ライフサイエンスコーパス: herpesvirus

1 ate responses are defective in controlling a herpesvirus.

2 tomegalovirus or Kaposi's sarcoma-associated herpesvirus.

3 n-Barr virus and Kaposi's sarcoma-associated herpesvirus.

4 gomyelinase playing a role in the entry of a herpesvirus.

5 n-Barr virus and Kaposi's sarcoma-associated herpesvirus.

6 c state investigated is infection by a human herpesvirus.

7 hanism appears to be common to several human herpesviruses.

8 tegument proteins compared with other human herpesviruses.

9 FS in TRs are positionally conserved between herpesviruses.

10 t is known to serve as a portal of entry for herpesviruses.

11 usion protein gB, which are conserved in all herpesviruses.

12 ely contributing to immunodeficiency against herpesviruses.

13 tribute to immune control of oncogenic gamma-herpesviruses.

14 d controlled trials of interventions against herpesviruses.

15 a key transition point in the life cycle of herpesviruses.

16 APOBEC3 evasion by large double-stranded DNA herpesviruses.

17 tunity for recombination between coinfecting herpesviruses.

18 tive for treating infections caused by these herpesviruses.

19 ns detected in nonpleocytic CSF samples were herpesviruses.

20 ganism for studying common properties of all herpesviruses.

21 pulations of DNA viruses and, in particular, herpesviruses.

22 on, transcription, and genome maintenance of herpesviruses.

23 teroid dexamethasone (DEX) stimulates bovine herpesvirus 1 (BoHV-1) and herpes simplex virus 1 (HSV-1

24 rpesvirinae subfamily members such as bovine herpesvirus 1 (BoHV-1) and herpes simplex virus 1 (HSV-1

25 Bovine herpesvirus 1 (BoHV-1) is an alphaherpesvirus that cause

26 ce of reproductive failure.IMPORTANCE Bovine herpesvirus 1 (BoHV-1) is the most frequently diagnosed

27 An important site for bovine herpesvirus 1 (BoHV-1) latency is sensory neurons within

28 ollowing stressful stimuli.IMPORTANCE Bovine herpesvirus 1 (BoHV-1), an important bovine pathogen, es

29 Equid herpesvirus 1 (EHV-1) is a viral pathogen of horse popul

30 tective against the diseases caused by equid herpesvirus 1 (EHV-1), especially the neurologic form.

31 Equine herpesvirus 1 (EHV1) replicates in the respiratory epith

32 Variants of the Ostreid herpesvirus 1 (OsHV-1) cause high losses of Pacific oyst

33 ntative polymerase chain reaction (qPCR) for herpesvirus 1-8 DNA in bile, blood and liver tissue of 7

34 n confined to one gammaherpesvirus (dasyurid herpesvirus 2 [DaHV-2]), for which captivity was identif

35 ricella-zoster virus (VZV; also called human herpesvirus 3 [HHV3]), the human alphaherpesvirus causin

36 otella spp., Streptococcus mutans, and Human herpesvirus 4 (Epstein-Barr virus [EBV]) were more preva

37 aherpesvirus Epstein-Barr virus (EBV) (human herpesvirus 4 [HHV4]) infects most adults and is an impo

38 is a tumor disease associated with chelonid herpesvirus 5 (ChHV5) that is an important cause of mort

39 ease associated with a herpesvirus (chelonid herpesvirus 5 [ChHV5]) that affects mainly green turtles

40 inct strains of human cytomegalovirus (human herpesvirus 5) and show that gene drive viruses can effi

41 ts detected were enterovirus (n = 38), human herpesvirus 6 (HHV-6) (n = 30), and Streptococcus pneumo

42 Human herpesvirus 6 (HHV-6) and cytomegalovirus (CMV) are popu

43 cerning the role of herpesviruses, and human herpesvirus 6 (HHV-6) in particular, in AD.

44 Human herpesvirus 6 (HHV-6) is an important cause of meningiti

45 Here, we show that human herpesvirus 6 (HHV-6, A or B) RNA was detected in 6.1% o

46 galovirus (CMV), Epstein-Bar virus and human herpesvirus 6 (HHV6), are known to infect neurons.

