ライフサイエンスコーパス: haemorrhage

1 ent-related death was reported (intracranial haemorrhage).

2 ry artery, myocardial infraction, and muscle haemorrhage).

3 e severe bleeding (traumatic and post-partum haemorrhage).

4 ncreasing the absolute risk of intracerebral haemorrhage).

5 otic demyelination syndrome and intracranial haemorrhage).

6 er and/or neuroretinal rim defects, and disc haemorrhages).

7 ood pressure lowering in acute intracerebral haemorrhage.

8 three of them (7.7%) demonstrated a meniscal haemorrhage.

9 inal delivery is a major cause of postpartum haemorrhage.

10 a good response 365 days after intracerebral haemorrhage.

11 oms (p=0.003) as risk factors for subsequent haemorrhage.

12 9%) demonstrated a concomitant periarticular haemorrhage.

13 itant central hypovolemia that occurs during haemorrhage.

14 ional outcome in patients with intracerebral haemorrhage.

15 e, with around half the risk of intracranial haemorrhage.

16 ould lead to a catastrophic gastrointestinal haemorrhage.

17 ppear to be at increased risk for subsequent haemorrhage.

18 ated in secondary injury after intracerebral haemorrhage.

19 50% of patients presented with intracerebral haemorrhage.

20 livery, perineal lacerations, and postpartum haemorrhage.

21 eath from bleeding in trauma and post-partum haemorrhage.

22 grade patients after aneurysmal subarachnoid haemorrhage.

23 ibrillation and one case of grade 3 or worse haemorrhage.

24 therapy or in the setting of a subarachnoid haemorrhage.

25 ith probable CAA without lobar intracerebral haemorrhage.

26 ) in cesarean section among women at risk of haemorrhage.

27 ity related to uterine rupture and extensive haemorrhage.

28 st corrected visual acuity owing to vitreous haemorrhage.

29 e (3%) patient with grade 2 bronchopulmonary haemorrhage.

30 haemorrhage size and thalamic intracerebral haemorrhage.

31 functional independence) after intracerebral haemorrhage.

32 or dependence by reducing the extent of the haemorrhage.

33 t risk factor for future lobar intracerebral haemorrhage.

34 n increased risk of symptomatic intracranial haemorrhage.

35 e between ischaemic stroke and intracerebral haemorrhage.

36 OE e2 significantly increased risk for gross haemorrhage.

37 ntions are investigational for intracerebral haemorrhage.

38 The key safety outcome was any intracranial haemorrhage.

39 mRS score 4-6), and symptomatic intracranial haemorrhage.

40 ch in UC patients with severe, acute colonic haemorrhage.

41 such as acute diverticulitis or diverticular haemorrhage.

42 ith greater risks of recurrent intracerebral haemorrhage.

43 51 patients in these three studies had major haemorrhages.

44 were 782 (55.1%) lobar and 596 (42.0%) deep haemorrhages.

45 e posterior pole, vascular abnormalities and haemorrhages.

46 al schisis with pre-retinal and intraretinal haemorrhages.

47 ies (WMH): 10 597 individuals; intracerebral haemorrhage: 1545 cases, 1481 controls].

48 rebral haemorrhage 28% [26-29], subarachnoid haemorrhage 16% [12-20], unspecified stroke type 15% [12

49 4] per 1000 patient-years vs 27 intracranial haemorrhages [17-41] per 1000 patient-years; and for >=2

50 7 (5%) in the late-OAC group (2 intracranial haemorrhages, 18 ischaemic strokes or TIAs and 31 deaths

51 chaemic stroke, 47% (46-48)for intracerebral haemorrhage, 19% (17-22; 52% for rural areas and 32% for

52 o 1.38) with increased risk of intracerebral haemorrhage (2.07, 95% CI 1.37 to 3.13) and early neurol

53 /day], haemoptysis [80 mg/day], intracranial haemorrhage [20 mg/day], ventricular fibrillation [120 m

54 8] per 1000 patient-years vs 39 intracranial haemorrhages [21-67] per 1000 patient-years).

