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1 into adulthood, we use the term 'alternating hemiplegia'.
2 ges were seen in six people with alternating hemiplegia.
3 explained premature mortality of alternating hemiplegia.
4 ctional correlates over and above effects of hemiplegia.
5 rk in the plegic lower limbs of persons with hemiplegia.
6 entia (35 [28.7%] vs 34 [11.4%]; p <0.0001), hemiplegia (14 [11.5%] vs 12 [4.0%]; p=0.004), malignanc
7            Clinically, 26.2% of children had hemiplegia, 34.4% had diplegia, 18.6% had quadriplegia,
8 I was used to study 11 patients with chronic hemiplegia after unilateral stroke that spared regions o
9              The syndrome of Anosognosia for Hemiplegia (AHP) can provide unique insights into the ne
10 Recent theories propose that anosognosia for hemiplegia (AHP) results from specific impairments in mo
11 terally during pedalling for 15 persons with hemiplegia and 12 neurologically intact age-matched cont
12 tory of severe diarrhea presented with right hemiplegia and altered mental status.
13 tis was stronger for diplegia (compared with hemiplegia and quadriplegia) and for cerebral palsy with
14                  All patients had unilateral hemiplegia and visual field loss.
15                 The study of anosognosia for hemiplegia, Anton's syndrome, and less dramatic disturba
16 rticoids (adjusted odds ratio [aOR], 31.50), hemiplegia (aOR, 28.48), neuralgia (aOR, 4.81), optic at
17                                   Stroke and hemiplegia are frequent complications of stroke.
18   The delusional features of anosognosia for hemiplegia can be explained as a failure of this re-repr
19 le brains, which could explain the prolonged hemiplegia, coma, and seizure phenotype in this variant
20 ee clinical forms of spastic cerebral palsy (hemiplegia, diplegia, and quadriplegia) and for cerebral
21 id) or according to the body parts involved (hemiplegia, diplegia, or quadriplegia).
22 dominant disorder characterized by transient hemiplegia during the aura phase of a migraine attack.
23 litis/trauma, and one each of hemiconvulsion-hemiplegia epilepsy and perinatal ischaemic insult) and
24 h a 5-day history of progressive right-sided hemiplegia, expressive aphasia, mild bulbar palsy, and r
25                               In adults with hemiplegia following stroke the threshold of the homonym
26                                         With hemiplegia following stroke, a person's movement respons
27 he treatment of epilepsy in association with hemiplegia for over 50 years.
28                         Stroke patients with hemiplegia frequently comment that their limbs feel heav
29 G recordings of 52 patients with alternating hemiplegia from nine countries: all had whole-exome, who
30 for hemiplegia, i.e. apparent unawareness of hemiplegia, have been clinically observed to show 'tacit
31 gram abnormalities are common in alternating hemiplegia, have characteristics reflecting those of inh
32           Some patients with anosognosia for hemiplegia, i.e. apparent unawareness of hemiplegia, hav
33 r awareness can occur called anosognosia for hemiplegia, i.e. the denial of motor deficits contralate
34                 Here we successfully modeled hemiplegia-induced protection from arthritis using the K
35                              Upper extremity hemiplegia is a common consequence of unilateral cortica
36                               Stroke-induced hemiplegia is a major cause of long-term disability, oft
37                                              Hemiplegia, leukemia, lymphoma, severe liver disease, an
38 diseases related to episodic ataxia, such as hemiplegia, migraine, and epilepsy.
39 ts in significant nerve dysfunction, such as hemiplegia, mood disorders, cognitive and memory impairm
40                                  Alternating hemiplegia of childhood (AHC) is a disorder that can res
41                                  Alternating hemiplegia of childhood (AHC) is a neurodevelopmental di
42                                  Alternating hemiplegia of childhood (AHC) is a rare, severe neurodev
43                                  Alternating hemiplegia of childhood (AHC) is typically distinguished
44 ases diagnosed with both COS and alternating hemiplegia of childhood (AHC), and for whom two distinct
45 miplegic migraine type 2 (FHM2), alternating hemiplegia of childhood (AHC), and rapid-onset dystonia
46 al hemiplegic migraine (ATP1A2), alternating hemiplegia of childhood (ATP1A2/A3), rapid-onset dystoni
47 plegic migraine (MIM#602481) and alternating hemiplegia of childhood (MIM#104290).
48 d-onset dystonia parkinsonism or alternating hemiplegia of childhood causes a dramatic reduction of N
49                                  Alternating hemiplegia of childhood is a rare disorder caused by de
50 ac investigation is warranted in alternating hemiplegia of childhood, as cardiac arrhythmic morbidity
51 lial Hemiplegic Migraine type 2, Alternating Hemiplegia of Childhood, Rapid-onset Dystonia Parkinsoni
52 -associated syndromes, including alternating hemiplegia of childhood, rapid-onset dystonia-parkinsoni
53 -onset dystonia parkinsonism and alternating hemiplegia of childhood.
54 tients expressing the R192Q mutation exhibit hemiplegia only.
55                Dysautonomia may occur during hemiplegia or in isolation.
56 ruited 8 adults with chronic stroke and left hemiplegia/paresis and 22 healthy adults.
57 bjects with chronic (> 6 months) post-stroke hemiplegia participated in the study.
58               Fourteen subjects with chronic hemiplegia participated in the study.
59 ks can be accompanied by seizures, coma, and hemiplegia; patients expressing the R192Q mutation exhib
60 motor performance observed when persons with hemiplegia pedal in a horizontal position is exacerbated
61                                              Hemiplegia remained unchanged following surgery in 22 ou
62 that, despite stroke producing contralateral hemiplegia, surviving regions of motor cortex actively p
63 antly more common in people with alternating hemiplegia than in an age-matched disease control group
64 ificantly shorter in people with alternating hemiplegia than in the disease control group.
65 r his 45th dose of natalizumab, he developed hemiplegia that evolved into quadriparesis.
66 tion following extensive lesions (pure motor hemiplegia) to incomplete basilar pontine syndrome and r
67  young man with migraine with aura including hemiplegia, we identified a novel SLC1A3 mutation that p
68 ubjects with stroke in adulthood and spastic hemiplegia were also studied.
69                 Abnormalities in alternating hemiplegia were more common in those >/=16 years old, co
70  CSF rhinorrhea occurred in one patient, and hemiplegia with homonymous hemianopsia developed as a co