ライフサイエンスコーパス: herpes simplex

1 t human cells, DNA viruses such as vaccinia, herpes simplex, and adenovirus induced increased IFN pro

2 Fatal herpes simplex encephalitis (HSE) results from immune pa

3 Herpes simplex encephalitis (HSE), caused by HSV type 1

4 cent reviews; these include the relevance of herpes simplex encephalitis and of epilepsy to AD, the a

5 Tumours, usually ovarian teratoma, and herpes simplex encephalitis are known triggers of NMDAR

6 infection (including cytomegalovirus [CMV], herpes simplex I/II or varicella zoster virus [HSV/VZV],

7 ree patients with a unilateral and relapsing herpes simplex keratitis (HSK group) that was quiescent

8 Ocular herpes simplex keratitis (HSK) is a consequence of viral

9 Herpes simplex keratitis (HSK), caused by herpes simplex

10 ity of life (QoL) in patients with quiescent herpes simplex keratitis compared with control patients

11 r clinical trial of 106 patients with active herpes simplex stromal keratitis who had not received an

12 flammation and in shortening the duration of herpes simplex stromal keratitis.

13 be structural organization of the virions of Herpes Simplex Type 1 viruses and bacteriophage MS2.

14 om patients with bacterial and viral (due to herpes simplex, varicella zoster, and enteroviruses) men

15 umanized monoclonal IgG antibody against the herpes simplex viral protein glycoprotein D (gD) was rad

16 We developed a herpes simplex viral vector to rapidly yet transiently o

17 erpesviridae-positive, which included 9 with herpes simplex virus (8.8%), 5 with varicella-zoster vir

18 this assessment include adenovirus 8 and 19, herpes simplex virus (HSV) 1 and 2, human immunodeficien

19 dy of neurotropic viruses in vitroIMPORTANCE Herpes simplex virus (HSV) affects millions of people wo

20 tions that US9 is neuron specific.IMPORTANCE Herpes simplex virus (HSV) and other alphaherpesviruses,

21 al fluid polymerase chain reaction (PCR) for herpes simplex virus (HSV) and varicella zoster virus wa

22 Herpes simplex virus (HSV) can cause severe infection in

23 Neonatal herpes simplex virus (HSV) disease results in unacceptab

24 the TG into the brain stem.IMPORTANCE Latent herpes simplex virus (HSV) DNA has been detected in the

25 erpesvirus entry mediator (HVEM) facilitates herpes simplex virus (HSV) entry through interactions wi

26 Herpes simplex virus (HSV) establishes latency in neuron

27 Herpes simplex virus (HSV) establishes lifelong latent i

28 Herpes simplex virus (HSV) glycoprotein C (gC) functions

29 We previously reported that herpes simplex virus (HSV) glycoprotein K (gK) binds to

30 of protein-protein interactions between four herpes simplex virus (HSV) glycoproteins (gD, gH/gL, and

31 The herpes simplex virus (HSV) heterodimer gE/gI and another

32 umcision was associated with reduced odds of herpes simplex virus (HSV) infection among MSM overall (

33 Numerous cases point to the link between herpes simplex virus (HSV) infection and multifocal CNS

34 Herpes simplex virus (HSV) infection is restricted to ep

35 Herpes simplex virus (HSV) infection is widespread in th

36 ance frequently complicates the treatment of herpes simplex virus (HSV) infections in immunocompromis

37 Herpes simplex virus (HSV) is a common and often benign

38 Herpes simplex virus (HSV) is a neuroinvasive virus that

39 Herpes simplex virus (HSV) is among the most prevalent v

40 The terminase of herpes simplex virus (HSV) is composed of three subunits

41 eity of cells expressing the LATs.IMPORTANCE Herpes simplex virus (HSV) is responsible for significan

42 Herpes simplex virus (HSV) is the main cause of viral en

43 f aqueous or vitreous humor was positive for herpes simplex virus (HSV) or varicella zoster virus (VZ

44 od to visualize pseudorabies virus (PRV) and herpes simplex virus (HSV) particles in living cells.

45 Despite frequent herpes simplex virus (HSV) reactivation, peripheral nerv

46 d microparticles and a replication-defective herpes simplex virus (HSV) recombinant vector.

