ライフサイエンスコーパス: handgrip

1 ty contractile exercise (25% and 80% maximal handgrip).

2 reased within 1 to 2 seconds of the onset of handgrip.

3 d all of the vasodilatation to contralateral handgrip.

4 ed by damage in brain areas activated during handgrip.

5 one (-54 +/- 11 ms), and also with PS during handgrip (+10 +/- 10 ms) compared with PS alone (-74 +/-

6 vascular resistance increases (basal versus handgrip, 17 +/- 1 versus 26 +/- 2 U; P = .01) in respon

7 l cortical vascular resistance (basal versus handgrip, 18 +/- 1 versus 25 +/- 3 U; P = .002); and (4)

8 e (BP; Finapres) were measured during static handgrip (20 s) at 10% and 70% of maximum voluntary cont

9 During mild to moderate handgrips (20-33% MVC) that do not evoke reflex-sympathe

10 l cortical vascular resistance (basal versus handgrip, 20 +/- 1 versus 25 +/- 2 U; P = .04); (3) the

11 er post-flight in all subjects before static handgrip (26 +/- 4 post- vs. 15 +/- 4 bursts min(-1) pre

12 pared with metoprolol and placebo (isometric handgrip -3.5 U for carvedilol versus -1.2 U for metopro

13 Likewise, when static handgrip (30 % MVC) was performed to fatigue, MSNA incre

14 During sustained handgrip (30% maximum voluntary contraction), peak renal

15 y decrease in renal blood flow (basal versus handgrip, 4.2 +/- 0.2 versus 3.5 +/- 0.3 mL.min-1.g-1; P

16 cortical blood flow decreases (basal versus handgrip, 4.4 +/- 0.1 versus 3.5 +/- 0.1 mL.min-1.g-1; P

17 1) 7-minute baseline; (2) 2-minute isometric handgrip (40% maximal voluntary contraction) or rhythmic

18 During high intensity handgrip, (45% MVC), contraction-induced decreases in mu

19 % maximal voluntary contraction) or rhythmic handgrip (50% and 30% maximal voluntary contraction) exe

20 During contralateral handgripping after stellate block, blood flow in the res

21 rcise circulatory occlusion at 40% isometric handgrip (all P<0.05) and HG only at 50% and 30% rhythmi

22 P<0.05) and HG only at 50% and 30% rhythmic handgrip (all P<0.05).

23 unexpectedly increased by 54 +/- 11% during handgrip alone.

24 peripheral afferent stimulation produced by handgrip and a cold pressor test in humans.

25 e subjects, vasomotor responses to isometric handgrip and cold pressor test did not differ between tr

26 m vascular resistance responses to isometric handgrip and cold pressor test were determined by plethy

27 55.3%), we compared the responses to dynamic handgrip and during a 3-minute period of posthandgrip re

28 Handgrip and forearm vasodilation yielded no haplotype d

29 i-Mental State Examination (MMSE) score, and handgrip and handheld dynamometer strength.

30 In addition, simulated handgrip and intermittent forearm compression produced b

31 e is greater in men than women during static handgrip and LBNP.

32 limb function and increased activations with handgrip and median nerve stimulation, but reduced activ

33 ty and blood pressure responses to fatiguing handgrip and post-exercise circulatory occlusion were si

34 he left primary motor cortex leg area during handgrip and the left primary sensory cortex face area d

35 al activation in response to right-sided (i) handgrip; and (ii) median and tibial nerve stimulation w

36 patient (I.G.) was quite unable to open her handgrip appropriately when directly reaching out to pic

37 unctional composite scores: muscle strength (handgrip, arm, and leg) and mobility (timed-up-and-go, c

38 d at rest and during right and left rhythmic handgrip before and 4 to 7 weeks after right TST.

