ライフサイエンスコーパス: hepatic artery

1 cted administration of microspheres into the hepatic artery.

2 of which also required reconstruction of the hepatic artery.

3 ere placed on the portal vein and the common hepatic artery.

4 artery thrombosis in the small-caliber left hepatic artery.

5 hypoglycemic detection do not reside in the hepatic artery.

6 uch more effective when administered via the hepatic artery.

7 e joined to the bifurcation of the recipient hepatic artery.

8 right hepatic artery and the recipient main hepatic artery.

9 ontain a bile duct, and 9% did not contain a hepatic artery.

10 infusion of ethanol and iodized oil into the hepatic artery.

11 ts were injected with [125I]IUdR through the hepatic artery.

12 mesenteric artery, celiac artery, or common hepatic artery.

13 pseudoaneurysm that developed from the left hepatic artery.

14 ia a microcatheter positioned in the desired hepatic artery.

15 overed stent between celiac trunk and common hepatic artery.

16 ayed and dispersed compared with that of the hepatic artery.

17 2-French catheter, which was secured in the hepatic artery.

18 enhancement was measured in both tumors and hepatic arteries.

19 r) microspheres, which are injected into the hepatic arteries.

20 NHBD with right replaced and left accessory hepatic arteries.

21 two portal vein stenoses, and four redundant hepatic arteries.

22 Resistive indexes were determined for the hepatic arteries.

23 end-to-side portacaval shunt and ligation of hepatic arteries.

24 ntrations were lower in the portal vein than hepatic artery (0.45 +/- 0.03 vs. 0.48 +/- 0.02, respect

25 % of hepatic veins (six of 40), and 14.2% of hepatic arteries (10 of 70) encompassed within the ablat

26 erlobular bile ducts was 13 +/- 4 microm, of hepatic arteries 12 +/- 5 microm, and of portal veins 35

27 ng beads (DEB-TACE), which was given via the hepatic artery 2-5 weeks after randomisation and accordi

28 dl1520 was infused into the hepatic artery (2 x 10(12) particles) on days 1 and 8 as

29 al segments included the coeliac trunk (50), hepatic artery (29), superior mesenteric artery (35), an

30 vein (97%), superior mesenteric vein (94%), hepatic artery (93%), and superior mesenteric artery (93

31 nhibitory antibodies to human F.IX following hepatic artery administration of an AAV-hF.IX vector, wh

32 scular conduits and performing microvascular hepatic artery anastomoses are the critical steps in imp

33 usly described requires a microvascular left hepatic artery anastomosis.

34 ts per portal tract, compared to 2.6 +/- 2.3 hepatic arteries and 0.7 +/- 0.7 portal veins.

35 ve index at 15 minutes was 12% in the proper hepatic arteries and 20% in the intrahepatic arteries.

36 VT occurred in 10 (9 hepatic artery and 1 portal vein) of 117 orthotopic live

37 administered AAV8-FIX in 16 macaques via the hepatic artery and assessed the effects of (1) preexisti

38 garding primary nonfunction and dysfunction, hepatic artery and biliary complications, and retranspla

39 Hepatic artery and liver glucose concentrations were sig

40 oped multiple mycotic aneurysms of the right hepatic artery and massive splenic infarction as rare co

41 grafts, vascular thrombosis (VT), including hepatic artery and portal vein (PVT), and the presence o

42 lly induced cholangiocyte proliferation, the hepatic artery and portal vein also undergo marked proli

43 ho fasted underwent duplex Doppler US of the hepatic artery and portal vein both before and after adm

44 ein, and femoral artery), perfusion rates in hepatic artery and portal vein were measured, and hepati

45 and portal vein) catheters and flow probes (hepatic artery and portal vein) implanted >16 days befor

46 formed using NMP via double perfusion of the hepatic artery and portal vein.

47 nce between the right main (or second-order) hepatic artery and the corresponding right main (or seco

48 yltransferase specifically terminated in the hepatic artery and the myenteric plexus of the stomach a

49 Doppler probe to monitor the patency of the hepatic artery and the portal vein during the early post

50 moses were generally between the donor right hepatic artery and the recipient main hepatic artery.

51 , there are two interlobular bile ducts, two hepatic arteries, and one portal vein per portal tract,

52 eaker expression detectable in portal veins, hepatic arteries, and the sinusoids.

53 ping the portal vein, 51% after clamping the hepatic artery, and 31% after clamping both.