47 Herpes simplex virus (HSV) requires fusion between the v

48 The herpes simplex virus (HSV) single-strand DNA binding pro

49 s, systemic antimicrobial use, imaging data, herpes simplex virus (HSV) testing, and overall hospital

50 h as immunotherapy with oncolytic engineered herpes simplex virus (HSV) therapy, are urgently warrant

51 The herpes simplex virus (HSV) type I alkaline nuclease, UL1

52 Presence of CMV, EBV, and herpes simplex virus (HSV) were independent predictors o

53 terial meningoencephalitis, 6% influenza, 6% herpes simplex virus (HSV), and 6% Mycoplasma pneumoniae

54 Anal swabs were collected to test HPV, herpes simplex virus (HSV), and 7 STIs.

55 to, HIV, respiratory syncytial virus (RSV), herpes simplex virus (HSV), and measles.

56 on of certain viruses, including poliovirus, herpes simplex virus (HSV), cytomegalovirus (CMV), and i

57 rather diverse glycan specificities such as Herpes Simplex Virus (HSV), Influenza A Virus (IAV), and

58 Other alphaherpesviruses, including herpes simplex virus (HSV), utilize low-pH-mediated endo

59 h diagnosed with-congenital cytomegalovirus, herpes simplex virus (HSV), varicella zoster virus (VZV)

60 The ubiquitous human pathogens, herpes simplex virus (HSV)-1 and HSV-2, are distinct vir

61 cally significant species of simplexviruses, herpes simplex virus (HSV)-1 and HSV-2, with estimated d

62 Herpes simplex virus (HSV)-1 proteins pUL7 and pUL51 for

63 ation in STING are unexpectedly resistant to Herpes Simplex Virus (HSV)-1, despite lacking STING-indu

64 In herpes simplex virus (HSV)-infected cells, ND10 bodies a

65 creased) particle-to-PFU ratio in vitro than herpes simplex virus (HSV).

66 The herpes simplex virus (HSV-1) latency-associated transcri

67 TDRD7 is a viral restriction factor against herpes simplex virus (HSV-1).

68 One compound was also active against herpes simplex virus (HSV1 and HSV2), and another compou

69 ineered to constitutively express the type I Herpes Simplex Virus (HSV1) HSV-1 receptor, nectin-1, to

70 alone mediates recognition and clearance of herpes simplex virus (HSV1)-infected cells.

71 Oncolytic herpes simplex virus (oHSV) selectively replicates in ca

72 ockout (KO) mice were infected ocularly with herpes simplex virus 1 (HSV-1) (strain McKrae).

73 In the case of the herpes simplex virus 1 (HSV-1) 0DeltaNLS vaccine, the co

74 DNA viruses and include the human pathogens herpes simplex virus 1 (HSV-1) and HSV-2 and are signifi

75 Herpes simplex virus 1 (HSV-1) and HSV-2 are large, doub

76 Herpes simplex virus 1 (HSV-1) and HSV-2 can efficiently

77 ng assembly of the neurotropic herpesviruses herpes simplex virus 1 (HSV-1) and pseudorabies virus (P

78 f UL37 homologs from two alphaherpesviruses, herpes simplex virus 1 (HSV-1) and pseudorabies virus (P

79 strate that these cells can be infected with herpes simplex virus 1 (HSV-1) at a multiplicity of infe

80 rtive infections in HeLa cells infected with herpes simplex virus 1 (HSV-1) at high multiplicity of i

81 ILCs isolated from mice can be infected with herpes simplex virus 1 (HSV-1) but that subsequent repli

82 Herpes simplex virus 1 (HSV-1) can induce damage in brai

83 Herpes simplex virus 1 (HSV-1) can infect virtually all

84 Herpes simplex virus 1 (HSV-1) causes a lifelong infecti

85 Herpes simplex virus 1 (HSV-1) causes significant morbid

86 Herpes simplex virus 1 (HSV-1) cycles between phases of

87 Herpes simplex virus 1 (HSV-1) encodes the multifunction

88 ined whether Cbl mediates the removal of the herpes simplex virus 1 (HSV-1) entry receptor Nectin-1 f

89 rs such as bovine herpesvirus 1 (BoHV-1) and herpes simplex virus 1 (HSV-1) establish and maintain li