55 rebral haemorrhage 44% [42-46], subarachnoid haemorrhage 22% [18-27], unspecified stroke type 40% [35

56 ischaemic stroke 16% [15-16], intracerebral haemorrhage 28% [26-29], subarachnoid haemorrhage 16% [1

57 ng diagnoses were perinatal intraventricular haemorrhage (35.3%) and malformations (33.9%) in infants

58 (ischaemic stroke 41% [41-42], intracerebral haemorrhage 44% [42-46], subarachnoid haemorrhage 22% [1

59 (77.8%), headache (16.7%), and intracerebral haemorrhage (5.55%), and 5.55% were asymptomatic.

60 -13.65]), and perioperative severe obstetric haemorrhage (5.87 [1.99-17.34]) or anaesthesia complicat

61 nrolled, of whom 525 (98%) had intracerebral haemorrhage: 507 (97%) were diagnosed on CT (252 assigne

62 lso ischaemic stroke; after an intracerebral haemorrhage, 56% of recurrent strokes were intracerebral

63 haemic stroke, 7440 (16%) were intracerebral haemorrhage, 702 (2%) were subarachnoid haemorrhage, and

64 sed eligible participants with intracerebral haemorrhage according to their treatment allocation in p

65 nsecutive UC patients diagnosed with massive haemorrhage admitted in a tertiary institution are repor

66 ic stroke; 2.45 (1.82-3.29) for intracranial haemorrhage and 1.23 (1.08-1.40) for ischaemic stroke.

67 PRETATION: In patients with intraventricular haemorrhage and a routine extraventricular drain, irriga

68 thrombosis in two patients, and intraluminal haemorrhage and active extravasation in one patient.

69 income countries and is driven by peripartum haemorrhage and anaesthesia complications.

70 Brain imaging features of intracerebral haemorrhage and cerebral small vessel diseases (such as

71 a largely untreatable cause of intracerebral haemorrhage and contributor to age-related cognitive dec

72 ents undergoing TIPSS insertion for variceal haemorrhage and correlate this with outcome.

73 verall our results indicate that the risk of haemorrhage and dire consequences is multifactorial.

74 th spontaneous (non-traumatic) intracerebral haemorrhage and elevated systolic blood pressure, withou

75 ing operation times and introducing risks of haemorrhage and injury to the heart and lungs during ste

76 T cells in the context of tPA-induced brain haemorrhage and investigated the underlying mechanisms o

77 s for association with primary intracerebral haemorrhage and ischaemic stroke subtypes.

78 0) for the composite outcome of intracranial haemorrhage and ischaemic stroke; 2.45 (1.82-3.29) for i

79 Primary intracerebral haemorrhage and lacunar ischaemic stroke are acute manif

80 , leading to increased primary intracerebral haemorrhage and lacunar stroke risk.

81 evels increase risk of primary intracerebral haemorrhage and lacunar stroke, directly implicate the m

82 ips was analysed over Days 0-3 after initial haemorrhage and magnetic resonance imaging studies were

83 8% died - one in the course of intracerebral haemorrhage and one due to other sustained injuries.

84 dexamethasone group (upper gastrointestinal haemorrhage and respiratory failure).

85 The major causes of SMOs were postpartum haemorrhage and severe pre-eclampsia/eclampsia.

86 evaluation of acute, active gastrointestinal haemorrhage and show its usefulness prior to embolizatio

87 Intracerebral haemorrhage and small vessel ischaemic stroke (SVS) are

88 ility and rate features of the intracerebral haemorrhage and surrounding brain.

89 After combining intracerebral haemorrhage and SVS datasets, our sample size included 2

90 oss-phenotype genetic study of intracerebral haemorrhage and SVS demonstrates novel genome-wide assoc

91 el genome-wide approach across intracerebral haemorrhage and SVS.

92 mine mesylate in patients with intracerebral haemorrhage and to establish whether the drug merits inv

93 ecause it precisely identifies the source of haemorrhage and vascular anatomy that helps the interven

94 ut significantly increased the risk of major haemorrhage and, unexpectedly, all-cause mortality.

