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1 urotropic coronavirus (rJ2.2 strain of mouse hepatitis virus).
2 ere highly susceptible to a murine CoV-mouse hepatitis virus.
3 ol acute infection with the cytopathic mouse hepatitis virus.
4 y presents compared to ALF from conventional hepatitis virus.
5 BV and of woodchucks infected with woodchuck hepatitis virus.
6 that was chronically infected with woodchuck hepatitis virus.
7 A synthesis in a related coronavirus, murine hepatitis virus.
8 fection of p85beta-deficient mice with mouse hepatitis virus.
9 ction by the neurotropic JHM strain of mouse hepatitis virus.
10 irus chikungunya virus and coronavirus mouse hepatitis virus.
11 capsid protein of a model coronavirus, mouse hepatitis virus.
12 gh-risk etiological challenges, most notably hepatitis virus.
13 ion units of blood are not tested for HIV or hepatitis viruses.
14 nodeficiency virus (HIV) and four species of hepatitis viruses.
15 CV vaccine and future research needs for the hepatitis viruses.
16 They are avian encephalomyelitis virus, duck hepatitis virus 1, duck picornavirus, porcine teschoviru
18 sses the recent advances in our knowledge of hepatitis viruses A through G, focusing on the literatur
21 Intracerebral infection of mice with mouse hepatitis virus, a member of the Coronaviridae family, r
22 the role of MDA5 during infection with mouse hepatitis virus, a murine coronavirus used to model vira
23 ity was examined in mice infected with mouse hepatitis virus, a well-described model of virus-induced
24 ly pathogenic neurotropic coronavirus (mouse hepatitis virus A59 strain [MHV-A59]) developed severe e
25 Lpro inhibitors in mice infected with murine hepatitis virus A59, a hepatotropic coronavirus, resulte
26 as helminths, mycobacteria, Plasmodium, and hepatitis viruses affect more than a third of the human
27 urprisingly, two beta-CoVs in group A, mouse hepatitis virus and HKU1, have evolved to use different
28 odchucks chronically infected with woodchuck hepatitis virus and vaccinated with woodchuck hepatitis
29 e estimated risk for transfusion transmitted hepatitis viruses and retroviruses is now vanishingly sm
30 adnaviruses (hepatitis B virus and woodchuck hepatitis virus), and an intron-retaining transcript enc
31 ic enzyme from two CoVs, SARS-CoV and murine hepatitis virus, and its monomeric homologue, XendoU fro
32 n the characterization of several fastidious hepatitis viruses, and we investigated the feasibility o
33 tis aplastic anemia, antibodies to the known hepatitis viruses are absent; the unknown infectious age
35 's murine encephalomyelitis virus and murine hepatitis virus, are used to induce infectious models of
36 splant recipients commonly are infected with hepatitis viruses, are immunosuppressed, and have other
37 sed from the replicase polyprotein of murine hepatitis virus as fusions with nonstructural protein 2
38 s within the replicase polyprotein of murine hepatitis virus as fusions with nonstructural proteins c
39 d HIV1 and HIV2, as well as of two different hepatitis viruses, attaining results of approximately 87
40 ucks chronically infected with the woodchuck hepatitis virus before and during 30 weeks of therapy wi
41 temperature-sensitive (ts) mutant of Murine hepatitis virus, Bristol ts31 (MHV-Brts31), that defines
42 al nervous system (CNS) by neurotropic mouse hepatitis virus but do not suffice to achieve sterile im
43 ntiviral protein IFN-alpha2 is used to treat hepatitis viruses but has proven rather ineffective agai
50 a monax) chronically infected with woodchuck hepatitis virus contained at least 100,000 clones of >1,
51 n development of HCV sequence databases, the Hepatitis Virus Database (Japan), euHCVdb (France), and
55 r results support the hypothesis that murine hepatitis virus ExoN activity is required for resistance
56 he 3' untranslated region (UTR) of the mouse hepatitis virus genome contains two essential and overla
57 (-/-) mice infected with a neurotropic mouse hepatitis virus had an impaired ability to control viral
58 end-stage liver disease (ESLD) secondary to hepatitis viruses has evolved rapidly during the last tw
59 of the SARS-associated coronavirus and mouse hepatitis virus have evolved to promote optimal frameshi
60 e livers chronically infected with woodchuck hepatitis virus, (i) hepadnavirus superinfection and cel
61 g RNA element (WPRE), derived from woodchuck hepatitis virus in combination with an antibody-cytokine
62 ncoded by ORF6, enhanced the growth of mouse hepatitis virus in tissue culture cells and in mice.
