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1 otential risks (including the possibility of high-grade prostate cancer).
2 men with systemic progression and death from high grade prostate cancer.
3 or SAPC as a potential prognostic marker for high grade prostate cancer.
4 ovel gene whose expression is inactivated in high grade prostate cancer.
5 gnosis of low-risk and the underdiagnosis of high-grade prostate cancer.
6 3.0% and 73.6%, respectively, for those with high-grade prostate cancer.
7 also associated with increased detection of high-grade prostate cancer.
8 n-7, could be involved in the development of high-grade prostate cancer.
9 hived specimens from untreated patients with high-grade prostate cancer.
10 There was no increase in high-grade prostate cancer.
11 enic, and eicosapentaenoic acids and risk of high-grade prostate cancer.
12 pendently associated with risk of lethal and high-grade prostate cancer.
13 d with decreased risk of prostate cancer and high-grade prostate cancer.
14 frequent allelic loss and downregulation in high-grade prostate cancer.
15 exual side effects and the increased risk of high-grade prostate cancer.
16 with an increased risk of advanced-stage and high-grade prostate cancer.
17 y end point was the incidence of adjudicated high-grade prostate cancer.
18 c alterations of KLF6 occur in a minority of high-grade prostate cancers.
19 expression is dramatically down-regulated in high grade prostate cancers (4/4) but is unaltered in lo
20 person-years of follow-up, the incidence of high-grade prostate cancer (5 of 2596 [0.19%] in the TRT
21 e 270, 148, and 88 cases of total, low-, and high-grade prostate cancers among African American men a
22 ification of both alternative treatments for high-grade prostate cancers and new biomarkers to predic
23 rmethylation is enriched in intermediate- to high-grade prostate cancers and not detectable in benign
25 risk-stratify patients with intermediate- or high-grade prostate cancer before prostatectomy based on
26 ty is associated with an increase in risk of high-grade prostate cancer but with a decrease in risk o
27 proanthocyanidins (P for trend = 0.04) with high-grade prostate cancer, but not with advanced prosta
30 hould be weighed with the potential risks of high-grade prostate cancer from 5-ARIs (although the maj
31 stry analysis revealed elevation of PROS1 in high grade prostate cancer (Gleason grade >/= 8), and fu
32 ade (HR, 1.14; 95% CI, 1.01 to 1.29) but not high-grade prostate cancer (HR, 0.83; 95% CI, 0.64 to 1.
33 index was positively associated with risk of high-grade prostate cancer in both non-Hispanic white me
35 ncidence (HR, 1.02; 95% CI, 0.96 to 1.08) or high-grade prostate cancer incidence (HR, 0.91; 95% CI,
37 nearly and inversely associated with risk of high-grade prostate cancer (quartile 4 vs. 1: TFA 18:1,
38 ds, flavan-3-ols, and proanthocyanidins with high-grade prostate cancer risk varied by follow-up time
39 and vitamin D intake and total, advanced, or high-grade prostate cancer risk, whether for total intak
43 may be associated with an increased risk of high-grade prostate cancer tumors that would ultimately
46 oreactivity was significantly upregulated in high-grade prostate cancer when compared to benign prost
47 men with a new diagnosis of intermediate- or high-grade prostate cancer who were candidates for prost
48 ferentiating low-grade from intermediate- or high-grade prostate cancer with diffusion-weighted MR im
50 Outcomes are also disparate among men with high-grade prostate cancer, with upwards of 65% having d