ライフサイエンスコーパス: herpes simplex virus

1 -Barr virus (EBV), cytomegalovirus (CMV) and Herpes simplex virus.

2 pesviruses such as varicella-zoster virus or herpes simplex viruses.

3 ockout (KO) mice were infected ocularly with herpes simplex virus 1 (HSV-1) (strain McKrae).

4 In the case of the herpes simplex virus 1 (HSV-1) 0DeltaNLS vaccine, the co

5 DNA viruses and include the human pathogens herpes simplex virus 1 (HSV-1) and HSV-2 and are signifi

6 Herpes simplex virus 1 (HSV-1) and HSV-2 are large, doub

7 Herpes simplex virus 1 (HSV-1) and HSV-2 can efficiently

8 f UL37 homologs from two alphaherpesviruses, herpes simplex virus 1 (HSV-1) and pseudorabies virus (P

9 ng assembly of the neurotropic herpesviruses herpes simplex virus 1 (HSV-1) and pseudorabies virus (P

10 strate that these cells can be infected with herpes simplex virus 1 (HSV-1) at a multiplicity of infe

11 rtive infections in HeLa cells infected with herpes simplex virus 1 (HSV-1) at high multiplicity of i

12 ILCs isolated from mice can be infected with herpes simplex virus 1 (HSV-1) but that subsequent repli

13 Herpes simplex virus 1 (HSV-1) can induce damage in brai

14 Herpes simplex virus 1 (HSV-1) can infect virtually all

15 Herpes simplex virus 1 (HSV-1) causes a lifelong infecti

16 Herpes simplex virus 1 (HSV-1) causes significant morbid

17 Herpes simplex virus 1 (HSV-1) cycles between phases of

18 Herpes simplex virus 1 (HSV-1) encodes the multifunction

19 ined whether Cbl mediates the removal of the herpes simplex virus 1 (HSV-1) entry receptor Nectin-1 f

20 rs such as bovine herpesvirus 1 (BoHV-1) and herpes simplex virus 1 (HSV-1) establish and maintain li

21 bility to reactivate from latency.IMPORTANCE Herpes simplex virus 1 (HSV-1) establishes a lifelong in

22 Herpes simplex virus 1 (HSV-1) establishes a lifelong la

23 Herpes simplex virus 1 (HSV-1) establishes latency in bo

24 Herpes simplex virus 1 (HSV-1) establishes latency withi

25 Following acute infection, herpes simplex virus 1 (HSV-1) establishes lifelong late

26 Herpes simplex virus 1 (HSV-1) establishes lifelong late

27 induced reactivation from latency.IMPORTANCE Herpes simplex virus 1 (HSV-1) establishes lifelong late

28 Chronic viruses such as herpes simplex virus 1 (HSV-1) evade the hosts' immune s

29 An earlier report showed that herpes simplex virus 1 (HSV-1) expresses two microRNAs (

30 During all stages of infection, herpes simplex virus 1 (HSV-1) expresses viral microRNAs

31 is one of several cellular receptors used by herpes simplex virus 1 (HSV-1) for cell entry.

32 Reactivation of herpes simplex virus 1 (HSV-1) from neurons in sensory g

33 The Us11 protein encoded by herpes simplex virus 1 (HSV-1) functions to impair autop

34 plication greatly enhances expression of the herpes simplex virus 1 (HSV-1) gamma2 late genes by stil

35 Herpes simplex virus 1 (HSV-1) genes are transcribed by

36 i derived from the long-arm component of the herpes simplex virus 1 (HSV-1) genome, (iv) pUL36 serves

37 gp120) construct fused to a small portion of herpes simplex virus 1 (HSV-1) glycoprotein D (gD) so th

38 Herpes simplex virus 1 (HSV-1) has infected more than 80

39 e predicted 80 open reading frames (ORFs) of herpes simplex virus 1 (HSV-1) have been intensively stu

40 emethylation of histone H3K9 associated with herpes simplex virus 1 (HSV-1) immediate early (IE) prom

41 The herpes simplex virus 1 (HSV-1) immediate early protein I

42 the role of CD8(+) T cells in the control of herpes simplex virus 1 (HSV-1) infection and disease is

43 es have established a potential link between herpes simplex virus 1 (HSV-1) infection and the develop