47 Murine roseolovirus, a homolog of human herpesvirus 6, can also be reactivated in the lung and o

48 nd conserved features in the genome of human herpesvirus 6.

49 with infectious causes (2 influenza; 2 human herpesvirus 6; 2 group B Streptococcus; 2 Streptococcus

50 r virus 6% in saliva and 3% in GCF; of human herpesvirus-6 (HHV-6) 6% in saliva and 2% in GCF; and HH

51 Human herpesvirus-6 (HHV-6) A and B are ubiquitous betaherpesv

52 Human herpesvirus 6A and 6B (HHV-6A and HHV-6B) are closely re

53 f people worldwide carry a copy of the human herpesvirus 6A or 6B (HHV-6A/B) in every cell of their b

54 as inherited chromosomally integrated human herpesvirus 6A/B (iciHHV-6A/B).

55 Human herpesvirus 6B (HHV-6B) belongs to the beta-herpesvirus

56 Human herpesvirus 6B (HHV-6B) DNA is frequently detected in hu

57 Human herpesvirus 6B (HHV-6B) frequently reactivates after all

58 analyses for HCT recipients.IMPORTANCE Human herpesvirus 6B (HHV-6B) is a DNA virus that infects most

59 +) T cells were enriched with DNA from human herpesvirus 6b.

60 athogens reported included bunyavirus, human herpesvirus 7, and enterovirus D-68, ultimately impactin

61 The human herpesvirus-7 (HHV-7) U21 glycoprotein binds to class I

62 Human herpesvirus 8 (HHV-8) encodes four viral interferon regu

63 tion through reactivation of recipient human herpesvirus 8 (HHV-8) infection or through donor-derived

64 Human herpesvirus 8 (HHV-8) is an oncogenic virus causally rel

65 Human herpesvirus 8 (HHV-8) viral interleukin-6 (vIL-6) is a c

66 Human herpesvirus 8 (HHV-8) viral interleukin-6 (vIL-6) locali

67 analyses of vIL-6 activity.IMPORTANCE Human herpesvirus 8 (HHV-8)-encoded viral interleukin-6 (vIL-6

68 sarcoma-associated herpesvirus (KSHV; human herpesvirus 8) is an oncogenic gammaherpesvirus that is

69 -8 latent and lytic biology.IMPORTANCE Human herpesvirus 8-encoded IRF homologues were the first to b

70 aposi's sarcoma-associated herpesvirus/human herpesvirus 8.

71 Herpesviruses acquire their membrane envelopes in the cy

72 The literature on the egress of different herpesviruses after secondary envelopment is contradicto

73 by its ability to restrict a broad range of herpesviruses and its profound upregulation during herpe

74 ected subjects, such as impaired immunity to herpesviruses and tumor surveillance.

75 of the viral protein kinase conserved across herpesviruses and two cellular proteins, ATM and KAP1, a

76 association between CMV (and possibly other herpesviruses) and HIV persistence are unclear.

77 gs of host-species interactions of poxvirus, herpesvirus, and influenza virus proteins, we propose a

78 odified to inhibit EBV and potentially other herpesviruses, and (ii) be developed into anticancer age

79 e tailed double-stranded DNA (dsDNA) phages, herpesviruses, and adenoviruses and, as such, is a viabl

80 d DNA bacteriophages, some archaeal viruses, herpesviruses, and adenoviruses.

81 al viruses, including tailed bacteriophages, herpesviruses, and archaeal viruses.

82 ve renewed the debate concerning the role of herpesviruses, and human herpesvirus 6 (HHV-6) in partic

83 ominated by papillomaviruses, anelloviruses, herpesviruses, and parvoviruses.

84 It is not known whether other herpesviruses are also implicated, nor whether a dose-re

85 roteins in tailed bacteriophages, with which herpesviruses are believed to share a common ancestry.

86 Viral tropism and transmission of herpesviruses are best studied in their natural host for

87 Beta-herpesviruses are common opportunistic pathogens that ca

88 Since herpesviruses are experts at coopting cellular functions

89 These herpesviruses are strictly host specific, and therefore

90 Herpesviruses are ubiquitous human pathogens that cause

91 Herpesviruses are ubiquitous, and infection by some, lik

92 Herpesviruses are ubiquitous, double-stranded DNA, envel

93 of pUL7 and pUL51 is conserved across human herpesviruses, as is their association with trans-Golgi

94 C is a uniquely useful model to study common herpesvirus assembly pathways and cell-specific pathways

95 role of the conserved pUL7:pUL51 complex in herpesvirus assembly.