39 thy volunteers performed fatiguing isometric handgrip before and after a local infusion of pyridoxine

40 e (FVC) during infusion of ADO to FVC during handgripping before and after infusion of dipyridamole (

41 es in peak heart rate response during static handgrip between groups (patients with HFpEF versus cont

42 ed exercise hyperaemia during heavy rhythmic handgripping, but vasodilator responses to exogenous ADO

43 MSNA during post-handgrip circulatory arrest was higher post- than pre- o

44 In contrast, during post-handgrip circulatory arrest, which isolates muscle metab

45 s during a 3-min sustained maximal voluntary handgrip contraction.

46 rticipants completed a series of 15-s static handgrip contractions at 20, 40 and 60% of maximal volun

47 lthy subjects in two tasks: (1) intermittent handgrip contractions at 20, 40, 60, and 80% of maximal

48 thy subjects performed repetitive unilateral handgrip contractions that induced significant muscle fa

49 ndgrip, whereas the pressor response to left handgrip did not change.

50 There was no effect at 6 months for handgrip (difference, 2.0 kg [95% CI, -1.3 to 5.4], P =

51 Handgrip strength was assessed with a handgrip dynamometer and skeletal muscle mass was estima

52 X-ray absorptiometry, muscle strength with a handgrip dynamometer, and blood biochemical indexes of n

53 sment (cognition), manual muscle testing and handgrip dynamometry (muscle and/or nerve function), and

54 ater reliability of handheld dynamometry and handgrip dynamometry was assessed in one study, and resu

55 Interrater reliability of handgrip dynamometry was very good in two studies (intra

56 subjective global assessment, anthropometry, handgrip dynamometry, biochemical and amino acid profile

57 arm (-12 +/- 1%) that were attenuated during handgrip exercise (-3 +/- 1%; P < 0.05).

58 2 agonist) in 10 healthy men during rhythmic handgrip exercise (10-15 % of maximum) and during a cont

59 energic receptor stimulation during rhythmic handgrip exercise (15% maximum voluntary contraction) an

60 (alpha(2)-agonist) during moderate rhythmic handgrip exercise (15% maximum voluntary contraction), d

61 etomidine (alpha(2)-agonist) during rhythmic handgrip exercise (15% MVC), a control non-exercise vaso

62 voluntary contraction; MVC); (iii) moderate handgrip exercise (15% MVC); and (iv) mild or moderate h

63 Seven subjects performed 2 min of isometric handgrip exercise (35% of maximal voluntary contraction)

64 ependent dilator) during resting conditions, handgrip exercise (5% maximum voluntary contraction) or

65 ACh or Protocol 2: low dose ATP); (ii) mild handgrip exercise (5% maximum voluntary contraction; MVC

66 measured at rest and during graded rhythmic handgrip exercise (5%, 15% and 25% of maximum voluntary

67 cle (forearm) blood flow (FBF) during graded handgrip exercise (5, 15, 25% maximal voluntary contract

68 alsalva manoeuvre (by approximately 45%) and handgrip exercise (by approximately 27%) with unaffected

69 sodilatory control, and (2) dynamic rhythmic handgrip exercise (EX; 15% maximal voluntary contraction

70 sed by vasomotor reactivity during isometric handgrip exercise (IHE), was recently quantified noninva

71 in contracting muscle during heavy rhythmic handgrip exercise (n = 4).

72 d when forearm ischemia was maintained after handgrip exercise (posthandgrip circulatory arrest).

73 xercise (15% MVC); and (iv) mild or moderate handgrip exercise + infusion of ACh or ATP to augment en

74 forearm vascular resistance at rest, during handgrip exercise and after transient arterial occlusion

75 (ml/min/100 ml) at rest and during rhythmic handgrip exercise and after transient arterial occlusion

76 during stress manoeuvres (mental arithmetic, handgrip exercise and cold pressor test).

