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1 a known ubiquitin pathway substrate in alpha haploid cells.
2 tory element that represses transcription in haploid cells.
3 f IME2, a meiotic gene normally repressed in haploid cells.
4  cell-specific promoter in meiotic and early haploid cells.
5  are dominant in diploid cells and lethal in haploid cells.
6 wide insertional mutagenesis screen in human haploid cells.
7 uring fission yeast karyogamy upon mating of haploid cells.
8  the use of forward genetic screens in human haploid cells.
9 s that escape sex chromosome inactivation in haploid cells.
10  and was manifested by delayed production of haploid cells.
11 PCR triggers events leading to the fusion of haploid cells.
12 breaks within the chromosomes of nondividing haploid cells.
13 lpha mating or from unisexual mating between haploid cells.
14 ks being required to isolate PGCs as well as haploid cells.
15 ation, and chromosome dosage compensation in haploid cells.
16 g balanced expression of the genome in these haploid cells.
17 re susceptible to copy number fluctuation in haploid cells.
18 d cells, was localized to the distal pole of haploid cells.
19  simultaneous cytokinesis yields a tetrad of haploid cells.
20  establish the physiological role of Ime4 in haploid cells.
21 th the effects of the tub2-T143G mutation in haploid cells.
22 y of approximately 0.1% in HR-competent Rad+ haploid cells.
23 kinase cascade that also regulates mating of haploid cells.
24 -activated cell sorting (FACS) enrichment of haploid cells.
25 s, expansion is limited to the post-meiotic, haploid cell and therefore cannot involve mitotic replic
26 te asexually within their mammalian hosts as haploid cells and are subject to DNA damage from the imm
27 cells are defective in bud site selection in haploid cells and bud in a bipolar fashion.
28 ar landmarks, such as Bud3 and Axl2/Bud10 in haploid cells and Bud8 and Bud9 in diploid cells.
29 ormed a genome-wide knockout screen in human haploid cells and identified the calcium pump SPCA1.
30 f SIN3 and RPD3 in that it represses IME2 in haploid cells and is necessary for sporulation in diploi
31 e chromosome segregation defect of bub1Delta haploid cells and restores viability to bub1Delta tetrap
32 d 7 in TTC7A-knockout (TTC7A-KO) HAP1 (human haploid) cells and reduce the susceptibility to apoptosi
33 e meiotically expressed transcripts in early haploid cells, and accumulation of NMD target mRNAs with
34  cells, these newly discovered C. tropicalis haploid cells are capable of undergoing filamentation, w
35      From this we demonstrate mating between haploid cells armed with hGPCRs and endogenous biosynthe
36 oteins were present in APC preparations from haploid cells arrested in G(1), S, and M phases and from
37 ed in both haploid and diploid cells, and in haploid cells arrested in G1 with alpha-factor or in S p
38                     Here, we exploited human haploid cells as a robust comparative screening platform
39  a sensitivity that allows the use of single haploid cells as starting material.
40                     Intron aI1 transposes in haploid cells at low frequency to target sites in mtDNA
41 rting approach also enables the isolation of haploid cells at low percentages, as well as the mainten
42                                 Nuclei of 32 haploid cells at various points in the cell cycle were m
43 that YAR1 is not an essential gene, but that haploid cells bearing a yar1 deletion grow significantly
44 ferently in haploid and diploid yeast cells: haploid cells bud in an axial manner, while diploid cell
45 regulated by mating type; it is expressed in haploid cells but not in diploids.
46 ingly, Bik1 is not required for viability in haploid cells, but becomes essential in polyploids.
47 scovered to influence mating-type control in haploid cells by locus-specific transcriptional silencin
48                                      Because haploid cells can spontaneously become diploid, their en
49             Ectopic expression of mei3(+) in haploid cells causes meiotic catastrophe.
50                     Saccharomyces cerevisiae haploid cells communicate with their opposite mating typ
51 on is due to a proliferative disadvantage of haploid cells compared with diploid cells.
52                                              Haploid cells containing the null allele are viable in m
53 component of the mouse meiotic nuage and its haploid cell counterpart, the chromatoid body.