90 bility to reactivate from latency.IMPORTANCE Herpes simplex virus 1 (HSV-1) establishes a lifelong in

91 Herpes simplex virus 1 (HSV-1) establishes a lifelong la

92 Herpes simplex virus 1 (HSV-1) establishes latency in bo

93 Herpes simplex virus 1 (HSV-1) establishes latency withi

94 Following acute infection, herpes simplex virus 1 (HSV-1) establishes lifelong late

95 Herpes simplex virus 1 (HSV-1) establishes lifelong late

96 induced reactivation from latency.IMPORTANCE Herpes simplex virus 1 (HSV-1) establishes lifelong late

97 Chronic viruses such as herpes simplex virus 1 (HSV-1) evade the hosts' immune s

98 An earlier report showed that herpes simplex virus 1 (HSV-1) expresses two microRNAs (

99 During all stages of infection, herpes simplex virus 1 (HSV-1) expresses viral microRNAs

100 is one of several cellular receptors used by herpes simplex virus 1 (HSV-1) for cell entry.

101 Reactivation of herpes simplex virus 1 (HSV-1) from neurons in sensory g

102 The Us11 protein encoded by herpes simplex virus 1 (HSV-1) functions to impair autop

103 plication greatly enhances expression of the herpes simplex virus 1 (HSV-1) gamma2 late genes by stil

104 Herpes simplex virus 1 (HSV-1) genes are transcribed by

105 i derived from the long-arm component of the herpes simplex virus 1 (HSV-1) genome, (iv) pUL36 serves

106 gp120) construct fused to a small portion of herpes simplex virus 1 (HSV-1) glycoprotein D (gD) so th

107 Herpes simplex virus 1 (HSV-1) has infected more than 80

108 e predicted 80 open reading frames (ORFs) of herpes simplex virus 1 (HSV-1) have been intensively stu

109 emethylation of histone H3K9 associated with herpes simplex virus 1 (HSV-1) immediate early (IE) prom

110 The herpes simplex virus 1 (HSV-1) immediate early protein I

111 the role of CD8(+) T cells in the control of herpes simplex virus 1 (HSV-1) infection and disease is

112 es have established a potential link between herpes simplex virus 1 (HSV-1) infection and the develop

113 Analysis of Herpes Simplex virus 1 (HSV-1) infection by population-a

114 onses following vaccination in resistance to herpes simplex virus 1 (HSV-1) infection continues to be

115 We have previously shown that herpes simplex virus 1 (HSV-1) infection results in the

116 We recently reported that herpes simplex virus 1 (HSV-1) infection suppresses CD80

117 (-/-) mice were highly susceptible to ocular herpes simplex virus 1 (HSV-1) infection, independent of

118 Herpes simplex virus 1 (HSV-1) infections afflict more t

119 Herpes simplex virus 1 (HSV-1) infects mucosal epithelia

120 Herpes simplex virus 1 (HSV-1) infects several types of

121 In our murine oro-ocular (OO) model, herpes simplex virus 1 (HSV-1) inoculation in one side o

122 Herpes simplex virus 1 (HSV-1) is a contagious neurotrop

123 The envelope glycoprotein I (gI) of herpes simplex virus 1 (HSV-1) is a critical mediator of

124 Herpes simplex virus 1 (HSV-1) is a leading cause of inf

125 t of proteolipids in this process.IMPORTANCE Herpes simplex virus 1 (HSV-1) is a neurotropic pathogen

126 Herpes simplex virus 1 (HSV-1) is one of the eight herpe

127 viral DNA from NPC-bound capsids.IMPORTANCE Herpes simplex virus 1 (HSV-1) is the causative agent of

128 amatic displacement of the portal.IMPORTANCE Herpes simplex virus 1 (HSV-1) is the causative agent of

129 Utilizing a mouse model of herpes simplex virus 1 (HSV-1) keratitis, we found that

130 Herpes simplex virus 1 (HSV-1) latency entails the repre

131 High rates of wild-type (WT) herpes simplex virus 1 (HSV-1) latency reactivation depe

132 lular insulator protein CTCF plays a role in herpes simplex virus 1 (HSV-1) latency through the estab