96 rylates RUNX2, recruiting the deubiquitinase herpesvirus-associated ubiquitin-specific protease (HAUS

97 been studied in dsDNA phages(6-9)-with which herpesviruses bear some similarities-a lack of high-reso

98 the prominent role of this mechanism in both herpesvirus biology and cancer, our screening assay may

99 Herpesviruses can rewire cellular signaling in host cell

100 Cytomegalovirus (CMV) is a herpesvirus capable of causing severe disease in neonate

101 DNA pressure of tens of atmospheres inside a herpesvirus capsid powers ejection of the viral genome i

102 Here we describe the first structure of herpesvirus capsids determined by sub-tomogram averaging

103 portal proteins of tailed bacteriophage and Herpesvirus capsids form dodecameric rings that occupy o

104 uman cytomegalovirus (HCMV) is a common beta-herpesvirus causing life-long latent infections.

105 is (FP) is a tumor disease associated with a herpesvirus (chelonid herpesvirus 5 [ChHV5]) that affect

106 ct infection and pathogenesis.IMPORTANCE How herpesviruses circumvent mucosal defenses to promote inf

107 significantly more enriched among genomes of herpesviruses compared to those of nonherpesviruses.

108 port the X-ray structures of beta- and gamma-herpesvirus core NECs obtained through an innovative rec

109 n imaging of individual virions of the human herpesvirus cytomegalovirus (CMV) showed that virion-to-

110 By simulating infection of the human herpesvirus, cytomegalovirus, we hypothesize that virus-

111 Of the human herpesviruses, cytomegalovirus (HCMV) and Epstein-Barr v

112 Studies with porcine herpesvirus demonstrated that primary enveloped particle

113 the cross-reactivity of antibodies to other herpesviruses, differences in viral epitopes, or differe

114 Evidence of HR between coinfecting herpesvirus DNA genomes can be found frequently both in

115 The exploitation of host innate defenses by herpesviruses during the early phase of host colonizatio

116 e core fusion machinery is conserved for all herpesviruses, each species uses distinct receptors and

117 Elephant endotheliotropic herpesvirus (EEHV) can cause lethal hemorrhagic disease

118 hs associated with elephant endotheliotropic herpesvirus (EEHV) infection result from primary infecti

119 As such, herpesviruses encode multiple viral proteins that antago

120 this Review, we discuss recent insights into herpesvirus entry by analysing the structures of entry g

121 microscopy studies have refined our model of herpesvirus entry into cells, clarifying both the conser

122 Previously, we reported that the absence of herpesvirus entry mediator (HVEM) decreases latency but

123 sis factor receptor superfamily member HVEM (herpesvirus entry mediator), which binds to the various

124 o this partially permissive phenotype of the herpesvirus Epstein-Barr virus (EBV) indicate that upon

125 enveloped viruses, examines what is known of herpesvirus ESCRT utilization in the nucleus and cytopla

126 Many viruses, including herpesviruses, express RNases that reduce host gene expr

127 gL, are the core functional proteins of the herpesvirus fusion complex.

128 ant from the gB fusion loops is critical for herpesvirus fusion, revealing a potential new target for

129 requires multiple functions: maintaining the herpesvirus genome in the nuclei of cells; partitioning

130 t a novel role for RUNX1 in directly binding herpesvirus genome, silencing the transcription of numer

131 CTCF and cohesin also bind to herpesvirus genomes at specific sites and regulate viral

132 es revealed unusually high RLFS densities in herpesvirus genomes, with RLFS densities particularly en

133 ne cytomegalovirus (MCMV), a prototypic beta-herpesvirus, harnesses the UPR to regulate its own life

134 Like all herpesviruses, HCMV infection is for life.

135 velopment during assembly of the neurotropic herpesviruses herpes simplex virus 1 (HSV-1) and pseudor

136 Structural studies of the prototypical herpesviruses herpes simplex virus 1 (HSV-1), HSV-2, hum

137 Like all the herpesviruses, herpes simplex virus encodes machinery th

138 a rare vascular tumor associated with human herpesvirus (HHV)-8 infection.