77 energic receptor stimulation during rhythmic handgrip exercise and during a control non-exercise vaso

78 Graded handgrip exercise and post-handgrip ischemic arrest were

79 r transduction was evaluated during ischemic handgrip exercise and postexercise ischemia, and it was

80 nce (FVC) responses during 5 min of rhythmic handgrip exercise at 20% maximal voluntary contraction i

81 In protocol 2, rhythmic handgrip exercise at 35% maximum voluntary contraction w

82 ary exercise testing (peak Vo(2)) and static handgrip exercise at 40% of maximal voluntary contractio

83 .5 tesla before, during, and after isometric handgrip exercise at a level that was 30 percent of the

84 Handgrip exercise changes ocular perfusion pressure free

85 mediated vasoconstriction was blunted during handgrip exercise compared to SNP (DeltaFVC: SNP: -32 +/

86 trictor response to PE was attenuated during handgrip exercise compared to SNP (DeltaFVC: SNP: -44 +/

87 ponses during hypoxia and moderate intensity handgrip exercise compared to young adults, and also ten

88 Wave energy also decreased during handgrip exercise due to increased heart rate.

89 c reactivity during mental stress and static handgrip exercise following 8 weeks of MBSR but not afte

90 rotocol 1, healthy volunteers performed mild handgrip exercise for 3 min, then transitioned to modera

91 y subjects performed maximal-effort rhythmic handgrip exercise for 5 min under control conditions (CO

92 vidence of an abnormal metabolic response to handgrip exercise in at least some women with chest pain

93 During sustained handgrip exercise in heart failure, both the magnitude a

94 conductance (CVC) decreases during isometric handgrip exercise in heat stressed individuals, and we h

95 the influence of blood flow occlusion during handgrip exercise on neuromuscular fatigue development a

96 (15% maximal voluntary contraction) rhythmic handgrip exercise or adenosine infusion.

97 oxia (80% SpO2 ), and during graded rhythmic handgrip exercise that was similar between groups (5, 15

98 tic traffic to the resting forearm, rhythmic handgrip exercise to fatigue followed by post-exercise i

99 rther aim was to examine the effect of local handgrip exercise training on radial artery L-FMC and fl

100 ted and randomly assigned to either a 6-week handgrip exercise training program (N=9) or a nonexercis

101 m vascular conductance (FVC) during rhythmic handgrip exercise under control conditions and during lo

102 re measured at baseline and during isometric handgrip exercise using a 3.0T magnetic resonance imagin

103 Reduced DeltaMSNA during handgrip exercise was also observed, while DeltaMSNA dur

104 e gap, nine healthy males performed rhythmic handgrip exercise with simultaneous measurements by NIRS

105 ng a sympatho-excitatory stimulus (isometric handgrip exercise) after either exercise (60 min cycling

106 e deoxygenation (systemic hypoxia and graded handgrip exercise) with age, which was caused by reduced

107 ed brachial artery blood flow during maximal handgrip exercise, 6-minute walk test, maximal oxidative

108 RI was performed before and during isometric handgrip exercise, an endothelial-dependent stressor, an

109 were quantified before and during isometric handgrip exercise, an endothelial-dependent stressor.

110 (2)) in cubital venous blood at rest, during handgrip exercise, and during recovery in 13 patients wi

111 forearm vascular conductance during ischemic handgrip exercise, despite a normal pressor response, su

112 Retina/choroid BF increases during brief handgrip exercise, paralleling increases in mean arteria

113 During handgrip exercise, patients with LVEF >60% had higher in

114 Graded handgrip exercise, posthandgrip circulatory arrest, and

115 r data indicate that during graded intensity handgrip exercise, the reduced FVC and subsequently lowe

116 rest and during moderate-intensity rhythmic handgrip exercise.

117 es of oxygen delivery and utilization during handgrip exercise.

118 ow during mild (10% MVC) continuous rhythmic handgrip exercise.

119 r of the muscle metaboreflex during ischemic handgrip exercise.

120 alva manoeuvre but remained unaltered during handgrip exercise.

121 also obtained during Valsalva manoeuvre and handgrip exercise.

122 ) restored skeletal muscle blood flow during handgrip exercise.