54 lamping the portal vein, during clamping the hepatic artery, and during clamping both.

55 at least one profile each of a portal vein, hepatic artery, and interlobular bile duct numbered 11 +

56 ensional reconstruction of the biliary tree, hepatic artery, and portal vein in normal rats and rats

57 nts, whereas the length of the biliary tree, hepatic artery, and portal vein remain unchanged.

58 The total volume of the biliary tree, hepatic artery, and portal vein was increased 18, 4, and

59 hrombosis of the intrahepatic portion of the hepatic artery, and sepsis.

60 00-300-mum SEBs (1.5 mg of sunitinib) in the hepatic artery, and six animals received saline injectio

61 derwent urgent surgical repair of a ruptured hepatic artery aneurysm.

62 Hepatic artery aneurysms (HAAs) constitute 14% to 20% of

63 Hepatic artery aneurysms represent a significant risk fo

64 Pseudoaneurysms of the hepatic artery are a rare complication of liver transpla

65 duct 10 mm or farther from the corresponding hepatic artery are at high risk for biliary complication

66 Hepatic artery area index was not significantly differen

67 he presence of a replaced or accessory right hepatic artery arising independently from the superior m

68 nt chemotherapy; four involved predominantly hepatic-artery-based chemotherapy or embolisation; and s

69 rs perfused via both the portal vein and the hepatic artery because the hepatic artery provides the b

70 By day 8, hepatic artery blood flow decreased by 50.0% (-59.08% to

71 th no significant changes in blood pressure, hepatic artery blood flow, serum hepatic enzyme levels,

72 ective radiological embolization of the left hepatic artery branches was necessary.

73 ductopenia but also by loss of portal tract hepatic artery branches.

74 d bicarbonate excretion when infused via the hepatic artery but not the portal vein.

75 The transplant hepatic artery, celiac trunk, superior mesenteric artery

76 patients had a variant origin of the common hepatic artery (CHA) arising from either the superior me

77 rgical sympathetic denervation of the common hepatic artery (CHADN) in improving glucose tolerance.

78 We conclude that systemic and hepatic-artery chemotherapy or chemoembolisation have no

79 h involved modalities other than systemic or hepatic-artery chemotherapy or embolisation.

80 3%) revascularized from the recipient common hepatic artery, compared with 2 of 51 grafts (3.9%) usin

81 The hepatic artery diameter was significantly larger in infa

82 d that administration of PRRT via the proper hepatic artery did not reproduce the increase in hepatic

83 the coil dislodged into the right branch of hepatic artery distal to the site of pseudoaneurysm.

84 nuous perfusion via both the portal vein and hepatic artery (dual perfusion), and (3) trapping viral

85 liably delivered to liver metastases via the hepatic artery, eliminating need for tumor embolization.

86 tide, the role of receptor-targeted therapy, hepatic-artery embolisation, and the arguments against c

87 Hepatic artery embolization accompanied an infusion of d

88 ceptor scintigraphy, long-acting octreotide, hepatic artery embolization, endoscopic mucosal resectio

89 vel therapies are being developed, including hepatic artery embolization, hepatic artery infusion of

90 heters and Doppler flow probes (portal vein, hepatic artery, external iliac artery) were implanted 17

91 lly unchanged during in situ infusions while hepatic artery flow nearly doubled (P=0.03).

92 teatosis increased, there was a reduction in hepatic artery flow volume, portal vein flow volume and

93 ), hepatic artery pulsatility index (PI) and hepatic artery flow volume.

94 rfusion injury, but had lower intraoperative hepatic artery flow with higher incidence of hepatic art

95 rols, high-dose focal liver irradiation with hepatic artery floxuridine prolongs survival in patients

96 with vena caval venting; and group 4 (n=19), hepatic artery flush with vena caval venting.

97 over time, as well as the increased use of 2 hepatic arteries for anastomosis during graft implantati

98 done by interruption of the portal vein and hepatic artery for 5, 10, and 20 min (5-10, 10-10, and 2

99 was induced by clamping the portal vein and hepatic artery for 9 min.

100 as conduit in combination with the recipient hepatic artery for renal graft inflow.

101 udy was to determine the contribution of the hepatic artery, gastroduodenal artery, and portal vein t

102 between the donor's right main bile duct and hepatic artery generally was small (mean distance, 3.8 m

103 established across the liver via a localized hepatic artery glucose infusion.

