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1 uced weight gain and adiposity in mice fed a high fat diet.
2 yeloid or lymphoid cells were subjected to a high fat diet.
3 e the progression of NAFLD in the setting of high fat diet.
4 alters the metabolic phenotype in mice fed a high fat diet.
5 burden in miR-144 knockout mice receiving a high fat diet.
6 ation in an obesity mouse model induced by a high fat diet.
7 improves metabolic parameters in mice fed a high fat diet.
8 skeletal muscle adaptions to training during high-fat diet.
9 xercise, mediate adaptations to exercise and high-fat diet.
10 ts showed the environment in vivo tends to a high-fat diet.
11 ocephalic arteries of ApoE-deficient mice on high-fat diet.
12 d diabetes phenotypes in mice fed a constant high-fat diet.
13 r compartments in fat and are activated by a high-fat diet.
14 hibit some, but not all, of the effects of a high-fat diet.
15 ion and peripheral glucose clearance after a high-fat diet.
16 al glucose tolerance in mice made obese with high-fat diet.
17 lial hyperplasia, which is an indicator of a high-fat diet.
18 ody weight and adiposity in obese mice fed a high-fat diet.
19 ic health of maternal mice challenged with a high-fat diet.
20 metabolism at resting state, even when fed a high-fat diet.
21 abolished in mice overexpressing SIRT1 fed a high-fat diet.
22 e, and greater inflammation in AT when fed a high-fat diet.
23 rosclerosis was quantified after 12 weeks of high-fat diet.
24 ior were also normal, even after exposure to high-fat diet.
25 pose and liver inflammation in response to a high-fat diet.
26 R deletion and were further increased by the high-fat diet.
27 nsgenerational inheritance of responses to a high-fat diet(9), thus raising the exciting possibility
28 microvesicles are enhanced by exposure to a high fat diet, a known risk factor for atherosclerosis.
29 ithelial T lymphocytes occurs within 7 wk of high-fat diet administration and is not dependent on chr
30 id metabolism resulting from gene mutations, high-fat diets, alcohol, or circadian disruption can con
31 sympathetic nerve activity were obtained in high fat diet and normal chow fed male C57BL/6J mice.
32 osis was induced in miR-144 knockout mice by high fat diet and vascular lesions were quantified by Oi
34 ient (Malat1(-/-)) mice that were fed with a high-fat diet and by studying the regulation of MALAT1 i
35 icient to improve liver damage in mice fed a high-fat diet and in mice fed a methionine-choline-defic
36 Cancer risk factors, such as high-sugar or high-fat diet and inflammation, impact cell competition-
37 stress in mice-elicited by a combination of high-fat diet and inhibition of constitutive nitric oxid
40 e are concerns about the association between high-fat diets and cognitive decline, this study aimed t
41 umulate in adipose and muscle tissues during high-fat diets and contribute to a state of local inflam
42 how that the convergence of dietary factors (high-fat diet) and dysregulated WNT signaling (APC mutat
43 ontrast, mice with comorbid diabetes (aging, high-fat diet, and streptozotocin-induced diabetes) had
44 protection from weight gain on standard and high-fat diets, and an adiposity-dependent improvement i
45 found that when chimeric animals were fed a high-fat-diet, animals with low levels of chimerism show
47 esoid X receptor null (Fxr(Delta/E)) mice on high-fat diet as well as wild-type C57BL/6 and glucagon-
50 Wild-type and PAI-1 knockout (KO) mice on a high-fat diet both became significantly heavier than lea
51 ced wheel running occurs when mice are fed a high-fat diet but is normalized when mice consume standa
52 al bacteria to offset the adverse effects of high fat diets, C57BL/6J mice were fed control/low fat (
53 tive of this study was to evaluate whether a high-fat diet can aggravate the liver disease caused by
54 demonstrate that both food restriction and a high-fat diet cause an endocannabinoid-dependent inhibit
56 ved choline-deficient, l-amino acid-defined, high-fat diet (CDAHFD) for 6 weeks or (d) 9 weeks (n = 8
57 al to subcutaneous fat (VAT/SAT) ratio after high-fat diet challenge, in comparison to their wild-typ
58 cantly less body weight and fat mass when on high-fat diets compared with littermate controls and wer
60 nction in male and female offspring Maternal high-fat diet consumption prior to and throughout pregna
61 dults at moderate CVD risk, consumption of a high-fat diet containing SFA-reduced, MUFA-enriched dair
62 ism by feeding Sirt5 knockout mice (Sirt5KO) high-fat diets containing either C(8)/C(10) fatty acids
63 rteries of endothelial Fto-deficient mice on high-fat diet; conversely, direct addition of prostaglan
64 abolism in mice fed either regular chow or a high-fat diet, demonstrating that these metabolic effect
66 ionally, ablation of Pnoc(CeA) cells reduces high-fat-diet-driven increases in bodyweight and adiposi
67 e previously determined that male mice fed a high-fat diet exhibit macrophage infiltration into the h
69 cise training protocol, in either low-fat or high-fat diet fed mice, did not require Bcl2-mediated au
70 ther increase insulin-positive islet area in high fat diet-fed mice and was unable to prevent or reve
78 used endothelium-specific knockout mice and high-fat diet-fed mice to assess the role of endothelial
81 of neuropathy and restores nerve function in high-fat diet-fed murine models of peripheral neuropathy
82 verexpressing mice and in serum and PGWAT of high-fat diet-fed RBP4-overexpressing mice vs. wild-type
83 atic vagal innervation, and are preserved in high-fat-diet-fed rats when the blood brain barrier is b
84 at CHRNA2 signaling is activated after acute high fat diet feeding and this effect is manifested thro
93 Compared with Foxp3cre mice, after 13 wk of high-fat diet, Foxp3creInsrfl/fl mice exhibited improved
94 usly, we found that PLA2G2A protects mice on high-fat diets from weight gain and insulin resistance.
