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1 hen catestatin variants were mixed in likely heterozygotic (1:1 M ratio) combinations, the inhibitory
2                          Here we show that a heterozygotic abrogation of FgfR2-exon 9 (IIIc) in mice
3                                         Both heterozygotic and homozygotic TSHR null mice are osteope
4  to meet the training criteria than those by heterozygotic and Long-Evans (wild type) rats.
5 cantly poorer attention accuracy compared to heterozygotic and Long-Evans rats.
6 ous mutant mice was compared to GC counts in heterozygotic and nonmutant siblings.
7 inal cortex, and hippocampus of 18-month-old heterozygotic animals.
8             This report describes a compound heterozygotic ASNSD child with two novel mutations in th
9           EIU was induced in wild-type (WT), heterozygotic (COX-2(+/-)) and COX-2 null (COX-2(-/-)) m
10 l cord explants from wild-type mice and mice heterozygotic for a null mutation in the neuregulin gene
11                                   DBA/2 mice heterozygotic for Chrna7 were bred together.
12 ry odorants than either individuals who were heterozygotic for this change or those who had the wild-
13 ression of all four mRNA species (PFKFB1-4), heterozygotic genomic deletion of the inducible PFKFB3 g
14 ved human neurons, a condition closer to the heterozygotic genotype of patients, revealed a dominant-
15  impaired performance on the IA task and OVX heterozygotic (HET) mice exhibited performance that was
16     EAE was seen in IFN-gamma knockout mice, heterozygotic (IFN-gamma +/-) mice, as well as wild-type
17 equimolar levels, mimicking the situation in heterozygotic individuals.
18 h wild-type and ALDH2-deficient, ALDH2*1/*2, heterozygotic knock-in mice.
19 nockdown of wild type (WT) Cdk2 with siWT in heterozygotic knockin cells resulted in the mutant Cdk2
20                             Whole-body SNARK heterozygotic knockout mice also had impaired contractio
21          We examined bone phenotype of Keap1 heterozygotic mice (Ht) in comparison with Keap1 wild ty
22 phthalmia and cataracts in the lens, whereas heterozygotic mice (VIM(WT/SA)) were indistinguishable f
23 e recently reported that neil1 knock-out and heterozygotic mice develop the majority of symptoms of m
24  widespread Abeta deposition in 18-month-old heterozygotic mice produces neuritic alterations and gli
25                                      In Lis1 heterozygotic mice, severe hippocampal disorganization a
26 d in certain amyloid plaque-prone regions of heterozygotic mice.
27 dative stress, neil1 knockout (neil1-/-) and heterozygotic (neil1+/-) mice develop severe obesity, dy
28 ed with elevated total serum IgE in subjects heterozygotic or homozygotic for this base exchange (p <
29                    Choline-supplemented mice heterozygotic or null-mutant for Chrna7 failed to show i
30                                   Cells from heterozygotic parents showed normal levels of unschedule
31                                              Heterozygotic perinatally infected twins with a marked d
32 ated with survival benefit in LPS-challenged heterozygotic PU.1-deficient mice, establishing a novel
33 ation but can survive until well after their heterozygotic siblings have pupariated.
34              These vectors were used to form heterozygotic virions containing RNAs of each parental v
35 can serve as a template for DNA synthesis in heterozygotic virions.
36 ant receptor properties are recapitulated in heterozygotic zebrafish bearing an L258P mutation in the