104 gation protocol, glucose was infused via the hepatic artery (HA protocol) to maintain liver normoglyc

105 Quality of hepatic artery (HA) and portal vein (PV) visualization,

106 Liver donation, procurement team origin, hepatic artery (HA) arising from the superior mesenteric

107 nsplantation (OLT) often represent a sign of hepatic artery (HA) complication (HAC).

108 s of 36 months revealed portal vein (PV) and hepatic artery (HA) complications of cryopreserved graft

109 e is known about the value of intraoperative hepatic artery (HA) flow measurement on the development

110 Little is known about hepatic artery (HA) patency and patient clinical course

111 ygen content and perfusion pressure than the hepatic artery (HA).

112 the route of input: portal vein (PV) versus hepatic artery (HA).

113 gle-vessel (portal vein; PV) or dual-vessel (hepatic artery [HA] + PV) perfusions during extracorpore

114 Historically, the hepatic artery has been considered mainly responsible fo

115 Anatomic variants in the origin of the right hepatic artery have led to various approaches at the tim

116 d after experimental distal occlusion of the hepatic arteries in the rats were bypassed by vessels si

117 tients, and the GDA originated distal to one hepatic artery in 25 (4.2%) patients in whom both hepati

118 Reconstruction of the hepatic artery in infants undergoing liver transplantati

119 ough catheters selectively placed within the hepatic artery in rodent models and subsequent distribut

120 The origin of APFs is the hepatic artery in the majority of patients (n = 56, 65%)

121 c by a 3-min ligation of the portal vein and hepatic artery in vivo before harvest and then cultured

122 n this model was exclusively supplied by the hepatic artery, in contrast to conventional orthotopic H

123 r trials have shown that surgery followed by hepatic artery infusion (HAI) of floxuridine (FUDR) alte

124 [125I]IUdR in hepatic tumors, thereby making hepatic artery infusion a suitable mode of delivery for

125 with resection and with or without adjuvant hepatic artery infusion chemotherapy were retrospectivel

126 therapies such as portal vein embolization, hepatic artery infusion chemotherapy, transarterial chem

127 520, also known as Onyx-015) administered by hepatic artery infusion in patients with gastrointestina

128 resection of primary colorectal cancer, and hepatic artery infusion in the setting of established he

129 Adjuvant hepatic artery infusion may mitigate the worse outcomes

130 rticularly in light of a patient death after hepatic artery infusion of a replication-defective adeno

131 oped, including hepatic artery embolization, hepatic artery infusion of chemotherapy, and radiofreque

132 We conducted a phase I study of a 30-minute hepatic artery infusion of melphalan via a percutaneousl

133 derwent this resection during laparotomy for hepatic artery infusion pump placement.

134 Adjuvant hepatic artery infusion was independently associated wit

135 approximately 1 cm) were subject to a single hepatic artery injection of LDL nanoparticles (2 mg/kg)

136 Each CSV hepatic artery interposition graft has been complicated

137 r portal vein interposition n=3, and CSV for hepatic artery interposition n=2) were utilized in 7 LRL

138 xpressing human F.IX was infused through the hepatic artery into seven subjects.

139 The hepatic artery is also a nonsurgical avenue for inducing

140 Hepatic artery is the main blood supply to bile duct and

141 d nineteen (19.8%) patients had variant left hepatic arteries (LHAs), and 89 (14.8%) had variant righ

142 umoniae poorly (50% mortality) compared with hepatic artery ligated controls (12% mortality) at 7 day

143 ), a hypoxia-activated cytotoxic agent, with hepatic artery ligation (HAL), which recapitulates trans

144 The patient underwent urgent right hepatic artery ligation and splenectomy.

145 Surgery (hepatic artery ligation n = 26; resection n = 13; emboli

146 Operative hepatic artery ligation was the initial method of contro

147 Control rats underwent laparotomy and hepatic artery ligation.

148 essment of significant fibrosis, while RI of hepatic artery may serve as a useful adjunct to serum bi

149 al modification obviates the need for a left hepatic artery microvascular anastomosis and should lowe

150 he hilar pathology was often associated with hepatic artery (n = 15) or portal vein thrombosis (n = 1

151 became clinically jaundiced with evidence of hepatic artery narrowing on ultrasound that resolved wit

152 hypoglycemic detection may also occur in the hepatic artery, normoglycemia was established across the

153 rotic debris and sludge were associated with hepatic artery occlusion in seven of nine (78%) and 16 o

154 bile duct necrosis are due to ischemia from hepatic artery occlusion, sludge may also have an ischem

155 s demonstrated by the administration of into hepatic arteries of a VX2 tumor-bearing rabbit under flu

156 rbable gelatin powder were injected into the hepatic artery of 50 rats.

