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1 idative stress/damage within the human scalp hair bulb.
2 dermal papilla (DP), that is embedded in the hair bulb.
3 yer of the IFE and in the dermal papilla and hair bulb.
4  bulge, and subsequently spreads towards the hair bulb.
5 haft fragility and untimely apoptosis in the hair bulb.
6 ted with increased cell proliferation in the hair bulb.
7 s, and the proliferative cells of the anagen hair bulb.
8  and, for the cyclic formation of new anagen hair bulbs.
9             TSP-1 expression was absent from hair bulb and dermal papilla cells during early to mid-a
10 type has been described as adendritic, since hair bulb and Harderian gland melanocytes appear to be r
11 stem cells jumped and targeted the bulge and hair bulb and other areas of the resident nude mouse pel
12 xpression onset at anagen IIIa above the new hair bulb and subsequent spreading towards the bulge.
13 mal tissues, except for keratinocytes of the hair bulb and syncytiotrophoblasts of the placenta, but
14 tion was detectable in DNA from the father's hair bulbs and buccal cells but not his white blood cell
15   Dermal papilla cells (DPCs) located in the hair bulb are the main site of androgen activity in the
16 (SC) are maintained in the bulge region, and hair bulbs at the base contain rapidly dividing, yet gen
17                            In the developing hair bulb, bcl-xL transgene expression was observed excl
18     After high-dose IR, when both matrix and hair bulb cells were depleted, the surviving outer root
19  significant reduction in apoptotic (TUNEL+) hair bulb cells, thus mitigating the degree of follicle
20              TSC2-null cells surrounding the hair bulb expressed markers of the dermal sheath and der
21 sequent hair cycle, i.e., after a new anagen hair bulb has been constructed, which points to the exis
22 mal hair follicles was virtually absent from hair bulbs, it was expressed by a subset of keratinocyte
23  and nuclear accumulation of beta-catenin in hair bulb keratinocytes.
24 rmal transfer of pigment granules to ectopic hair bulb locations, extrafollicular melanin incontinenc
25 cted in progenitor cell compartments; in the hair bulb matrix and basal epidermal layer, a small subc
26 can generate epidermis, sebaceous gland, and hair bulb matrix cells.
27                       After 3-5 d, surviving hair bulb melanocytes express c-kit receptor, proliferat
28                               After 24-48 h, hair bulb melanocytes expressing Fas undergo apoptosis.
29 nt) and phaeomelanin (yellow/red pigment) by hair bulb melanocytes.
30 genitor population that resides in the lower hair bulb of anagen-phase follicles and expresses Hopx.
31 genically-active melanocytes from the anagen hair bulbs of affected human scalp remains unclear, oxid
32 , and a novel role for Hopx-expressing lower hair bulb progenitor cells in contributing to stem cell
33 cally, after low-dose IR, keratin 5(+) basal hair bulb progenitors, rather than bulge SCs, were quick
34 e full transcriptome profile analysis of the hair bulb region of normal and miniaturized hair follicl
35 ted in formation of fewer HFs with decreased hair bulb size and thinner hair production.
36                                         When hair bulb tissues were examined separately, epithelial m