133 Corneal infection with herpes simplex virus 1 (HSV-1) leads to infection of tri

134 ve previously shown that the live-attenuated herpes simplex virus 1 (HSV-1) mutant lacking the nuclea

135 pectrometry approach, we have shown that the herpes simplex virus 1 (HSV-1) neurovirulence- and autop

136 le effect on cell-intrinsic immunity against herpes simplex virus 1 (HSV-1) or gammaherpesvirus 68 (g

137 ering number of the world population harbors herpes simplex virus 1 (HSV-1) potentially leading to bl

138 stimulates bovine herpesvirus 1 (BoHV-1) and herpes simplex virus 1 (HSV-1) reactivation.

139 was to reexamine the requirement of UL21 for herpes simplex virus 1 (HSV-1) replication.

140 Ocular infection with herpes simplex virus 1 (HSV-1) sets off an inflammatory

141 terograde transneuronal tracers derived from herpes simplex virus 1 (HSV-1) strain 129 (H129) are imp

142 The features of herpes simplex virus 1 (HSV-1) strain 129 (H129), includ

143 nd ICP34.5 are among the proteins encoded by herpes simplex virus 1 (HSV-1) that modulate type I IFN

144 e ability of the prototypic alphaherpesvirus herpes simplex virus 1 (HSV-1) to enter neurons via axon

145 We used herpes simplex virus 1 (HSV-1) to infect the human DRG-d

146 stressful stimuli.IMPORTANCE The ability of herpes simplex virus 1 (HSV-1) to periodically reactivat

147 nd transmission to nerve endings, capsids of herpes simplex virus 1 (HSV-1) travel retrogradely withi

148 Herpes simplex virus 1 (HSV-1) triggers both the cyclic

149 Herein, we report that the live-attenuated herpes simplex virus 1 (HSV-1) VC2 vaccine strain, which

150 Here, we show that herpes simplex virus 1 (HSV-1) virions travel in associa

151 Entry of the human alphaherpesvirus herpes simplex virus 1 (HSV-1) was independent of both h

152 sidering the three groups, the prevalence of herpes simplex virus 1 (HSV-1) were 9% in saliva and 5%

153 e proportion of the world population harbors herpes simplex virus 1 (HSV-1), a major cause of infecti

154 protein 0 (ICP0), an E3 ubiquitin ligase of herpes simplex virus 1 (HSV-1), can derepress viral gene

155 Oncolytic herpes simplex virus 1 (HSV-1), devoid of the gamma(1)34

156 In cells infected with herpes simplex virus 1 (HSV-1), hnRNPA2B1 was quantitati

157 roinvasive alphaherpesviruses, such as human herpes simplex virus 1 (HSV-1), HSV-2, and veterinarian

158 al studies of the prototypical herpesviruses herpes simplex virus 1 (HSV-1), HSV-2, human cytomegalov

159 ocused on understanding the biology of human herpes simplex virus 1 (HSV-1), no tool has been develop

160 zuelan equine encephalitis virus (VEEV), and herpes simplex virus 1 (HSV-1), suggesting that LIMK inh

161 phages and viruses, including T4, Phi29, and herpes simplex virus 1 (HSV-1), the portal forms a nucle

162 sosome-terminal endocytic pathway.IMPORTANCE Herpes simplex virus 1 (HSV-1), the prototype alphaherpe

163 For herpes simplex virus 1 (HSV-1), we and others have previ

164 In herpes simplex virus 1 (HSV-1)-infected cells, hnRNPA2B1

165 Although most herpes simplex virus 1 (HSV-1)-infected individuals shed

166 the more distantly related alphaherpesvirus herpes simplex virus 1 (HSV-1).

167 5beta, are activated early in infection with herpes simplex virus 1 (HSV-1).

168 GLF5 in Epstein-Barr virus (EBV), and vhs in herpes simplex virus 1 (HSV-1).

169 irus that can enter via the plasma membrane, herpes simplex virus 1 (HSV-1).

170 SP141), bound to RIG-I during infection with herpes simplex virus 1 (HSV-1).

171 between AAV2 and one of its helper viruses, herpes simplex virus 1 (HSV-1).

172 f mice that have been ocularly infected with herpes simplex virus 1 (HSV-1).