157 le pumps were used to deliver FdUrd into the hepatic artery of animals at a rate of 0.3 mg/kg/day in

158 vo, administration of fusogenic VSV into the hepatic artery of Buffalo rats bearing syngeneic multifo

159 virus administered in the portal vein or the hepatic artery of nude rats bearing intrahepatic LoVo co

160 Injection of LDL-DHA into the hepatic artery of rats selectively deregulated redox rea

161 F) is an abnormal, direct connection between hepatic artery or its branch and the portal vein.

162 There was no incidence of hepatic artery or portal vein thrombosis.

163 ic artery in 25 (4.2%) patients in whom both hepatic arteries originated from the CHA.

164 the 12 donors had a completely replaced left hepatic artery originating from the left gastric artery,

165 )Y-DOTATOC were administered into the proper hepatic artery over 30 min.

166 e follow-up period, during which we analyzed hepatic artery patency with Doppler ultrasound at 1, 3,

167 n of right portal vein maintaining the right hepatic artery patent.

168 In two patients, the proper hepatic artery (PHA) was the first branch of the SMA and

169 ic bile ducts, whereas NTPDase1 was found in hepatic arteries, portal veins, and hepatic central vein

170 demonstrate that the dendritic nature of the hepatic artery, portal vein and hepatic vein can be pred

171 nary artery), perfusion rates were measured (hepatic artery, portal vein, and cardiac output), and ph

172 On the other hand, hepatic artery, portal vein, and hepatic vein cortisol c

173 The incidence of hepatic artery, portal vein, and hepatic vein thrombosis

174 g image masks and texture mapping of tumors, hepatic artery, portal vein, and the hepatic veins was d

175 er injection of liposomal doxorubicin in the hepatic artery, portal vein, or tail vein.

176 pseudoaneurysm in the vicinity of the right hepatic artery probably originating from the cystic arte

177 rtal vein and the hepatic artery because the hepatic artery provides the blood supply to bile ducts.

178 We describe a 70-year-old woman who had a hepatic artery pseudoaneurysm after orthotopic liver tra

179 Ruptured hepatic artery pseudoaneurysm is a rare condition that i

180 Ruptured hepatic artery pseudoaneurysm stands as a deadly conditi

181 e present a case of a spontaneously ruptured hepatic artery pseudoaneurysm that occurred after a blun

182 study demonstrates a spontaneously ruptured hepatic artery pseudoaneurysm which emerged following a

183 stenoses, six hepatic artery thromboses, two hepatic artery pseudoaneurysms, two splenic artery aneur

184 volume, hepatic artery resistive index (RI), hepatic artery pulsatility index (PI) and hepatic artery

185 10 mCi [131I]IUdR through the sideport of a hepatic artery pump.

186 ET/CT images obtained after 65 right or left hepatic artery radioembolizations in 59 patients.

187 t (defined as a previously constructed aorto-hepatic artery remnant using donor iliac artery), and CM

188 w portal flow (<10 cm/s) combined with lower hepatic artery resistance index (<0.65) are strong warni

189 An elevation of hepatic artery resistance that is probably due to arteri

190 vein peak velocity, portal vein flow volume, hepatic artery resistive index (RI), hepatic artery puls

191 blinded) fashion, nine sonographers measured hepatic artery resistive index and systolic acceleration

192 ies (LHAs), and 89 (14.8%) had variant right hepatic arteries (RHAs).

193 e severe hepatosteatosis group, although the hepatic artery RI and PI values were not statistically s

194 In the mild hepatosteatosis group, hepatic artery RI and PI values were statistically signi

195 with volume rendering technique detected 10 hepatic artery stenoses, six hepatic artery thromboses,

196 However, the incidence of both hepatic artery stenosis (16.6% versus 5.4%; P = 0.001) a

197 ntional radiology treatment of patients with hepatic artery stenosis (HAS) after liver transplantatio

198 ar complications included three instances of hepatic artery stenosis (NS compared with non-BCS liver

199 Hepatic artery stenosis and thrombosis are common compli

200 Two patients developed hepatic artery stenosis and were treated with balloon an

201 Screening for the development of hepatic artery stenosis prior to late thrombosis may be

202 MATERIAL/METHODS: Hepatic artery stenosis was diagnosed and treated by end

203 computed tomography (CT) detected a moderate hepatic artery stenosis, while conventional angiography

204 eased incidence of biliary complications and hepatic artery stenosis.