63 against CCL5 to mice with established mouse hepatitis virus-induced demyelination and impaired motor
64 opathogenesis, is essential for the study of hepatitis virus-induced liver disease and for therapeuti
65 ined with significant receptor expression in hepatitis virus-infected livers, suggests that IL-29 may
66 us (OR = 9.4; 95% CI = 2.7-32.7) and chronic hepatitis virus infection (OR = 31.2; 95% CI = 6.3-153.2
67 etween heavy alcohol consumption and chronic hepatitis virus infection (OR, 53.9; 95% CI, 7.0-415.7)
73 d models were used to estimate the effect of hepatitis virus infection, and adjusted for potential co
78 roduction in all viral hepatitis infections: Hepatitis virus infections did not alter NK cell differe
81 V-JHM strain of the murine coronavirus mouse hepatitis virus is much more neurovirulent than the MHV-
84 ase, but the intrahepatic immune response to hepatitis viruses is poorly understood because of a lack
85 ample, C57BL/6 (B6) mice infected with mouse hepatitis virus (JHM strain, JHMV) develop severe enceph
86 ontaining CNS cells were infected with mouse hepatitis virus-JHM, which causes fatal encephalitis in
87 ice with the neurotropic JHM strain of mouse hepatitis virus (JHMV) (a member of the Coronaviridae fa
88 omyelitis induced by the JHM strain of mouse hepatitis virus (JHMV) and sustained during viral persis
89 ted with the neurotropic JHM strain of mouse hepatitis virus (JHMV) develop acute and chronic demyeli
90 (CNS) by the neurotropic JHM strain of mouse hepatitis virus (JHMV) induces an acute encephalomyeliti
91 ion with the neurotropic JHM strain of mouse hepatitis virus (JHMV) into the central nervous system (
93 ion with the neurotropic JHM strain of mouse hepatitis virus (JHMV) resulted in an acute encephalomye
94 CNS with the neurotropic JHM strain of mouse hepatitis virus (JHMV) results in an immune-mediated dem
99 we demonstrate that NHC inhibits both murine hepatitis virus (MHV) (50% effective concentration [EC(5
102 The endoribonuclease activity of the mouse hepatitis virus (MHV) A59 Nsp15 was also increased by pR
103 he outcome of viral encephalomyelitis [mouse hepatitis virus (MHV) A59, Theiler's encephalomyelitis v
104 infection with the murine coronavirus mouse hepatitis virus (MHV) activated the NLRP3 inflammasome a
105 Infection with the murine coronavirus mouse hepatitis virus (MHV) activates the pattern recognition
106 uses within the genus Betacoronavirus, mouse hepatitis virus (MHV) and MERS-CoV, encode 2',5'-phospho
107 -D; four nucleotide substitutions) in murine hepatitis virus (MHV) and severe acute respiratory syndr
108 5' untranslated regions (5'UTRs) from mouse hepatitis virus (MHV) and severe acute respiratory syndr
110 acute respiratory syndrome (SARS)-CoV, mouse hepatitis virus (MHV) and the human CoV OC43 S2 subunits
115 ) of the 5'-untranslated region of the mouse hepatitis virus (MHV) contains a highly conserved pental
116 glycoprotein from strain RSA59 (PP) of mouse hepatitis virus (MHV) contains two central, consecutive
119 h the recombinant JHM (RJHM) strain of mouse hepatitis virus (MHV) elicits a weak CD8(+) T-cell respo
122 Most strains of murine coronavirus mouse hepatitis virus (MHV) express a cleavable spike glycopro
123 ing the recombinant murine coronavirus mouse hepatitis virus (MHV) expressing the T cell-chemoattract
127 us work with the prototype coronavirus mouse hepatitis virus (MHV) has shown that a major determinant
128 Many strains of the murine coronavirus mouse hepatitis virus (MHV) have distinct, S-dependent organ a
129 revious studies have demonstrated that mouse hepatitis virus (MHV) hepatotropism is determined largel
131 ucted mutants of the model coronavirus mouse hepatitis virus (MHV) in which all or part of the M prot
132 nts of a severely defective mutant of murine hepatitis virus (MHV) in which the N gene was replaced w
135 determine the requirement of nsp4 for murine hepatitis virus (MHV) infection in culture, engineered d
139 e replacements were engineered into a murine hepatitis virus (MHV) infectious clone in place of conse
141 synthesis by the prototype coronavirus mouse hepatitis virus (MHV) is carried out by a replicase-tran
143 ive roles of specific ISGs against the mouse hepatitis virus (MHV) members of the coronaviruses are l
145 monstrated that the murine coronavirus mouse hepatitis virus (MHV) nonstructural protein 2 (ns2) is a