173 titer than other alphaherpesviruses, such as herpes simplex virus 1 (HSV1) or pseudorabies virus (PRV

174 In human cells, herpes simplex virus 1 (HSV1) UL39-encoded ICP6 blocks R

175 that are derived from the DNA polymerase of herpes simplex virus 1 (Pol peptides).

176 aherpesviruses (pseudorabies virus [PRV] and herpes simplex virus 1 [HSV-1]).

177 EBV], human herpesvirus 6A [HHV-6A], HHV-6B, herpes simplex virus 1 [HSV-1], HSV-2, JC virus [JCV], a

178 ncestral alphaherpesvirus that is related to herpes simplex virus 1 and causes respiratory, reproduct

179 This VZV study also complemented prior herpes simplex virus 1 and pseudorabies virus studies in

180 presence of brefeldin A, while studies with herpes simplex virus 1 documented an impaired secondary

181 -oligosaccharyltransferase with NGI-1 causes herpes simplex virus 1 dysfunction.

182 This disease can occur after reactivation of herpes simplex virus 1 in the trigeminal ganglia, leadin

183 Furthermore, time-lapse imaging of herpes simplex virus 1 infected epithelial cells enabled

184 e the molecular organization of chromatin in herpes simplex virus 1 infection and its effect on the t

185 ns independently of this activity.IMPORTANCE Herpes simplex virus 1 invades the nervous system by ent

186 owth within tumor cells.IMPORTANCE Oncolytic herpes simplex virus 1 is a promising agent for cancer i

187 polyfunctional antibody responses.IMPORTANCE Herpes simplex virus 1 is the leading cause of infectiou

188 er replication of BKV, whereas influenza and herpes simplex virus 1 replication were clearly reduced.

189 study focuses on two tegument proteins from herpes simplex virus 1 that are conserved in all herpesv

190 selectively kill senescent cells expressing herpes simplex virus 1 thymidine kinase (HSV-TK).

191 a direct association between infection with herpes simplex virus 1, a ubiquitous human pathogen gene

192 virus, influenza virus, dengue virus type 2, herpes simplex virus 1, and nonenveloped human adenoviru

193 stomatitis virus, Semliki Forest virus, and herpes simplex virus 1, elicit the neuronal expression o

194 e, whereas infection with influenza A virus, herpes simplex virus 1, or cytomegalovirus induced a str

195 and higher incidence of cytomegalovirus and herpes simplex virus 1, possibly influenced by demograph

196 Random walk modelling of herpes simplex virus 1-sized particles in a three-dimens

197 adults worldwide are latently infected with herpes simplex virus 1.

198 Human immunodeficiency virus (HIV-1) and herpes simplex virus 2 (HSV-2) affect hundreds of millio

199 Herpes simplex virus 2 (HSV-2) can be transmitted in the

200 Reactivation of herpes simplex virus 2 (HSV-2) from latency causes viral

201 after the last virus inoculation.IMPORTANCE Herpes simplex virus 2 (HSV-2) infects nearly 500 millio

202 Herpes simplex virus 2 (HSV-2) is a common sexually tran

203 Reactivation of herpes simplex virus 2 (HSV-2) results in infection of e

204 y(I.C) double-stranded RNA or infection with herpes simplex virus 2 (HSV-2).

205 Herpes simplex virus 2 (HSV2) causes genital herpes in >

206 in addition to one of three TAP inhibitors: herpes simplex virus 2 ICP47, bovine herpes virus 1 UL49

207 nced by certain microbial stimuli, including herpes simplex virus 2, and blocked by antibodies agains

208 Instead, upon secondary challenge with herpes simplex virus 2, circulating memory B cells that

209 Thirteen patients (62%) had herpes simplex virus and 8 patients (38%) had varicella

210 ital specimens each containing >/=105 copies herpes simplex virus DNA/ml collected a median of 5 mont

211 ciation, presence of ovarian teratoma, prior herpes simplex virus encephalitis, and isolated psychiat

212 revious reports linking TLR3 deficiency with herpes simplex virus encephalitis.

213 Like all the herpesviruses, herpes simplex virus encodes machinery that enables it t

214 ly in stromal fibroblasts restores oncolytic herpes simplex virus function.

