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1 those antibodies were employed to isolate a heparin binding protein.
2 ment leads to degranulation and secretion of heparin-binding protein.
3 e serine proteinase inhibitor (serpin) and a heparin-binding protein.
4 (PF4) is an abundant platelet alpha-granule heparin-binding protein.
5 eins, including many previously unidentified heparin-binding proteins.
6 lly related, extracellular matrix-associated heparin-binding proteins.
7 inity comparable to that of other identified heparin-binding proteins.
8 alpha-Chemokines are known heparin-binding proteins.
11 racellular matrix protein tenascin-R and two heparin-binding proteins, amphoterin and the heparin-bin
12 We have reported previously that Noggin is a heparin-binding protein and associates with the cell sur
13 recently showed that thyroglobulin (Tg) is a heparin-binding protein and that heparin inhibits bindin
14 vestigated, for example, as aptamers against heparin-binding proteins and as purine-motif triplex-for
15 bility-increasing protein, azurocidin (CAP37/heparin-binding protein), and neutrophil elastase were e
16 its target protease kallikrein 7 (KLK7) are heparin-binding proteins, and inhibition of KLK7 by vasp
20 eparin release, we demonstrated that Tg is a heparin-binding protein, as are several megalin ligands.
21 ple ionophores to model recognition sites of heparin-binding proteins at liquid/liquid interfaces.
22 we characterized one of the newly-discovered heparin-binding proteins, C-type lectin 14a (CLEC14A), a
23 peptide sequence analysis revealed that this heparin-binding protein corresponded to the extravesicul
26 phosphodiester oligonucleotides can bind to heparin-binding proteins (eg, basic fibroblast growth fa
30 studies have demonstrated elevated levels of heparin-binding protein (HBP) in patients with sepsis an
32 udies have shown that the neutrophil-derived heparin-binding protein (HBP), also known as CAP37 or az
34 tivated polymorphonuclear leukocytes release heparin-binding protein (HBP; also known as CAP37 or azu
36 nding sites for a diverse array of mammalian heparin binding proteins, including adhesive glycoprotei
37 ents presenting at the emergency department, heparin-binding protein is an early indicator of infecti
38 sic fibroblast growth factor (bFGF), another heparin-binding protein is increased in Alzheimer's dise
40 o wounding was demonstrated by the fact that heparin-binding proteins isolated from wounded, but not
41 f the latter patients had an elevated plasma heparin-binding protein level (>30 ng/mL) prior to devel
42 n polarized myeloma cells are incubated with heparin-binding proteins, like hepatocyte growth factor
43 studies indicated that Tia, as a prokaryotic heparin binding protein, may also interact via sulfated
48 ivity (>300-fold) for HPSE compared to other heparin-binding proteins (platelet factor 4 and antithro
53 atform is used to assess binding of specific heparin-binding protein targets at very high sensitivity
54 We demonstrate that this holds true for all heparin binding proteins tested but not for epidermal gr
55 way was able to interact with four different heparin-binding proteins tested, i.e., TSG-6, chemokines
57 early gene Cyr61, a secreted, cysteine-rich, heparin binding protein that promotes endothelial cell a
58 ed Cyr61 protein is a secreted, cystein-rich heparin-binding protein that associates with the cell su
59 rly gene, CYR61 is a secreted, cysteine-rich heparin-binding protein that associates with the extrace
60 Cyr61 is an extracellular matrix-associated heparin-binding protein that can mediate cell adhesion,
61 , and subsequent studies identified a second heparin-binding protein that co-purified with FGF-1.
62 mily, is an extracellular matrix-associated, heparin-binding protein that mediates cell adhesion, pro
63 ducible midkine gene encodes a highly basic, heparin-binding protein that possesses potential functio
64 f amphoterin and HB-GAM (Kd = 0.3-8 nM), two heparin-binding proteins that are developmentally regula
66 lthough they are traditionally classified as heparin-binding proteins, under normal physiological con
68 modimer, we sought to determine whether this heparin-binding protein was involved in the release of F
70 or glypican-1 insofar as no other identified heparin-binding proteins were isolated using our affinit
73 ere designed based on consensus sequences in heparin-binding proteins: XBBXBX and XBBBXXBX, where X a