95 DBS complications, with fewer intracerebral haemorrhages and infections with general anaesthesia (p<

96 vitritis associated with large paravascular haemorrhages and yellow necrotic borders, involving the

97 or patients with non-CAA deep intracerebral haemorrhage) and patients with Alzheimer's disease.

98 primary (recurrent symptomatic intracerebral haemorrhage) and secondary (ischaemic stroke) outcomes f

99 bral haemorrhage, 702 (2%) were subarachnoid haemorrhage, and 1002 (2%) were an unspecified stroke ty

100 areas and 32% for urban areas) subarachnoid haemorrhage, and 24% (22-27) for unspecified stroke.

101 nine [<1%] vs seven [<1%]) had grade 3-5 CNS haemorrhage, and 40 (19 [1%] vs 21 [1%]) had grade 3-5 c

102 56% of recurrent strokes were intracerebral haemorrhage, and 41% of recurrent strokes were ischaemic

103 e (including ischaemic stroke, intracerebral haemorrhage, and myocardial infarction) by linkage with

104 r ischaemic stroke, symptomatic intracranial haemorrhage, and symptomatic ischaemic stroke.

105 oke, intracerebral haemorrhage, subarachnoid haemorrhage, and unspecified type).

106 with ischaemic stroke and 3059 intracerebral haemorrhage] and 13 472 controls).

107 Paramount in the clot's capability to stem haemorrhage are its changing mechanical properties, the

108 in synergy with direct ischaemic effects of haemorrhage as mechanisms of infarct development.

109 n in the context of ageing and intracerebral haemorrhage, as well as in Alzheimer's and other dementi

110 us seen in traditional GWAS of intracerebral haemorrhage, as well as the rediscovery of 13q34, which

111 After aneurysmal subarachnoid haemorrhage (aSAH), extracellular haemoglobin (Hb) in th

112 ide associations for non-lobar intracerebral haemorrhage at 2q33 and 13q34.

113 Excessive haemorrhage at cesarean section requires donor (allogene

114 erebrospinal fluid leakage and intracerebral haemorrhage) at days 3-7 after AAV2 gene therapy, and we

115 riovenous malformation-related intracerebral haemorrhage (AVICH) score showed better outcome predicti

116 s in up to 30% of patients with subarachnoid haemorrhage but the incidence of ALI after ischemic stro

117 cerebral microbleed burden for intracranial haemorrhage but this effect was less marked for ischaemi

118 Non-contrast CT can exclude haemorrhage, but the addition of CT perfusion imaging an

119 lerosis), we performed GWAS of intracerebral haemorrhage by location in 1813 subjects (755 lobar and

120 s outnumbered warfarin-related intracerebral haemorrhages by about 15-fold (280 vs 19), rising to 50-

121 scovery cohort of 1409 primary intracerebral haemorrhage cases and 1624 controls from three studies,

122 hologies, including old and recent infarcts, haemorrhages, cerebral amyloid angiopathy (CAA) and arte

123 risk of recurrent symptomatic intracerebral haemorrhage compared with avoiding antiplatelet therapy.

124 risk of recurrent symptomatic intracerebral haemorrhage compared with avoiding antiplatelet therapy.

125 s (96 healthy elderly, 42 deep intracerebral haemorrhage controls) and 72 patients with Alzheimer's d

126 : Acute non-traumatic convexity subarachnoid haemorrhage (cSAH) is increasingly recognised in cerebra

127 bolism) and delayed symptomatic intracranial haemorrhage (d-sICH) within 90 days.

128 cluded the rates of symptomatic intracranial haemorrhage, device-related complications, and other sev

129 safe, the observed reduction in intracranial haemorrhage did not lead to improved clinical outcome co

130 ts and neurological lesions in foetuses like haemorrhages, diffuse cerebral edema, necrotizing enceph

131 the significance of delayed intraventricular haemorrhage (dIVH) is less well defined.

132 the significance of delayed intraventricular haemorrhage (dIVH) is less well defined.

