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1 tional status to heme biosynthesis and acute hepatic porphyria.
2 ice, hepatomegaly, acute hepatic failure and hepatic porphyria.
3 ntermittent porphyria, the most common acute hepatic porphyria.
4  treatment of the acute attacks of the acute hepatic porphyrias.
5 ute heme-deficient states, such as the acute hepatic porphyrias.
6 d often fatal clinical symptoms of the acute hepatic porphyrias.
7                                        Acute hepatic porphyrias (acute intermittent porphyria, varieg
8                                        Acute hepatic porphyria (AHP), a rare genetic disorder, causes
9 llular carcinoma (HCC) is increased in acute hepatic porphyrias (AHP).
10                                    The acute hepatic porphyrias (AHPs) are a group of rare, inherited
11                                    The acute hepatic porphyrias (AHPs) are inherited disorders of hem
12   We conclude that iron potentiates both the hepatic porphyria and toxicity of TCDD in susceptible mi
13 -nonresponsive) mice, iron overcame the weak hepatic porphyria and toxicity responses in BALB/c and S
14 etic testing to determine the specific acute hepatic porphyria, and a complete medical history and ph
15                                    The acute hepatic porphyrias are a group of four inherited disorde
16                                    The acute hepatic porphyrias are inherited disorders of heme biosy
17                                    Inducible hepatic porphyrias are inherited genetic disorders of en
18 ograms/kg) markedly potentiated not only the hepatic porphyria but also general hepatocellular damage
19                                        Acute hepatic porphyria comprises a group of rare genetic dise
20      Eligible patients had a confirmed acute hepatic porphyria diagnosis and had experienced >=3 atta
21 derstanding of the pathogenesis of the acute hepatic porphyrias, identification of the iron overload-
22  iron, previously implicated in TCDD-induced hepatic porphyria, in mice with alleles of Ahr that enco
23 ttent porphyria (AIP), the most common acute hepatic porphyria, is an autosomal dominant inborn error
24 the acute attacks in patients with the acute hepatic porphyrias, perform the chronic phlebotomies to
25 framework for monitoring patients with acute hepatic porphyrias to ensure optimal outcomes.
26 own to induce porphyrin accumulation causing hepatic porphyria, up-regulate ABCB6 expression in both
27 d clinical management in patients with acute hepatic porphyria who experience recurrent attacks.