147 Here, we engineered mutations in murine hepatitis virus (MHV) nsp14 N7-MTase at residues D330 an
148 ween the endoribonuclease activity of murine hepatitis virus (MHV) Nsp15 (mNsp15) and its role in vir
149 magnetic resonance (NMR) structure of mouse hepatitis virus (MHV) nsp3a and show, using isothermal t
150 en shown previously that mutations in murine hepatitis virus (MHV) nsp4 loop 1 that alter nsp4 glycos
151 everse genetic mutagenesis of the CoV murine hepatitis virus (MHV) nsp5, we identified a new temperat
158 cultured cells with murine coronavirus mouse hepatitis virus (MHV) resulted in activation of the mito
159 t infection of rat oligodendrocytes by mouse hepatitis virus (MHV) resulted in apoptosis, which is ca
160 ) A1 has previously been shown to bind mouse hepatitis virus (MHV) RNA at the 3' end of both plus and
161 to function as regulatory factors for mouse hepatitis virus (MHV) RNA synthesis as a result of their
165 important for clearance of neurotropic mouse hepatitis virus (MHV) strain A59, although their possibl
167 temperature-sensitive (TS) mutants of murine hepatitis virus (MHV) strain A59, proposes that an inter
169 We observed that the nonfusogenic mouse hepatitis virus (MHV) strain MHV-2 reached a titer of ap
171 Sequencing and reversion analysis of murine hepatitis virus (MHV) temperature-sensitive (ts) viruses
172 previously generated E gene mutants of mouse hepatitis virus (MHV) that had marked defects in viral g
173 urrent studies, neurotropic strains of mouse hepatitis virus (MHV) that induce meningitis, encephalit
174 usly generated E gene point mutants of mouse hepatitis virus (MHV) that were defective in growth and
175 previously generated E gene mutants of mouse hepatitis virus (MHV) that were defective in viral growt
176 at infection by the murine coronavirus mouse hepatitis virus (MHV) triggers the proximal UPR transduc
181 kaging signal (PS) for the coronavirus mouse hepatitis virus (MHV) was originally identified as an el
183 hR is activated in cells infected with mouse hepatitis virus (MHV), a coronavirus (CoV), and contribu
184 In previous work with the coronavirus mouse hepatitis virus (MHV), a highly defective M protein muta
188 stion, we used the rJHM strain (rJ) of mouse hepatitis virus (MHV), a neurotropic coronavirus that ca
189 erminal domain (NTD) of N protein from mouse hepatitis virus (MHV), a virus most closely related to S
190 d by the prototypical Betacoronavirus, mouse hepatitis virus (MHV), and by Middle East respiratory sy
192 vely, including the murine coronavirus mouse hepatitis virus (MHV), Haemophilus influenzae, Neisseria
194 pe of a highly neurovirulent strain of mouse hepatitis virus (MHV), JHM, is thought to be essential f
196 nfected with a prototypic coronavirus, mouse hepatitis virus (MHV), resulting in the expression of se
198 ed virus entry and cell-cell fusion of mouse hepatitis virus (MHV), suggesting the importance of lipi
201 y, we exploited the model coronavirus, mouse hepatitis virus (MHV), to investigate the genotype and p
202 ort genetic analysis of a ts strain of mouse hepatitis virus (MHV), tsNC11, focusing on the role of m
203 the N protein of the model coronavirus mouse hepatitis virus (MHV), we constructed mutants in which e
204 f N protein domains in the coronavirus mouse hepatitis virus (MHV), we replaced the MHV N gene with i
205 ation and expression is altered due to mouse hepatitis virus (MHV)-A59 infection both in vivo and in
206 activated protein kinases (MAPKs) in a mouse hepatitis virus (MHV)-infected macrophage-derived J774.1
217 titution mutations into the genome of murine hepatitis virus (MHV-A59) containing ExoN activity [ExoN
219 nto a heterologous murine coronavirus (mouse hepatitis virus [MHV]) but is not essential for optimal
220 with a recombinant murine coronavirus (mouse hepatitis virus [MHV]) expressing the T-cell chemoattrac
221 of RNase L during murine coronavirus (mouse hepatitis virus [MHV]) infection of myeloid cells correl
224 In this report, we show that the murine hepatitis virus nsp2 sequence was tolerated in ORF1b wit
225 the interaction between coronaviruses (mouse hepatitis virus) of different neurovirulences with prima
227 during clearance of the JHM strain of mouse hepatitis virus, only few virus-specific Ab-secreting ce
228 zation is critical for maintaining JHM mouse hepatitis virus persistence within the central nervous s
229 neurotropic coronavirus JHM strain of mouse hepatitis virus persists in oligodendroglia despite the