44 Analysis of Herpes Simplex virus 1 (HSV-1) infection by population-a

45 onses following vaccination in resistance to herpes simplex virus 1 (HSV-1) infection continues to be

46 Herpes simplex virus 1 (HSV-1) infection is widespread a

47 We have previously shown that herpes simplex virus 1 (HSV-1) infection results in the

48 We recently reported that herpes simplex virus 1 (HSV-1) infection suppresses CD80

49 (-/-) mice were highly susceptible to ocular herpes simplex virus 1 (HSV-1) infection, independent of

50 Herpes simplex virus 1 (HSV-1) infections afflict more t

51 Herpes simplex virus 1 (HSV-1) infects mucosal epithelia

52 Herpes simplex virus 1 (HSV-1) infects several types of

53 In our murine oro-ocular (OO) model, herpes simplex virus 1 (HSV-1) inoculation in one side o

54 The immediate early protein ICP0 of herpes simplex virus 1 (HSV-1) interacts with CIN85, an

55 Herpes simplex virus 1 (HSV-1) is a contagious neurotrop

56 The envelope glycoprotein I (gI) of herpes simplex virus 1 (HSV-1) is a critical mediator of

57 Herpes simplex virus 1 (HSV-1) is a leading cause of inf

58 t of proteolipids in this process.IMPORTANCE Herpes simplex virus 1 (HSV-1) is a neurotropic pathogen

59 Human herpes simplex virus 1 (HSV-1) is a widespread pathogen,

60 The UL16 tegument protein of herpes simplex virus 1 (HSV-1) is conserved among all he

61 Herpes simplex virus 1 (HSV-1) is one of the eight herpe

62 viral DNA from NPC-bound capsids.IMPORTANCE Herpes simplex virus 1 (HSV-1) is the causative agent of

63 amatic displacement of the portal.IMPORTANCE Herpes simplex virus 1 (HSV-1) is the causative agent of

64 Utilizing a mouse model of herpes simplex virus 1 (HSV-1) keratitis, we found that

65 Herpes simplex virus 1 (HSV-1) latency entails the repre

66 High rates of wild-type (WT) herpes simplex virus 1 (HSV-1) latency reactivation depe

67 lular insulator protein CTCF plays a role in herpes simplex virus 1 (HSV-1) latency through the estab

68 Corneal infection with herpes simplex virus 1 (HSV-1) leads to infection of tri

69 ve previously shown that the live-attenuated herpes simplex virus 1 (HSV-1) mutant lacking the nuclea

70 pectrometry approach, we have shown that the herpes simplex virus 1 (HSV-1) neurovirulence- and autop

71 le effect on cell-intrinsic immunity against herpes simplex virus 1 (HSV-1) or gammaherpesvirus 68 (g

72 ering number of the world population harbors herpes simplex virus 1 (HSV-1) potentially leading to bl

73 stimulates bovine herpesvirus 1 (BoHV-1) and herpes simplex virus 1 (HSV-1) reactivation.

74 was to reexamine the requirement of UL21 for herpes simplex virus 1 (HSV-1) replication.

75 Ocular infection with herpes simplex virus 1 (HSV-1) sets off an inflammatory

76 terograde transneuronal tracers derived from herpes simplex virus 1 (HSV-1) strain 129 (H129) are imp

77 The features of herpes simplex virus 1 (HSV-1) strain 129 (H129), includ

78 nd ICP34.5 are among the proteins encoded by herpes simplex virus 1 (HSV-1) that modulate type I IFN

79 e ability of the prototypic alphaherpesvirus herpes simplex virus 1 (HSV-1) to enter neurons via axon

80 We used herpes simplex virus 1 (HSV-1) to infect the human DRG-d

81 stressful stimuli.IMPORTANCE The ability of herpes simplex virus 1 (HSV-1) to periodically reactivat

82 nd transmission to nerve endings, capsids of herpes simplex virus 1 (HSV-1) travel retrogradely withi

