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1 for children and young adults without severe hemodynamic instability.
2 , and when significant enough, can result in hemodynamic instability.
3 gy for early detection of progression toward hemodynamic instability.
4 brile neutropenia, and empiric treatment for hemodynamic instability.
5 er noncardiac surgery (MINS) without causing hemodynamic instability.
6  mortality, resuscitated cardiac arrest, and hemodynamic instability.
7 physiological process by which the RV causes hemodynamic instability.
8 rs of end-organ dysfunction, and profiles of hemodynamic instability.
9 in intrathoracic pressure can lead to severe hemodynamic instability.
10 s typically presenting as abdominal pain and hemodynamic instability.
11 ardial infarction and death without inducing hemodynamic instability.
12 y in patients with sepsis, such as fever and hemodynamic instability.
13 using severe acidosis, renal impairment, and hemodynamic instability.
14 ated archiving method as early indicators of hemodynamic instability.
15 ta are limited in children and patients with hemodynamic instability.
16 tion of severe liver disease results in more hemodynamic instability.
17 en with cessation of neurologic function and hemodynamic instability.
18 c arrest (CA) have been low, presumably from hemodynamic instability.
19 mitted to the intensive care unit because of hemodynamic instability.
20  hypercapnia was associated with significant hemodynamic instability.
21  patients required immediate exploration for hemodynamic instability.
22 e due to hypercapnia, and seven secondary to hemodynamic instability.
23  did not translate into increased cardiac or hemodynamic instability.
24 baseline values, leading to life-threatening hemodynamic instability.
25 ion has been corroborated by data indicating hemodynamic instability.
26 fewer early terminations of sustained VT for hemodynamic instability (1.0 vs. 4.0; p = 0.001).
27 l treatment included poor prognosis (33.7%), hemodynamic instability (19.8%), death before surgery (2
28 nts were registered) the most frequent being hemodynamic instability (26.5%) and severe hypoxemia (20
29        The main reason for ICU admission was hemodynamic instability (58%), predominantly related to
30 atients with severe injuries (mean ISS = 23; hemodynamic instability = 70%; hospital/ICU stay = 12 d;
31 r transplant recipients with coagulopathy or hemodynamic instability after allograft reperfusion.
32                                              Hemodynamic instability after liver graft reperfusion in
33                     In multivariate analysis hemodynamic instability, age >/= 75 years, history of st
34 riteria were CMV+ donors to CMV- recipients, hemodynamic instability, age >50, size mismatch (donor w
35                    The incidence of clinical hemodynamic instability and bleeding complications tende
36  related to suggested detrimental effects on hemodynamic instability and enhanced oxidative stress.
37 clinical evaluation of patients for pain and hemodynamic instability and evaluation of MR images for
38 neonatal cardiac surgery, where pre-existing hemodynamic instability and metabolic abnormalities are
39 ve strategy to salvage patients with extreme hemodynamic instability and multiorgan injury.
40                            They are prone to hemodynamic instability and must be monitored with vario
41                                              Hemodynamic instability and myocardial dysfunction are m
42 and the incidence of sepsis that can lead to hemodynamic instability and organ failure.
43                                              Hemodynamic instability and oxygenation failure as trach
44 tion in 10 patients; only those (n = 3) with hemodynamic instability and relatively low plasma argini
45      Addition of 15 mg/kg of DEA resulted in hemodynamic instability and thus DFT was not obtained.
46 grafts from suicidal hanging donors (without hemodynamic instability and with downward trend in the d
47 -day mortality, resuscitated cardiac arrest, hemodynamic instability, and 90-day readmission.
48 salvage patients with cardiac arrest, severe hemodynamic instability, and multiorgan failure results
49 volume and location, serum hemoglobin level, hemodynamic instability, and presence of active bleeding
50 of low clinical risk included no evidence of hemodynamic instability, arrhythmias or electrocardiogra
51                              Peri-intubation hemodynamic instability but not severe hypoxemia was ide
52 in patients with complex coronary disease or hemodynamic instability, but not at high-volume hospital
53         Propofol-based sedation may increase hemodynamic instability by decreasing vascular tone and
54 uence of RV infarction is thought to produce hemodynamic instability by reducing left ventricular (LV
55              Severe CRS was characterized by hemodynamic instability, capillary leak, and consumptive
56 enced an HBAT-related serious adverse event (hemodynamic instability characterized by bradycardia, ta
57      Approximately 2% to 9% of patients have hemodynamic instability, characterized by inability to m
58 own source, post surgical); patient factors (hemodynamic instability, coagulopathy, multi-organ failu
59  resulting in hemoglobin decreases >=2 g/dL, hemodynamic instability, colonoscopy, angiography, or su
60 scuing patients from tamponade and reversing hemodynamic instability complicating invasive cardiac ca
61 jor exclusions were end-stage renal disease, hemodynamic instability, concurrent COVID-19 infection,
62 edictors of adverse outcome were measures of hemodynamic instability, disease severity, demographics
63 irculatory support for patients experiencing hemodynamic instability due to myocardial infarction, ca
64 IL-6 levels may be a predictor for recipient hemodynamic instability during liver reperfusion.
65 g physiological stress that may translate to hemodynamic instability during LT.
