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1 80% of strokes are ischaemic, as opposed to haemorrhagic.
4 er alone or in combination: grade 3 or worse haemorrhagic adverse events; platelet counts of less tha
5 tion of these venom components to the severe haemorrhagic and coagulopathic pathology of envenoming a
6 of interests were 15 prespecified AESIs (non-haemorrhagic and haemorrhagic stroke, acute myocardial i
8 y emphasise the counter-balancing effects of haemorrhagic and ischaemic complications after stent imp
9 ripheral vessel AVMs, stroke (separately for haemorrhagic and ischaemic), transient ischaemic attack
10 setts General Hospital (Boston, MA, USA) for haemorrhagic and non-haemorrhagic CAA markers, and brain
13 ere the first diagnosis of thrombocytopenic, haemorrhagic and thromboembolic events in primary or sec
14 r, the actual risk stratification models for haemorrhagic and thrombotic events are not validated in
19 fatal stroke, and death from cardiovascular, haemorrhagic, and unknown causes (Antiplatelet Trialists
20 e and small infarcts, many of which appeared haemorrhagic; and microglial activation with microglial
21 ions, increased NCAM expression and produced haemorrhagic angiogenesis in mouse matrigel implants.
22 ith OCCC-like histopathology, culminating in haemorrhagic ascites and a median survival period of 7.5
23 verity of any recurrent stroke (ischaemic or haemorrhagic; assessed using the modified Rankin Scale)
24 erebrovascular physiology, (stage three) non-haemorrhagic brain injury, and (stage four) appearance o
27 l (Boston, MA, USA) for haemorrhagic and non-haemorrhagic CAA markers, and brain tissue samples were
28 cardiac arrest (n=1 in apixaban group), and haemorrhagic cerebral sinus vein thrombosis (n=1 in the
30 in regional blood volumes during a simulated haemorrhagic challenge imposed via lower-body negative p
31 ons in the capacity to withstand a simulated haemorrhagic challenge; however, this capacity is normal
35 Escherichia coli O157:H7 causes diarrhoea, haemorrhagic colitis, and the haemolytic uraemic syndrom
36 asymptomatic shedding, non-bloody diarrhoea, haemorrhagic colitis, haemolytic uraemic syndrome, and d
37 EC) serotype O157:H7, the causative agent of haemorrhagic colitis, has been shown to utilize a cell-t
38 and has been implicated in many outbreaks of haemorrhagic colitis, some of which included fatalities
39 y practiced by recognizing the presence of a haemorrhagic collection and how it predominates in deter
41 ed data on predictors of early ischaemic and haemorrhagic complications after AF-associated ischaemic
42 ad substantially lower 30-day rates of major haemorrhagic complications and net adverse clinical even
44 also associated with early recanalisation or haemorrhagic complications, and early neurologic deterio
49 man who had an ischaemic stroke with massive haemorrhagic conversion requiring a decompressive hemicr
52 topenia (BNP; previously known as idiopathic haemorrhagic diathesis and commonly known as bleeding ca
56 apoptosis.Importance Responsible for severe haemorrhagic disease in domestic pigs and wild boar, cla
57 an swine fever virus (ASFV) causes a lethal, haemorrhagic disease in domestic swine that threatens pi
58 nce that causes severe leukopenia, fever and haemorrhagic disease in domesticated pigs and wild boar
61 known as bleeding calf syndrome) is a novel haemorrhagic disease of young calves which has emerged i
68 had recurrent strokes (12 ischaemic and two haemorrhagic) during a median follow-up of 2.8 years (IQ
69 io 0.51; 95% CI 0.31-0.85) but not for major haemorrhagic events (0.88, 0.74-1.06) or death (0.82, 0.
70 vein thrombosis, or pulmonary embolism) and haemorrhagic events (symptomatic intracranial or signifi
74 -PCI infusion or heparin (Minimizing Adverse Haemorrhagic Events by TRansradial Access Site and Syste
75 led in the MATRIX-Access (Minimizing Adverse Haemorrhagic Events by Transradial Access Site and Syste
76 sub-study of the MATRIX (Minimizing Adverse Haemorrhagic Events by Transradial Access Site and Syste
79 uartiles of predicted risk of thrombotic and haemorrhagic events with random effect meta-analysis.