123 ympathetic vasomotor outflow during rhythmic handgrip exercise.

124 alva manoeuvre but remained unchanged during handgrip exercise.

125 uctance catheter during basal conditions and handgrip exercise.

126 nce imaging (MRI) at rest and during maximal handgrip exercise.

127 s 26 +/- 2 U; P = .01) in response to static handgrip exercise; (2) central command and/or the mechan

128 KCl) at rest; (2) mild or moderate intensity handgrip exercise; and (3) combined mild exercise + ACh,

129 adrenergic stimulation achieved through post-handgrip-exercise ischaemia (PEI) and beta1 -adrenergic

130 The ergoreceptor test involved two 5-minute handgrip exercises.

131 During handgrip, femoral vascular conductance was reduced 47.2

132 ollowing leg cycling exercise, (3) isometric handgrip followed by PEI.

133 n was assessed using standing broad jump and handgrip for strength, and the shuttle-run test for card

134 The handgrip force and electromyograms (EMG) of the finger f

135 f 50% MVC contractions resulted in decreased handgrip force in the contracting hand, and decreased RT

136 acting hand, and decreased RTs and increased handgrip force in the contralateral hand.

137 The task involved trading handgrip force production against monetary benefits.

138 ings of scalp electroencephalographic (EEG), handgrip force, and finger flexor surface electromyograp

139 Both handgrip (HG) and disengagement of baroreflexes with low

140 total LBM, appendicular lean mass (ALM), and handgrip (HG) and knee extension (KE) strength were incl

141 (BMI) and lean body mass (LBM) depletion on handgrip (HG) force and inspiratory muscle function (IMF

142 During PEI following handgrip, HR was similarly elevated from rest under cont

143 Interestingly, post-handgrip hyperaemia is greater in women than men and is,

144 ate were measured during fatiguing isometric handgrip (IHG) at 30% maximum voluntary contraction and

145 Isometric handgrip (IHG) exercise increases sweat rate and arteria

146 arm flow and autonomic responses to ischemic handgrip in young and older subjects.

147 as they executed their fastest and strongest handgrips in response to a visual cue, which was accompa

148 Graded handgrip exercise and post-handgrip ischemic arrest were used to clarify the reflex

149 ilt, during the Valsalva maneuver and during handgrip isometric exercise.

150 um and phentolamine prior to another bout of handgripping, little or no vasodilatation was seen eithe

151 Secondary outcomes included handgrip maximal strength, functional performance, blood

152 Eight participants performed 200 handgrip maximal voluntary contractions (MVCs) with simu

153 ry responses to stressful stimuli (sustained handgrip, maximal forearm ischemia, mental stress, and t

154 Measurements included quadriceps and handgrip maximum voluntary isometric force and the relax

155 n SD HGS in the 1809 patients with available handgrip measurement was 17.0 7.1 kg for females and 28.

156 During handgrip, MSNA increased in all groups (all P<0.05); in

157 lly from those obtained in a 2-min sustained handgrip MVC published in a recent report, in which the

158 icant fatigue were induced; (2) intermittent handgrip MVCs (100 trials) that resulted in significant

159 man participants performed approximately 100 handgrip MVCs (each 2-s contraction was followed by a 1-

160 The fMRI data from the task of intermittent handgrip MVCs differed dramatically from those obtained

161 CI, -4.07 to -1.30; P < .001) and decreased handgrip myotonia on clinical examination (mexiletine, 0

162 Handgrip myotonia was seen in three-quarters of particip

163 myotonia assessment; quantitative measure of handgrip myotonia; and Individualized Neuromuscular Qual

164 h partial flow restriction and PEI following handgrip (P > 0.05 vs. rest).