83 Herpes simplex virus 1 (HSV-1) triggers both the cyclic

84 Herpes simplex virus 1 (HSV-1) UL20 plays a crucial role

85 Herein, we report that the live-attenuated herpes simplex virus 1 (HSV-1) VC2 vaccine strain, which

86 Here, we show that herpes simplex virus 1 (HSV-1) virions travel in associa

87 Entry of the human alphaherpesvirus herpes simplex virus 1 (HSV-1) was independent of both h

88 sidering the three groups, the prevalence of herpes simplex virus 1 (HSV-1) were 9% in saliva and 5%

89 e proportion of the world population harbors herpes simplex virus 1 (HSV-1), a major cause of infecti

90 protein 0 (ICP0), an E3 ubiquitin ligase of herpes simplex virus 1 (HSV-1), can derepress viral gene

91 Oncolytic herpes simplex virus 1 (HSV-1), devoid of the gamma(1)34

92 In cells infected with herpes simplex virus 1 (HSV-1), hnRNPA2B1 was quantitati

93 roinvasive alphaherpesviruses, such as human herpes simplex virus 1 (HSV-1), HSV-2, and veterinarian

94 al studies of the prototypical herpesviruses herpes simplex virus 1 (HSV-1), HSV-2, human cytomegalov

95 ocused on understanding the biology of human herpes simplex virus 1 (HSV-1), no tool has been develop

96 zuelan equine encephalitis virus (VEEV), and herpes simplex virus 1 (HSV-1), suggesting that LIMK inh

97 phages and viruses, including T4, Phi29, and herpes simplex virus 1 (HSV-1), the portal forms a nucle

98 sosome-terminal endocytic pathway.IMPORTANCE Herpes simplex virus 1 (HSV-1), the prototype alphaherpe

99 For herpes simplex virus 1 (HSV-1), we and others have previ

100 In herpes simplex virus 1 (HSV-1)-infected cells, hnRNPA2B1

101 Although most herpes simplex virus 1 (HSV-1)-infected individuals shed

102 the more distantly related alphaherpesvirus herpes simplex virus 1 (HSV-1).

103 5beta, are activated early in infection with herpes simplex virus 1 (HSV-1).

104 GLF5 in Epstein-Barr virus (EBV), and vhs in herpes simplex virus 1 (HSV-1).

105 irus that can enter via the plasma membrane, herpes simplex virus 1 (HSV-1).

106 SP141), bound to RIG-I during infection with herpes simplex virus 1 (HSV-1).

107 between AAV2 and one of its helper viruses, herpes simplex virus 1 (HSV-1).

108 f mice that have been ocularly infected with herpes simplex virus 1 (HSV-1).

109 titer than other alphaherpesviruses, such as herpes simplex virus 1 (HSV1) or pseudorabies virus (PRV

110 In human cells, herpes simplex virus 1 (HSV1) UL39-encoded ICP6 blocks R

111 that are derived from the DNA polymerase of herpes simplex virus 1 (Pol peptides).

112 aherpesviruses (pseudorabies virus [PRV] and herpes simplex virus 1 [HSV-1]).

113 EBV], human herpesvirus 6A [HHV-6A], HHV-6B, herpes simplex virus 1 [HSV-1], HSV-2, JC virus [JCV], a

114 ncestral alphaherpesvirus that is related to herpes simplex virus 1 and causes respiratory, reproduct

115 This VZV study also complemented prior herpes simplex virus 1 and pseudorabies virus studies in

116 presence of brefeldin A, while studies with herpes simplex virus 1 documented an impaired secondary

117 -oligosaccharyltransferase with NGI-1 causes herpes simplex virus 1 dysfunction.

118 This disease can occur after reactivation of herpes simplex virus 1 in the trigeminal ganglia, leadin

119 Furthermore, time-lapse imaging of herpes simplex virus 1 infected epithelial cells enabled

120 e the molecular organization of chromatin in herpes simplex virus 1 infection and its effect on the t

121 ns independently of this activity.IMPORTANCE Herpes simplex virus 1 invades the nervous system by ent

122 owth within tumor cells.IMPORTANCE Oncolytic herpes simplex virus 1 is a promising agent for cancer i

123 polyfunctional antibody responses.IMPORTANCE Herpes simplex virus 1 is the leading cause of infectiou

124 er replication of BKV, whereas influenza and herpes simplex virus 1 replication were clearly reduced.

