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1  knockout mutant and analyzing the resulting heterokaryon.
2 self-fertile strain, which was shown to be a heterokaryon.
3 fusion with a different individual to form a heterokaryon.
4 het locus (11 in N. crassa) to form a stable heterokaryon.
5 thin the common cytoplasm of a multinucleate heterokaryon.
6 on in normal matings and in the self-fertile heterokaryon.
7 ons, while those of the same VCG form viable heterokaryons.
8  nucleus and shuttle between nuclei in yeast heterokaryons.
9 ls to initiate somatic cell reprogramming in heterokaryons.
10 al fusion with different individuals to form heterokaryons.
11 oration within the somatic partner nuclei in heterokaryons.
12 ramming of somatic cells in experimental ESC-heterokaryons.
13 ith ~0.4% of Purkinje cells being binucleate heterokaryons.
14 FTR channels, in control NHBEs and hMSC/NHBE heterokaryons.
15 BEs) and observed the formation of hMSC/NHBE heterokaryons.
16 s a role in the formation of Purkinje neuron heterokaryons.
17 ns resulting in the formation of binucleated heterokaryons.
18 r cells after fusion in stable multinucleate heterokaryons.
19 n endothelial cells in stable, non-dividing, heterokaryons.
20  assembly in human cells and in human-simian heterokaryons.
21 uman and mouse transcripts in these chimeric heterokaryons.
22 elic expression of selected Xi-genes by many heterokaryons (30-50%).
23 omorphs (a and A), resulting in self-fertile heterokaryons (a type of sexual reproduction termed pseu
24 t for heterokaryon), they cannot form stable heterokaryons after vegetative fusion.
25 dicate that CpRbp1 is an essential gene, and heterokaryons allowed the fungus to maintain lethal CpRb
26                            Neuroblast:glioma heterokaryon analyses revealed that loss of neurovirulen
27 Nucleocytoplasmic shutting was determined by heterokaryon analyses.
28               Both ATP depletion studies and heterokaryon analysis demonstrated that TRbeta rapidly s
29                                Neuron-glioma heterokaryon analysis implicates neuronal trans-dominant
30                                              Heterokaryon analysis indicates that Cca1p is a nucleus/
31                                    Combining heterokaryon analysis with fluorescence in situ hybridiz
32                             Using a modified heterokaryon analysis, we have localized the Vpr nuclear
33 tly reprogram lymphocytes and fibroblasts in heterokaryon and hybrid assays.
34                                              Heterokaryon and in situ hybridization experiments revea
35                                              Heterokaryon and transformation tests showed that nonsel
36 t the fact that matings can produce unstable heterokaryons and disrupt the pseudohomothallic life cyc
37 uman keratinocytes and mouse muscle cells in heterokaryons, and extensive changes are observed within
38 lasmic shuttling activity in an interspecies heterokaryon assay and for the ability to associate with
39         Examination of Mta localization in a heterokaryon assay provided evidence that Mta shuttles b
40 aller than 42 kDa shuttle efficiently in the heterokaryon assay via a crm-1-independent mechanism.
41  not demonstrate detectable shuttling in the heterokaryon assay yet still coactivates EBNA2 similarly
42        Using a series of SENP2 mutants and a heterokaryon assay, we demonstrate that SENP2 shuttles b
43           Furthermore, using an interspecies heterokaryon assay, we found that QKI-5, but not another
44 uclear shuttling of v-Rel in an interspecies heterokaryon assay.
45 ttled between the nucleus and cytoplasm in a heterokaryon assay.
46 her confirmed the functionality of NES2 by a heterokaryon assay.
47                                              Heterokaryon assays confirmed directly that ACF shuttles
48                                     However, heterokaryon assays demonstrated that ZBP-89 retained p5
49                               We carried out heterokaryon assays using cells that endogenously produc
50 RM1-specific inhibitor leptomycin B (LMB) in heterokaryon assays, suggesting a role for an export rec
51 0 is unable to shuttle out of the nucleus in heterokaryon assays, suggesting the existence of specifi
52 -1 was cloned by a novel procedure employing heterokaryon-assisted transformation and ligation-mediat
53                                 In addition, heterokaryons at the MAT locus were observed in field is
54                                  Analysis of heterokaryons between CsA-dependent HeLa-P4 cells and Cs
55                               Interestingly, heterokaryons between primary monocytes and a human embr
56 y macrophages and macrophage-epithelial cell heterokaryons, but not epithelial cell lines.