133 flows and outflow diameter) of intracerebral haemorrhage due to intracranial dural arteriovenous fist

134 a patient with recurrent post-tonsillectomy haemorrhage due to pseudoaneurysm of the facial artery,

135 symptoms to be independently associated with haemorrhage during follow-up (HR 9.39, CI 1.86 to 170.8,

136 ong this latter group, 16 (25.0%) suffered a haemorrhage during follow-up and 11 (17.2%) required sur

137 verage of uterotonics to prevent post-partum haemorrhage, early initiation of breastfeeding (within 1

138 associated with a lower risk of intracranial haemorrhage (eight [3%] of 253 vs 152 [14%] of 1089; inc

139 anaemia (eight [4%]), upper gastrointestinal haemorrhage (eight [4%]), pneumonia (seven [3%]), gastri

140 ns were observed for non-lobar intracerebral haemorrhage enhanced by SVS with rs2758605 [MTAG P-value

141 , lacunar infarcts, microhaemorrhage, larger haemorrhage, fibrinoid necrosis, microaneurysms, perivas

142 haemorrhage (six [3%]), and gastrointestinal haemorrhage (five [2%]) in the trastuzumab emtansine 2.4

143 chaemic stroke exceeded that of intracranial haemorrhage (for ten or more cerebral microbleeds, 64 is

144 ed with a Hunt and Hess grade 3 subarachnoid haemorrhage from a ruptured aneurysm.

145 puted tomography scan showed active arterial haemorrhage from ascendant or sigmoid colon; subsequent

146 the Genetics and Observational Subarachnoid Haemorrhage (GOSH) Study, a retrospective multicentre co

147 ansfusion (primary outcome) and fetomaternal haemorrhage >/=2 ml in RhD-negative women with RhD-posit

148 Intracerebral haemorrhage GWAS results by location were meta-analysed

149 uctural cerebral cause for the intracerebral haemorrhage, had a low score (3-5) on the Glasgow Coma S

150 ociated with stroke recurrence, intracranial haemorrhage (ICH) and acenocoumarol maintenance dose in

151 botic agents increase risks of intracerebral haemorrhage (ICH) and associated adverse outcomes.

152 rning system that detects acute intracranial haemorrhage (ICH) and classifies five ICH subtypes from

153 Patients with primary intracerebral haemorrhage (ICH) are at increased long-term risks of re

154 ticoagulants (NOAC)-associated intracerebral haemorrhage (ICH) are largely unknown.

155 ment of spontaneous cerebellar intracerebral haemorrhage (ICH) differ.

156 r statins increase the risk of intracerebral haemorrhage (ICH) in patients with a previous stroke rem

157 mined injury progression after intracerebral haemorrhage (ICH) induced by collagenase in mice using a

158 Intracerebral haemorrhage (ICH) is a life-threatening emergency, the i

159 ure (BP) at the acute phase of intracerebral haemorrhage (ICH) is beneficial.

160 Intraventricular extension of intracerebral haemorrhage (ICH) predicts poor outcome, but the signifi

161 Intraventricular extension of intracerebral haemorrhage (ICH) predicts poor outcome, but the signifi

162 ous malformation (AVM)-related intracerebral haemorrhage (ICH) than other AVM or ICH scores.

163 ebrovascular disease including intracerebral haemorrhage (ICH), and common collagen IV variants are a

164 quality of life (HRQoL) after intracerebral haemorrhage (ICH).

165 er spontaneous (non-traumatic) intracerebral haemorrhage (ICH).

166 Type I fistulas were associated with haemorrhage in 1/3 of all cases.

167 kine release syndrome and one from pulmonary haemorrhage in a patient with persistent cytopenia.

168 iated with an increased risk of intracranial haemorrhage in adults with cerebral cavernous malformati

169 disseminated encephalomyelitis (n = 9), with haemorrhage in five, necrosis in one, and myelitis in tw

170 nance of arterial blood pressure (BP) during haemorrhage in humans.

171 xed reports on the incidence of intracranial haemorrhage in patients with dural arteriovenous fistula

172 fter acute spontaneous primary intracerebral haemorrhage in people taking antiplatelet therapy might

173 iplatelet therapy on recurrent intracerebral haemorrhage in primary subgroup analyses of cerebral mic

174 may alleviate thrombolytic treatment-induced haemorrhage in stroke victims.