95 DHEA in male iWAT and eWAT in response to a high-fat diet further strengthen the inference regarding
96 ler increases in FGF21 after the low-protein high-fat diet gained more weight after 6 months in free-
100 r low-fat counterparts (LF mice), mice fed a high-fat diet (HF mice) had impairments in inflammatory
102 een obesity and AD by feeding APP/PS1 mice a high fat diet (Hfd) and evaluating behavioral, physiolog
103 ve been implicated in the mechanism by which high fat diet (HFD) and saturated fatty acids (SFA) modu
107 female mice were fed a normal chow (NC) or a high fat diet (HFD) for 5 weeks before mating, then also
109 sly reported that GDNF is protective against high fat diet (HFD)-induced hepatic steatosis in mice th
110 a growing body of evidence illustrating that high fat diet (HFD)-induced maternal obesity can regulat
117 s demonstrate that in comparison with males, high-fat diet (HFD) allergic female mice exhibit a reduc
119 developed mild insulin resistance when fed a high-fat diet (HFD) and had reduced food intake during r
120 ed into two groups fed either a control or a high-fat diet (HFD) and then the mice on each diet were
121 red these with the effects of a prototypical high-fat diet (HFD) as well as cessation of exposure on
123 PTOA was induced in C57Bl/6 mice fed with high-fat diet (HFD) by surgically destabilising the meni
124 Previous studies showed that 12 weeks of high-fat diet (HFD) consumption caused not only prediabe
125 fatty liver progress to NASH, and mice fed a high-fat diet (HFD) develop fatty liver but are resistan
126 t et al. showed that feeding maternal mice a high-fat diet (HFD) during lactation attenuated the acti
128 rate in a murine prostate cancer model, that high-fat diet (HFD) enhances the MYC transcriptional pro
129 In mice, we previously found that maternal high-fat diet (HFD) exposure results in reduced fetal gr
130 s of a non-pharmacological intervention in a high-fat diet (HFD) fed mouse model, capable of recapitu
132 Control and ACC2 iKO mice were subjected to high-fat diet (HFD) feeding for 24 weeks to induce obesi
134 ngs exist regarding the impact of short-term high-fat diet (HFD) feeding on the development of allerg
148 d L2-IL1B mice) were fed a chow (control) or high-fat diet (HFD) or were crossbred with mice that exp
149 lobule membrane (MFGM-PL) supplementation to high-fat diet (HFD) rats during pregnancy and lactation
152 fed a control diet, plasmas from mice fed a high-fat diet (HFD) showed delayed PG and reduced PG vel
153 aluation paradigm, we found that exposure to high-fat diet (HFD) suppresses the intake of nutritional
154 b/ob (obese) or heterozygote (lean) mice fed high-fat diet (HFD) that received either 17beta-Estradio
155 -/-) .Leiden mice received 16 weeks either a high-fat diet (HFD) to induce obesity, or chow as refere
157 icantly increased in NT(+/+) mice fed with a high-fat diet (HFD) which were improved in NT-deficient
165 ance to obesity and fatty liver induced by a high-fat diet (HFD), whereas liver-specific IMP2 overexp
167 r and cellular aspects of atherosclerosis in high-fat diet (HFD)-fed L13a KO and intact (control) mic
171 ney (uninephrectomy [UniNx]) in mice reduced high-fat diet (HFD)-induced adipose tissue inflammation,
174 ial transcription factor A (TFAM) attenuates high-fat diet (HFD)-induced fat gain and IR in mice in c
194 otein E knockout mice (ApoE(-/-)) were fed a high-fat-diet (HFD) for up to four-months prior to MRI o
195 e report that cardiac dysfunction induced by high-fat-diet (HFD) persists for two subsequent generati
197 KO mice), we showed that AKT1 is involved in high-fat-diet (HFD)-induced growth and survival of beta
201 ption factor A (TFAM) and inhibited maternal high fat diet-impaired placental efficiency and glucose
203 nating feeding mice with a low-fat diet or a high-fat diet in a 1-week switch protocol caused further
207 ed in obese patients and after 24 weeks of a high-fat diet in mice, accompanying signs of AT inflamma
208 ormin prevented weight gain in response to a high-fat diet in wild-type mice but not in mice lacking
210 aloric control diet and a 3-day hypercaloric high-fat diet (increase of 75% in energy, 81-83% energy
213 g Rebaudioside A and sucralose on NASH using high fat diet induced obesity mouse model by substitutin
215 etabolic stress induced in mice by feeding a high-fat diet induced greater DNA damage in osteoblast o
216 in alveolar and interstitial macrophages in high-fat diet induced obese mice were lower than regular
217 on, we treated regular chow diet-fed mice or high-fat diet induced obese mice with lipopolysaccharide
219 ammation in both models, disturbed flow- and high fat diet-induced atherosclerosis, whereas Nck2 dele
221 e conclude that LCN2 is dispensable for both high fat diet-induced obesity and its therapeutic reduct
222 mic overexpression of SH2B1 protects against high fat diet-induced obesity and metabolic syndromes.
