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1 asion of the retina and brain, organs seeded hematogenously.
2 pread occurs via both lymphatic channels and hematogenously.
3 her opportunistic pathogens that disseminate hematogenously.
4 l adhesion molecule A (JAM-A) to disseminate hematogenously.
5                           As it disseminates hematogenously and invades a wide range of tissues, T. p
6 r propensity for the bacteria to disseminate hematogenously, and a strong splenic T cell cytokine res
7  that extrapulmonary tissues may be infected hematogenously by RSV and harbor this virus allowing the
8 sity for treponemes to migrate through skin, hematogenously disseminate, and invade targeted tissues.
9 roviding durable protective immunity against hematogenously disseminated C. albicans and C. auris and
10  IFN-gamma augments the host defense against hematogenously disseminated candidal infections.
11 sphosphate, which was shown to contribute to hematogenously disseminated candidiasis (DC) after sever
12   Using RNA-seq analysis of a mouse model of hematogenously disseminated candidiasis (HDC) and episod
13 t Hyr1p significantly protected mice against hematogenously disseminated candidiasis (P = .001).
14 e findings indicate that the murine model of hematogenously disseminated candidiasis accurately recap
15 n (SSK21) was avirulent in a murine model of hematogenously disseminated candidiasis and less able to
16  required for virulence in a murine model of hematogenously disseminated candidiasis and that strains
17                                    Mice with hematogenously disseminated candidiasis died of progress
18                          The murine model of hematogenously disseminated candidiasis is the standard
19 l adults, but its role in host resistance to hematogenously disseminated candidiasis is unknown.
20 , required for virulence in a mouse model of hematogenously disseminated candidiasis, and for potenti
21                                       During hematogenously disseminated candidiasis, bloodborne Cand
22 or caPLB1, as assessed in a murine model for hematogenously disseminated candidiasis, was significant
23           The incidence of life-threatening, hematogenously disseminated candidiasis, which is predom
24  significant attenuation in a mouse model of hematogenously disseminated candidiasis, while the doubl
25 t system is important for host resistance to hematogenously disseminated candidiasis.
26 ation of MBL increased resistance of mice to hematogenously disseminated candidiasis.
27 ion (HR), was evaluated in a murine model of hematogenously disseminated candidiasis.
28  identify some virulence factors relevant to hematogenously disseminated candidiasis.
29 plenic immune responses in a murine model of hematogenously disseminated candidiasis.
30 e of infections, from superficial mucosal to hematogenously disseminated candidiasis.
31  a successful infection in a murine model of hematogenously disseminated candidiasis.
32 irulence of C. albicans in a murine model of hematogenously disseminated candidiasis.
33 aplb1-2 mutant were indistinguishable during hematogenously disseminated candidiasis.
34                                       During hematogenously disseminated infection, blood-borne Candi
35 culature to invade the deep tissues during a hematogenously disseminated infection.
36 rmation is critical for C. albicans to cause hematogenously disseminated infections.
37                                              Hematogenously disseminated M. tuberculosis infection wa
38 uction in virulence using the mouse model of hematogenously disseminated systemic candidiasis.
39 ugh the skin, the organism causes disease by hematogenously disseminating to multiple organs.
40 e been speculated that platelets may protect hematogenously disseminating tumor cells from NK-depende
41 is mediated by infected B cells that traffic hematogenously from draining lymph nodes to peripheral l
42 -borne disease Lyme borreliosis, disseminate hematogenously from the tick bite site to the joints, th
43                                      Animals hematogenously infected with the C. albicans clinical is
44    The ability of tumor cells to metastasize hematogenously is regulated by their interactions with p
45                                          4T1 hematogenously metastasizes to the lung, liver, bone, an
46 gy of ovarian carcinoma differs from that of hematogenously metastasizing tumors because ovarian canc
47  may represent TNF-alpha protein released by hematogenously recruited macrophages.
48 o form locomotory organelles and spread, and hematogenously seed distant organs.
49 ens gaining access to surgical wounds either hematogenously, through drains, or through slowly healin
50 g it to cause disease locally or disseminate hematogenously throughout the body.
51 sition into skin, Lyme borreliae disseminate hematogenously to other organs, resulting in extracutane
52 y and tends to metastasize lymphatically and hematogenously to the lung, liver, brain, and bone; it i
53 vidence that F. nucleatum may be transmitted hematogenously to the placenta and cause adverse pregnan
54 ection, only the tPMP(r) strain disseminated hematogenously to the spleen (15 of 15 rabbits) versus 0