83 recurrent clinically diagnosed ischaemic or haemorrhagic (excluding subarachnoid haemorrhage) stroke
86 ultiorgan dysfunction in COVID-19 and dengue haemorrhagic fever (DHF) are two diseases that can assoc
89 equently and results in Lassa fever, a viral haemorrhagic fever (VHF) associated with a high case fat
98 nd Marburg filoviruses cause a rapidly fatal haemorrhagic fever in humans for which no approved antiv
100 e causative agents of a severe form of viral haemorrhagic fever in man, designated Ebola haemorrhagic
101 us, was associated with an outbreak of acute haemorrhagic fever in Mangala, Democratic Republic of th
102 mic Large DNA Virus that causes an incurable haemorrhagic fever in pigs with a high impact on global
108 vary within Africa, with regions where viral haemorrhagic fever outbreaks have previously occurred (e
109 gests that the coagulopathy of Crimean-Congo haemorrhagic fever relates to platelet dysfunction.
111 ation testing in patients with Crimean-Congo haemorrhagic fever to increase our understanding of the
112 Virus detection using MSSPE arboviral or haemorrhagic fever viral panels was comparable in sensit
116 ses like Rift Valley fever and Crimean-Congo haemorrhagic fever viruses continue to emerge in new geo
117 arito, Junin and Sabia viruses are New World haemorrhagic fever viruses that do not use alpha-dystrog
118 of tick-borne encephalitis and Crimean-Congo haemorrhagic fever viruses, have changed their geographi
119 15, 65 patients with confirmed Crimean-Congo haemorrhagic fever were recruited and had blood taken at
120 ied by muroid rodents and cause the diseases haemorrhagic fever with renal syndrome (HFRS) and hantav
121 egative association between the incidence of haemorrhagic fever with renal syndrome and the species r
122 ecies richness and incidence of rodent-borne haemorrhagic fever with renal syndrome in the human popu
125 haemorrhagic fever in man, designated Ebola haemorrhagic fever, and are endemic in regions of centra
126 haemorrhagic fevers in Africa, Crimean-Congo haemorrhagic fever, Ebola virus disease, Lassa fever, an
142 ubnational pandemic potential for four viral haemorrhagic fevers in Africa, Crimean-Congo haemorrhagi
144 Of the multiple arenaviruses that cause haemorrhagic fevers in the Americas, all lack reliable t
145 apies for emerging diseases, including viral haemorrhagic fevers such as those caused by Ebola virus
146 onse syndromes in bacterial sepsis and viral haemorrhagic fevers, and anticoagulants can be effective
149 was associated with several inflammatory and haemorrhagic gastrointestinal diseases, including oesoph
150 icant differences were found between the non-haemorrhagic group and the haemorrhagic group regarding
151 d between the non-haemorrhagic group and the haemorrhagic group regarding the number of outflows (p =
152 h approximately 15% of strokes in adults are haemorrhagic, half of incident strokes in children are h
153 6.3%) in children, and tumours (24.6%), post-haemorrhagic hydrocephalus (16.2%) and idiopathic normal
154 stream regulator of Yap that can cause fetal haemorrhagic hydrocephalus, deregulates Yap in the devel
155 ral CB1 cannabinoid receptors contributes to haemorrhagic hypotension, and anandamide produced by mac
156 Lassa virus (LASV) can cause Lassa fever, a haemorrhagic illness with an estimated fatality rate of
157 and cortical superficial siderosis-a new CAA haemorrhagic imaging signature and (b) whether acute cSA
159 in 88 (29.3%) patients, among which 62 were haemorrhagic infarction and 26 were parenchymal haemorrh