66 ic variables for early detection of imminent hemodynamic instability during progressive central hypov
67                                Prevention of hemodynamic instability during renal replacement therapy
68 ngth <320 ms) isthmuses is often hindered by hemodynamic instability during sustained FVT and by rate
69 etomidate, ketamine probably results in more hemodynamic instability during the peri-intubation perio
70 In patients with high-risk PE and refractory hemodynamic instability, ECMO may be a valuable supporti
71                Secondary end points included hemodynamic instability events, procedure time, serious
72                                              Hemodynamic instability (HDI) during liver transplantati
73 s in brain white matter were associated with hemodynamic instability (higher mean arterial pressure e
74 owed significantly increased mortality, more hemodynamic instability, higher nitric oxide levels, and
75 ntly perceived barriers to mobilization were hemodynamic instability, hypoxemia, and dependency on ve
76 rs; range, 4.5-102 hours), and the cause was hemodynamic instability in 10 of the 12 patients (83.3%)
77 oninferiority trial to compare perioperative hemodynamic instability in 92 steroid-treated IBD patien
78 ause of respiratory failure with hypoxia and hemodynamic instability in critically ill patients.
79 cardiomyopathy, ventricular arrhythmias, and hemodynamic instability in the absence of obstructive co
80 all risk of the Norwood operation and sudden hemodynamic instability in the intensive care unit.
81  with etomidate, ketamine probably increases hemodynamic instability in the peri-intubation period (r
82 currence of GM-IVH is highly associated with hemodynamic instability in the premature brain, yet the
83                                              Hemodynamic instability in the trauma patient is most co
84 t studies on the use of steroids in treating hemodynamic instability in these children.
85 sk of complications such as hypocalcemia and hemodynamic instability, limiting their widespread adopt
86 r tachycardia (VT) is frequently hampered by hemodynamic instability, long procedure duration, and hi
87 on models resulted in the greatest degree of hemodynamic instability (mean [SD] arterial pressure dec
88        Fifty organ donors were evaluated for hemodynamic instability, (mean arterial pressure [MAP]</
89 g/dL), active bleeding (n = 479; 27.7%), and hemodynamic instability (n = 406 [23.5%]).
90 ort with electrocardiographic changes, acute hemodynamic instability, newly recognized left ventricul
91 oncentrations of stress hormones, as well as hemodynamic instability, occurred after brain death.
92  bleeding, and detected episodes of clinical hemodynamic instability occurring as long as 4 weeks aft
93 ium, or other systemic bleeding resulting in hemodynamic instability or blood transfusions.
94 rvived with lung injury combined with either hemodynamic instability or hepatic and renal failure.
95 rameters, including duration and severity of hemodynamic instability or hypoxia might be a better pre
96          Cooling was well tolerated, with no hemodynamic instability or increase in arrhythmia.
97                                Patients with hemodynamic instability or liver dysfunction were exclud
98 on is indicated when the arrhythmia leads to hemodynamic instability or myocardial ischemia.
99 racheal intubations when the child had acute hemodynamic instability or oxygen failure and when the c
100                   Patients were excluded for hemodynamic instability or significant renal or hepatic
101         No complications occurred, including hemodynamic instability or uncontrollable decreases in h
102 rdial infarction (OR = 2.5), CHF (OR = 2.4), hemodynamic instability (OR = 2.8), cardiopulmonary bypa
103 rmed in 44 cases because of complex anatomy, hemodynamic instability, or failed percutaneous coronary
104  and mortality is mediated by fluid balance, hemodynamic instability, or low potassium or phosphate b
105 ment groups in the prevalence of barotrauma, hemodynamic instability, or mucus plugging.
106                       Multiple morphologies, hemodynamic instability, or noninducibility may limit ve
107                                              Hemodynamic instability, organ malperfusion, increasing
108 s affected by emergent status (P < .001) and hemodynamic instability (P = .04) but not by age, sex, b
109                                Patients with hemodynamic instability, peritonitis, or an unevaluable
110 e short-term support to patients with severe hemodynamic instability, permit recovery of multiorgan i
111 atment-refractory electrical storm and their hemodynamic instability prevents emergency catheter abla
112 cute myocardial infarction frequently causes hemodynamic instability, pulmonary edema, and cardiogeni
113       The hospital course was complicated by hemodynamic instability, renal failure, pneumonia, and a
114                                In 1 patient, hemodynamic instability required an intra-aortic balloon
115  of CPB for those patients with preoperative hemodynamic instability requiring a salvage CABG operati
116 ation success, and major adverse events (new hemodynamic instability, severe hypoxemia, and cardiac a
117 tion, preoperative intraaortic balloon pump, hemodynamic instability, shock, intravenous nitroglyceri
118 igestive content, oxygen desaturation, major hemodynamic instability, sustained arrhythmia, cardiac a
119  14 (group I) had a cardiac arrest or severe hemodynamic instability (systolic blood pressure </=75 m
120     Women had a higher incidence of shock or hemodynamic instability than men (25% versus 17%, P<0.05
121 opriate SR responses were more predictive of hemodynamic instability than VT rate and ejection fracti
122  injury (SCI) causes immediate and sustained hemodynamic instability that threatens neurological reco
123 k fluid draining from the bronchial orifice, hemodynamic instability, thrombocytopenia, and coagulopa
124      Pediatric patients meeting criteria for hemodynamic instability underwent serial echocardiograms
125 criteria included patients with peritonitis, hemodynamic instability, unreliable physical examination
126 dysfunction, especially when associated with hemodynamic instability unresponsive to conventional tre
127                                              Hemodynamic instability was not predictive of fatal outc
128                          An adverse event of hemodynamic instability was recorded in 19 of 575 patien
129 well tolerated in a normal heart often cause hemodynamic instability when they occur in the immediate
130 olam in two nonsurviving patients because of hemodynamic instability, which persisted despite the cha
131 cations of renal replacement therapy include hemodynamic instability with ensuing shortened treatment
132                                          New hemodynamic instability within 30 minutes after intubati
133 ysaccharide administration induced transient hemodynamic instability without significant impact on mo

 
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