175 context of neutropenic fever, infection, and haemorrhage in the 5-day group, and in the 10-day group

176 ies included data for the incidence of major haemorrhage in the ECMO group.

177 re we studied acute sequelae of subarachnoid haemorrhage in the gyrencephalic brain of propofol-anaes

178 igher prevalence of symptomatic intracranial haemorrhage in the intervention group.

179 antly increased risk of acute/subacute gross haemorrhage in the presence of CAA.

180 iplatelet therapy on recurrent intracerebral haemorrhage in the presence of cerebral microbleeds.

181 e stroke (ischaemic or primary intracerebral haemorrhage) in England and Wales between April 1, 2013,

182 1193 ischaemic strokes and 191 intracerebral haemorrhages) in patients admitted during the same time

183 es (81 ischaemic strokes and 5 intracerebral haemorrhages) in patients with evidence of COVID-19 at t

184 rative studies to calculate the intracranial haemorrhage incidence rate ratio according to antithromb

185 the risk of potentially harmful intracranial haemorrhage, including haemorrhagic transformation of th

186 Intraventricular haemorrhage is a subtype of intracerebral haemorrhage, w

187 Intraplaque haemorrhage is accepted by neurologists and radiologists

188 Severe haemorrhage is an uncommon but life-threatening complica

189 e stroke due to supratentorial intracerebral haemorrhage is associated with high morbidity and mortal

190 tack in whom the rate of future intracranial haemorrhage is likely to exceed that of recurrent ischae

191 Haemorrhage is the leading cause of battlefield and civi

192 s, with dIVH defined as new intraventricular haemorrhage (IVH) on the latter scan.

193 d: CNS involvement, a stroke or intracranial haemorrhage less than 12 months before enrolment, clinic

194 Because intracerebral haemorrhage location is an adequate surrogate for distin

195 management of mothers at risk of peripartum haemorrhage might improve maternal and neonatal outcomes

196 For moderate to large intracerebral haemorrhage, MISTIE did not improve the proportion of pa

197 lacenta previa/abruptio placenta/ante-partum haemorrhage; multiple birth; pre-delivery LoS >= 3 days;

198 = 11) or were made anaemic by isovolumetric haemorrhage (n = 12) for 1 week prior to myocardial cont

199 epatic failure (n=1), upper gastrointestinal haemorrhage (n=1), neurological decompensation (n=1), an

200 f ischaemic stroke (n=14 930), intracerebral haemorrhage (n=3496), and acute myocardial infarction (n

201 outnumbered disabling or fatal intracerebral haemorrhage (n=45 vs n=18), with an absolute risk of 9.1

202 enom-induced pathophysiological changes like haemorrhage, necrosis, nephrotoxicity and often develop

203 Perioperative haemorrhage negatively affects patient outcomes and resu

204 ridioides difficile infection and procedural haemorrhage); neither was assessed by the investigator a

205 arrest, pneumonia, sepsis, and subarachnoid haemorrhage), none were deemed treatment related.

206 age volume less than 30 mL, intraventricular haemorrhage obstructing the 3rd or 4th ventricles, and n

207 Late seizures after intracerebral haemorrhage occur after the initial acute haemorrhagic i

208 Haemorrhage occurred in 13 (20%; 95% CI 11.1-31.8) of 65

209 Intracranial haemorrhage occurred in 33 (0.6%) and 31 (0.6%) patients

210 Intracranial haemorrhage occurred in 48 (36%) of 134 in the aspiratio

211 obable treatment-related death (intracranial haemorrhage) occurred 41 days after starting open-label

212 more likely to have pseudoexfoliation, disc haemorrhages, ocular medication changes, and IOP-lowerin

213 ental abruption, ruptured uterus, antepartum haemorrhage (odds ratio 4.47 [95% CI 1.46-13.65]), and p

214 nce of primary intramedullary damage such as haemorrhage, oedema, post-traumatic cystic cavities, and

215 non-traumatic, supratentorial intracerebral haemorrhage of 30 mL or more.

216 3 patients vs five [3%] of 184 patients) and haemorrhage of any type (17 [4%] of 403 vs one [<1%] of

217 utcomes in patients with acute intracerebral haemorrhage of predominantly mild-to-moderate severity.