230 us-mouse phenylalanine hydroxylase-woodchuck hepatitis virus post-transcriptional response element (r
232 s sc counterpart, which lacked the woodchuck hepatitis virus posttranscriptional regulatory element (
233 NS) with the neurotropic JHM strain of mouse hepatitis virus produces acute and chronic demyelination
235 duced high titer viremia and acute resolving hepatitis; viruses recovered from both animals lacked th
236 und that proteasome inhibitors blocked mouse hepatitis virus replication at an early step in the vira
238 CXCL10-expressing murine coronavirus (mouse hepatitis virus) resulted in protection from disease and
240 ligation of T cells in vitro with the murine hepatitis virus spike protein, a natural ligand for the
241 Combining risk-score tertile levels and hepatitis virus status to stratify participants was more
242 ped using accessible variables combined with hepatitis virus status, which allows selection of asympt
243 n.) infection of A/J mice with the CoV mouse hepatitis virus strain 1 (MHV-1) induces an acute respir
245 ne coronaviruses expressing wild-type murine hepatitis virus strain 4 (MHV-4) or MHV-A59 spike glycop
249 cently established for the coronavirus mouse hepatitis virus strain A59 (MHV-A59), in which cDNA frag
260 e to high-fat diet-caused obesity and murine hepatitis virus strain-3 (MHV-3)-induced fulminant hepat
261 Mice infected with the coronavirus mouse hepatitis virus, strain JHM (JHM) develop a disease that
262 infected with the murine coronavirus, mouse hepatitis virus, strain JHM (MHV) develop an immune-medi
263 ce infected with attenuated strains of mouse hepatitis virus, strain JHM, develop a chronic infection
264 cognized in C57BL/6 mice infected with mouse hepatitis virus, strain JHM, or lymphocytic choriomening
265 epatitis virus and vaccinated with woodchuck hepatitis virus surface antigen could be induced to reco
266 s are likely to be related to differences in hepatitis viruses that are most prevalent in a populatio
269 recently been shown that cell entry of mouse hepatitis virus type 2 (MHV-2) is mediated through endoc
271 y of PLPs from SARS-CoV, MERS-CoV, and mouse hepatitis virus was evaluated against seven ISG15s origi
272 importance of the CD (SWWSFNPETNNL) in mouse hepatitis virus was investigated with a panel of mutant
273 s mice infected with the JHM strain of mouse hepatitis virus, which exhibit CTL escape variants with
274 he cis-acting element found in the woodchuck hepatitis virus (WHV) (the WHV posttranscriptional regul
275 onically infected with HBV-related woodchuck hepatitis virus (WHV) and already developed HCCs were us
276 her animal hepadnaviruses, such as woodchuck hepatitis virus (WHV) and duck hepatitis B virus (DHBV).
277 ed that the X-deficient mutants of woodchuck hepatitis virus (WHV) are not completely replication def
278 odchucks chronically infected with woodchuck hepatitis virus (WHV) are superinfected with HDV, they p
279 Woodchucks infected at birth with woodchuck hepatitis virus (WHV) cleared viremia and developed anti
281 ucks chronically infected with the woodchuck hepatitis virus (WHV) elicited differential T-cell respo
282 odchucks chronically infected with woodchuck hepatitis virus (WHV) induces a transient decline in vir
283 atal woodchucks with self-limiting woodchuck hepatitis virus (WHV) infection to those woodchucks prog
284 issue from woodchucks with chronic woodchuck hepatitis virus (WHV) infection was assayed for randomly
288 (Marmota monax) infected with the woodchuck hepatitis virus (WHV) represent the best animal model fo
289 en the immunogenicity of an analog woodchuck hepatitis virus (WHV) surface antigen (WHsAg) pDNA vacci
290 ting the ability of the virions of woodchuck hepatitis virus (WHV) to induce productive acute infecti
292 monax) is naturally infected with woodchuck hepatitis virus (WHV), a hepadnavirus closely related to
293 odchucks chronically infected with woodchuck hepatitis virus (WHV), a hepadnavirus closely related to
294 n an estimated 240 million people; woodchuck hepatitis virus (WHV), an HBV homologue, has been an imp
297 rmota monax ) harbors a DNA virus (Woodchuck hepatitis virus [WHV]) that is similar in structure and
298 e (M) protein of the model coronavirus mouse hepatitis virus with its counterpart from a heterologous
299 ation of the neurotropic JHM strain of mouse hepatitis virus within the central nervous system is con