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1 a possible treatment-related grade 5 event (hemoptysis).
2 onary status and prevent further episodes of hemoptysis.
3 cidence, BAA represents a potential cause of hemoptysis.
4 sease (COPD), who presented with non-massive hemoptysis.
5 Twenty-seven percent of patients experienced hemoptysis.
6 He did not report fever, night sweats, or hemoptysis.
7 ns; minor mucocutaneous hemorrhage and major hemoptysis.
8 7 (39%) reported cough, and 5 (28%) reported hemoptysis.
9 of pneumothorax, chest drain placement, and hemoptysis.
10 lied vigorously to all patients with massive hemoptysis.
11 of choice in operable patients with massive hemoptysis.
12 derwent 36 BAE procedures for the control of hemoptysis.
13 on (BAE) is one of the treatment options for hemoptysis.
14 o patient had chest discomfort, coughing, or hemoptysis.
15 along with intermittent episodes of streaky hemoptysis.
17 h as cough (40%), shortness of breath (34%), hemoptysis (10%), or metastases with corresponding local
18 yzed a total of 55 consecutive patients with hemoptysis (14 mild, 31 moderate, and 10 massive) treate
24 ing one case of hematoma and 4 cases of mild hemoptysis, and 30-day mortality (2%-3%) did not differ
25 tcomes were pulmonary hemorrhage, documented hemoptysis, and bleeding complications necessitating int
26 rosis, pulmonary edema, alveolar hemorrhage, hemoptysis, and death, hallmark clinical features of fat
28 thm (clinical signs of deep-vein thrombosis, hemoptysis, and pulmonary embolism as the most likely di
29 g toxicities observed included hypertension, hemoptysis, and stomatitis and were seen primarily at th
37 gery is recommended in patients with massive hemoptysis caused by thoracic vascular injury, arteriove
45 include nonproductive cough usually without hemoptysis, dyspnea, hypoxemia, a decrease in hematocrit
46 ciated with the Eisenmenger syndrome include hemoptysis, gout, cholelithiasis, hypertrophic osteoarth
47 s hospitalized (HR 2.8; CI 1.3-5.9), massive hemoptysis (HR 2.1; CI 1.1-3.9), and relative drop in FE
48 s were evaluated for pain, pneumothorax, and hemoptysis immediately following and at 1 day, 1 week, a
54 commendations for life-threatening pediatric hemoptysis incorporating underlying disease pathophysiol
61 r, including concurrent influenza infection, hemoptysis, multilobar infiltrates, and prehospital anti
62 plications included pulmonary edema (n = 1), hemoptysis (n = 1) and contralateral stent compression (
64 of CF with respiratory failure (RF, n = 65), hemoptysis (n = 33), antibiotic desensitization (n = 30)
66 a, no prior venous thromboembolism event, no hemoptysis, no unilateral leg swelling, and no estrogen
68 Mortality was independently associated with hemoptysis onset location, underlying diagnosis, and sev
69 , including those who were critically ill at hemoptysis onset or who became critically ill as a resul
70 ronchiectasis, renal dysfunction, inhospital hemoptysis onset, and higher Pediatric Logistic Organ Dy
72 rs, brain metastases, clinically significant hemoptysis, or inadequate organ function or performance
73 ay present with wheeze, chest pain, dyspnea, hemoptysis, or symptoms attributable to metastatic disea
79 ory diseases, such as COPD, in patients with hemoptysis, TAE of the BAA and of the pathological bronc
80 erall efficacy of BAE for initial control of hemoptysis was 75% (n = 22) after one session, 89% (n =
81 ition and pneumothorax, drain placement, and hemoptysis was assessed by using multivariable logistic
83 e occurred in 483 of the 1175 TTLBs (41.1%); hemoptysis was documented in 21 of the 1175 TTLBs (1.8%)
85 or non-household individuals with cough, and hemoptysis were modestly associated with closer resident
87 hold exposure to individuals with cough, and hemoptysis, were modestly associated with closer proximi
88 She had no fevers, chills, night sweats, hemoptysis, wheezing, chest pain, palpitations, orthopne
89 on could cause fever, cough, chest pain, and hemoptysis with signs consistent with severe pneumonia 1