215 (UL16-binding protein 4)(27); MICA(41); the herpes simplex virus glycoprotein HSVgI(33); ATPase-apol

216 h inhibits HIV-1, HIV-2, Influenza virus and herpes simplex virus infection, and enhances the potenti

217 variety of tests are available for detecting herpes simplex virus infection, but only a subset are us

218 monly found bacterial pigment in controlling herpes simplex virus infection, for which diverse and mu

219 During Herpes Simplex Virus infection, viral replication compar

220 icase-primase inhibitor for the treatment of herpes simplex virus infections, was prepared.

221 r development of antiviral agents.IMPORTANCE Herpes simplex virus is a major pathogen, and although n

222 prevention of neonatal infections.IMPORTANCE Herpes simplex virus is among the most serious infection

223 ovir decreased the recurrence of any type of herpes simplex virus keratitis by approximately half.

224 h clinically diagnosed ARN, PCR-positive for herpes simplex virus or varicella zoster virus and evalu

225 activity during HSV-1 infections.IMPORTANCE Herpes simplex virus persists lifelong in neurons and ca

226 transgenic mice expressing the suicide gene, herpes simplex virus thymidine kinase (HSVtk), driven by

227 w that respiratory syncytial virus (RSV) and herpes simplex virus type 1 (HSV-1) accumulate a rich an

228 Here we report that GA inhibits Herpes simplex virus type 1 (HSV-1) by inhibition of bot

229 This study aimed at characterizing herpes simplex virus type 1 (HSV-1) epidemiology in the

230 Herpes simplex virus type 1 (HSV-1) has the ability to d

231 Infectious titers of EBOV or herpes simplex virus type 1 (HSV-1) in detergents-treate

232 Orolabial herpes simplex virus type 1 (HSV-1) infection has a wide

233 Herpes simplex keratitis (HSK), caused by herpes simplex virus type 1 (HSV-1) infection, is the co

234 In herpes simplex virus type 1 (HSV-1) infection, the coupl

235 ls associated with protective and pathogenic herpes simplex virus type 1 (HSV-1) infections remains u

236 Glycoprotein D (gD) of herpes simplex virus type 1 (HSV-1) is one of four glyco

237 lly occurs during delivery from mothers with herpes simplex virus type 1 (HSV-1) or type 2 (HSV-2) ge

238 Each herpes simplex virus type 1 (HSV-1) replication compartm

239 ological stress.IMPORTANCE Like all viruses, herpes simplex virus type 1 (HSV-1) reproduction relies

240 Here we use direct RNA-seq to profile the herpes simplex virus type 1 (HSV-1) transcriptome during

241 we reported a new series of highly defective herpes simplex virus type 1 (HSV-1) vectors that were fu

242 tion and spread of vaccinia virus (VACV) and herpes simplex virus type 1 (HSV-1) were enhanced in HDA

243 asis of genome packaging and organization in herpes simplex virus type 1 (HSV-1), we developed sequen

244 ormational approach to genome engineering of herpes simplex virus type 1 (HSV-1), which has a large D

245 Herpes simplex virus type 1 (HSV-1)-infected corneas can

246 (mDCs) are not permissive for infection with herpes simplex virus type 1 (HSV-1).

247 t protective functions during infection with herpes simplex virus type 1 (HSV-1).

248 Herpes simplex virus type 1 (HSV1) infection has been as

249 unravel the complexity of the interactome of herpes simplex virus type 1 (HSV1), the prototypical her

250 Support for the concept that herpes simplex virus type 1 (HSV1), when present in the

251 Herpes simplex virus type 1 and Alzheimer's disease: pos

252 athway are associated with susceptibility to herpes simplex virus type 1 encephalitis (HSE).

253 Recent infection with, or reactivation of, herpes simplex virus type 1 or type 1/2 unspecified, cyt

254 The herpes simplex virus type 1 thymidine kinase (HSV1-tk) g

255 CMV, herpes simplex virus type 1, and human herpesvirus 6 inf

256 e association of 4 human herpesviruses (CMV, herpes simplex virus type 1, human herpesvirus type 6, a

257 une responses to chronic infections, such as herpes simplex virus type 2 (HSV-2) in HIV/HSV-coinfecte

258 f infection were seen in smokers, those with herpes simplex virus type 2 (HSV-2) infection, men who h

259 n to be at least tripled in individuals with herpes simplex virus type 2 (HSV-2) infection.

