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1 idative stress/damage within the human scalp hair bulb.
2 dermal papilla (DP), that is embedded in the hair bulb.
3 yer of the IFE and in the dermal papilla and hair bulb.
4 bulge, and subsequently spreads towards the hair bulb.
5 haft fragility and untimely apoptosis in the hair bulb.
6 ted with increased cell proliferation in the hair bulb.
7 s, and the proliferative cells of the anagen hair bulb.
8 and, for the cyclic formation of new anagen hair bulbs.
10 type has been described as adendritic, since hair bulb and Harderian gland melanocytes appear to be r
11 stem cells jumped and targeted the bulge and hair bulb and other areas of the resident nude mouse pel
12 xpression onset at anagen IIIa above the new hair bulb and subsequent spreading towards the bulge.
13 mal tissues, except for keratinocytes of the hair bulb and syncytiotrophoblasts of the placenta, but
14 tion was detectable in DNA from the father's hair bulbs and buccal cells but not his white blood cell
15 Dermal papilla cells (DPCs) located in the hair bulb are the main site of androgen activity in the
16 (SC) are maintained in the bulge region, and hair bulbs at the base contain rapidly dividing, yet gen
19 significant reduction in apoptotic (TUNEL+) hair bulb cells, thus mitigating the degree of follicle
21 sequent hair cycle, i.e., after a new anagen hair bulb has been constructed, which points to the exis
22 mal hair follicles was virtually absent from hair bulbs, it was expressed by a subset of keratinocyte
24 rmal transfer of pigment granules to ectopic hair bulb locations, extrafollicular melanin incontinenc
25 cted in progenitor cell compartments; in the hair bulb matrix and basal epidermal layer, a small subc
30 genitor population that resides in the lower hair bulb of anagen-phase follicles and expresses Hopx.
31 genically-active melanocytes from the anagen hair bulbs of affected human scalp remains unclear, oxid
32 , and a novel role for Hopx-expressing lower hair bulb progenitor cells in contributing to stem cell
33 cally, after low-dose IR, keratin 5(+) basal hair bulb progenitors, rather than bulge SCs, were quick
34 e full transcriptome profile analysis of the hair bulb region of normal and miniaturized hair follicl