139 The Kaposi's sarcoma-associated herpesvirus homologue, ORF61, also bound APOBEC3B and me

140 The herpesvirus human cytomegalovirus (HCMV) is a leading ca

141 tein complexes throughout infection with the herpesvirus, human cytomegalovirus (HCMV).

142 d persistence of Kaposi's sarcoma-associated herpesvirus/human herpesvirus 8.

143 NKT cell subsets iNKT1, iNKT2, and iNKT17 in herpesvirus immunity remains to be fully elucidated.

144 o evaluate the prevalence and correlation of herpesviruses in bile, blood and liver tissue and to inv

145 orting the roles of the JAK-STAT pathway and herpesviruses in mediating the adverse drug reaction.

146 uating the periodontal disease and detecting herpesviruses in patients with CKD as the inflammatory p

147 s study was to analyse the presence of human herpesviruses in saliva and gingival crevicular fluid (G

148 n reminiscent of most DNA viruses.IMPORTANCE Herpesviruses infect nearly all humans and persist quies

149 Herpesviruses infect virtually all humans and establish

150 viruses infecting bacteria and archaea, and herpesviruses infecting animals and humans, where naked

151 Early during ocular herpesvirus infection (i.e., day 2), the gamma interfero

152 We found that early during ocular herpesvirus infection (i.e., on day 2 postinfection), IF

153 cell subsets using the mouse model of ocular herpesvirus infection and disease.

154 S impaired interferon response during murine herpesvirus infection and that the inhibition occurred d

155 viruses and its profound upregulation during herpesvirus infection as part of a germline-specific tra

156 of the innate immune network during chronic herpesvirus infection remains poorly defined.

157 he findings suggest that early during ocular herpesvirus infection, cornea-resident IFN-gamma-produci

158 During herpesvirus infection, egress of nascent viral capsids f

159 of iNKT cell subsets in asymptomatic ocular herpesvirus infection.

160 nding of cellular factors that contribute to herpesvirus infection.

161 preformed capsids is a critical step during herpesvirus infection.

162 data will be important for future studies of herpesvirus infection.

163 Thus, endogenous HSATII RNA synthesis after herpesvirus infections appears to have functionally impo

164 as well as with increased susceptibility to herpesvirus infections.

165 p us better understand the complexity behind herpesvirus infections.

166 ed with symptomatic and asymptomatic corneal herpesvirus infections.

167 is known to play a protective role in other herpesvirus infections; yet, ADCC has never been investi

168 KSHV, like other herpesviruses, intermittently reactivates from latency a

169 .IMPORTANCE Kaposi's sarcoma (KS)-associated herpesvirus is the causative agent of multiple malignanc

170 packaging motor of tailed bacteriophages and herpesviruses is a powerful nanomachine built by several

171 recruiting p53 to Zp.IMPORTANCE EBV, a human herpesvirus, is latently present in most nasopharyngeal

172 double-stranded DNA viruses, from phages to herpesviruses, is strongly conserved.

173 Like other herpesviruses, it has latent and lytic repertoires.

174 us (KSHV) are two of the classical oncogenic herpesviruses known to induce the oncogenic phenotype.

175 The Kaposi's sarcoma-associated herpesvirus (KSHV) alkaline exonuclease SOX, encoded by

176 Kaposi's sarcoma-associated herpesvirus (KSHV) and Epstein-Barr Virus (EBV) establis

177 R-M4, encoded by Kaposi's sarcoma-associated herpesvirus (KSHV) and Marek's disease virus (MDV), resp

178 n controlling Kaposi sarcoma (KS)-associated herpesvirus (KSHV) and preventing disease development, t

179 Both Kaposi's sarcoma-associated herpesvirus (KSHV) and the closely related rhesus macaqu

180 gammaherpesvirus Kaposi's sarcoma-associated herpesvirus (KSHV) and the more distantly related alphah

181 virus (EBV) and Kaposi's sarcoma-associated herpesvirus (KSHV) are human gammaherpesviruses and are