205 In the hepatic artery, the concentration of gadolinium was much

206 stases derive the majority of blood from the hepatic artery, the regional delivery of chemotherapy ca

207 Despite having a patent hepatic artery, the remaining six patients (35%) died fr

208 Porcine livers were cannulated via the hepatic artery, then perfused with PBS, followed by succ

209 s derive most of their blood supply from the hepatic artery; therefore, for patients with hepatic met

210 had prior perioperative portal vein and/or 5 hepatic artery thrombectomies.

211 que detected 10 hepatic artery stenoses, six hepatic artery thromboses, two hepatic artery pseudoaneu

212 suffered from portal vein, hepatic vein, or hepatic artery thrombosis

213 An increased rate of graft failure due to hepatic artery thrombosis <=14 days from initial LT was

214 he most common etiologies of graft loss were hepatic artery thrombosis (33.4%), acute or chronic reje

215 days following primary transplant) included hepatic artery thrombosis (5), chronic rejection (4), se

216 and the main cause for retransplantation was hepatic artery thrombosis (6 [2%] vs 17 [6%]).

217 Early hepatic artery thrombosis (eHAT) after liver transplanta

218 ansplant recipients were found to have early hepatic artery thrombosis (HAT) after a median of 7 post

219 tation, and outcome of management of delayed hepatic artery thrombosis (HAT) after liver transplant (

220 Hepatic artery thrombosis (HAT) after liver transplantat

221 Hepatic artery thrombosis (HAT) after liver transplantat

222 Whilst causes of hepatic artery thrombosis (HAT) after liver transplantat

223 igate whether center volume impacts the rate hepatic artery thrombosis (HAT) and patient survival aft

224 Although the clinical features of early hepatic artery thrombosis (HAT) are well defined, the fe

225 Hepatic artery thrombosis (HAT) can be a devastating com

226 Hepatic artery thrombosis (HAT) did not occur in any pat

227 The incidence of hepatic artery thrombosis (HAT) following orthotopic liv

228 Hepatic artery thrombosis (HAT) increases morbidity and

229 Hepatic artery thrombosis (HAT) is a cause of morbidity

230 trahepatic biliary strictures (IHBS) without hepatic artery thrombosis (HAT) is a serious complicatio

231 Hepatic artery thrombosis (HAT) is a significant cause o

232 Hepatic artery thrombosis (HAT) remains a devastating co

233 The most common predisposing factor was hepatic artery thrombosis (HAT), which occurred in eight

234 ventional angiography or surgery: transplant hepatic artery thrombosis (n = 3) or stenosis (n = 3), p

235 hepatic artery flow with higher incidence of hepatic artery thrombosis (P = 0.043) and biliary compli

236 titis C (P<0.0001), as well as occurrence of hepatic artery thrombosis (P=0.0018) and prolonged cold

237 reasons (primary graft nonfunction (PGNF) or hepatic artery thrombosis [HAT]), survival was significa

238 ow Protein S may be causally associated with hepatic artery thrombosis after OLT.

239 ecurrent cytomegalovirus activation, one has hepatic artery thrombosis and one is likely to have pers

240 In an attempt to decrease the incidence of hepatic artery thrombosis and to increase collaboration

241 ed (more than 4 weeks after transplantation) hepatic artery thrombosis are less clearly defined.

242 ve a significantly higher incidence of early hepatic artery thrombosis compared with non-FAP transpla

243 The incidence of hepatic artery thrombosis has decreased from 22% to 0% w

244 Other causes for re-OLT included hepatic artery thrombosis in 10 (2.6%), chronic rejectio

245 e believe that emergent revascularization of hepatic artery thrombosis in asymptomatic patients and r

246 ical implications in the prevention of early hepatic artery thrombosis in FAP patients after liver tr

247 A single vascular complication occurred (one hepatic artery thrombosis in group B).

248 nastomosis and should lower the incidence of hepatic artery thrombosis in the small-caliber left hepa

249 Reoperations identified two cases of hepatic artery thrombosis not previously identified by d

250 ant centers in North America with the lowest hepatic artery thrombosis rate and biliary complication

251 en advocated and has resulted in a decreased hepatic artery thrombosis rate in both the adult and ped

252 Complications included one case each of hepatic artery thrombosis requiring retransplantation, b

253 reas 97 patients required retransplantation; hepatic artery thrombosis was the most common indication

254 biliary strictures, primary nonfunction and hepatic artery thrombosis were observed in the total coh

255 he primary graft and 1 from complications of hepatic artery thrombosis).

