ライフサイエンスコーパス: hepatic vein

1 not adequately drained by a remaining major hepatic vein.

2 ory hepatic vein, and one had a double right hepatic vein.

3 an intrahepatic portal vein and an adjacent hepatic vein.

4 percutaneous access route through the fetal hepatic vein.

5 ) via a quadripolar catheter positioned in a hepatic vein.

6 tions of ammonia, glutamine, and urea in the hepatic vein.

7 des but were not detected egressing into the hepatic vein.

8 ar catheter positioned in a subdiaphragmatic hepatic vein.

9 ol subjects by direct catheterization of the hepatic vein.

10 and sampled from femoral artery and vein and hepatic vein.

11 %) patients had involvement of the portal or hepatic veins.

12 he short, nongrafted portions of the outflow hepatic veins.

13 treatment for BCS with diffuse occlusion of hepatic veins.

14 inserted in the superior mesenteric and left hepatic veins.

15 6 nmol/min), and cortisol appearance in the hepatic vein after oral cortisone was unchanged.

16 ostprocessing depicted arterial, portal, and hepatic vein anatomy traversing the anticipated surgical

17 Left hepatic vein anatomy was classified into 3 types: type 1

18 min with blood sampling from catheters in a hepatic vein and a radial artery (concentrations of (18)

19 reased the plasma LEAP2 concentration in the hepatic vein and abdominal aorta but not in the portal v

20 Samples were obtained from the hepatic vein and an arterialized hand vein at steady sta

21 egmentectomy with reconstruction of the left hepatic vein and one patient died at 3 months after rese

22 effective for controlling bleeding from the hepatic vein and safer than increasing pneumoperitoneum

23 Plasma was collected from the hepatic vein and superior vena cava and underwent protei

24 hepatectomies extended to include the middle hepatic vein and the caudate lobe but preserving the maj

25 SHAPE data were collected from a portal and hepatic vein and were compared with invasive measurement

26 Conclusion During microwave ablation of HCC, hepatic veins and arteries were resistant to vessel occl

27 e visualized from the inferior vena cava and hepatic veins and corroborated.

28 ns in three, portal tracts in five, and both hepatic veins and portal tracts in two patients.

29 asurements were performed after clamping the hepatic veins and recipient hepatectomy.

30 the lungs, caused by altered diameter of the hepatic veins and sinusoids in Bmpr2(+/-) mice.

31 8 years) with fatty infiltration surrounding hepatic veins and/or portal tracts were retrospectively

32 ar predisposition to fat accumulation around hepatic veins and/or portal tracts.

33 Dogs had sampling (artery, portal vein, and hepatic vein) and infusion (vena cava and portal vein) c

34 simultaneous sampling from the portal vein, hepatic vein, and an arterialized peripheral vein.

35 ed (ie., gastroduodenal artery, portal vein, hepatic vein, and femoral artery), perfusion rates in he

36 The portal vein (PV), TIPS, right hepatic vein, and IVC were segmented on MRI scans to cre

37 xteen had at least one significant accessory hepatic vein, and one had a double right hepatic vein.

38 obtained (i.e., portal vein, carotid artery, hepatic vein, and pulmonary artery), perfusion rates wer

39 data were collected from a portal vein and a hepatic vein, and the difference was compared with HVPG

40 Complete occlusion of the portal veins, hepatic veins, and hepatic arteries within and directly

41 ein, superior mesenteric vein, splenic vein, hepatic veins, and inferior vena cava (IVC) were evaluat

42 myography-guided PN monitoring using a novel hepatic vein approach for prevention of PNP.

43 Anatomical variations of left hepatic vein are observed in nearly a third of left late

44 na cava (SVC), inferior vena cava (IVC), and hepatic veins are connected to the pulmonary arteries in

45 uinaud's segment, in which the corresponding hepatic veins are exposed on the raw surface.

46 The pattern of amino acids appearing in hepatic vein blood was affected by changes in amino acid

47 he first 10 did not include the right middle hepatic vein branches in the graft.

48 ine medially to incorporate the right middle hepatic vein branches into the donor graft.