125 study focuses on two tegument proteins from herpes simplex virus 1 that are conserved in all herpesv

126 tein domains, as well as a truncated form of herpes simplex virus 1 thymidine kinase (HSV-TK).

127 selectively kill senescent cells expressing herpes simplex virus 1 thymidine kinase (HSV-TK).

128 a direct association between infection with herpes simplex virus 1, a ubiquitous human pathogen gene

129 virus, influenza virus, dengue virus type 2, herpes simplex virus 1, and nonenveloped human adenoviru

130 stomatitis virus, Semliki Forest virus, and herpes simplex virus 1, elicit the neuronal expression o

131 e, whereas infection with influenza A virus, herpes simplex virus 1, or cytomegalovirus induced a str

132 and higher incidence of cytomegalovirus and herpes simplex virus 1, possibly influenced by demograph

133 Random walk modelling of herpes simplex virus 1-sized particles in a three-dimens

134 adults worldwide are latently infected with herpes simplex virus 1.

135 Herpes simplex viruses 1 and 2 (HSV-1 and HSV-2) infect

136 transport of enveloped particles.IMPORTANCE Herpes simplex viruses 1 and 2 and varicella-zoster viru

137 alphaherpesvirus related to human pathogens herpes simplex viruses 1 and 2 and varicella-zoster viru

138 rus, mumps virus, measles virus, lyssavirus, herpes simplex viruses 1 and 2, Epstein-Barr virus, ente

139 Herpes simplex virus-1 (HSV-1) encephalitis (HSE) is typ

140 Herpes simplex virus-1 (HSV-1) establishes a latent infe

141 iviral immunity in mouse and human models of herpes simplex virus-1 (HSV-1) infections.

142 Herpes Simplex Virus-1 (HSV-1) is a ubiquitous human pat

143 gy is a powerful host defense that restricts herpes simplex virus-1 (HSV-1) pathogenesis in neurons.

144 Herpes simplex virus-1 (HSV-1) replicates within the nuc

145 Neurotropic viruses, such as herpes simplex virus-1 (HSV-1), also rely on cellular AK

146 Infection by viruses, including herpes simplex virus-1 (HSV-1), and cellular stresses ca

147 Concordantly, GPX4 deficiency inhibited herpes simplex virus-1 (HSV-1)-induced innate antiviral

148 f several other pathogenic viruses including Herpes Simplex Virus-1 and-2, Vaccinia virus, and Zika v

149 cytomegalovirus (MCMV) or the human pathogen herpes simplex virus-1 compared with littermate control

150 ers in the lung, and are resistant to lethal herpes simplex virus-1 infection due to enhanced product

151 Here, we address this deficit, focusing on a herpes simplex virus-1 protein, ICP27.

152 ared to class B infections (cytomegalovirus, herpes simplex virus-1/2, human herpesvirus 8, hepatitis

153 Human immunodeficiency virus (HIV-1) and herpes simplex virus 2 (HSV-2) affect hundreds of millio

154 Herpes simplex virus 2 (HSV-2) can be transmitted in the

155 Reactivation of herpes simplex virus 2 (HSV-2) from latency causes viral

156 after the last virus inoculation.IMPORTANCE Herpes simplex virus 2 (HSV-2) infects nearly 500 millio

157 Herpes simplex virus 2 (HSV-2) is a common sexually tran

158 Reactivation of herpes simplex virus 2 (HSV-2) results in infection of e

159 y(I.C) double-stranded RNA or infection with herpes simplex virus 2 (HSV-2).

160 Herpes simplex virus 2 (HSV2) causes genital herpes in >

161 in addition to one of three TAP inhibitors: herpes simplex virus 2 ICP47, bovine herpes virus 1 UL49

162 nced by certain microbial stimuli, including herpes simplex virus 2, and blocked by antibodies agains

163 Instead, upon secondary challenge with herpes simplex virus 2, circulating memory B cells that

164 e mechanisms underlying rapid elimination of herpes simplex virus-2 (HSV-2) in the human genital trac

165 erpesviridae-positive, which included 9 with herpes simplex virus (8.8%), 5 with varicella-zoster vir

166 ed virus, often an adeno-associated virus or herpes simplex virus, among many other types.