260 HIV and herpes simplex virus type 2 (HSV-2) infections cause a s

261 Genital infection with herpes simplex virus type 2 (HSV-2) is common and increa

262 This study investigated herpes simplex virus type 2 (HSV-2) seroprevalence utili

263 Herpes simplex virus type 2 (HSV-2; herpes) exacerbates

264 against cervical cancer, cervical dysplasia, herpes simplex virus type 2, chlamydia, and syphilis.

265 Interestingly, cells infected with herpes simplex virus type-1 (HSV-1) incorporated EdC and

266 Herpes simplex virus type-1 (HSV-1), one of the most wid

267 CB) women, including bacterial vaginosis and herpes simplex virus type-2 (HSV-2) infection.

268 ded controls for sociodemographic variables, herpes simplex virus type-2 status, and recreational dru

269 rr virus (EBV), human herpesvirus 6 (HHV-6), herpes simplex virus types 1 (HSV-1) and 2 (HSV-2), and

270 ed by the development of acyclovir-resistant herpes simplex virus viremia, primary graft failure, and

271 lium, Trichomonas vaginalis, adenovirus, and herpes simplex virus were absent.

272 ed virus, often an adeno-associated virus or herpes simplex virus, among many other types.

273 es were investigated for Epstein-Barr virus, herpes simplex virus, and HCMV-specific immunoglobulin G

274 iency virus, hepatitis B virus, and neonatal herpes simplex virus, from which lessons for the evaluat

275 papilloma virus, hepatitis B and C viruses, herpes simplex virus, norovirus, rotavirus, parvovirus,

276 For herpes simplex virus, this process requires the products

277 al response to HCMV, but not Epstein-Barr or herpes simplex virus, was associated with increased risk

278 Herpes simplex virus-1 (HSV-1) encephalitis (HSE) is typ

279 Herpes simplex virus-1 (HSV-1) establishes a latent infe

280 iviral immunity in mouse and human models of herpes simplex virus-1 (HSV-1) infections.

281 gy is a powerful host defense that restricts herpes simplex virus-1 (HSV-1) pathogenesis in neurons.

282 Herpes simplex virus-1 (HSV-1) replicates within the nuc

283 Neurotropic viruses, such as herpes simplex virus-1 (HSV-1), also rely on cellular AK

284 Infection by viruses, including herpes simplex virus-1 (HSV-1), and cellular stresses ca

285 Concordantly, GPX4 deficiency inhibited herpes simplex virus-1 (HSV-1)-induced innate antiviral

286 f several other pathogenic viruses including Herpes Simplex Virus-1 and-2, Vaccinia virus, and Zika v

287 cytomegalovirus (MCMV) or the human pathogen herpes simplex virus-1 compared with littermate control

288 ared to class B infections (cytomegalovirus, herpes simplex virus-1/2, human herpesvirus 8, hepatitis

289 e mechanisms underlying rapid elimination of herpes simplex virus-2 (HSV-2) in the human genital trac

290 ells.IMPORTANCE We study the pathogenesis of herpes simplex virus-mediated corneal disease.

291 -Barr virus (EBV), cytomegalovirus (CMV) and Herpes simplex virus.

292 that include the significant human pathogens herpes simplex viruses (HSV) and varicella zoster virus

293 significant effect.IMPORTANCE Infections by herpes simplex viruses (HSV) cause painful cold sores or

294 ntain the same genetic variations.IMPORTANCE Herpes simplex viruses (HSV) infect a majority of adults

295 ults in Finland are seropositive carriers of herpes simplex viruses (HSV).

296 transport of enveloped particles.IMPORTANCE Herpes simplex viruses 1 and 2 and varicella-zoster viru

297 alphaherpesvirus related to human pathogens herpes simplex viruses 1 and 2 and varicella-zoster viru

298 rus, mumps virus, measles virus, lyssavirus, herpes simplex viruses 1 and 2, Epstein-Barr virus, ente

299 Herpes simplex viruses bud into the nuclear membrane of

300 pesviruses such as varicella-zoster virus or herpes simplex viruses.