54 pecifically increased in the G1 stage of the haploid cell cycle, as well as by the glucose depletion-
55 is of elevated spontaneous mutation rates of haploid cells deleted for this gene.
56                                              Haploid cells deleted of MMP37 are viable but display a
57 oexpression of MATa1 and MATalpha2 in Sir(+) haploid cells did not lead to lethality from the HO-indu
58 mportant in regulation of diploid as well as haploid cell differentiation in a variety of tissues.
59   Diploid cells divide at a larger size than haploid cells do, but they maintain SA-based divisions,
60 membranes (PSMs), the structures that engulf haploid cells during meiosis II (MII).
61 zed process of cell division that results in haploid cells (e.g., gametes).
62                                           In haploid cells, each mutant results in rapid mtDNA deplet
63 rowth is determined genetically: a and alpha haploid cells exhibit an axial budding pattern, and a/al
64 ind that diploid cells of equivalent size to haploid cells exhibit lower CDK activity in response to
65 ious environmental conditions, revealed that haploid cells experienced higher rates of silencing loss
66                       Here we show that MATa haploid cells exposed to low levels of the alpha-factor
67          We generated loss-of-function human haploid cells for FA complementation group C (FANCC), a
68 aling via a MAP kinase pathway that prepares haploid cells for mating.
69 ssue, providing an abundant supply of single haploid cells for transformation.
70  transcriptional mechanisms leading to early haploid cell formation are unknown.
71 divisions without DNA replication, producing haploid cells from diploid ones.
72 complex developmental process that generates haploid cells from diploid progenitors.
73  fusion in yeast is the process by which two haploid cells fuse to form a diploid zygote.
74 e higher eukaryotes, the first mitosis after haploid cell fusion in budding yeast may forgo cell cycl
75 r and controls normal temporal expression of haploid cell genes during spermiogenesis.
76                          Here, we describe a haploid-cell genetic screen for NMD effectors that has u
77 elopment of forward genetic screens in human haploid cells has the potential to transform our underst
78                                     Although haploid cells have been observed in egg cylinder stage p
79 ssion of DLGAP4 mRNAs and non-coding RNAs in haploid cells having the translocation.
80 stranded breaks that cannot be repaired by a haploid cell if induced before replication, does not inv
81 nduce ploidy plasticity and the formation of haploid cells in C. tropicalis.
82 d 1-butanol, stimulate filamentous growth in haploid cells in which this differentiation is normally
83 functions as an activator site in vegetative haploid cells, it seemed likely that the factors binding
84                                              Haploid cells lacking cdc24(+) are inviable, indicating
85  is needed for proper bud site selection, as haploid cells lacking Dbm1p bud predominantly in a bipol
86 ide, topotecan, and paclitaxel) using a near-haploid cell line (HAP1) and then analyzed the genomes o
87                           We have isolated a haploid cell line of N. plumbaginifolia, hNP 588, that i
88                    The recent development of haploid cell lines has facilitated forward genetic scree
89 and the development of genetic tools such as haploid cell lines, allowing high-throughput screening t
90 at this panel can be used to confirm doubled-haploid cell lines.
91 amental genetic feature in mammals, in which haploid cells normally arise only as post-meiotic germ c
92                     A sexual cycle involving haploid cells of a and alpha mating types has been repor
93      It has a defined sexual cycle involving haploid cells of alpha and a mating types, yet the vast
94 re we report that simultaneous expression in haploid cells of both MATa and MATalpha information supp
95                                              Haploid cells of budding yeast Saccharomyces cerevisiae
96 myces cerevisiae, the pheromones that induce haploid cells of opposite cell types to mate activate th
97       To this end, mitochondrial proteins in haploid cells of opposite mating type were labeled with
98                                         When haploid cells of Saccharomyces cerevisiae are crossed, p
99                                              Haploid cells of the budding yeast Saccharomyces cerevis
100                                              Haploid cells of the budding yeast Saccharomyces cerevis
101                                              Haploid cells of the fission yeast Schizosaccharomyces p
102 yces cerevisiae form after the mating of two haploid cells of the opposite mating type.