167 Thirteen patients (62%) had herpes simplex virus and 8 patients (38%) had varicella

168 es were investigated for Epstein-Barr virus, herpes simplex virus, and HCMV-specific immunoglobulin G

169 Herpes simplex viruses bud into the nuclear membrane of

170 ital specimens each containing >/=105 copies herpes simplex virus DNA/ml collected a median of 5 mont

171 ciation, presence of ovarian teratoma, prior herpes simplex virus encephalitis, and isolated psychiat

172 revious reports linking TLR3 deficiency with herpes simplex virus encephalitis.

173 Like all the herpesviruses, herpes simplex virus encodes machinery that enables it t

174 iency virus, hepatitis B virus, and neonatal herpes simplex virus, from which lessons for the evaluat

175 ly in stromal fibroblasts restores oncolytic herpes simplex virus function.

176 (UL16-binding protein 4)(27); MICA(41); the herpes simplex virus glycoprotein HSVgI(33); ATPase-apol

177 this assessment include adenovirus 8 and 19, herpes simplex virus (HSV) 1 and 2, human immunodeficien

178 dy of neurotropic viruses in vitroIMPORTANCE Herpes simplex virus (HSV) affects millions of people wo

179 tions that US9 is neuron specific.IMPORTANCE Herpes simplex virus (HSV) and other alphaherpesviruses,

180 al fluid polymerase chain reaction (PCR) for herpes simplex virus (HSV) and varicella zoster virus wa

181 a broad antimicrobial factor that restricts herpes simplex virus (HSV) by activating type I interfer

182 Herpes simplex virus (HSV) can cause severe infection in

183 Neonatal herpes simplex virus (HSV) disease results in unacceptab

184 the TG into the brain stem.IMPORTANCE Latent herpes simplex virus (HSV) DNA has been detected in the

185 erpesvirus entry mediator (HVEM) facilitates herpes simplex virus (HSV) entry through interactions wi

186 Herpes simplex virus (HSV) establishes latency in neuron

187 Herpes simplex virus (HSV) establishes lifelong latent i

188 Herpes simplex virus (HSV) glycoprotein C (gC) functions

189 We previously reported that herpes simplex virus (HSV) glycoprotein K (gK) binds to

190 of protein-protein interactions between four herpes simplex virus (HSV) glycoproteins (gD, gH/gL, and

191 The herpes simplex virus (HSV) heterodimer gE/gI and another

192 umcision was associated with reduced odds of herpes simplex virus (HSV) infection among MSM overall (

193 Numerous cases point to the link between herpes simplex virus (HSV) infection and multifocal CNS

194 Neuron-virus interactions that occur during herpes simplex virus (HSV) infection are not fully under

195 Herpes simplex virus (HSV) infection is restricted to ep

196 Herpes simplex virus (HSV) infection is widespread in th

197 ance frequently complicates the treatment of herpes simplex virus (HSV) infections in immunocompromis

198 Herpes simplex virus (HSV) infections of the central ner

199 Herpes simplex virus (HSV) is a common and often benign

200 Herpes simplex virus (HSV) is a neuroinvasive virus that

201 Herpes simplex virus (HSV) is among the most prevalent v

202 The terminase of herpes simplex virus (HSV) is composed of three subunits

203 eity of cells expressing the LATs.IMPORTANCE Herpes simplex virus (HSV) is responsible for significan

204 Herpes simplex virus (HSV) is the main cause of viral en

205 f aqueous or vitreous humor was positive for herpes simplex virus (HSV) or varicella zoster virus (VZ

206 od to visualize pseudorabies virus (PRV) and herpes simplex virus (HSV) particles in living cells.

207 Despite frequent herpes simplex virus (HSV) reactivation, peripheral nerv

208 d microparticles and a replication-defective herpes simplex virus (HSV) recombinant vector.

209 Herpes simplex virus (HSV) requires fusion between the v

210 The herpes simplex virus (HSV) single-strand DNA binding pro

211 s, systemic antimicrobial use, imaging data, herpes simplex virus (HSV) testing, and overall hospital

212 h as immunotherapy with oncolytic engineered herpes simplex virus (HSV) therapy, are urgently warrant

213 The herpes simplex virus (HSV) type I alkaline nuclease, UL1

214 Presence of CMV, EBV, and herpes simplex virus (HSV) were independent predictors o

215 terial meningoencephalitis, 6% influenza, 6% herpes simplex virus (HSV), and 6% Mycoplasma pneumoniae

216 Anal swabs were collected to test HPV, herpes simplex virus (HSV), and 7 STIs.

217 to, HIV, respiratory syncytial virus (RSV), herpes simplex virus (HSV), and measles.

