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1 rom the biliary tree and develops within the hepatic parenchyma.
2 tructure, which shows similarities to normal hepatic parenchyma.
3 hal insult to tumors while preserving normal hepatic parenchyma.
4 th in the portal triad that effaced adjacent hepatic parenchyma.
5 of amastigotes from foci of infection in the hepatic parenchyma.
6 ns, angiogenesis, and regeneration of normal hepatic parenchyma.
7  was no collateral damage to the neighboring hepatic parenchyma.
8 oth in portal regions and extending into the hepatic parenchyma.
9 ccompanying enhancement abnormalities of the hepatic parenchyma.
10 er the risk of toxic effects of drugs on the hepatic parenchyma.
11 e requirement for the formation of an active hepatic parenchyma.
12 foci of increased vascularization within the hepatic parenchyma.
13 neum (11 sites), lymph nodes (10 sites), and hepatic parenchyma.
14  negligible inflammation/necrosis within the hepatic parenchyma.
15 us activated Kupffer cells were noted in the hepatic parenchyma.
16 enous inflow block with hypoperfusion of the hepatic parenchyma.
17 the number of neutrophils sequestered in the hepatic parenchyma.
18 te out of the vessels and integrate into the hepatic parenchyma.
19 ounding the bile ductules and throughout the hepatic parenchyma.
20 whereas the portal vein supplies most normal hepatic parenchyma.
21 n response to direct injury or injury to the hepatic parenchyma.
22 r cells, which are rarely observed in normal hepatic parenchyma.
23 inflammation and the pathogenic processes in hepatic parenchyma.
24  suggested the infiltration of HSCs into the hepatic parenchyma after stem cell mobilization.
25 der of the tumor nodule from the surrounding hepatic parenchyma) also was measured.
26 ultifocal accumulations of leukocytes in the hepatic parenchyma and around portal tracts and central
27 inflammation leads to fibrosis involving the hepatic parenchyma and biliary tree, which can lead to c
28 lation, with less neutrophils present in the hepatic parenchyma and far more neutrophils adherent to
29 enges in establishing long-term viability of hepatic parenchyma and incorporating structurally and fu
30 xpressing monocytes and Kupffer cells in the hepatic parenchyma and perivascular sites and absence of
31 ductular reactive (DR) cells extend into the hepatic parenchyma and promote inflammation and fibrosis
32 teract with HSCs on their migration into the hepatic parenchyma, and a depletion or deactivation of H
33 ivo recruitment of NK cells to pulmonary and hepatic parenchyma, and s.c. tumor after treatment of mi
34       Volumes of total liver, hepatic cysts, hepatic parenchyma, and total kidney were measured by a
35 se the numbers of ECFC persisting within the hepatic parenchyma are needed in order to enhance ECFC t
36  profiles are associated with changes in the hepatic parenchyma as reflected in increased scores for
37  chemokine induction was infiltration of the hepatic parenchyma by neutrophils.
38 atic segments, limiting the volume of normal hepatic parenchyma exposed to radiation.
39 c +/- 3 (P < .0001 and P < .01) and those in hepatic parenchyma in healthy volunteers (745 msec +/- 6
40 y liver functions and can integrate into the hepatic parenchyma in vivo.
41 ed hepatocytes is essential for a functional hepatic parenchyma, including genes encoding several apo
42 ed the extent of the biliary tree within the hepatic parenchyma, including identification of the cana
43 auses of oval cell proliferation in that the hepatic parenchyma is relatively unaffected.
44 ere clinically equivocal (myocardium, lungs, hepatic parenchyma, jejunum, and renal cortex/medulla) a
45 s characterized by cystic replacement of the hepatic parenchyma, leading to hepatic dysfunction, port
46  hepatocellular tumors was identified in the hepatic parenchyma of A/JCr mice.
47 carrier family members present in the entire hepatic parenchyma of the DEN-induced rats.
48 zing lesion was progressive and involved the hepatic parenchyma, portal triads, and intralobular venu
49 ously shown that the microenvironment of the hepatic parenchyma regulates hepatic tumor formation fro
50 ematoma was punctured to relieve pressure on hepatic parenchyma, retrieving 300 mL of blood.
51     Autoimmune hepatitis is a disease of the hepatic parenchyma that can present in acute or chronic
52  pancreatic islet autotransplantation in the hepatic parenchyma (the intrahepatic [IH] group, n = 9)
53 bacterial invasion or translocation into the hepatic parenchyma through portal system.
54                     Recovery/regeneration of hepatic parenchyma was assessed by histologic assessment
55                                              Hepatic parenchyma was normal, whereas apoptosis and cel
56  stage 2 metastases mimicked the surrounding hepatic parenchyma, whereas stage 3 metastases exhibited
57 uminous subcapsular hematoma compressing the hepatic parenchyma, which appeared ischemic with a flatt
58                             The thickness of hepatic parenchyma with intense perilesional enhancement
59                             The thickness of hepatic parenchyma with intense perilesional enhancement
60                       The mean percentage of hepatic parenchyma with nontumorous perfusion defects wa
61                          Uniform necrosis of hepatic parenchyma within the cryolesion was confirmed.
62       FT NPs were found to accumulate in the hepatic parenchyma without tumor nodules of Wistar rats