218 nts aged 18 years and older with parenchymal haemorrhage on the first CT scan.

219 etroperitoneal haemorrhage, one subarachnoid haemorrhage, one respiratory distress, and one from dise

220 unrelated to treatment (one retroperitoneal haemorrhage, one subarachnoid haemorrhage, one respirato

221 usions for 3 consecutive days within 24 h of haemorrhage onset.

222 the syndrome is complicated by intracranial haemorrhage or brain infarction.

223 with a lower risk of subsequent intracranial haemorrhage or focal neurological deficit (one [2%] of 6

224 associated with a lower risk of intracranial haemorrhage or focal neurological deficit from cerebral

225 CAA versus patients with deep intracerebral haemorrhage or healthy controls.

226 a composite of any symptomatic intracranial haemorrhage or ischaemic stroke, symptomatic intracrania

227 amine does not impair tolerance to simulated haemorrhage or mechanisms responsible for maintenance of

228 sentation and the occurrence of intracranial haemorrhage or persistent or progressive focal neurologi

229 failure of aneurysm treatment, intracranial haemorrhage or residual aneurysm on 1-year imaging.

230 uency of clinically symptomatic intracranial haemorrhage or surrogate haemorrhagic lesions on MRI sca

231 ded 241 024 participants (6255 intracerebral haemorrhage or SVS cases and 233 058 control subjects).

232 p (209 [18.7%] of 1115) had any intracranial haemorrhage (OR 0.75, 0.60-0.94, p=0.0137).

233 OR 1.25, 95% CI 1.06-1.48), gastrointestinal haemorrhage (OR 1.26, 95% CI 1.11-1.43) and intestinal E

234 o -0.02), but a higher risk of intracerebral haemorrhage (OR: 1.64, 95% CI = 1.26-2.13).

235 IA, stroke (ischaemic stroke or intracranial haemorrhage) or death within 90 days of the qualifying s

236 PMCTA was better at identifying trauma and haemorrhage (p=0.008), whereas autopsy was better at ide

237 higher ISCM size (p=0.024), history of prior haemorrhage (p=0.013) and presence of symptoms (p=0.003)

238 Deaths from post-partum haemorrhage peaked 2-3 h after childbirth.

239 acute kidney injury, cardiac arrest, gastric haemorrhage, peritonitis, pneumothorax, septic shock, an

240 ulmonary dysplasia, sepsis, intraventricular haemorrhage, periventricular leukomalacia, and necrotisi

241 ssed included neurological status and annual haemorrhage rates.

242 survivors for association with intracerebral haemorrhage recurrence.

243 c stroke is higher than that of intracranial haemorrhage, regardless of cerebral microbleed presence,

244 Haemorrhage related to pseudoaneurysm occurs most common

245 h risk, which was stronger for intracerebral haemorrhage (relative risk [RR] per 280 g per week 1.58,

246 Haemorrhage remains the most frequent and serious compli

247 eater frequency of complete intraventricular haemorrhage removal via alteplase produces gains in func

248 ing acute ischaemic stroke and intracerebral haemorrhage, results in neuronal cell death and the rele

249 1.07, P = 0.04) and recurrent intracerebral haemorrhage risk (hazards ratio, 1.45, P = 0.006).

250 e associated with both primary intracerebral haemorrhage risk (odds ratio, 1.07, P = 0.04) and recurr

251 s of periprocedural symptomatic intracranial haemorrhage (RR=0.59, 95% CI 0.43 to 0.81; p=0.001) and

252 UIA and the risk of subsequent subarachnoid haemorrhage (SAH) by follow-up on intensive medical trea

253 on the woman was diagnosed with subarachnoid haemorrhage (SAH) in the region of the previously treate

254 l and radiological intensity of subarachnoid haemorrhage (SAH) was evaluated by using the Hunt-Hess a

255 Obstetric haemorrhage, sepsis, and hypertensive disorders of pregn

256 tcomes included cesarean section, postpartum haemorrhage, severe perineal tear, Apgar score at 5 minu

257 ta on predictors of symptomatic intracranial haemorrhage (sICH) in patients who underwent mechanical

258 eight [4%]), pneumonia (seven [3%]), gastric haemorrhage (six [3%]), and gastrointestinal haemorrhage

259 found after adjustment for intraventricular haemorrhage size and thalamic intracerebral haemorrhage.