165 to-stand-10 (p = 0.027), right and left hand handgrip (p = 0.044, p < 0.001), one-heel left leg raise

166 line leg power (p trend = 0.046), and poorer handgrip (p trend = 0.005) were associated with higher a

167 sing post-exercise ischaemia (PEI) following handgrip partially maintains exercise-induced increases

168 (PEI-M) and high (PEI-H) intensity isometric handgrip performed at 25% and 40% maximum voluntary cont

169 In controls, 2 min of static handgrip performed at 33 % or 45 % of maximal voluntary

170 rested for 1 minute followed by 1 minute of handgrip, repeating three times, while maintaining stabl

171 uding the cold pressor test (CPT), sustained handgrip (SHG), and mental stress (MS).

172 e activity (SSNA) during intermittent static handgrip (SHG; at 45% of maximal voluntary contraction;

173 Handgrip significantly increased retina/choroid BF by 25

174 gh muscle volume (3.6%; 95% CI: 0.2%, 7.0%), handgrip strength (2.3 kg; 95% CI: 0.8, 3.7 kg), and 1-R

175 tegory (0.5 [95% CI, 0.2-0.8]; P = .01), and handgrip strength (2.6 kg [95% CI, 0.9-4.2 kg]; P = .002

176 16; 95% CI, -11.31 to -7.00; P < .001), left handgrip strength (6 months: beta = 2.69; 95% CI, 0.96-4

177 = 5.52; 95% CI, 3.70-7.33; P < .001), right handgrip strength (6 months: beta = 2.75; 95% CI, 1.01-4

178 Athletes had superior handgrip strength (Athletes: 55.92 +/- 17.06 vs. CONTROL

179 ity, higher intake was associated with lower handgrip strength (B: -1.4 0.7 kg; P = 0.041).

180 min B-6 tended to be associated with greater handgrip strength (B: 1.5 0.8 kg; P = 0.051), whereas in

181 appendicular skeletal muscle mass (ASM) and handgrip strength (HGS) are key components of sarcopenia

182 Handgrip strength (HGS) has been proposed as an easy-to-

183 Handgrip strength (HGS) is one of the measures of muscul

184 idarm and calf circumference, serum albumin, handgrip strength (HGS), and patient-self assessment of

185 Outcomes included handgrip strength (kPa), knee extensor strength (kg), an

186 elivery (n = 41), protein delivery (n = 35), handgrip strength (n = 40), and other nutritional-relate

187 9), android distribution of fat (P = 0.021), handgrip strength (P = 0.001), standardized summary scor

188 Longer ICU stay was associated with lower handgrip strength (P<0.01) and lower aromatic amino acid

189 ntage (PR, 2.33; 95% CI, 1.21-4.50), reduced handgrip strength (PR, 2.71; 95% CI, 1.44-5.10) and low

190 summary score was related to improvement in handgrip strength (r = -0.51; P < 0.001).

191 e relationship between physical activity and handgrip strength among older adults.

192 th a few additional tests that often include handgrip strength and arm-muscle circumference.

193 Handgrip strength and body mass index (BMI; in kg/m(2))

194 Lower preoperative handgrip strength and branched chain amino acid levels a

195 -providing snacks was associated with better handgrip strength and knee extensor strength at T4 and l

196 Handgrip strength and phase angle improved after LTx (P<

197 cant increase of about 11% in quadriceps and handgrip strength at mid-cycle compared with both the fo

198 3 years improved appendicular lean mass and handgrip strength compared to no treatment, whereas bisp

199 h better physical performance at T4 and less handgrip strength decline.

200 th and knee extensor strength at T4 and less handgrip strength decline.

201 he sum of both hands) and relative (absolute handgrip strength divided by body mass index) handgrip s

202 Lower handgrip strength has been linked to memory impairment,

203 tified analysis, LTL was not associated with handgrip strength in either men or women.

204 We noted no difference in handgrip strength in ROMANA 1 (-1.10 kg [-1.69 to -0.40]

205 Between discharge and 6 months, handgrip strength increased 6.2 times more than did Pi(m

206 t discharge, Pi(max) did not change, whereas handgrip strength increased by 34.8% (P < 0.001).