218 on of certain viruses, including poliovirus, herpes simplex virus (HSV), cytomegalovirus (CMV), and i

219 itro nanomolar irreversible activity against herpes simplex virus (HSV), human papilloma virus, respi

220 rather diverse glycan specificities such as Herpes Simplex Virus (HSV), Influenza A Virus (IAV), and

221 Other alphaherpesviruses, including herpes simplex virus (HSV), utilize low-pH-mediated endo

222 h diagnosed with-congenital cytomegalovirus, herpes simplex virus (HSV), varicella zoster virus (VZV)

223 The ubiquitous human pathogens, herpes simplex virus (HSV)-1 and HSV-2, are distinct vir

224 cally significant species of simplexviruses, herpes simplex virus (HSV)-1 and HSV-2, with estimated d

225 Herpes simplex virus (HSV)-1 proteins pUL7 and pUL51 for

226 ation in STING are unexpectedly resistant to Herpes Simplex Virus (HSV)-1, despite lacking STING-indu

227 In herpes simplex virus (HSV)-infected cells, ND10 bodies a

228 creased) particle-to-PFU ratio in vitro than herpes simplex virus (HSV).

229 The herpes simplex virus (HSV-1) latency-associated transcri

230 TDRD7 is a viral restriction factor against herpes simplex virus (HSV-1).

231 that include the significant human pathogens herpes simplex viruses (HSV) and varicella zoster virus

232 significant effect.IMPORTANCE Infections by herpes simplex viruses (HSV) cause painful cold sores or

233 ntain the same genetic variations.IMPORTANCE Herpes simplex viruses (HSV) infect a majority of adults

234 ults in Finland are seropositive carriers of herpes simplex viruses (HSV).

235 One compound was also active against herpes simplex virus (HSV1 and HSV2), and another compou

236 ineered to constitutively express the type I Herpes Simplex Virus (HSV1) HSV-1 receptor, nectin-1, to

237 alone mediates recognition and clearance of herpes simplex virus (HSV1)-infected cells.

238 h inhibits HIV-1, HIV-2, Influenza virus and herpes simplex virus infection, and enhances the potenti

239 variety of tests are available for detecting herpes simplex virus infection, but only a subset are us

240 monly found bacterial pigment in controlling herpes simplex virus infection, for which diverse and mu

241 During Herpes Simplex Virus infection, viral replication compar

242 icase-primase inhibitor for the treatment of herpes simplex virus infections, was prepared.

243 r development of antiviral agents.IMPORTANCE Herpes simplex virus is a major pathogen, and although n

244 prevention of neonatal infections.IMPORTANCE Herpes simplex virus is among the most serious infection

245 ovir decreased the recurrence of any type of herpes simplex virus keratitis by approximately half.

246 ells.IMPORTANCE We study the pathogenesis of herpes simplex virus-mediated corneal disease.

247 papilloma virus, hepatitis B and C viruses, herpes simplex virus, norovirus, rotavirus, parvovirus,

248 Oncolytic herpes simplex virus (oHSV) selectively replicates in ca

249 h clinically diagnosed ARN, PCR-positive for herpes simplex virus or varicella zoster virus and evalu

250 activity during HSV-1 infections.IMPORTANCE Herpes simplex virus persists lifelong in neurons and ca

251 For herpes simplex virus, this process requires the products

252 transgenic mice expressing the suicide gene, herpes simplex virus thymidine kinase (HSVtk), driven by

253 Furthermore, an unrelated herpes simplex virus-thymidine kinase (HSV-TK) promoter

254 w that respiratory syncytial virus (RSV) and herpes simplex virus type 1 (HSV-1) accumulate a rich an

