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1 er neurons, which project to the M-cell axon hillock.
2 at the presumptive domain boundary, the axon hillock.
3 ing were present in the vicinity of the axon hillock.
4  forward along the dendrite to the soma-axon hillock.
5  opposed to the planar regions surrounding a hillock.
6 ated with the formation of well-defined etch hillocks.
7 autophagy effector ULK1 accumulate at axonal hillocks.
8 he accumulation of the mRNA both at the axon hillock after the treatment with 5-HT and at the opposit
9                          The cell body, axon hillock and basal dendritic compartments achieve unique
10 like features that coexpress basal, luminal, hillock and club marker genes, before transitioning to K
11 eins abolished both accumulation at the axon hillock and long-term facilitation.
12 in mRNA and protein accumulation at the axon hillock and number of syntaxin granules in the SN axon w
13 he accumulation of sensorin mRNA at the axon hillock and the stability of sensorin mRNA exported to d
14 onal cell bodies, primary dendrites and axon hillocks and function in neuronal protection and stabili
15 h is also accompanied with the formations of hillocks and granules due to the dewetting of Au films a
16  We discuss the experimental observations of hillocks and pinhole-type defects producing inter-layer
17 electrical activation occurred near the axon hillock, and that dendrites contributed little.
18 ion of syntaxin mRNA and protein at the axon hillock, and the increase in syntaxin granules in SN axo
19 and protein were targeted away from the axon hillock, and the number of syntaxin granules in the SN a
20                    Furthermore, we show that hillocks are one origin of 'squamous metaplasia', which
21                                       Airway hillocks are stratified epithelial structures of unknown
22      This AMPK activation triggers localized hillock autophagosome accumulation and mitophagy, ultima
23                                              Hillock basal stem cells are capable of massive clonal e
24                                              Hillock basal stem cells continually replenish overlying
25 use hillock squamous cells shield underlying hillock basal stem cells from injury.
26                                              Hillock basal stem cells preferentially stratify and ker
27 f atomically flat Si(100) terminated by etch hillocks bounded by {111}- and {110}-oriented microfacet
28 ugh morphological phases defined as placode, hillock, bud, and bulb stages followed by branching morp
29 nsities, cells in serum-coated dishes formed hillocks, but cells in noncoated dishes did not.
30                             The induction of hillocks by NAC was correlated with downregulation of al
31                                              Hillocks capped by different pentameric proteins spontan
32 f genes defining this mixed basal, club, and hillock cell identity in DNPC models.
33 signaling cell populations analogous to Club/Hillock cells enriched in the aged human prostate.
34 e reminiscent of previously described mouse "hillock" cells and with squamous cells expressing SCEL a
35 oduces multifocal nodular structures, i.e., "hillocks," consisting of cells and extracellular matrix.
36  GaN:Mg films grown by MOCVD on high and low hillock density GaN template layers.
37 e conductivity of GaN:Mg films grown on high hillock density template layers is verified by optical a
38 urons, syntaxin mRNA accumulates at the axon hillock during long-term facilitation of sensory-motor n
39 ells express genes defining basal, club, and hillock epithelial cells from benign prostate.
40                      Here we show that mouse hillock expansion is the cause of vitamin A deficiency-i
41                     In summary, key steps in hillock formation are: (1) migration, (2) secretion of f
42            Antibodies to fibronectin blocked hillock formation by cells on serum-coated substrata and
43        Smooth muscle cell and mesangial cell hillock formation have been proposed as in vitro models
44                                              Hillock formation is associated with induction of a diff
45 r and how the cellular redox state modulates hillock formation.
46 cer of glutathione, dramatically facilitated hillock formation.
47  pericellular fibronectin-fibrils was key to hillock formation.
48  relocalization of syntaxin mRNA to the axon hillock from the opposite pole in the cell body of the s
49 for CRPC displaying a mixed basal, club, and hillock identity.
50 ore examined how changes in R(a) at the axon hillock impact the voltage threshold (V(th)) of the soma
51 action potentials initiate first in the axon hillock/initial segment (AH-IS) region because of a loca
52     The goldfish M-cell initial segment/axon hillock is surrounded by a composite axon cap consisting
53  ciliated, AZGP1(high) goblet, and KRT13(+) "hillock"-like cells, and we identify genes associated wi
54  were found surrounding cell bodies and axon hillocks most often in the buccal and abdominal ganglia.
55 xin mRNA and protein accumulated at the axon hillock of SNs during the initial phase of synapse forma
56  of high levels of sensorin mRNA in the axon hillock of the SN and in SN neurites contacting L7.
57         At the nodes of Ranvier and the axon hillocks of central neurons, VGSCs are associated with t
58 lse can be initiated in either the soma-axon hillock or in the distal primary dendrite, and that the
59 e at a fixed location, typically at the axon hillock or initial segment, although action potentials,
60   A high density of Na+ channels in the axon hillock, or initial segment, is believed to determine th
61 ial to SI >/= 1.0 to generate/develop growth hillocks, or other factors.
62                                              Hillocks persist for months and have a unique population
63                             The existence of hillocks reframes our understanding of airway epithelial
64 t the impulse always arises in the soma-axon hillock region, with back-propagation through excitable
65 ts were located near the nucleus or the axon hillock region.
66  generating spikes of depolarization at axon hillock regions and propagating the initial rising phase
67 Ca2+ release within the perinuclear and axon hillock regions provide a mechanism for preferential ini
68                                              Hillocks resist a remarkably broad spectrum of injuries,
69 ilayered ridges and nodules, termed hills or hillocks, separated by less populated areas termed valle
70 llock structure is comparable to that within hillock sidewall facets measured at 1.3 x 10(19) cm(-3)
71 he Mg cluster radius is decreased within the hillock sidewall.
72 ron including an axon, initial segment, axon hillock, soma, and simplified dendritic tree was used to
73 he odorant transduction process and the axon hillock spiking mechanism of the olfactory sensory neuro
74  infection, acid and physical injury because hillock squamous cells shield underlying hillock basal s
75 oncentration in planar regions surrounding a hillock structure is comparable to that within hillock s
76                            Within individual hillock structures a decreased Mg cluster density is obs
77 reased Mg cluster density is observed within hillock structures as opposed to the planar regions surr
78 f magnesium dopants into facets of hexagonal hillock structures in N-polar GaN, studied by comparativ
79 Ga during growth of semi-polar facets of the hillock structures.
80 on potential (AP) successfully from the axon hillock to a synaptic terminal.
81 e and menadione inhibited the development of hillocks triggered by either NAC, glutathione, or prolon
82 y distributed arrays of "tips" (tall conical hillocks) upon oxidation of palladium (Pd) thin films.
83                   Finally, we identify human hillocks whose basal stem cells generate functional squa