223 n wild-type mice, NaHS treatment ameliorates high fat diet-induced obesity and metabolism disorders,
225 of streptozocin-induced type 1 diabetes and high fat diet-induced type 2 diabetes mouse models and l
227 ity and dyslipidemia, it protected mice from high-fat diet-induced glucose intolerance and insulin re
230 Here we show that CSE knockout exacerbated high-fat diet-induced mouse obesity as well as its relat
234 mogenic genes in BAT, and are protected from high-fat diet-induced obesity and development of insulin
235 ic transfer of the EAT gene to mice prevents high-fat diet-induced obesity, insulin resistance and fa
241 eased energy expenditure and amelioration of high-fat-diet-induced obesity and markedly improved gluc
243 e-specific MyD88 or IRAK2 deficiency reduced high-fat-diet-induced weight gain, increased energy expe
244 Herein we investigated whether gestational high fat diet influences the offspring susceptibility to
245 diac-specific deletion of PKBalpha/beta or a high fat diet inhibits insulin-induced phosphorylation o
249 When mice with CrAT deletion were fed a high-fat diet, kidney disease was more severe and develo
250 chromosome complement in combination with a high-fat diet led to enhanced weight gain in the presenc
251 these data provide the first evidence that a high-fat diet may be a risk factor for the development o
252 Here, we investigate the effects of maternal high-fat diet (MHFD) at different stages of pre- or post
255 om the SFA-rich high-fat diet to a MUFA-rich high-fat diet; nerve conduction velocities and intraepid
257 fection negated the deleterious effects of a high-fat diet on cardiac function and remodeling, and ac
258 Triple transgenic dams were administered high fat diet or regular chow throughout 3 weeks gestati
260 bunit up-regulation, and in the setting of a high-fat diet, p110gamma ablation failed to protect agai
262 he injection of P5 peptide into WT mice on a high-fat diet prevents macrophage accumulation in adipos
265 ght and body fat than did control mice fed a high-fat diet, resulting in ameliorated glucose toleranc
266 We have previously described that mice fed a high-fat diet rich in polyunsaturated fatty acids (HFD-P
267 f neutrophils in bone marrow than mice fed a high-fat diet rich in saturated fatty acids (HFD-S).
271 2(S587A) mice that were fed either a chow or high-fat diet showed similar weight gain as the wild-typ
272 rnal metformin treatment along with maternal high fat diet significantly increased mouse placental ab
275 n Tgr5(-/-), but not Fxr(Delta/E) mice fed a high-fat diet, suggesting a role for intestinal Fxr.
276 or 16-wks) on either a low-fat, high-fat, or high-fat diet supplemented with 1.5X branched chain amin
277 testinal serotonin of mice fed for 9 wk on a high-fat diet supplemented with different sources of fib
279 oma, and metabolic derangements induced by a high-fat diet; therefore, elucidating the intracellular
280 t when female Ppp1r15a mutant mice are fed a high fat diet they gain less weight than wild type litte
281 rsed by switching the mice from the SFA-rich high-fat diet to a MUFA-rich high-fat diet; nerve conduc
285 I, Tan and Hang et al. report that feeding a high-fat diet to mice compromised the function of the or
287 hepatic transcriptional response in mice on high-fat diet treated with metformin was largely ablated
291 eding the hep-LAL-ko mice a vitamin A excess/high-fat diet (VitA/HFD) further increased hepatic chole
292 ry of antibiotics in individuals consuming a high-fat diet was associated with the greatest risk for
294 tro from male and female mice fed control or high-fat diet, we demonstrated that macrophages derived
295 ce (C57BL/6J mice, 33 weeks old), fed with a high fat diet which increases adipose tissue favouring o
296 s collected from mice on a choline-deficient high-fat diet, which developed chronic liver inflammatio
297 were randomly assigned to receive chow diet, high fat diet with sugar in drinking water (Western diet
298 late the effect of dietary fat, we generated high-fat diets with varying fatty acid balance and type.
300 liver steatosis that developed in mice fed a high-fat diet, with or without combination with an inhib