255 Here we report that GA inhibits Herpes simplex virus type 1 (HSV-1) by inhibition of bot

256 This study aimed at characterizing herpes simplex virus type 1 (HSV-1) epidemiology in the

257 Herpes simplex virus type 1 (HSV-1) has the ability to d

258 Infectious titers of EBOV or herpes simplex virus type 1 (HSV-1) in detergents-treate

259 Orolabial herpes simplex virus type 1 (HSV-1) infection has a wide

260 Herpes simplex keratitis (HSK), caused by herpes simplex virus type 1 (HSV-1) infection, is the co

261 In herpes simplex virus type 1 (HSV-1) infection, the coupl

262 ls associated with protective and pathogenic herpes simplex virus type 1 (HSV-1) infections remains u

263 Glycoprotein D (gD) of herpes simplex virus type 1 (HSV-1) is one of four glyco

264 lly occurs during delivery from mothers with herpes simplex virus type 1 (HSV-1) or type 2 (HSV-2) ge

265 Each herpes simplex virus type 1 (HSV-1) replication compartm

266 ological stress.IMPORTANCE Like all viruses, herpes simplex virus type 1 (HSV-1) reproduction relies

267 Here we use direct RNA-seq to profile the herpes simplex virus type 1 (HSV-1) transcriptome during

268 we reported a new series of highly defective herpes simplex virus type 1 (HSV-1) vectors that were fu

269 tion and spread of vaccinia virus (VACV) and herpes simplex virus type 1 (HSV-1) were enhanced in HDA

270 asis of genome packaging and organization in herpes simplex virus type 1 (HSV-1), we developed sequen

271 ormational approach to genome engineering of herpes simplex virus type 1 (HSV-1), which has a large D

272 Herpes simplex virus type 1 (HSV-1)-infected corneas can

273 (mDCs) are not permissive for infection with herpes simplex virus type 1 (HSV-1).

274 t protective functions during infection with herpes simplex virus type 1 (HSV-1).

275 Herpes simplex virus type 1 (HSV1) infection has been as

276 unravel the complexity of the interactome of herpes simplex virus type 1 (HSV1), the prototypical her

277 Support for the concept that herpes simplex virus type 1 (HSV1), when present in the

278 Herpes simplex virus type 1 and Alzheimer's disease: pos

279 athway are associated with susceptibility to herpes simplex virus type 1 encephalitis (HSE).

280 Recent infection with, or reactivation of, herpes simplex virus type 1 or type 1/2 unspecified, cyt

281 The herpes simplex virus type 1 thymidine kinase (HSV1-tk) g

282 CMV, herpes simplex virus type 1, and human herpesvirus 6 inf

283 e association of 4 human herpesviruses (CMV, herpes simplex virus type 1, human herpesvirus type 6, a

284 fumarate (TDF) has in vitro activity against herpes simplex virus type 2 (HSV-2) and reduced HSV-2 ac

285 une responses to chronic infections, such as herpes simplex virus type 2 (HSV-2) in HIV/HSV-coinfecte

286 f infection were seen in smokers, those with herpes simplex virus type 2 (HSV-2) infection, men who h

287 n to be at least tripled in individuals with herpes simplex virus type 2 (HSV-2) infection.

288 HIV and herpes simplex virus type 2 (HSV-2) infections cause a s

289 Genital infection with herpes simplex virus type 2 (HSV-2) is common and increa

290 This study investigated herpes simplex virus type 2 (HSV-2) seroprevalence utili

291 Herpes simplex virus type 2 (HSV-2; herpes) exacerbates

292 against cervical cancer, cervical dysplasia, herpes simplex virus type 2, chlamydia, and syphilis.

293 Interestingly, cells infected with herpes simplex virus type-1 (HSV-1) incorporated EdC and

294 Herpes simplex virus type-1 (HSV-1), one of the most wid

295 CB) women, including bacterial vaginosis and herpes simplex virus type-2 (HSV-2) infection.

296 ded controls for sociodemographic variables, herpes simplex virus type-2 status, and recreational dru

297 rr virus (EBV), human herpesvirus 6 (HHV-6), herpes simplex virus types 1 (HSV-1) and 2 (HSV-2), and

298 ed by the development of acyclovir-resistant herpes simplex virus viremia, primary graft failure, and

299 al response to HCMV, but not Epstein-Barr or herpes simplex virus, was associated with increased risk

300 lium, Trichomonas vaginalis, adenovirus, and herpes simplex virus were absent.