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1 use these genes to predict the likelihood of hepatic metastasis.
2 ls in HEC of Wls(HEC-KO) mice did not affect hepatic metastasis.
3 tumor growth, retroperitoneal invasion, and hepatic metastasis.
4 eases with tumor progression, and relates to hepatic metastasis.
5 ions for the role of CEA as a facilitator of hepatic metastasis.
6 lain CEA-induced enhancement of experimental hepatic metastasis.
7 be clinically targeted for the treatment of hepatic metastasis.
8 Three of them died, and 1 is alive with hepatic metastasis.
9 cantly decreased pancreatic tumor growth and hepatic metastasis and completely inhibited retroperiton
10 enesis completely abolished establishment of hepatic metastasis and formation of secondary metastasis
11 to be highly expressed in a cohort of human hepatic metastasis and their primary colorectal tumors,
12 ferring selective potential for formation of hepatic metastasis, as well as a possible target to prev
13 ement in the CT-based response assessment of hepatic metastasis between the two observers was good (k
15 the results indicate that RHAMM(B) promotes hepatic metastasis by islet tumor cells, perhaps through
16 Data on demographics, primary tumor, and hepatic metastasis characteristics, as well as details o
18 ol weight form, yet not sensitive enough for hepatic metastasis detection; and (b) for radioimmunothe
20 ropositive for aquaporin-4 IgG and who had a hepatic metastasis from a small-bowel neuroendocrine tum
21 mbryonic antigen (CEA) causes enhancement of hepatic metastasis from colorectal cancer is not defined
31 ory response in the liver that is needed for hepatic metastasis, not only through direct, paracrine e
32 tumor growth, retroperitoneal invasion, and hepatic metastasis of human pancreatic carcinomas in mic
34 indings, one patient had colon cancer with a hepatic metastasis, one patient had a hepatic angioma, o
38 s for migration, adhesion, angiogenesis, and hepatic metastasis showed that C. albicans promotes a mo
39 in which APOBEC3G promotes colorectal cancer hepatic metastasis through inhibition of miR-29-mediated
42 ild-type mice confirming that Notch controls hepatic metastasis via modulation of HEC adhesion molecu
46 wth on the cecum as well as the frequency of hepatic metastasis was significantly higher in control m
47 mor transplantation models, and inhibits the hepatic metastasis when tumor cells were transplanted in
48 ld-type (WT) CEA also increases experimental hepatic metastasis, whereas the delPELPK CEA does not.