162 al haemorrhage occur after the initial acute haemorrhagic insult subsides, and represent one of its m
165 was fatal or disabling stroke (ischaemic or haemorrhagic), intracranial haemorrhage, or clinically s
166 the periods post-first dose of BNT162b2 for haemorrhagic (IRR 1.47, 95%CI: 1.04-2.08) and idiopathic
167 S-CoV-2 infection the risk was increased for haemorrhagic (IRR 1.49, 95%CI: 1.15-1.92), VTE (IRR 5.63
168 (ARIA-E) in brain parenchyma or sulci or as haemorrhagic lesions (ARIA-H) in the form of cerebral mi
170 hat cerebral amyloid angiopathy (CAA) causes haemorrhagic lesions in cerebellar cortex as well as sub
171 omatic intracranial haemorrhage or surrogate haemorrhagic lesions on MRI scans, whereas later DOAC-ad
172 y with prolonged bleeding and progression of haemorrhagic lesions, the risk of hypercoagulopathy with
173 rosis and other white-matter diseases (acute haemorrhagic leucoencephalitis, leucodystrophies and cen
174 cular death (both overall and in subgroups), haemorrhagic major adverse cardiovascular events, and fu
175 investigated associations with ischaemic and haemorrhagic manifestations of small vessel disease [sma
176 tro-orbital pain, myalgia, arthralgia, rash, haemorrhagic manifestations, and leukopenia; fever and a
180 bsequent collapse of tumours, similar to non-haemorrhagic necrosis in ischaemia and unlike haemorrhag
181 aemorrhagic necrosis in ischaemia and unlike haemorrhagic necrosis induced by tumour necrosis factor.
184 getative cultures of wild-type CN3685 caused haemorrhagic necrotizing enteritis in rabbit ileal loops
185 2 QS system, is required for CN3685 to cause haemorrhagic necrotizing enteritis, apparently because t
186 ical superficial siderosis is an established haemorrhagic neuroimaging marker of cerebral amyloid ang
188 duals experienced either a thrombocytopenic, haemorrhagic or thromboembolic event during the study pe
192 incidence of childhood stroke (ischaemic and haemorrhagic) range from 2.3 to 13.0 per 100,000 childre
195 rct growth, enhance reperfusion, or decrease haemorrhagic risk has gained renewed interest with the h
200 t late seizure following an immediately post-haemorrhagic seizure; and (ii) investigate the effect of
203 Adults (age >=16 years) with trauma-related haemorrhagic shock and hypotension (defined as systolic
204 olar epithelium in the early phase following haemorrhagic shock by attenuating NO-mediated oxidative
205 ort across the alveolar epithelium following haemorrhagic shock is mediated by reactive nitrogen spec
207 rease in blood pressure in rats subjected to haemorrhagic shock, whereas similar treatment of normote
211 and there were significant reductions in non-haemorrhagic stroke (131 [2.8%] vs 174 [3.8%]; RR 0.75,
212 number of people with incident ischaemic and haemorrhagic stroke (37% and 47% increase, respectively)
214 stroke (RR 68%, 95% CI 1.52-1.87, P < .001), haemorrhagic stroke (RR 114%, 95% CI 1.74-2.64, P < .001
215 significant (0.20%vs 0.21% per year, p=0.4: haemorrhagic stroke 0.04%vs 0.03%, p=0.05; other stroke
216 tated regimens than in primary intervention (haemorrhagic stroke 15 [0.7%] vs two [0.1%], p=0.0014; t
217 mg), and one death (<1%; upadacitinib 15 mg, haemorrhagic stroke [ruptured aneurysm]) were reported i
219 icant increase of 22% (5-30) in incidence of haemorrhagic stroke and a 6% (-7 to 18) non-significant
220 e (cSVD) is a leading cause of ischaemic and haemorrhagic stroke and a major contributor to dementia.
222 amyloid angiopathy, an untreatable cause of haemorrhagic stroke and vascular cognitive impairment.