182 virus (EBV) and Kaposi's sarcoma-associated herpesvirus (KSHV) are two of the classical oncogenic he

183 Assembly of Kaposi's sarcoma-associated herpesvirus (KSHV) begins at a bacteriophage-like portal

184 s of the host in Kaposi's sarcoma-associated herpesvirus (KSHV) biology helped discover that KSHV inf

185 er the oncogenic Kaposi's sarcoma-associated herpesvirus (KSHV) deregulates the activity of APC/C dur

186 Detectable Kaposi's sarcoma-associated herpesvirus (KSHV) DNA in blood and increased antibody t

187 Reactivation of Kaposi's sarcoma-associated herpesvirus (KSHV) from latency requires the viral trans

188 pstein-Barr virus (EBV) and Kaposi's sarcoma herpesvirus (KSHV) human DNA tumor viruses.

189 sarcoma is a tumor caused by Kaposi sarcoma herpesvirus (KSHV) infection and is thought to originate

190 hat it restricts Kaposi's sarcoma-associated herpesvirus (KSHV) infection.

191 sarcoma who met criteria for Kaposi sarcoma herpesvirus (KSHV) inflammatory cytokine syndrome.

192 sed by oncogenic Kaposi's sarcoma-associated herpesvirus (KSHV) is a highly angiogenic and invasive v

193 Kaposi's sarcoma-associated herpesvirus (KSHV) is a human oncogenic nuclear DNA viru

194 Kaposi's sarcoma-associated herpesvirus (KSHV) is a human oncogenic virus, which mai

195 Kaposi's sarcoma-associated herpesvirus (KSHV) is a human oncogenic virus.

196 Kaposi sarcoma-associated herpesvirus (KSHV) is an emerging pathogen and is the ca

197 ation.IMPORTANCE Kaposi's sarcoma-associated herpesvirus (KSHV) is an oncogenic gammaherpesvirus that

198 ature.IMPORTANCE Kaposi's sarcoma-associated herpesvirus (KSHV) is associated with a substantial dise

199 lance.IMPORTANCE Kaposi's sarcoma-associated herpesvirus (KSHV) is associated with several cancers, i

200 enses.IMPORTANCE Kaposi's sarcoma-associated herpesvirus (KSHV) is causally linked to Kaposi's sarcom

201 Kaposi's sarcoma-associated herpesvirus (KSHV) is etiologically associated with endo

202 Kaposi sarcoma-associated herpesvirus (KSHV) is necessary but not sufficient for p

203 Kaposi's sarcoma-associated herpesvirus (KSHV) is the causal agent for Kaposi's sarc

204 avity.IMPORTANCE Kaposi's sarcoma-associated herpesvirus (KSHV) is the causal agent for Kaposi's sarc

205 Kaposi's sarcoma-associated herpesvirus (KSHV) is the causative agent for Kaposi sar

206 Kaposi's sarcoma-associated herpesvirus (KSHV) is the causative agent of Kaposi's sa

207 Kaposi's sarcoma-associated herpesvirus (KSHV) is the causative agent of two B-cell

208 Kaposi's sarcoma-associated herpesvirus (KSHV) is the etiologic agent of Kaposi's sa

209 Kaposi's sarcoma-associated herpesvirus (KSHV) is the etiologic agent of Kaposi's sa

210 The DNA virus Kaposi's sarcoma-associated herpesvirus (KSHV) is the etiological agent of Kaposi's

211 thway.IMPORTANCE Kaposi's sarcoma-associated herpesvirus (KSHV) is the etiological agent of three hum

212 Kaposi's sarcoma (KS)-associated herpesvirus (KSHV) is tightly linked with KS, primary ef

213 ant roles in the Kaposi's sarcoma-associated herpesvirus (KSHV) latent and lytic gene replication.

