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1 pleural effusions, intra-alveolar edema, and hemorrhagic lesions.
2 number (p = 0.007) and volume (p = 0.008) of hemorrhagic lesions.
3 ism for the evolution of atherosclerotic and hemorrhagic lesions.
4 -type CN3758 MDS lysates induced significant hemorrhagic lesions and luminal fluid accumulation, MDS
5 g neuroinflammatory responses accompanied by hemorrhagic lesions and neuronal impairment, which are l
6                             Age, temporal or hemorrhagic lesions, and cerebral spinal fluid lymphocyt
7 todermal lineages upon differentiation, form hemorrhagic lesions, and contribute to developing placen
8  is characterized by neurological disorders, hemorrhagic lesions, and frequent fatal outcomes.
9  in the deep cortical layers adjacent to the hemorrhagic lesions, and in the hippocampus.
10 avior of taxis toward serum, colonization of hemorrhagic lesions, and the consumption of serum nutrie
11 lacenta failed to occur in VHL -/- mice, and hemorrhagic lesions developed in the placenta.
12 s with variable enhancement, with associated hemorrhagic lesions in 11 of 37 patients (30%; 95% CI: 1
13 ent IM strain caused rapid bursal atrophy or hemorrhagic lesions in the bursa, as seen with the varia
14 egion in the vaccine strain failed to induce hemorrhagic lesions in the bursa.
15 is showed that the cats generating myoclonus hemorrhagic lesions in the retrorubral nucleus (RRN) and
16 tin-induced uveitis, cocaine-related retinal hemorrhagic lesions in utero, deferoxamine-related decre
17 elial restitution occurred faster (even over hemorrhagic lesions) in control rats.
18              Six eyes (7%) had predominantly hemorrhagic lesions initially, whereas this developed in
19 d echography in assessing the volume of late hemorrhagic lesions (intraclass correlation coefficient,
20 he sensor additionally detected ischemic and hemorrhagic lesions located deep inside the brain, and i
21 nti-VEGF therapy suggests that predominantly hemorrhagic lesions may develop within 3.5 years of init
22 r beta-catenin in brain endothelial cells of hemorrhagic lesions of hemorrhagic stroke patients.
23 re subjected ICH and neurologic deficits and hemorrhagic lesion outcomes were evaluated at 72 hours a
24 30) at 6 to 12 months had, on average, fewer hemorrhagic lesions (p = 0.003) and lower volume (p = 0.
25                                          The hemorrhagic lesions, quantitated both macroscopically an
26    The CMRglc of cortical areas (remote from hemorrhagic lesions), striatum, thalamus, brain stem, ce
27  MR imaging depicts significantly more small hemorrhagic lesions than does conventional GRE MR imagin
28 W MR imaging appeared to depict much smaller hemorrhagic lesions than GRE MR imaging.
29 hantom by helping identify proteinaceous and hemorrhagic lesions through assessment of their distinct
30 sive craniectomy to assess the size of acute hemorrhagic lesions, to measure midline structures and t
31            Furthermore, only in animals with hemorrhagic lesions was EHDV-2 isolated and/or erythrocy
32 ning cells and, to a lesser extent, to brain hemorrhagic lesions was identified.
33 nguishing endometriomas from nonendometrioma hemorrhagic lesions were calculated.
34                                              Hemorrhagic lesions were found preferentially in the tem
35                                              Hemorrhagic lesions were much more visible on SW MR imag
36 f patients (20 of 37, 54%) had intracerebral hemorrhagic lesions with a more severe clinical presenta