207 Handgrip strength increased by a mean adjusted differenc

208 Handgrip strength is used as a marker for physical capab

209 Studies have suggested that handgrip strength might be a marker for cardiometabolic

210 were the median change in lean body mass and handgrip strength over 12 weeks and were measured in all

211 y (self-reported) was associated with higher handgrip strength over time in men (coefficient [coef]:

212 ombined resting energy expenditure (REE) and handgrip strength provided a valuable assessment in data

213 was assessed by means of a maximal isometric handgrip strength test and a test of lower-back trunk mu

214 Handgrip strength test results and discomfort ratings di

215 patency of the ulnopalmar arches, as well as handgrip strength tests to examine the isometric strengt

216 We designed an effort measure based on handgrip strength to assess the motivation to earn money

217 .4-43.2) cm H(2)O (53.1% predicted), whereas handgrip strength was 16.4 (95% confidence interval, 14.

218 Baseline handgrip strength was a mediator between HEI and ADL man

219 Mediation by handgrip strength was also tested.

220 Handgrip strength was assessed at baseline.

221 Handgrip strength was assessed with a handgrip dynamomet

222 Handgrip strength was measured at 12 and 25 y, expressed

223 were at least 30% lower than predicted, but handgrip strength was relatively preserved.

224 Handgrip strength was tested by a grip dynamometer and u

225 Gait speed and handgrip strength were obtained, and patients were dicho

226 Females had lower relative handgrip strength when their plasma glucose (p = 0.001)

227 were at risk (p = 0.021), and lower relative handgrip strength when their plasma glucose (p = 0.034)

228 Males had lower absolute handgrip strength when their triglycerides levels were a

229 ance, Medical Research Council sum score and handgrip strength), a full pulmonary function test, and

230 , systolic and diastolic blood pressure, and handgrip strength), behavioural (smoking, alcohol consum

231 physical function assessed (i.e., 4 m walk, handgrip strength).

232 thyroid dysfunction, hearing impairment, and handgrip strength).

233 of the indirect effect was explained through handgrip strength).

234 blood pressure, 0.90 (95% CI, 0.82-0.99) for handgrip strength, and 2.19 (95% CI, 1.96-2.46) for the

235 , cardiorespiratory fitness, blood pressure, handgrip strength, and a combined risk z score in late a

236 by anthropometric assessment, bioimpedance, handgrip strength, and dietary intake (before and 30, 90

237 y symptoms, cognitive functioning, mobility, handgrip strength, and health-related quality of life.

238 mpedance, quadriceps, respiratory muscle and handgrip strength, and physical performance with the Sho

239 he secondary outcomes were body composition, handgrip strength, and physical performance.

240 ests: "Timed Up and Go" test, walking speed, handgrip strength, and standing heel-rises.

241 no statistically significant differences in handgrip strength, delirium rate, intensive care unit mo

242 evaluated by the Fried index, incorporating handgrip strength, gait speed, physical activity, uninte

243 iency, high C-reactive protein levels, lower handgrip strength, hearing impairment, orthostatic hypot

244 lementation affected appendicular lean mass, handgrip strength, knee extension strength, physical per

245 Appendicular lean mass, handgrip strength, leg strength, physical performance, a

246 level of education, percentage body fat, and handgrip strength, low ALM remained independently associ

247 Subjective Global Assessment, handgrip strength, multifrequency bioelectrical impedanc

248 Thigh muscle volume, handgrip strength, one-repetition maximum (1-RM) lower-

249 outcomes were cancer-related fatigue levels, handgrip strength, peak expiratory flow rate, and qualit

250 In Wave 3, a direct assessment of handgrip strength, standing long-jump, and 6-min walk te

251 Physical performance was assessed by handgrip strength, the Short Physical Performance Batter

252 We compared handgrip strength, usual gait speed, timed up and go (TU

253 We find unique evidence of ID for handgrip strength, waist/hip ratio, and visual and audit

254 ical activity was positively associated with handgrip strength, with a moderate dose-response relatio

255 These results suggest that having adequate handgrip strength-a proxy of overall strength capacity-m

256 period in both physical activity levels and handgrip strength.