214 ells, can induce Kaposi's sarcoma-associated herpesvirus (KSHV) lytic replication and directly activa

215 sions.IMPORTANCE Kaposi's sarcoma-associated herpesvirus (KSHV) manipulates several cellular pathways

216 ar mechanisms of Kaposi's sarcoma-associated herpesvirus (KSHV) reactivation have been studied primar

217 Kaposi sarcoma (KS)-associated herpesvirus (KSHV) subtype depends mostly on patient ori

218 Kaposi's sarcoma-associated herpesvirus (KSHV) transcribes a long noncoding polyaden

219 Kaposi's sarcoma-associated herpesvirus (KSHV) transcription is regulated by CTCF an

220 virus (EBV) and Kaposi's sarcoma-associated herpesvirus (KSHV) utilize chromatin insulators to order

221 wever, the role of Kaposi sarcoma-associated herpesvirus (KSHV), also endemic in Africa, has not been

222 arr virus (EBV), Kaposi's sarcoma-associated herpesvirus (KSHV), and human papillomavirus (HPV) share

223 rus (EBV), human Kaposi's sarcoma-associated herpesvirus (KSHV), and murine gammaherpesvirus 68 (MHV6

224 gammaherpesvirus Kaposi's sarcoma-associated herpesvirus (KSHV), and the closely related rhesus macaq

225 Kaposi sarcoma-associated herpesvirus (KSHV), one of the human oncogenic viruses,

226 The ability for Kaposi's sarcoma-associated herpesvirus (KSHV), the causative agent of Kaposi's sarc

227 ood infection with Kaposi sarcoma-associated herpesvirus (KSHV), the maternal immune response remains

228 In Kaposi's sarcoma-associated herpesvirus (KSHV), these vTAs are encoded by ORF18, ORF

229 we show that the Kaposi's sarcoma-associated herpesvirus (KSHV)-encoded LANA protein enhances the ubi

230 The Kaposi's sarcoma-associated herpesvirus (KSHV)-encoded latency-associated nuclear an

231 ddicted cancers, Kaposi's sarcoma-associated herpesvirus (KSHV)-transformed cells depend on glutamine

232 Bcl-2 promoter of Kaposi sarcoma-associated herpesvirus (KSHV).

233 ma infected with Kaposi's sarcoma-associated herpesvirus (KSHV).

234 infection with the Kaposi sarcoma-associated herpesvirus (KSHV).

235 re associated with Kaposi sarcoma-associated herpesvirus (KSHV).

236 s and is caused by Kaposi sarcoma-associated herpesvirus (KSHV).

237 oters.IMPORTANCE Kaposi's sarcoma-associated herpesvirus (KSHV; human herpesvirus 8) is an oncogenic

238 In most cases, herpesviruses lacking CHPK can propagate in cell culture

239 in structures of the dsDNA tailed phages and herpesviruses make phages ideal models to understand cap

240 ne responses to common coinfections, such as herpesviruses, may sustain HIV tissue reservoirs during

241 inst all enveloped viruses tested, including herpesviruses, Measles virus, influenza, and SARS-CoV-2.

242 latency to the lytic phase is necessary for herpesvirus-mediated pathology as well as viral spread a

243 reactivation.IMPORTANCE The reactivation of herpesviruses, most commonly varicella-zoster virus (VZV

244 factors required for infection with the beta-herpesvirus murine cytomegalovirus (MCMV).

245 Herpesviruses must amplify their DNA to load viral parti

246 RISPR-based screen harnessed the capacity of herpesvirus mutants that trigger antiviral necroptotic c

247 severe disease outcomes, abortion and equine herpesvirus myeloencephalopathy (EHM).

248 differ considerably in these beta- and gamma-herpesvirus NECs, the binding free energy contributions

249 Herpesvirus of turkeys (HVT) is a nonpathogenic alphaher

250 5% sequence similarity to the chicken Nr-13, herpesvirus of turkeys (HVT) vNr-13, encoded by the HVT0

251 hways for membrane reorganization.IMPORTANCE Herpesvirus particles are complex and contain many diffe

252 There was histologic evidence of acute herpesvirus pathology, including acantholysis in the squ

253 Among 1039 viral infections, herpesviruses predominated (51%) in kidney, liver, and h

254 IMPORTANCE Human cytomegalovirus (HCMV) is a herpesvirus present in up to 85% of some populations.

255 r virus (EBV) lytic phase, like those of all herpesviruses, proceeds via an orderly cascade that inte

256 any conserved genes, including the conserved herpesvirus protein kinase (CHPK) that has multifunction

257 Thus, although a large number of herpesvirus proteins have been assigned roles in envelop

258 tiorgan inflammatory disease associated with herpesvirus reactivation and subsequent onset of autoimm

259 n of some viruses, little is known about how herpesviruses regulate mitochondrial homeostasis during

260 ng a reliable natural host in order to study herpesvirus replication and pathogenesis in animals.