57                                              Heterokaryon cells generated by fusion of HeLa and permi
58 s round 1 genomic exclusion, resulted in two heterokaryon clones of different mating types which have
59                             These rad51 null heterokaryons complete all of the early and middle stage
60 erozygous loci to homozygosity, resulting in heterokaryons containing highly diverse populations of d
61                                           In heterokaryons containing Mdm2 and p19(ARF), the longer t
62 afficking was analyzed by using interspecies heterokaryons containing nuclei from infected and uninfe
63                                          The heterokaryons created ectopic ventricular pacemaker acti
64 nalysis of LPS-induced NF-kappaB activity in heterokaryons derived from polyethylene glycol-fused cel
65                                        These heterokaryons display a T-cell-like phenotype with respe
66 ed somatic cells were poorly reprogrammed in heterokaryons, due in part to defective DNA replication.
67 leocytoplasmic shuttling was demonstrated by heterokaryon experiments and energy-dependent blockade o
68 ion maintenance 1 (CRM1) overexpression, and heterokaryon experiments indicate that Bright actively s
69                                              Heterokaryon experiments revealed that v-ErbA did not un
70                                      Using a heterokaryon export assay in transfected cultured cells,
71                                As assayed in heterokaryons, export of SLBP from the nucleus is depend
72                                          The heterokaryons expressed several proteins characteristic
73                                              Heterokaryons expressed the alpha- and gamma-ENaC subuni
74 man Jurkat T leukemia cells and formation of heterokaryons failed to result in a complementation of t
75 compatibility, minimizing the possibility of heterokaryon formation and mitotic recombination.
76                                Within 4 h of heterokaryon formation and with protein synthesis inhibi
77 , het genes play crucial roles by regulating heterokaryon formation between different individuals.
78                              However, viable heterokaryon formation between individuals of the same V
79                         The observation that heterokaryon formation in the cerebellum occurs as part
80 e, we have explored Purkinje cell fusion and heterokaryon formation in the human brain and the influe
81 1 does not greatly affect vegetative growth, heterokaryon formation or male fertility in either matin
82                           We have shown that heterokaryon formation takes place in control subjects,
83 ween nucleus and cytoplasm was also shown by heterokaryon formation upon cell fusion.
84             Purkinje cells were analysed for heterokaryon formation using immunohistochemistry techni
85 o tightly linked genes in barrage formation, heterokaryon formation, and asymmetric, gene-specific in
86 related increase in Purkinje cell fusion and heterokaryon formation.
87 ransplanted BMDCs predominantly derives from heterokaryon formation.
88                                BMDC-Purkinje heterokaryons formation may reflect an endogeneous neuro
89 activity of influenza A viral polymerases in heterokaryons formed between avian (DF1) and human (293T
90 ivo experiments examining lysosome fusion in heterokaryons formed between cells containing fluorescen
91                                              Heterokaryons formed by the fusion of anergic murine T c
92  in reprogramming, we generated interspecies heterokaryons (fused mouse embryonic stem (ES) cells and
93 uttle between the nucleus and cytoplasm in a heterokaryon fusion assay, suggesting the presence of nu
94 ttles from the nucleus to the cytoplasm in a heterokaryon fusion assay.
95 tep gene replacement and by a new procedure, heterokaryon gene replacement.
96 e transcription products, and is dominant in heterokaryons generated by fusion of permissive and nonp
97 fungal individuals are unable to form viable heterokaryons if they differ in allelic specificity at a
98                                   In mitotic heterokaryons, INCENP was detected in association with s
99                                              Heterokaryon incompatability in vegetatively growing fun
100 e growth in filamentous fungi is mediated by heterokaryon incompatibility (het) loci.
101 ns is dependent upon genetic constitution at heterokaryon incompatibility (het) loci.
102 nserved programmed cell death pathway called heterokaryon incompatibility (HI).
103 n filamentous ascomycete fungi and is termed heterokaryon incompatibility (HI).
104 s required for het-c nonself recognition and heterokaryon incompatibility (HI).
105  seemed plausible that N. tetrasperma avoids heterokaryon incompatibility by maintaining compatible a
106  prion-driven neurodegenerative diseases and heterokaryon incompatibility in fungi, is discussed.
107  is conferred by genetic differences at het (heterokaryon incompatibility) loci.
108 ses the question of how this organism avoids heterokaryon incompatibility.
109 spora anserina, and is involved in mediating heterokaryon incompatibility.
110 in a self/nonself recognition process called heterokaryon incompatibility.
111  the evolved biological function of HET-s in heterokaryon incompatibility.
112  self/non-self recognition phenomenon called heterokaryon incompatibility.