257 tional indices, anthropometric variables and handgrip strength.

258 Timed Up and Go (TUG) test, and dynamometer handgrip strength.

259 d isokinetic strength of the lower limbs and handgrip strength.

260 In children, the handgrip strength/body mass levels for a low metabolic r

261 from Colombia, using MS relative to weight (handgrip strength/body mass).

262 nificantly increased lean body mass, but not handgrip, strength in patients with advanced non-small-c

263 absolute (r = - 0.17, p = 0.03) and relative handgrip strengths (r = - 0.28, p < 0.01).

264 andgrip strength divided by body mass index) handgrip strengths were collected in 173 Hispanic/Latino

265 suspected myocardial ischemia underwent MRS handgrip stress testing and follow-up evaluation.

266 MSNA) were recorded before and during static handgrip sustained to fatigue at 40 % of maximum volunta

267 ography during a visually cued, incentivized handgrip task in subjects with Parkinson's disease (n =

268 sure by 25%+/-6% (averaged across the entire handgrip task) (P<0.01), but did not change intraocular

269 e by 22%+/-5% (measured at the middle of the handgrip task), and ocular perfusion pressure by 25%+/-6

270 forming an isometric, dynamic visually paced handgrip task.

271 the reference group (normal BMI and highest handgrip tertile), the risk of all-cause mortality incre

272 For participants in the lowest handgrip tertile, there was little difference in the ris

273 ot affected by melatonin during an isometric handgrip test (30% maximum voluntary contraction) and a

274 Basic fitness measures included a handgrip test and 3-minute step test.

275 Isometric handgrip testing (IHGT), a well-established laboratory-b

276 179 +/- 4 cm) completed four constant power handgrip tests to exhaustion under conditions of control

277 Most patients had markedly impaired TUG and handgrip tests, and 21% recalled having fallen more than

278 Subjects performed rhythmic handgrip (thirty 1-sec contractions/min) at 30% maximal

279 more avidly an action involving squeezing a handgrip to earn money.

280 ubjects (3 women, 7 men) performed ischaemic handgripping to fatigue before and after acute local ana

281 Manoeuvres such as contralateral ischaemic handgripping to fatigue that cause vasoconstriction in t

282 excitation evoked by contralateral ischaemic handgripping to fatigue.

283 orearm vasoconstriction during contralateral handgripping to fatigue.

284 trations may not rise enough during rhythmic handgripping to have a major impact on these responses.

285 cBRS was unchanged during handgrip under free-flow conditions, handgrip with parti

286 In females, absolute handgrip was related to fasting plasma glucose (r = - 0.

287 In males, absolute handgrip was related to triglycerides (r = - 0.25, p < 0

288 des (r = - 0.25, p < 0.05), whereas relative handgrip was related to waist circumference (r = - 0.32,

289 ose (r = - 0.28, p = 0.03), whereas relative handgrip was related to waist circumference (r = - 0.38,

290 Pressor responses to static handgrip were also attenuated in patients compared to co

291 Increases in CVR with handgrip were greater in men vs. women (1.25 +/- 0.49 vs

292 Changes in CBV with handgrip were linked to the myocardial oxygen consumptio

293 In the patients, MSNA responses to static handgrip were markedly attenuated (33 +/- 14 % at 33 % M

294 ure and mean arterial pressure during static handgrip were not different before, during and after spa

295 fter practice, I.G. became able to scale her handgrip when grasping a real target object that she had

296 ced mean arterial pressure response to right handgrip, whereas the pressor response to left handgrip

297 coronary vasoconstriction occurs with static handgrip with a time course that suggests a sympathetic

298 during handgrip under free-flow conditions, handgrip with partial flow restriction and PEI following

299 After handgrip, women had a greater rise in conductance than m

300 unilateral thigh-cuff release and isometric handgrip) would be greater after the administration of t