261 e novel mechanisms provide new insights into herpesvirus respiratory tract infection and pathogenesis

262 re human (and/or mammalian) hosts, including herpesviruses, retroviruses, Mycobacterium tuberculosis,

263 cteriophages, some archaeal viruses, and the herpesviruses share a structural motif, the HK97 fold.

264 implex virus type 1 (HSV1), the prototypical herpesvirus species.

265 l for cell culture-based replication of most herpesviruses studied.

266 herpesvirus 6B (HHV-6B) belongs to the beta-herpesvirus subfamily of the Herpesviridae.

267 pUL47 may have a similar function in human herpesviruses such as varicella-zoster virus or herpes s

268 Core stabilising structures are conserved in herpesviruses suggesting their ancestral origin.

269 We found homologs to herpesvirus surface glycoprotein gB in cytoplasmic virus

270 Epstein-Barr virus (EBV) is a human herpesvirus that can cause lymphomas, epithelial cancers

271 MDV is a deadly and contagious herpesvirus that can kill infected animals in less than

272 r virus (VZV) is a medically important human herpesvirus that causes chickenpox and shingles, but its

273 Human cytomegalovirus (HCMV) is a ubiquitous herpesvirus that causes disease in immunosuppressed popu

274 Barr virus (EBV) is a ubiquitous human gamma-herpesvirus that establishes life-long infection and inc

275 Cytomegalovirus (CMV) is a beta-herpesvirus that establishes lifelong latency in infecte

276 Human cytomegalovirus (HCMV) is a beta-herpesvirus that has co-evolved with the host immune sys

277 ression.IMPORTANCE MDV is a potent oncogenic herpesvirus that induces T-cell lymphoma in infected chi

278 n cytomegalovirus (HCMV) is an opportunistic herpesvirus that is asymptomatic for healthy individuals

279 Human cytomegalovirus (HCMV) is a large DNA herpesvirus that is highly prevalent in the human popula

280 IMPORTANCE Human cytomegalovirus (HCMV) is a herpesvirus that leads to serious health consequences in

281 -Barr virus, one of the most prevalent human herpesviruses that also causes cancer, we have discovere

282 response.IMPORTANCE HHV-6A and -6B are human herpesviruses that have the unique property of being abl

283 pstein-Barr virus (EBV) is one of nine human herpesviruses that persist latently to establish permane

284 alysis yields targets for the design of anti-herpesvirus therapeutic strategies across all alphaherpe

285 Curiously, some viruses, including herpesviruses, thrive despite the induction of ROS, sugg

286 nd viral proteins that could link enveloping herpesviruses to cellular ESCRT components.

287 suggests this strategy may be used by other herpesviruses to reinforce latency in a cell-specific ma

288 Equine herpesvirus type 1 (EHV-1) outbreaks continue to occur d

289 We used the alphaherpesvirus equine herpesvirus type 1 (EHV1) and equine respiratory tissues

290 trate how a central alphaherpesvirus, equine herpesvirus type 1 (EHV1), actually exploits beta-defens

291 y Epstein-Barr virus (EBV) positive or human herpesvirus type-8 (HHV-8) positive.

292 Herpesviruses uniquely express two essential nuclear egr

293 he hexon-capping SCP-the largest among human herpesviruses-uses its N-terminal half to bridge hexon M

294 The herpesviruses varicella-zoster virus (VZV) and human cyt

295 y studied vCD200 molecule, however, the only herpesvirus vCD200 molecule to be examined in vivo is th

296 ility in protein abundance within individual herpesvirus virion particles enables probabilistic bet h

297 l systems has precluded understanding of how herpesvirus virions overcome the abundant mucosal beta-d

298 stantially smaller than those of other human herpesviruses, VZV has a similarly sized capsid, consist

299 Compared to blood donors, human herpesviruses were more prevalent in all MSM groups (P <

300 ction are frequently coinfected with chronic herpesviruses, which periodically replicate and produce