113  gene products of Podospora anserina trigger heterokaryon incompatibility.
114 egulation, inducibility of other prions, and heterokaryon incompatibility.
115               However, the viability of such heterokaryons is dependent upon genetic constitution at
116 reprogramming towards pluripotency in single heterokaryons is initiated without cell division or DNA
117    We conclude that nuclear reprogramming in heterokaryons is rapid, extensive, bidirectional, and di
118     The advantages of asexual persistence of heterokaryons may have been one of the drivers of select
119 ted with Crm1 and showed nuclear export in a heterokaryon nucleocytoplasmic shuttling assay.
120                                        Since heterokaryons obtained by somatic fusion of an inf1-sile
121 d antigen is reversible by immunization with heterokaryons of dendritic cells and MUC1-positive carci
122       Findings from studies involving forced heterokaryons of opposite mating-type strains show that
123 s well as the subsequent formation of stable heterokaryons, offers a tantalizing potential solution t
124 ort of proteins and RNAs has been studied in heterokaryons or by microinjecting test substrates into
125                                              Heterokaryons or partial diploids that contain het-c all
126 e of proliferation (embryonic stem [ES] cell heterokaryons) or DNA replication (nuclear transfer).
127 In this work, transport experiments based on heterokaryons, photobleaching, and micronucleation demon
128 development in zebrafish and mice as well as heterokaryons point to a role beyond immunology.
129 ed for hyphal elongation and survival of the heterokaryon produced by cell fusion.
130 I(f)-mediated pacemaker activity arises from heterokaryons rather than electrotonic coupling.
131                                       In the heterokaryon rescue technique, gene deletions are carrie
132  are essential and can only be propagated by heterokaryon rescue.
133                                              Heterokaryon shuttling assays with NIH3T3 (mouse) cells
134 otein from the nucleus is greatly reduced in heterokaryon shuttling assays.
135                       Analysis of reciprocal heterokaryons suggested the absence of an inhibitor of v
136      This study demonstrates that our fungal heterokaryon system can be applied effectively to determ
137 e function of SRE was further confirmed by a heterokaryon test and by gene complementation.
138 ele is maintained in spontaneously generated heterokaryons that consist of two genetically distinct t
139 o autologous DC resulted in the formation of heterokaryons that express the CA-125 Ag and DC-derived
140 attern of gene expression in the ESCs of the heterokaryons that recapitulated ontogeny, with early me
141                                              Heterokaryons that silently harbor homozygous recessive
142 genic homokaryotic progeny from the silenced heterokaryons, thereby demonstrating that the presence o
143 at one or more of these loci (termed het for heterokaryon), they cannot form stable heterokaryons aft
144 at Tox1B; (2) the ability of Tox1A- + Tox1B- heterokaryons to complement for T-toxin production; and
145    We show that, in addition to shuttling in heterokaryons, TRalpha shuttles rapidly in an unfused mo
146 n het specificity undergo hyphal fusion, the heterokaryon undergoes a programmed cell death reaction
147                  In trans complementation of heterokaryons using a full-length wild-type allele of th
148        Moreover, in trans complementation of heterokaryons using chimeric structures of the CpRbp1 ge
149 nts showed that the membrane surface area of heterokaryons was similar to that of NHBEs.
150                    By forming HeLa-L929 cell heterokaryons, we demonstrate that HuR shuttles between
151                   By using human-simian cell heterokaryons, we show that the inhibition of assembly i
152      No mononucleate polyploid Purkinje cell heterokaryons were found.
153                                   When these heterokaryons were mated, the exconjugant progeny cells
154 that the vif-deleted virions released by the heterokaryons were noninfectious whereas the wild-type v
155 ase defect in HeLa versus T cells, transient heterokaryons were produced between HeLa cells and the J
156  of the block to HIV-1 assembly, mouse-human heterokaryons were tested for ability to assemble and re
157                               These knockout heterokaryons were used to demonstrate that gene transfe
158 is mobility of Nup2p was also detected using heterokaryons where, unlike nucleoporins, Nup2p was obse
159 ls of different VCGs fail to form productive heterokaryons, while those of the same VCG form viable h
160 mating of gamma-tubulin gene, GTU1, knockout heterokaryons with a GTU1 gene inserted into the MTT1 lo
161 n of human cultured cells to produce mitotic heterokaryons with two spindles fused in a V conformatio
162 protein gp120-gp41, and we asked whether the heterokaryons would release infectious HIV gpt virions.
163 nfined to the nucleus, we created binucleate heterokaryon yeast cells in which one nucleus suffered a

 
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