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1  well as neurons of the respiratory network (heteroreceptors).
2 as a presynaptic inhibitory autoreceptor and heteroreceptor.
3 EpoR and betacR comprise a tissue-protective heteroreceptor.
4  modulate dopamine transmission via mGluR2/3 heteroreceptors.
5 and post-synaptic serotonergic receptors and heteroreceptors.
6                            However, distinct heteroreceptors acting during adulthood are involved in
7 ptor activity without also changing 5-HT(1B) heteroreceptor activity.
8                        In contrast, 5-HT(1A) heteroreceptors affect responses to forced swim stress,
9                         Co-activation of the heteroreceptor also resulted in synergistic increases in
10  aimed to determine the role of alpha(2a)-AR heteroreceptors and BNST G(i)-GPCR signaling in stress-i
11 findings demonstrate a role for alpha(2a)-AR heteroreceptors and BNST G(i)-GPCR signaling in stress-i
12 central 2alpha-adrenergic autoreceptors, and heteroreceptors and is an antagonist of serotonin 5-HT2
13 ibits dual functionality as autoreceptor and heteroreceptor, and this enables H3Rs to modulate the hi
14 e induction of signaling from p185(neu).EGFR heteroreceptor assemblies requires the ectodomain for li
15 r, these data suggest that mGlu7 serves as a heteroreceptor at inhibitory synapses in area CA1 and th
16 one on GABA(B) presynaptic autoreceptors and heteroreceptors because blocking GABA(B) receptors produ
17 t dopamine stimulation by cocaine enhances a heteroreceptor complex formation between dopamine D2 rec
18               We define therein an axonal D1 heteroreceptor component, apparently mediating volume ne
19                                          All heteroreceptor crosslinking DFMT combinations demonstrat
20 nd subsequent crosslinking of both targets ("heteroreceptor crosslinking"), can further enhance the a
21 nstrate in vivo the enhanced efficacy of the heteroreceptor-crosslinking DFMT design versus single-ta
22                    We show that alpha(2a)-AR heteroreceptor deletion disrupts stress-induced reinstat
23 al mice to differentiate autoreceptor versus heteroreceptor effects of 8-OH-DPAT on hypoglossal nerve
24 ur results demonstrate that forebrain 5-HT1B heteroreceptors expressed during an early postnatal peri
25 he cells while enhancing postsynaptic 5-HT1A heteroreceptor expression in nonserotonergic neurons.
26  effect of activation of downstream 5-HT(1a) heteroreceptors from that of autoreceptors.
27 ift in the balance between dopamine auto and heteroreceptor function may contribute to the therapeuti
28                     Furthermore, the GABA(B) heteroreceptor (GABA(B)R), a G(i/o)-coupled G-protein-co
29 rocircuits heterosynaptically target GABA(B) heteroreceptors (GABA(B)R) on glutamate terminals.
30 s in raphe nuclei without affecting 5-HT(1A) heteroreceptors, generating mice with higher (1A-High) o
31 s that expresses the IL-4Ralpha/IL-13Ralpha1 heteroreceptor (HR) and commits only to the myeloid line
32 ve previously shown that ETPs expressing the heteroreceptor (HR) comprising IL-4Ralpha and IL-13Ralph
33  fine tunes signaling through the IL-4/IL-13 heteroreceptor (HR) in early thymic progenitors (ETPs),
34                  The IL-4Ralpha/IL-13Ralpha1 heteroreceptor (HR) serves both IL-4 and IL-13 cytokines
35 13, namely the IL-4Ralpha/IL-13Ralpha1 (13R) heteroreceptor (HR), is compromised and determined wheth
36 hese compartments, m2 receptors appear to be heteroreceptors, i.e., they are associated predominantly
37 g Th2 programs, IL-4 is captured by the IL-4 heteroreceptor (IL-4Ralpha/IL-13Ralpha1) expressed on de
38 s chain associates with IL-4Ralpha to form a heteroreceptor (IL-4Ralpha/IL-13Ralpha1) that marks the
39 othesis that mGluR4a serves as a presynaptic heteroreceptor in the globus pallidus, where it may play
40                                           As heteroreceptors in GABAergic terminals in the same lamin
41 (dorsal raphe nucleus) and local inhibition (heteroreceptors in projection areas).
42 s, it also activates excitatory G(i)-coupled heteroreceptors in the bed nucleus of the stria terminal
43 mechanisms of activation of presynaptic NMDA heteroreceptors in the rat central nervous system.
44 eurons by presynaptic muscarinic and GABA(B) heteroreceptors in the spinal cord probably contributes
45 ) and a 5-HT1A agonist, and dependent on the heteroreceptor interface.
46 cific D2R knock-out mice, we uncover that D2 heteroreceptors located on non-DAergic medium spiny neur
47                             Activation of D2 heteroreceptors, located on STN axon terminals, provides
48 um, the presynaptic GABA-B autoreceptors and heteroreceptors may exhibit distinct receptor-effector c
49 utyric acid type B (GABA-B) autoreceptor and heteroreceptor-mediated inhibition to the sulfhydryl alk
50                                      This D2 heteroreceptor-mediated mechanism is more efficient in t
51 ition and refine mechanisms by which GABA(B) heteroreceptors modulate mesolimbic circuit function.SIG
52 the expression of an IL-4Ralpha/IL-13Ralpha1 heteroreceptor on Th1 cells.
53 the C1 area of the RVL function primarily as heteroreceptors on presynaptic axons and terminals of no
54 ty, and its control by presynaptic auto- and heteroreceptors on presynaptic terminals.
55 ammation), acting on inhibitory histamine H3 heteroreceptors on serotonin terminals.
56 ndently manipulate 5-HT(1A) autoreceptor and heteroreceptor populations.
57 roughout the brain that overlaps with 5-HT1B heteroreceptors (receptors located on non-serotonergic n
58 icated in autoregulation and also may play a heteroreceptor role in regulation of the output of the d
59 (a brain area with high expression of 5-HT1A heteroreceptors sensitive to cholinergic effects on affe
60                      Thus, BNST alpha(2a)-AR heteroreceptor signaling might decrease the beneficial e
61 evoked HA signaling in the NAc recruits H(3) heteroreceptor signaling to shift thalamocortical input
62 , we mimicked BNST G(i)-coupled alpha(2a)-AR heteroreceptor signaling using a G(i)-coupled designer r
63  5-HT release throughout the brain and (2) a heteroreceptor that mediates inhibitory responses to rel
64 ells rapidly produce IL-4 which utilizes the heteroreceptor to drive apoptosis of Th1 cells, thus yie
65 opmentally expressed IL-4Ralpha/IL-13Ralpha1 heteroreceptor to endow IFN regulatory factor 1 (IRF-1)
66 (1B) receptors function as autoreceptors and heteroreceptors to exert presynaptic inhibition of trans
67 rst define the contributions of alpha(2a)-AR heteroreceptors to stress-induced reinstatement.
68 ha1, leading to an unusual expression of the heteroreceptor, which will serve as a death marker for t
69 mechanistic insight into the role of GABA(B) heteroreceptors within reward circuitry, elucidate a nov