260 baseline covariates (stability intracerebral haemorrhage size, age, Glasgow Coma Scale, stability int

261 asgow Coma Scale, stability intraventricular haemorrhage size, and clot location).

262 ection of pain medications administered to a haemorrhaging soldier are based upon limited scientific

263 For a haemorrhaging soldier, there are several pain medication

264 emic or haemorrhagic (excluding subarachnoid haemorrhage) stroke 5-42 days before randomisation, who

265 ded by type (ischaemic stroke, intracerebral haemorrhage, subarachnoid haemorrhage, and unspecified t

266 spective cohort of 174 primary intracerebral haemorrhage survivors for association with intracerebral

267 sure (LBNP; a validated model for simulating haemorrhage) test was conducted following the administra

268 ive hazard (aHR) for subsequent intracranial haemorrhage than for ischaemic stroke, but the absolute

269 ents at higher absolute risk of intracranial haemorrhage than ischaemic stroke.

270 der donor age (P = 0.010), and intracerebral haemorrhage/thrombosis in donor (P = 0.023).

271 ategies in patients with acute intracerebral haemorrhage to determine the strength of associations be

272 from the onset of symptoms of intracerebral haemorrhage to randomisation of 3.6 h (2.7-4.4).

273 e Blood Pressure Reduction in Acute Cerebral Haemorrhage Trial (INTERACT2) and the second Antihyperte

274 e Blood Pressure Reduction in Acute Cerebral Haemorrhage Trials (INTERACT 1 and 2).

275 e Blood Pressure Reduction in Acute Cerebral Haemorrhage Trials (INTERACT 1 and 2).

276 e Blood Pressure Reduction in Acute Cerebral Haemorrhage Trials (INTERACT1 and 2).

277 nit with stable, non-traumatic intracerebral haemorrhage volume less than 30 mL, intraventricular hae

278 aHR 4.55 [95% CI 3.08-6.72] for intracranial haemorrhage vs 1.47 [1.19-1.80] for ischaemic stroke; fo

279 Median time to haemorrhage was 2.3 years (0.1-12.3).

280 The overall annual risk of haemorrhage was 5.5% per person year.

281 One death due to gastric haemorrhage was considered treatment related.

282 Haemorrhage was defined as any lesion that was either T1

283 Haemorrhage was defined as clinical worsening in tandem

284 Postpartum haemorrhage was diagnosed in one woman allocated to oxyt

285 The prevalence of symptomatic intracranial haemorrhage was higher in the alteplase group than among

286 The rate of fetomaternal haemorrhage was higher with the intervention (10.5% in t

287 e with frontal infarcts and/or intracerebral haemorrhage were both significantly more likely to have

288 A total of 872 survivors of intracerebral haemorrhage were enrolled and followed for a median of 3

289 Association estimates for intracerebral haemorrhage were negatively correlated with those for sm

290 y, spontaneous, supratentorial intracerebral haemorrhage were randomly assigned (1:1) to receive defe

291 tients with alcoholic cirrhosis and variceal haemorrhage were studied prior to and 1-hour after TIPSS

292 ons of this particular sample, no additional haemorrhages were observed that were missed on magnetic

293 21 intracranial haemorrhages were reported during the 5 years of follow-

294 d magnesium sulphate and 67% with antepartum haemorrhage who needed blood received it.

295 hether attempting to remove intraventricular haemorrhage with alteplase versus saline irrigation impr

296 Hereditary cerebral haemorrhage with amyloidosis-Dutch type (HCHWA-D) is a g

297 ociation between CAA and lobar intracerebral haemorrhage (with its high recurrence risk) is now well

298 ar haemorrhage is a subtype of intracerebral haemorrhage, with 50% mortality and serious disability f

299 occlusion of the tumour vessels followed by haemorrhage within the tissue and the eventual collapse

300 angiopathy can cause life-threatening brain haemorrhages; yet, there is no proof that the transmissi