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1 esters as determined in incubations with rat hepatic microsomes.
2 d 6-hydroxy-2-nitropyrene from 2-NP in human hepatic microsomes.
3 mers were more metabolically stable in human hepatic microsomes.
4               Incubation of 17AAG with human hepatic microsomes and cyclohexene oxide, a known inhibi
5 nal properties observed previously in intact hepatic microsomes and displayed the highest specific ac
6 ssentially similar to those found with human hepatic microsomes and in human cells.
7  and vandetanib) were characterized by using hepatic microsomes and recombinant proteins.
8 y the same metabolites as produced by murine hepatic microsomes, but the 2-min metabolite was the maj
9  L1 preadipocyte cells, TBMEHP inhibited rat hepatic microsome deiodinase activity and was an agonist
10 effect on CCl4-induced lipid peroxidation of hepatic microsomes, exceeds that produced by alpha-tocop
11 asured by LC-MS/MS methods.CYP3A activity in hepatic microsomes exhibited a significant decrease in M
12      The recovery of sEH inhibitors from rat hepatic microsomes, fortified at levels of 50, 100, and
13  reaction for CYP3A4) was reduced by >50% in hepatic microsomes from CYP3A4-HBN mice compared with co
14 tro glucuronidation of SN-38 was screened in hepatic microsomes from normal rats (n = 4), normal huma
15 le epoxide hydrolase (sEH) inhibitors in rat hepatic microsomes is described.
16 e formation of the HMM bands was observed in hepatic microsomes isolated from rats treated 1 week or
17                                     With rat hepatic microsomes or purified CPR, the presence of NADP
18                     Metabolism studies using hepatic microsomes suggest that this procedure would be
19 ected in rat, hamster, dog, monkey, or human hepatic microsomes, suggesting the lack of oral toxicity
20 of retinol metabolism have been described in hepatic microsomes that involve, in part, cytochrome P45
21 shown to be specific in human plasma and rat hepatic microsomes, was further combined with the SRM tr
22                                        Human hepatic microsomes were competent in metabolizing all th
23 on and butanol extraction, we found that all hepatic microsomes were competent to activate 3-NBA.
24                                              Hepatic microsomes were loaded with 45Ca2+, and superfus
25                                              Hepatic microsomes were prepared by standard sucrose den
26 reduced accumulation in small intestinal and hepatic microsomes, which influenced the rate of ezetimi
27                12 was rapidly metabolized by hepatic microsomes, which supports its topical use.
28 sized sEH inhibitors were extracted from rat hepatic microsomes with ethyl acetate and were determine
29 tification of residual sEH inhibitors in rat hepatic microsomes without interference.