49 were scored for visualization of portal and hepatic vein branches, liver edge sharpness, cardiac pul

50 monitoring using a catheter positioned in a hepatic vein can aid in preventing phrenic nerve palsy (

51 ature of the hepatic artery, portal vein and hepatic vein can be predicted, together with their geome

52 Liver resection with reconstruction of the hepatic veins can be performed in selected cases.

53 pneumoperitoneum pressure, bleeding from the hepatic vein cannot be controlled under high airway pres

54 ght-matched control subjects by means of the hepatic vein catheterization (HVC) technique.

55 e obtained in 12 control subjects undergoing hepatic vein catheterization and infusion of identical t

56 We used isotope dilution/hepatic vein catheterization techniques to examine wheth

57 on, directly measured using the arterial and hepatic vein catheters, did not differ (67 +/- 3 vs. 71

58 Hepatic vein-cava stenoses occurred after a mean of 37.2

59 s occurred in 32 LRD, 3 RSS, and 3 FS, while hepatic vein-cava stenoses occurred in 2 LRD, 8 RSS, and

60 tified in 49 patients (38 portal vein and 12 hepatic vein-cava stenoses).

61 contrast to noise in the azygos vein, right hepatic vein, common bile duct, and superior mesenteric

62 significant reduction in the number of large hepatic veins compared to embryonic wild-type (WT) liver

63 n inflammatory cells surrounding the central hepatic vein, compared with WT.

64 even consecutive patients with tumors at the hepatic vein confluence were prospectively evaluated wit

65 pects of the procedure, including the middle hepatic vein controversy and the "small for size syndrom

66 other hand, hepatic artery, portal vein, and hepatic vein cortisol concentrations did not differ (0.3

67 quires no retrocaval, hepatic vein, or short hepatic vein dissection, and the inferior vena cava can

68 we present a liver graft with abnormal left hepatic vein draining directly to the right atrium of th

69 mental system used for this study mimics the hepatic vein draining into the inferior vena cava and al

70 often employed to control bleeding from the hepatic vein during pure laparoscopic hepatectomy; howev

71 d metabolite concentrations over time in the hepatic vein during toxin-induced liver damage and regen

72 re collected from the inferior vena cava and hepatic veins during right heart catheterization from 3

73 ation, portal vein endothelial inflammation, hepatic vein endothelial inflammation, and centrilobular

74 bnormalities, including aberrant subcapsular hepatic veins, enlarged glomeruli, intestinal polyps con

75 al mitral annular e' velocity, and prominent hepatic vein expiratory diastolic flow reversals are ind

76 ombination with either medial e'>/=9 cm/s or hepatic vein expiratory diastolic reversal ratio >/=0.79

77 ial mitral annular e' to lateral e', and (5) hepatic vein expiratory diastolic reversal ratio.

78 septal shift, (2) medial mitral e', and (3) hepatic vein expiratory diastolic reversal ratio.

79 Portal vein flow was greater than hepatic vein flow in 25% of patients.

80 stal trocars to expose the root of the right hepatic vein for segmentectomy VII and VIII.

81 inuous spectral doppler ultrasonography of a hepatic vein from 20 s before to 3 min after a periphera

82 P2 concentration was in the following order: hepatic vein > abdominal aorta > portal vein.

83 ari syndrome (BCS) with diffuse occlusion of hepatic veins has a high mortality rate and remains chal

84 artery (RA) catheter, portal vein (PV), and hepatic vein (HV) during the dissection phase and was re

85 The donor's left hepatic vein (HV) was anastomosed to the confluence of t

86 s from 4 different vascular sites, including hepatic vein (HV), peripheral artery (PA), peripheral ve

87 nt via percutaneous access through the fetal hepatic vein in a sheep model.

88 ary artery, carotid artery, portal vein, and hepatic vein in swine infused with PGE1 (range, 0.67-4.9

89 sed to the confluence of the left and middle hepatic veins in all cases.

90 ) in level 1-2 buds and becomes CD34(-) near hepatic veins in level 3-4 buds.

91 t hepatic vein (RHV) confluence of accessory hepatic veins in the surgical plane.