256 function, 50% for chronic rejection, 60% for hepatic artery thrombosis, and 60% for recurrent HCV.

257 -grafts were constructed for recipients with hepatic artery thrombosis, and double donor arteries wer

258 ence of ischemic type biliary strictures and hepatic artery thrombosis, and evaluated the causes of g

259 uded primary nonfunction, chronic rejection, hepatic artery thrombosis, and recurrent disease.

260 transplants; mainly for primary nonfunction, hepatic artery thrombosis, and recurrent primary disease

261 he main indication for retransplantation was hepatic artery thrombosis, and the major cause of death

262 Although duplex US remains a good screen for hepatic artery thrombosis, angiography is strongly recom

263 er recipients who smoke have higher rates of hepatic artery thrombosis, biliary complications, and ma

264 ibed as an independent risk factor for early hepatic artery thrombosis, more studies to understand th

265 acute rejection at day 7, the development of hepatic artery thrombosis, nonanastomotic biliary strict

266 In addition, we reported two cases of hepatic artery thrombosis, one case of wound dehiscence

267 The incidence of hepatic artery thrombosis, portal vein stenosis/thrombos

268 There were no significant differences in hepatic artery thrombosis, portal vein thrombosis, prima

269 The occurrence is, partly, attributed to hepatic artery thrombosis, which is considered to be the

270 Early detection of hepatic artery thrombosis, with subsequent correction an

271 lure due to chronic rejection and late onset hepatic artery thrombosis.

272 nsplantation, was frequently associated with hepatic artery thrombosis.

273 ation is associated with a high incidence of hepatic artery thrombosis.

274 thrombosis) enable prediction of subsequent hepatic artery thrombosis.

275 No patient had evidence of hepatic artery thrombosis.

276 +/- 0.1 microg/dl (P < 0.001 by ANOVA) from hepatic artery to portal vein to hepatic vein, respectiv

277 udy, we described the first procedure of via hepatic artery transplantation of human fetal biliary tr

278 Median hepatic artery velocity was greatest in diffuse lesions

279 31%; thrombocytopenia n = 12, 10%).Increased hepatic artery velocity was seen in 63 (56%).

280 tion of the resistive index (P < .01) of the hepatic arteries was observed after ingestion of oral CT

281 A selective embolization of the right hepatic artery was carried out.

282 In one case the left hepatic artery was divided and the celiac trunk was main

283 The right hepatic artery was divided, and the celiac trunk was mai

284 transcatheter delivery of the MTC-DOX to the hepatic artery was monitored by using intraprocedural MR

285 donor's second-order duct and corresponding hepatic artery was more variable (mean distance, 6.6 mm;

286 ylactic acid (1-5 microns) injected into the hepatic artery was seen circulating through the sinusoid

287 The recipient common hepatic artery was used to provide arterial inflow in 22

288 Two right sectorial inferior hepatic arteries were embolized.

289 In a retrospective review, hepatic arteries were evaluated on the basis of arterial

290 Double hepatic arteries were seen in 22 (3.7%) patients.

291 dules delivered by an infusion pump into the hepatic artery were mathematically investigated.

292 Intraoperative flows of the portal vein and hepatic artery were routinely measured.

293 dy negative) produced aneurysms of renal and hepatic arteries, whereas small vessel vasculitis affect

294 patients (75%) were asymptomatic with patent hepatic artery, which was confirmed by multislice comput

295 ver tumors, catheters were positioned in the hepatic arteries with conventional angiographic guidance

296 ives blood from both the portal vein and the hepatic artery, with the peak of the portal vein time-ac

297 sion of the portal veins, hepatic veins, and hepatic arteries within and directly abutting the ablati

298 es were significantly correlated with patent hepatic arteries within the ablation zone (P = .02) but

299 esions were dysplastic nodules with unpaired hepatic arteries within.

300 ntly results in an increase in the number of hepatic arteries without affecting bile duct formation.