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1 enal, pulmonary, neurological, skeletal, and hepatic damage.
2 patients with HCC in NCL lacked any sign of hepatic damage.
3 indicators of ongoing viral replication and hepatic damage.
4 nocytes in triggering liver inflammation and hepatic damage.
5 e detrimental host inflammatory response and hepatic damage.
6 ng their potential as a sensitive measure of hepatic damage.
7 disease because of the nature of preexisting hepatic damage.
8 ion plays a critical role in immune-mediated hepatic damage.
9 ups, which correlated with a lower degree of hepatic damage.
10 n of CXCR3(+) lymphocytes into the liver and hepatic damage.
11 identify candidate genes that contribute to hepatic damage.
12 nt, showing increased activation of pAKT and hepatic damage.
13 a cytokine environment conducive to limited hepatic damage.
14 mice against endotoxin- and ischemia-induced hepatic damage.
15 lalanine, or tyrosine) resulted in renal and hepatic damage.
16 ansaminase (ALT) levels at 6 hours confirmed hepatic damage.
17 contribution to PMN and platelet adhesion in hepatic damage.
18 of lethally injected mice, indicating severe hepatic damage.
19 ared with females, male mice had more severe hepatic damage.
20 are potential markers for Geniposide-induced hepatic damage.
22 ion, PAI-1 protein is a negative effector of hepatic damage after HS-R through its influence on class
24 r instance, older organisms showed extensive hepatic damage, along with increased morbidity and morta
27 ction, pronounced weight loss and markers of hepatic damage and disease were observed exclusively in
28 e hepatoprotective feature of STAT3 prevents hepatic damage and fibrosis under the condition of persi
29 vels positively correlate with the degree of hepatic damage and serum TNF-alpha, MIP-1alpha, and MIP-
30 Moreover, AM/AMBP-1 significantly attenuated hepatic damage and the elevation of plasma lactate, and
31 over, administration of AM/AMBP-1 attenuated hepatic damage and the increase in plasma lactate and pr
32 Circulating and hepatic GH and EGF levels, hepatic damage, and regeneration parameters were evaluat
34 he absence of IL-12p40 induction and serious hepatic damage are involved in the death of IRF-1-/- mic
37 polysaccharide (LPS)-induced endotoxemia and hepatic damage associated with decreased proinflammatory
38 a1-antitrypsin Z (ATZ) in hepatocytes causes hepatic damage by a gain-of-function, "proteotoxic" mech
42 bility of IRF-1-/- and ICSBP-/- mice; and 3) hepatic damage caused by Brucella virulence contributes
43 expression by combining 2-AAF with selective hepatic damage caused by either carbon tetrachloride (CC
44 IL-10 is known to suppress the extent of hepatic damage caused by parasite ova during natural inf
45 Nase, a NET inhibitor, significantly reduced hepatic damage despite prior administration of IL-17A, a
49 mice had decreased HO-1 levels, exacerbated hepatic damage/frequency of TUNEL+ cells, increased mRNA
50 vivo-morpholino in Mdr2(-/-)-mice decreased hepatic damage; H2HR protein expression and MC presence
53 then pass through the liver, contributing to hepatic damage, impaired microbial clearance, and impair
63 lso accompanied by a significant increase in hepatic damage shown as more severe fat accumulation, he
64 causes a range of toxic responses, including hepatic damage, steatohepatitis, and a lethal wasting sy
65 ing high levels of FasL on the initiation of hepatic damage through analysis of chemokine and chemoki
72 evaluate the degree of primary and secondary hepatic damage, we generated a transgenic mouse with liv
73 pool can be instilled by BS feeding, without hepatic damage, which makes Hrn mice an attractive model
74 evalence of exposure to factors potentiating hepatic damage with acute hepatitis B contributed to the