ライフサイエンスコーパス: hard tissue

1 "soft tissue") to bone (a stiff, structural "hard tissue").

2 ective reparative process that generates new hard tissue.

3 them were blue-green colored that indicates hard tissue.

4 p, which later leads to uneven deposition of hard tissue.

5 mmatory response in the periodontal soft and hard tissues.

6 profound effects on the biomineralization of hard tissues.

7 There were no harmful effects to soft or hard tissues.

8 g stream of water effectively removes dental hard tissues.

9 ing osseous alterations and anomalies within hard tissues.

10 expression of PIEZO1/2 in the dentoalveolar hard tissues.

11 four orders of magnitude higher than that of hard tissues.

12 challenging in the biomimetic engineering of hard tissues.

13 progression were confirmed for both soft and hard tissues.

14 systemic impact on the peri-implant soft and hard tissues.

15 uring cell differentiation and production of hard tissues.

16 ival toward optimal preservation of soft and hard tissues.

17 ession presents destruction of both soft and hard tissues.

18 reference to Clopton Havers, a 17th Century hard tissue anatomist, and Franz Halberg, a long-time ex

19 Periodontists' knowledge of soft and hard tissue anatomy and their ability to manage soft tis

20 Intensities of dental caries, hard tissue and pulp were measured and calculated as aSN

21 e facilitates the remineralization of dental hard tissues and affects bacterial activities.

22 inerals by a vast array of organisms to form hard tissues and shape them in various forms.

23 gmentation was able to rebuild stable facial hard-tissue and soft-tissue contours that were esthetica

24 dentin, teleost fish enameloid (enamel-like hard tissue), and tetrapod enamel.

25 Digit fusion involves both soft and hard tissues, and is associated with reduced apoptosis a

26 ntal procedures is the creation of soft- and hard-tissue architecture that is consistent with periodo

27 study reported a minimal loss of mineralized hard tissue around dental implants placed non-submerged

28 palaeometabolomes from fossilized mammalian hard tissues as a molecular ecological strategy to provi

29 ires the coordinated responses from soft and hard tissues as well as the soft tissue-to-bone interfac

30 ls to treat tooth defects involving multiple hard tissues, as in the case of noncarious cervical lesi

31 sed as an interface or surface for soft- and hard-tissue attachment and integration are commercially

32 notype modification therapy (PhMT) involving hard tissue augmentation (PhMT-b) or soft tissue augment

33 Evidence at this moment supports CAOT with hard tissue augmentation accelerated tooth movement.

34 GR), clinical attachment level (CAL), dental hard tissues, basic medical history data and oral hygien

35 t differences regarding the loss of marginal hard tissues between mesial and distal surfaces or the m

36 portant shifts reflected in aspects of human hard tissue biology and the archeological record.

37 Hard tissue biopsies of 7 sites in 6 patients were obtai

38 Hard tissue biopsies were taken from 10 representative c

39 issue by measuring leukocyte recruitment and hard tissues by measuring osteoclastogenesis.

40 s a positive effect on marginal peri-implant hard-tissue changes.

41 ols and a superior stability of peri-implant hard tissue compared with immediate implant placement.

42 sue height over time, and 3) if the marginal hard tissue could be detected on the implant platform at

43 ue height over time, and 3) whether marginal hard-tissue could be detected on the implant platform at

44 BSP is present in cementum, the hard tissue covering the tooth root that anchors periodo

45 e challenge comes with the analyses of their hard tissues: current methods are time-consuming, destru

46 Often, in these patients, soft and hard tissue defects result from a variety of causes, suc

47 to repair challenging interproximal soft and hard tissue deficiencies.

48 We infer that metabolites preserved in hard tissues derive from an extravasated vasculature ser

49 tential to contribute to excessive soft- and hard-tissue destruction.

50 ole in the biological mechanisms controlling hard tissue development, but the details of molecular re

51 e of diagnostic disagreement, progression of hard tissue diagnoses in the right TMJ occurred in 15.2%

52 Of 794 joints with baseline joint-specific hard tissue diagnoses of DJD, progression was observed i

53 oft tissue diagnoses and 71% of the baseline hard tissue diagnoses remained stable.