223 <0.0001), with a non-significant increase in haemorrhagic stroke but reductions of about a fifth in t
225 of grip strength at 4 weeks post-stroke and haemorrhagic stroke explained the underestimated classif
226 mortality data suggest that the incidence of haemorrhagic stroke has fallen in the past 20 years, but
227 andardised mortality rates for ischaemic and haemorrhagic stroke have decreased in the past two decad
229 country-specific estimates for ischaemic and haemorrhagic stroke incidence, mortality, mortality-to-i
231 clinical endpoints (all cause mortality, non-haemorrhagic stroke or systemic embolism, and major blee
233 e.g. new-onset diabetes mellitus or possibly haemorrhagic stroke) and long-term data are needed to ad
234 eg, neuroprotective drugs and treatments for haemorrhagic stroke) options in the prehospital setting,
235 ity, myocardial infarction, and ischaemic or haemorrhagic stroke), hospital events for heart failure
237 15 prespecified AESIs (non-haemorrhagic and haemorrhagic stroke, acute myocardial infarction, deep v
238 d systemic embolic events, ischaemic stroke, haemorrhagic stroke, all-cause mortality, myocardial inf
239 e, 0.58 million deaths (0.57 to 0.60) due to haemorrhagic stroke, and 0.34 million deaths (0.32 to 0.
241 management, reversal of warfarin effects in haemorrhagic stroke, and management of cerebral emergenc
242 g ischaemic heart disease, ischaemic stroke, haemorrhagic stroke, and other cardiovascular diseases)
244 tients had CNS events (six ischaemic and one haemorrhagic stroke, eight transient ischaemic attacks,
246 lure, atrial fibrillation, all-cause stroke, haemorrhagic stroke, ischaemic stroke, hypertension, aor
248 myocardial infarction or coronary death, non-haemorrhagic stroke, or any arterial revascularisation p
249 hough migraine appears to be associated with haemorrhagic stroke, the migraine aura status has a smal
250 orta, atrial fibrillation, ischaemic stroke, haemorrhagic stroke, venous thromboembolism, cardiovascu
251 troke events, including ischaemic stroke and haemorrhagic stroke, were identified through hospital an
259 ) for ischaemic stroke; 1.56 (1.19-2.05) for haemorrhagic stroke; 1.84 (1.59-2.13) for unclassified s
260 nts aged 18-83 years with ischaemic (ie, non-haemorrhagic) stroke with a baseline National Institutes
261 e were no apparent differences in numbers of haemorrhagic strokes (24 [0.4%] vs 25 [0.4%]) or deaths
262 n encountered in patients with ischaemic and haemorrhagic strokes, subarachnoid haemorrhage, cerebrov
268 enesis and has been implicated in hereditary haemorrhagic telangiectasia (HHT), atherosclerosis, tumo
269 es on a human vascular dysplasia, hereditary haemorrhagic telangiectasia (HHT), wherein arterial and
272 gene with the inherited disorder Hereditary Haemorrhagic Telangiectasia Type 1 (HHT1), a disease cha
273 mutations in the ALK1 gene cause hereditary haemorrhagic telangiectasia type 2 (HHT2), a disabling d
274 vascular lesions associated with hereditary haemorrhagic telangiectasia, especially occult arteriove
276 ardiovascular disorders including hereditary haemorrhagic telangiectasia, pulmonary arterial hyperten
282 mia (HR 2.91, 95% CI 1.08 to 7.84) and early haemorrhagic transformation (HR 5.35, 95% CI 2.22 to 12.
284 Complications of rt-PA therapy, such as haemorrhagic transformation and angio-oedema, are review
285 administration of tPA (10 mg/kg) resulted in haemorrhagic transformation in the ischaemic territory 1
288 ilable to rescue ischaemic brain tissue; the haemorrhagic transformation that can cause severe functi
289 ter delayed tPA treatment in ischaemic mice, haemorrhagic transformation was significantly decreased,
290 l venous thrombosis, head trauma, or tumour; haemorrhagic transformation within an infarct; and refer
291 gical complications, such as brain oedema or haemorrhagic transformation, occur earlier than do medic
292 t-emergent adverse event was non-symptomatic haemorrhagic transformation, which occurred in 17 (31%)
298 e clearest association was with the familial haemorrhagic variant where all four families tested had
299 oke was subdivided into fatal, ischaemic and haemorrhagic, with higher magnitude associations for fat