92 Fatty infiltration surrounded hepatic veins in three, portal tracts in five, and both

93 ying the galactose infusion point (simulated hepatic vein) in a 15-cm conduit was 1.7 to 2.8 mm, or 1

94 1) but did not differ in the portal vein and hepatic vein, indicating net uptake across the viscera b

95 a common trunk which drains into the middle hepatic vein/inferior vena cava (IVC), subtype 1a length

96 Hepatic vein involvement by hepatic malignancy does not

97 provide comparable results for assessment of hepatic vein involvement by tumor.

98 ameters were measured 15-20 mm caudal to the hepatic vein junction and recorded by bidimensional imag

99 terial blood supply, leucine export into the hepatic vein, leucine oxidation and transamination, and

100 Anatomy of the left hepatic vein (LHV) was studied in a series of 53 consecu

101 del that PH in combination with right median hepatic vein ligation (RMHV-L) caused confluent parenchy

102 hepatectomy plane to the right of the middle hepatic vein (MHV) and separating the right and left lob

103 located liver tumor, with or without middle hepatic vein (MHV) invasion.

104 fts (AVGs) used for reconstruction of middle hepatic vein (MHV) tributaries in living donor liver tra

105 the individualized management of the middle hepatic vein (MHV).

106 ws adequate venous outflow through the right hepatic vein more than 1 cm, which is demonstrated by th

107 oportal vein (n = 24), vena cava (n = 3), or hepatic vein (n = 3).

108 Sixteen hepatic veins (nine right, four middle, three left) were

109 rly and transient increase in posttransplant hepatic vein nitrate levels (pretransplant, 90 microM; 2

110 Monocrotaline caused hepatic vein NO to decrease by 30% at 24 hours and by 70

111 e nodularity (SN), lobar redistribution, and hepatic vein nodularity.

112 Hepatic vein NOx was significantly higher than circulati

113 f interventional treatments in patients with hepatic vein obstruction (Budd-Chiari Syndrome) and in t

114 Cellular overgrowth from the hepatic vein occluded the end of one graft at 3 months,

115 SSPCS in BCS with hepatic vein occlusion alone results in reversal of live

116 In the 32 patients with BCS resulting from hepatic vein occlusion alone, SSPCS had a surgical death

117 Hepatic vein occlusion was significantly correlated with

118 The right hepatic vein of the donor graft was anastomosed to the c

119 and have a patent main portal vein and major hepatic veins on duplex ultrasonography.

120 vein drainage to the inferior vena cava and hepatic vein or of the inferior phrenic vein (n = 7).

121 two veins closely merging toward the median hepatic vein, or (C) a double outflow.

122 al hemorrhage, or suffered from portal vein, hepatic vein, or hepatic artery thrombosis

123 cavaplasty technique requires no retrocaval, hepatic vein, or short hepatic vein dissection, and the

124 een proximal FHVP (obtained at 2 cm from the hepatic vein outlet) and IVC (measured at the level of t

125 Levels were also determined from hepatic vein, portal vein, and systemic arterial blood i

126 s were located immediately adjacent to major hepatic veins, portal veins, or both; thus, they were no

127 he difference between wedged (WHVP) and free hepatic vein pressure (FHVP), predicts survival in patie

128 reening inflammatory serum biomarkers of the hepatic vein pressure gradient (HVPG) is based on the fa

129 The hepatic vein pressure gradient (HVPG) is the standard us

130 subjects with compensated cirrhosis and PH (hepatic vein pressure gradient [HVPG] >5 mm Hg).

131 underwent a left hepatectomy without middle hepatic vein procurement.

132 donor liver transplantation that necessitate hepatic vein reconstruction can be applied to hepatic re

133 ts who underwent hepatic resection requiring hepatic vein reconstruction from 1996-2001 were reviewed

134 ous or cadaveric graft for hepatic artery or hepatic vein repair were the most recommended (89%).

135 Involvement of the hepatic veins requiring reconstruction has traditionally

136 While middle hepatic vein resection caused higher postresection PVP a

137 , 6.8%): V2 and V3 drain into IVC and middle hepatic vein respectively.

138 ANOVA) from hepatic artery to portal vein to hepatic vein, respectively, indicating 8 +/- 3 and 28 +/

139 d by prominent sprouting angiogenesis of the hepatic vein, restricted to the periphery of the organ.