54 Baseline hard tissue diagnoses showed no statistical association

55 e soft tissue diagnoses were associated with hard tissue diagnostic changes at follow-up ( P < 0.0001

56 ective compares the preservation of soft and hard tissue dimensional changes after alveolar ridge pre

57 During this study, the hard tissue dimensional changes were measured at 12-mont

58 nt ridge preservation techniques on soft and hard tissue dimensions.

59 and minimal change of peri-implant soft and hard tissue dimensions.

60 overing, the other a morphologically diverse hard tissue distributed at over 200 sites in the body.

61 cal and precisely orchestrated deposition of hard tissue during tooth formation in acrodont dentition

62 involved in initiating the mineralization of hard tissue ECMs.

63 sms is likely to be important for successful hard tissue engineering.

64 ation preclinical model is an ideal tool for hard tissue evaluation by micro-computed tomography, his

65 Safety was assessed through oral soft/hard tissue exam, adverse events, and routine laboratory

66 butions of known and potential biomarkers in hard tissues, facilitating exposome research.

67 Other hard tissue findings showed similar clinically superior

68 le that statherin plays in the regulation of hard tissue formation in humans, the surface recognition

69 ling processes including nerve sprouting and hard tissue formation.

70 atrix proteins as well as their control over hard tissue formation.

71 derstanding this process would shed light on hard-tissue formation and guide efforts to develop bioma

72 ling semi-automated segmentation of soft and hard tissue from muCT data.

73 sive picture of the development of a complex hard tissue, from the secretion of its organic macromole

74 Extracellular matrix proteins regulate hard tissue growth by acting as adhesion sites for cells

75 oactive peptide ligands direct both soft and hard tissue growth in a hierarchically organized manner.

76 hat these proteins play in the regulation of hard tissue growth in humans, the exact mechanism used b

77 sight into the recognition and regulation of hard tissue growth.

78 ons, reproducing a key feature of biological hard-tissue growth and assembly.

79 In addition, these implants had hard tissue healing that extended onto the implant shoul

80 Hard tissue healing was determined histomorphometrically

81 Hard-tissue healing was evaluated by microcomputed tomog

82 oning of the implant, 2) changes of marginal hard tissue height over time, and 3) if the marginal har

83 oning of the implant, 2) changes of marginal hard-tissue height over time, and 3) whether marginal ha

84 Hard-tissue histology-the analysis of thin two-dimension

85 eliability and diagnostic power of classical hard-tissue histology.

86 Hard tissue improvements resulted in the complete closur

87 Both treatments provided soft and hard tissue improvements when compared to baseline (P <

88 tion involvements, both in terms of soft and hard tissue improvements.

89 ears to represent a new strategy for forming hard tissue in animals.

90 dictably led to the preservation of soft and hard tissue in extraction sites.

91 first time the ultrastructure of the dental hard tissues in an archival case of intrinsically pigmen

92 important clinical features of both soft and hard tissues in various anatomical joints.

93 in family is required for the development of hard tissues including bone, cartilage and teeth.

94 However, stability of peri-implant soft and hard tissues indicates the need to take measures that mi

95 o far, studies have predominantly focused on hard tissue integration.

96 behaviour and structural features of soft-to-hard tissue interfaces treated by CESS.

97 ryloyloxy) ethyl] phosphate (BMEP) of dental hard tissue, interfacial characteristics, and inhibition

98 apillary deficiency associated with soft and hard tissue interproximal defects.

99 sis of the biopsied specimen revealed dental hard tissue interspersed in a field of odontogenic epith

100 for dealing with conditions where healing of hard tissues is compromised by bacterial contamination.

101 Because restoration of both soft and hard tissues is crucial to oral tissue physiology, this

102 ingiva do not contribute to the formation of hard tissue, it is theoretically possible that under app

103 Degree of hard tissue loss and type of final prosthetic restoratio

104 sthetic restoration as well as the degree of hard tissue loss on 7-y clinical performance of ETT rest

105 therapeutic modality for severe peri-implant hard tissue loss.

106 n the groups regarding marginal peri-implant hard-tissue loss.

107 and used treatment option for both soft and hard tissue malignancies.

