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1 l epithelial EBV in the pathogenesis of oral hairy leukoplakia.
2  not sufficient for the pathogenesis of oral hairy leukoplakia.
3  of the tongue epithelium in lesions of oral hairy leukoplakia.
4 t of EBV-associated diseases other than oral hairy leukoplakia.
5 om NPC biopsies and a BL biopsy but not from hairy leukoplakia, a permissive EBV infection.
6 -seropositive subjects with and without oral hairy leukoplakia, a replicative EBV-associated epitheli
7 odds ratio (OR) = 0.68, p = 0.0035) and oral hairy leukoplakia (adjusted OR = 0.67, p = 0.033) when c
8 n-Barr virus (EBV) replication characterizes hairy leukoplakia, an oral epithelial lesion typically o
9 and patients with either oral candidiasis or hairy leukoplakia and a low CD4:CD8 cell ratio should be
10         EBV replication was detected in both hairy leukoplakia and normal tongue tissues.
11 ription from Zp, with all Z(+) cells in oral hairy leukoplakia being BLIMP1(+).
12                                              Hairy leukoplakia (HL) is a proliferative lesion of the
13              Epstein-Barr virus (EBV) causes hairy leukoplakia (HL), a benign lesion of oral epitheli
14 sis (EC), pseudomembranous candidiasis (PC), hairy leukoplakia (HL), and warts was computed over foll
15 ent membrane protein (LMP)-1 is expressed in hairy leukoplakia (HL), but data on LMP-1 sequence varia
16 immunodeficiency virus (HIV)-associated oral hairy leukoplakia (HLP) and Epstein-Barr virus (EBV) rep
17 his study, EBV strains were identified in 25 hairy leukoplakia (HLP) biopsies and six matched periphe
18 F-7 protein expression in vivo in lesions of hairy leukoplakia (HLP) in which there is abundant EBV r
19              The immunodeficiency-associated hairy leukoplakia (HLP) lesion is the only pathologic ma
20 nes associated with the pathogenesis of oral hairy leukoplakia (HLP).
21  virus (EBV) replicates productively in oral hairy leukoplakia (HLP).
22 duce epithelial cell lesions resembling oral hairy leukoplakia in AIDS patients.
23 found a high coprevalence of candidiasis and hairy leukoplakia in these subjects.
24 immunodeficiency virus (HIV)-candidiasis and hairy leukoplakia-in 152 HIV-infected blood transfusion
25 s, such as nasopharyngeal carcinoma and oral hairy leukoplakia, indicating that the virus can infect
26 ence of HHV-8 DNA in both the EBV-associated hairy leukoplakia lesions and in the EBV-associated AIDS
27 cells lytically infected with EBV (from oral hairy leukoplakia lesions) express much more FAS than un
28         EBV was detected by Southern blot in hairy leukoplakia lesions, in a subset of AIDS-related l
29  as nasopharyngeal carcinoma (NPC), and oral hairy leukoplakia (OHL) lesions that have lytic infectio
30 ses are due to lytic infection (such as oral hairy leukoplakia) or latent infection (such as nasophar
31 roduce diverse pathologies ranging from oral hairy leukoplakia to nasopharyngeal carcinoma, from infe
32                                         Oral hairy leukoplakia tongue tissue, which contains the lyti
33                                 A history of hairy leukoplakia was less predictive of persistence of
34 lication in vivo, resulting in resolution of hairy leukoplakia when it was present.