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1  a decline in ALDH1A1 activity, a protective hepatic enzyme.
2 hematological dysfunction, coagulopathy, and hepatic enzyme.
3 llular damage as revealed by serum levels of hepatic enzymes.
4 rmore, GBR diet can also reduce the level of hepatic enzymes.
5 epatocellular damage and elevation of plasma hepatic enzymes.
6 erlying sex differences in the expression of hepatic enzymes active in the metabolism of drugs, stero
7 ed by comparing results from measurements of hepatic enzyme activities in mice fed either deficient o
8 astric for 4 days), whereas the elevation of hepatic enzyme activities was less pronounced.
9 prague Dawley rats inhibited the increase in hepatic enzyme activity and mRNA levels of CYP1A1, 1A2,
10  disruption has pleiotropic implications for hepatic enzyme activity, lipid processing, nuclear recep
11 resent study was to examine whether elevated hepatic enzymes (alanine aminotransferase [ALT], asparta
12 of lipid peroxidation levels and activity of hepatic enzymes (alanine transferase, ALT and aspartate
13 lbutyrate increases the proportion of active hepatic enzyme and unphosphorylated form over the inacti
14 ic grade 3 to 4 toxicity, including elevated hepatic enzymes and hyperbilirubinemia, was less common.
15          In patients on each diet, levels of hepatic enzymes and markers of inflammation decreased, i
16 atment significantly reduced serum levels of hepatic enzymes and TNF-a, IL-1-B, and IL-6.
17 ey require replacement of a single deficient hepatic enzyme, and multiple small-animal models exist f
18 rbital, and primidone are potent inducers of hepatic enzymes, and decrease the plasma concentration o
19 of atherosclerosis, although the CE from the hepatic enzyme appeared to promote more atherosclerosis
20                       Serum concentration of hepatic enzymes are linked to liver dysfunction, metabol
21 ta remains controversial, although screening hepatic enzymes are recommended in the evaluation of non
22 ncy of PDA on ARG2 rather than the principal hepatic enzyme ARG1 opens a therapeutic window for obesi
23 asting total cholesterol, uric acid, and the hepatic enzymes aspartate transaminase and alanine trans
24 r plasma concentrations of the intracellular hepatic enzyme, aspartate aminotransferase.
25 es, plasma proteins, transporters, and other hepatic enzymes at levels equal to adult liver tissue.
26 lirubinemia resulting from deficiency of the hepatic enzyme bilirubin-UDP-glucuronosyltransferase.
27 erload disease, weakness, fatigue, increased hepatic enzyme concentrations, right upper quadrant pain
28                  The NH2-terminal peptide of hepatic enzyme contained 18 amino acids not found in enz
29 PV are both substrates and inhibitors of the hepatic enzyme, cytochrome P450 3A, and the drug transpo
30                    Monogenic diseases due to hepatic enzymes deficiency result in chronic hyperoxalur
31 rutinib group than in the placebo group were hepatic enzyme elevations (four [6%] vs 0), headache (th
32 f an immune-based mechanism for the observed hepatic enzyme elevations.
33  and transcriptional alterations but without hepatic enzyme elevations.
34 tein kinase C (aPKC) in liver and muscle and hepatic enzyme expression in mice consuming a moderate h
35 ostane receptor (hCAR) and its regulation of hepatic enzyme expression.
36                                          The hepatic enzyme glycine N-methyltransferase (GNMT) plays
37                      DLTs comprised elevated hepatic enzymes, hypophosphatemia, and thrombocytopenia.
38 d in four of seven patients (phase 1b study: hepatic enzyme increased, dizziness, aphthous ulcer, art
39 terin (BH4) is an important cofactor for two hepatic enzymes, inducible nitric oxide synthase (iNOS)
40             As has been observed previously, hepatic enzyme induction with PB or betaNF shifted the l
41  genetic polymorphisms encoding CYP2C19, the hepatic enzyme involved in biotransformation of clopidog
42 These data suggest that UGT2B10 is the major hepatic enzyme involved in nicotine/cotinine glucuronida
43 ochrome P450 1A2 (CYP1A2) is a predominantly hepatic enzyme known to be important in the metabolism o
44 dverse events were anemia (65%) and elevated hepatic enzyme levels (49%).
45        During the initial 24 weeks, elevated hepatic enzyme levels (P<.001), gastrointestinal tract c
46 en suspected in patients with abnormal serum hepatic enzyme levels and obstruction of the biliary sys
47  most pronounced in individuals with reduced hepatic enzyme levels, as occurs in approximately 10% of
48              Recipient survival rates, serum hepatic enzyme levels, coagulation, and metabolic parame
49 d pressure, hepatic artery blood flow, serum hepatic enzyme levels, or in weight compared with contro
50 ncreases in serum creatinine, uric acid, and hepatic enzyme levels.
51 ncreases in serum creatinine, uric acid, and hepatic-enzyme levels were also observed.
52 ncreases in serum creatinine, uric acid, and hepatic-enzyme levels.
53 t from BMI in predicting increased levels of hepatic enzymes, likely as a result of unrecognized fatt
54 e encoding MAT(alpha)1, the subunit of major hepatic enzymes MAT I ([alpha1]4) and III([alpha1]2).
55 this condition, but replacement of deficient hepatic enzymes might restore excretion of porphyrin pre
56 clerosis of the aorta, and upregulation of a hepatic enzyme of lipoprotein secretion, stearoyl coenzy
57 he identity, function, and regulation of the hepatic enzymes potentially involved in catalyzing the h
58 ion combined to control some, but not other, hepatic enzyme programs.
59 r weight gain and increases in the levels of hepatic enzymes, prolactin, fasting glucose, fasting tot
60 rosmolar rats were observed to have elevated hepatic enzyme release and decreased bile production com
61       Both MP regimens significantly reduced hepatic enzyme release and improved bile production, cle
62 ity of tryptophan 2,3-dioxygenase (TDO), the hepatic enzyme responsible for tryptophan catabolism.
63 tophan 2,3-dioxygenase (TDO) is an unrelated hepatic enzyme that also degrades tryptophan along the k
64  I iodothyronine 5'-deiodinase (5' D-I), the hepatic enzyme that converts thyroxine to T3 and that is
65 hosphate synthetase I (CPSI) is an essential hepatic enzyme that initiates the urea cycle.
66 fluxes and changes in the gene expression of hepatic enzymes that closely resemble those of fasting.
67 iciency on the expressions and activities of hepatic enzymes that regulate transmethylation reactions
68 ctor aryl hydrocarbon receptor (AhR) and the hepatic enzyme tryptophan 2,3-dioxygenase, which provide
69 A nephropathy was determined by blocking the hepatic enzyme uricase with oxonic acid (CSA-OA).
70 hat regulation by environmental compounds of hepatic enzymes via CAR and PXR may have impact on the m
71                                Elevations in hepatic enzymes were detected in a number of in trial pa
72  C-reactive protein level were elevated, and hepatic enzymes were normal in the laboratory findings.
73  Twelve patients demonstrated an increase in hepatic enzymes without jaundice during the pregnancy.