108 of mineral leading to dissolution of dental hard tissues may result in a caries lesion that can be s

109 Intrasurgical hard tissue measurements consisted of the vertical open

110 Analysis of soft and hard tissue measurements demonstrated a statistically si

111 Hard tissue measurements included defect depth, alveolar

112 Hard tissue measurements included height of the alveolar

113 at surgical reentry (9 to 13 months later), hard tissue measurements included: stent to defect base,

114 Hard tissue measurements obtained at surgical reentry we

115 e was surgically re-entered and all soft and hard tissue measurements repeated.

116 Hard tissue measurements were recorded during the initia

117 The following soft and hard tissue measurements were recorded prior to initial

118 Both soft and hard tissue measurements were taken the day of surgery (

119 Soft and hard tissue measurements, cone beam computed tomography

120 assembly of matrix proteins is a key step in hard tissue mineralization, developing an understanding

121 ol' of structural-bound water that occurs in hard tissue niches(7).

122 Dentin is the major hard tissue of teeth formed by differentiated odontoblas

123 human form, information about both soft and hard tissues of our ancestors is needed.

124 ations and suggesting that patterning of the hard tissues of the mandible is instructed by the epithe

125 l for the development and maintenance of the hard tissues of the skeleton.

126 77% of the implants in Group B demonstrated hard tissue on the implant platform.

127 The mineralized hard tissue on the implant shoulder was found in 69% of

128 This study aims to compare hard tissue outcomes of VBA, with and without recombinan

129 ly higher in carious lesions than in healthy hard tissue (p < 0.001).

130 Baseline and 6-month reentry soft and hard tissue parameter measurements were made by calibrat

131 d similar favorable improvements in soft and hard tissue parameters in the treatment of human intraos

132 ally significant improvement to the soft and hard tissue parameters measured.

133 t using conservative flap reentry surgeries; hard tissue parameters were also assessed at this time.

134 indicate that there may be an enhancement of hard tissue parameters when enamel matrix derivative is

135 d trial was to compare peri-implant soft and hard tissue parameters, esthetic ratings of, and patient

136 herefore comparison of peri-implant soft and hard tissue parameters, esthetic ratings, and patient-re

137 iographic findings were non-contributory for hard tissue pathoses.

138 ar origin of the instructive information for hard tissue patterning of the jaws has been the subject

139 y odontogenesis and later differentiation of hard tissue-producing cells.

140 nt position and loss of marginal mineralized hard tissue (r = 0.15; P >0.05).

141 s in structures that can be used as soft and hard tissue-regenerating materials, biosensors, and ener

142 ising findings in various models of soft and hard tissue regeneration such as myocardial infarction,

143 on and demonstrate their value for promoting hard tissue regeneration.

144 ization have the potential to be utilized in hard tissue regeneration.

145 al-dose doxycycline, also indirectly promote hard-tissue regeneration.

146 recession, probing, clinical attachment) and hard tissue (resorption, defect fill) parameters 6 month

147 The evaluation of the hard tissue response at the 12-month re-entry demonstrat

148 omposites in combination with bonding to the hard tissue result in stress transfer and inward deforma

149 In contrast, micro-CT analysis of hard tissues revealed that deposition of dentin and enam

150 cium sulphate (CS) based cement to produce a hard tissue scaffold with the ability to inhibit bacteri

151 ssociated with microdamage and repair at the hard tissue-soft tissue interface.

152 cessfully applied our new approach to assess hard-tissue specimens of different species.

153 biological systems regulate the formation of hard tissue structures.

154 he root canal, uninterrupted by the soft and hard tissues surrounding the teeth in an ex vivo model.

155 the cellular/molecular integrity of mini pig hard tissues, then demonstrated that the results of thes

156 erative medicine is to restore oral soft and hard tissues through cell, scaffold, and/or signaling ap

157 signal to noise ratio) and aHTMCNR (apparent hard tissue to muscle contrast to noise ratio) in both s

158 In Biology, this process enables soft and hard tissues to coexist in the same organism with relati

159 lysis and mass spectrometry imaging (MSI) of hard tissues (tooth and hair) for the detection and mapp

160 2 mm, without loss of interproximal soft and hard tissue, treated with the CAF procedure and evaluate

161 months, upon flap reflection, no regenerated hard tissue was found.

162 The mean marginal loss of hard tissues was 0.11 +/- 0.30 mm for Group A and 0.08 +

163 The mean marginal loss of hard tissues was 0.11 +/- 0.30 mm.

164 al and radiographic measurements of soft and hard tissues were recorded by means of a stent before an