103 yeast Schizosaccharomyces pombe normally has haploid cells of two mating types, which differ at the c
104 ump2 eliminated the filamentous phenotype of haploid cells on low ammonium, while ump1 disruption onl
105                            Meiosis generates haploid cells or spores for sexual reproduction.
106 tures, telomerase activity becomes limiting: haploid cell populations senesce and generate aneuploid
107  stage of land plants develops from a single haploid cell produced by meiosis - the spore.
108 nstable state, so that cultures of mammalian haploid cells rapidly become enriched in diploids.
109 that determine the specification and fate of haploid cells remain unknown.
110 plex system for switching the mating type of haploid cells, requiring the genome to have three mating
111                     Saccharomyces cerevisiae haploid cells respond to extrinsic mating signals by for
112  the budding yeast Saccharomyces cerevisiae, haploid cells respond to mating pheromone through a G-pr
113                                         Once haploid cells respond to pheromone, the mating-specific
114 genesis in vegetatively growing cells and in haploid cells responding to mating pheromone.
115 olarized mating projection characteristic of haploid cells responding to pheromone.
116                       In this study, using a haploid cell screening platform, we identify the guanine
117 e simulations, colonies initiated by an aged haploid cell show declined mating probability at an earl
118 ase is similar in haploid and diploid cells, haploid cells spend longer in mitosis, indicative of pro
119 iosis involved in the packaging of resultant haploid cells (spores) into asci.
120 plete arrest of spermatogenesis at the early haploid cell stage.
121                                           In haploid cells suffering an HO endonuclease-induced, doub
122 f the M double-stranded RNA viral segment in haploid cells that are "healed" in diploids.
123 ls undergo meiosis to produce sperm or eggs, haploid cells that are primed to meet and propagate life
124             Disruption of GPI1 yields viable haploid cells that are temperature-sensitive for growth,
125                                              Haploid cells that lack Erv14p are viable but display a
126        When DSBs were made on chromosomes in haploid cells that retain their mating type, sir Delta m
127              During the prior conjugation of haploid cells, the four vegetatively expressed septins (
128                                           In haploid cells, the relevant landmark proteins are concen
129 es are brought together by the fusion of two haploid cells, the Y and Z proteins from different matin
130       Diploid cells of budding yeast produce haploid cells through the developmental program of sporu
131           We performed a gene-trap screen in haploid cells to identify host factors for adenovirus (A
132 d a loss-of-function genetic screen in human haploid cells to identify host factors important in C. t
133 vern the ability of Saccharomyces cerevisiae haploid cells to mate.
134 esults demonstrate the utility of screens in haploid cells to study interactions of human cells with
135                             Subsequently, in haploid cells, TUBD1 works in partnership with the micro
136 d cooperatively to sites in the promoters of haploid cell-type-specific genes (hsg) to repress their
137  large number of distinctly regulated genes: haploid cell-type-specific genes, G2-cell-cycle-regulate
138                       Mating between the two haploid cell types (a and alpha) of the yeast Saccharomy
139  and efficient transitions between alternate haploid cell types allow the organism to access the adva
140          We sequenced the methylome of three haploid cell types from developing pollen: the sperm cel
141 es can also stimulate filamentation, whereas haploid cells undergo a similar invasive growth response
142    In eukaryotes, diploid cells give rise to haploid cells via meiosis, a program of two cell divisio
143 tasac6/GG-actin cells from either diploid or haploid cells was unsuccessful.
144 ow glucose influences budding pattern in the haploid cell, we examined the roles of bud-site-selectio
145                                    Moreover, haploid cells were not detected by flow cytometry.
146      These include the lethality of tub1-724 haploid cells when the beta-tubulin-binding protein Rbl2
147 sts that non-homologous end-joining, even in haploid cells where homologous chromosomes are not prese
148                                       Unlike haploid cells, which arrest growth as unbudded cells, te
149 sive synaptonemal complexes and post-meiotic haploid cells with a similar pattern of ACROSIN staining
150 lates to a high level in mitotically growing haploid cells, yet transposition occurs at very low freq

 
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