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1  crucial factor reducing function in chronic hemiparesis.
2  were: epilepsy, intellectual disability and hemiparesis.
3         Unfortunately, there was one case of hemiparesis.
4 f migraine with aura that is associated with hemiparesis.
5 of grasping behaviour in patients with right hemiparesis.
6 ncluding a syndrome of delayed contralateral hemiparesis.
7  nerve palsy and one increase in preexisting hemiparesis.
8 tients within 10 hours of the onset of acute hemiparesis.
9 herotomies) invariably lead to contralateral hemiparesis.
10 of the slower gait speed seen in people with hemiparesis.
11 ining of individuals affected by post-stroke hemiparesis.
12 ral asymmetry may be optimal for people with hemiparesis.
13 stic gait patterns in 20 patients with acute hemiparesis.
14 consciousness, expressive aphasia, and right hemiparesis.
15 vided tone reduction and clinical benefit in hemiparesis.
16                            Most CP cases had hemiparesis (40%) or quadriparesis (38%); 87% had been d
17 wever it is difficult to understand how this hemiparesis affects movement patterns as it often presen
18                   Twenty-three patients with hemiparesis after first-time stroke were scanned at 2.0
19                Seven days later, severity of hemiparesis and cerebral infarct size were examined.
20 iorgan IgG4-RD developed progressive spastic hemiparesis and dementia.
21                       Two patients developed hemiparesis and in two cases epilepsy recurred.
22 monstrate that persons with mild to moderate hemiparesis and no measured sensory or perceptual defici
23  patients suffering from motor CD (nine with hemiparesis and six with paraparesis) and 25 age- and ge
24                                              Hemiparesis and tetraparesis developed in 1 patient each
25 owly progressive dementia, seizures, ataxia, hemiparesis, and decreased vision without neuropathy hav
26 sease 2019 who presented with seizure, right hemiparesis, and dysarthria with positive findings for s
27 (most commonly memory/cognition impairment), hemiparesis, and visual defects.
28 ) children had focal features (most commonly hemiparesis) at presentation.
29 40% and 60% weakness models, suggesting that hemiparesis can account for a portion of the slower gait
30                                              Hemiparesis, defined as unilateral muscle weakness, ofte
31 a that, while subjects with mild-to-moderate hemiparesis demonstrate differences in the feature of a
32 d deficits after small focal lesions (ataxic hemiparesis, dysarthria-clumsy hand syndrome, dysarthria
33   Eight subjects with mild to moderate right hemiparesis following a stroke and seven age and gender
34 rome and to increase the movement ability in hemiparesis following stroke.
35 The results suggest that patients with right hemiparesis from a subcortical lesion of the corticospin
36 egarded as intractable such as phantom pain, hemiparesis from stroke and complex regional pain syndro
37 a more comprehensive understanding of stroke hemiparesis gait patterns and suggests considering both
38                        (ii) Do patients with hemiparesis have a learning deficit?
39            Many patients with a pre-existing hemiparesis, however, experience no deterioration in mot
40 emorrhagic stroke with clinically meaningful hemiparesis (ie, a total score of >=3 points on the foll
41 tients (33%) in the surgery group, including hemiparesis in 15 (26%).
42 portion of patients had subdural empyema and hemiparesis in 2011-2013.
43        Stroke patients with mild to moderate hemiparesis in either the left or right arm because of c
44 symptoms, a transient contralateral hindlimb hemiparesis in rats.
45  abnormal reaching movements in persons with hemiparesis is important to the development of rehabilit
46 balloon occlusion demonstrated more profound hemiparesis, larger infarct sizes, lower Spetzler neurol
47  use of a fictional patient with obesity and hemiparesis limit generalizability.
48 e group; and memory loss, dizziness, ataxia, hemiparesis, loss of consciousness and hemisensory sympt
49 tent with HSV encephalitis, including fever, hemiparesis, meningitis, and hemorrhage in the basal gan
50 nd hand motor control in chronic post-stroke hemiparesis ( NCT04512690 ).
51    Twenty-four stroke survivors with chronic hemiparesis of the hand participated in the trials, alon
52 rate was 6% (wound separation and vocal cord hemiparesis, one each).
53  diagnosed retrospectively, when evidence of hemiparesis or postneonatal seizures leads to later eval
54 cognised only retrospectively, with emerging hemiparesis or seizures after the early months of life.
55      Thirty-two individuals with post-stroke hemiparesis performed a grasp-and-lift task, while their
56 doses of motor therapy in chronic poststroke hemiparesis result in better outcomes, compared to lower
57 t first-ever stroke patients presenting with hemiparesis resulting from cerebral infarction sparing t
58 te whether, and to what extent, persons with hemiparesis retain the ability to exploit motor abundanc
59 ent cycle demonstrated that the persons with hemiparesis showed different patterns of joint couplings
60  follows (in descending order of frequency): hemiparesis, vertigo/dizziness, diplopia, dysarthria, ny
61 py for a stroke patient with upper extremity hemiparesis, we propose a cortico-basal ganglia model ca
62 nal capsule resulted in 2 cases (8%) of mild hemiparesis, which improved and prompted monitoring of a
63 d adults and one individual with post-stroke hemiparesis while walking in shoes-only and with zero-st