140 presence, size, and distance from the right hepatic vein (RHV) confluence of accessory hepatic veins

141 nferior vena cava, through drainage into the hepatic vein, right atrium or left atrium.

142 d laceration or contusion extending into the hepatic vein(s), inferior vena cava, porta hepatis, or g

143 he present study, arterial, portal vein, and hepatic vein sampling catheters were surgically placed i

144 monitoring using a catheter positioned in a hepatic vein seems feasible and effective to prevent PNP

145 a catheter positioned in a subdiaphragmatic hepatic vein seems feasible during cryoballoon ablation.

146 aped (H-type portal-caval)], 2 had portal-to-hepatic vein shunts (portohepatic), and 2 had a persiste

147 nd US yielded identical findings at 26 of 27 hepatic vein sites.

148 n 39.7% of portal veins (29 of 73), 15.0% of hepatic veins (six of 40), and 14.2% of hepatic arteries

149 rrent variceal bleeding or ascites than were hepatic vein stenoses (odds ratio, 3.6; P = .02).

150 ted after 30 days; and in 19 patients (16%), hepatic vein stenoses were detected after 30 days.

151 Although common, hepatic vein stenoses were rarely associated with recurr

152 d including all interventional treatments of hepatic vein stenosis post major liver resection since 2

153 erved that during the first 48 h, most major hepatic veins, such as the portal and umbilical veins, n

154 (1.33 +/- 0.11; P < 0.001) were lower in the hepatic vein than in the portal vein, indicating product

155 ficantly higher leptin concentrations in the hepatic vein than lean sham-operated, fa/fa BDL, or fa/f

156 ly the splitting of the vena cava and middle hepatic vein, the parenchymal transection, and the venou

157 PAI-1 play pivotal and antagonistic roles in hepatic vein thrombosis and that PAI-1 is a potential ta

158 ncidence of hepatic artery, portal vein, and hepatic vein thrombosis is 0%, 0%, and 0%, respectively.

159 or vascular disorders such as portal vein or hepatic vein thrombosis.

160 ccessful deceased donor liver transplant for hepatic vein thrombosis.

161 re largely protected from the development of hepatic vein thrombosis.

162 ws showed no difference in the occurrence of hepatic vein thrombosis/stenosis, major morbidity (P = .

163 ribution was similar between portal vein and hepatic vein to cava, as was the mean patient age.

164 shunts were created from the middle or left hepatic vein to the left portal vein, and none of the su

165 rease of 10% above baseline levels indicated hepatic vein transit time (HVTT).

166 ups: the first had occlusion confined to the hepatic veins treated by direct side-to-side portacaval

167 nt graft (segments 2, 3, and 4 of the middle hepatic vein trunk) and left portal vein graft to the re

168 isease (M), macrovascular involvement of all hepatic veins (V) or portal bifurcation (P), contiguous

169 negative gradient of M30 from the portal to hepatic vein was demonstrated in patients with acetamino

170 Injury involving at least one major hepatic vein was found in 15 (88%) of 17 patients who re

171 ays of gestation (term, 147 days), the fetal hepatic vein was punctured percutaneously under ultrasou

172 x patients with HCC and cirrhosis, the right hepatic vein was reconstructed to provide venous outflow

173 tumors, hepatic artery, portal vein, and the hepatic veins was developed.

174 The SHAPE gradient between the portal and hepatic veins was in good overall agreement with the hep

175 The SHAPE gradient between the portal and hepatic veins was in good overall agreement with the HVP

176 truction of either the inferior vena cava or hepatic veins was performed in five patients.

177 of the inferior vena cava, portal vein, and hepatic veins, was successfully managed by aggressive ch

178 spectral Doppler signals of right and middle hepatic veins were analyzed.

179 Forty-four accessory hepatic veins were identified in 40 patients; seven drai

180 Significant accessory hepatic veins were preserved, brought together in a comm

181 (PV) injection of plasmid DNA in mice whose hepatic veins were transiently occluded.

182 ch in treating BCS with diffuse occlusion of hepatic veins, which should be performed in time.

183 confluence of a venous tributary (i.e., the hepatic vein) with a major vascular channel (i.e., the v

184 d reconstruction of the vena cava and middle hepatic vein, with dual arterial and portal hypothermic