytotoxicity and haemolysis of the progenitor peptide is ameliorated with
88 iglyceridaemia, but there was no evidence of haemolysis or microangiopathy.
89 se (SCD), including vaso-occlusion, anaemia, haemolysis, organ injury and pain.
90 RSA) and fungi, without inducing significant haemolysis over a wide range of concentrations.
91 ignificantly reduced listeriolysin O-induced haemolysis (p < 0.05), and ameliorated H(2)O(2)-induced
92 that at temperatures < or = 15 degrees C the haemolysis rate was significantly inhibited with little
93           These findings are consistent with haemolysis requiring HbS polymerisation and support the
94 r varieties, as assessed by the ORAC and the haemolysis resistance assays.
95 induced systemic inflammation and apoptosis, haemolysis, rhabdomyolysis, smoke inhalation injury, dru
96   Many acute and chronic anaemias, including haemolysis, sepsis and genetic bone marrow failure disea
97 tion, microneutralisation, and single radial haemolysis (SRH).
98                            The intravascular haemolysis that is the clinical hallmark of PNH is a con
99  the serious adverse events were significant haemolysis (three in the 7-day group and one in the 14-d
100 ale-biased macrophage concentration, BKA and haemolysis titers, but only during the breeding season.
101 sferrin concentration, haemagglutination and haemolysis titres increased 12 weeks into the dry season
102 in concentrations, and haemagglutination and haemolysis titres) over two annual cycles of wet and dry
103 in concentrations, and haemagglutination and haemolysis titres), body mass and primary moult, fortnig
104 r [11%] of 37), chest pain (two [5%] of 37), haemolysis (two [5%] of 37), and neutropenia (two [5%] o
105  designed to characterise primaquine-induced haemolysis using a holistic Bayesian analysis of all pub
106                   When alpha toxin-dependent haemolysis was measured on erythrocytes at various tempe
107                                              Haemolysis was reduced in patients on long-term (>5 mont
108                                              Haemolysis was substantially greater and a larger propor
109                                              Haemolysis was temperature- and pH-dependent.
110                                  Patterns of haemolysis were compared between G6PD wild-type and G6PD
111 e rate of sample laboratory rejection due to haemolysis when commonly practiced deviations from the g
112 s has been the appearance of complete (beta) haemolysis when grown in the presence of blood.
113 adherence to the vascular endothelium and by haemolysis, which results in endothelial cell activation
114 rent work addresses the hypothesis that this haemolysis will provide a novel diagnostic and prognosti
115       The peel extract inhibit the oxidative haemolysis, with IC(50) values of 255 and 381 mug/mL for
116       The peel extract inhibit the oxidative haemolysis, with IC(50) values of 255 and 381 ug/mL for

 
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