ライフサイエンスコーパス: hind leg

1 ond to tactile stimulation or movements of a hind leg.

2 l melanoma cells after s.c. injection in the hind leg.

3 uropil regions of the ipsilateral middle and hind legs.

4 retracted the denervated front, middle, and hind legs.

5 are primarily achieved by their two powerful hind legs.

6 ke approximately fourfold and also increased hind leg 1-MX metabolism by 50%, suggesting increased ex

7 cts in chimeric mice, characterized by short hind legs, aberrant limb features, split lumbar vertebra

8 ve, trip duration, presence of pollen on the hind legs and mass upon return to the hive, during the l

9 stimulus, orienting behavior (rearing on the hind legs), and food cup behavior (placing the head insi

10 dominant, causes progressive weakness of the hind legs, and there is severe demyelination in the peri

11 lts demonstrate that insulin increases total hind leg blood flow and metabolism of 1-MX, suggesting a

12 cterized by a distinctive dysfunction of the hind legs, causing uncoordinated movements.

13 radiation-induced (single dose, 35 or 45 Gy) hind leg contraction in C3H/Hen mice.

14 umping was generated by slow contractions of hind leg depressor muscles and then stored by bending sp

15 on of the giant muscles that pulled back the hind leg during walking.

16 High-speed microscopy recordings of hind legs during the acceleration phase of jumps reveale

17 - (front), meso- (middle), and metathoracic (hind) leg during stick insect (Carausius morosus) walks.

18 e cross-sectional area of the tendons of the hind leg extensor-tibiae muscle by 31%.

19 e's center of mass, pollen is carried on the hind legs, farther from the center of mass.

20 Adding mass to increase hind leg flailing kinetic energy increased the animal's

21 this study, behaviors (open field, grooming, hind-leg gait, water maze, and acoustic startle reflex)

22 vels, muscle interstitial oxygen saturation, hind leg glucose extraction, and muscle insulin clearanc

23 Insulin treatment increased hind leg glucose uptake approximately fourfold and also

24 ously, orienting behavior (rearing up on the hind legs) habituated across trials in normo-active cont

25 Rats were trained to maintain a hind leg in a flexed position to avoid noxious stimulati

26 cic (T2) transection can learn to maintain a hind leg in a flexed position to minimize exposure to a

27 Spinalized rats given shock whenever 1 hind leg is extended learn to maintain that leg in a fle

28 ve suggested that drag-based thrust by their hind legs is responsible for their propulsion(1)(,)(2)(,

29 In addition, hind leg kick force, produced by stimulating the extenso

30 ogical and life history traits: body weight, hind leg length, parasite burden, horn length, horn grow

31 nents of shape: body width, body height, and hind-leg length.

32 ssive responses thus likely act during mouse hind leg locomotion, but their amplitude and even sign m

33 Finally, based on the diving beetle's hind-leg movement pattern, a kinematic model was constru

34 Blood flow distribution within the hind leg muscles was assessed by measuring the metabolis

35 Metabolism of intact hind-leg muscles from young rats was studied in vitro un

36 a recruitment of capillary blood flow in rat hind leg not mimicked by epinephrine.

37 tumors transplanted into the subcutis of the hind leg of Nembutal-anesthetized (50 mg/kg) Fischer 344

38 .OH-induced structural changes in DNA of the hind leg of the BALB/c mouse.

39 n two identified motoneurons innervating the hind leg of the locust: the FETi-FlTi synapse (fast exte

40 cells were intramuscularly injected into the hind legs of 18 nude mice.

41 kdown resulted in permanent breakages in the hind legs of 29% of knockdown locusts as tested by elect

42 MCA, injected into the hind legs of mice, produced a variety of significant str

43 ividuals, we compared the front, middle, and hind legs of multiple flies using scanning electron micr

44 he air preceded by the animal rearing on its hind legs or raising its head.

45 with Poly:ICLC plus OVA protein in the neck, hind leg, or foreleg for drainage into the cervical, ing

46 ese mice exhibit motor defects which lead to hind leg paralysis and tremor.

47 sin A(-/-) mice developed slowly progressive hind leg paralysis with clinical onset at approximately

48 Hind-leg paralysis preceded the death of the mice.

49 is issue, we measured, in anesthetized mice, hind leg passive movements in response to changes in ani

50 ies-specific perfume mixtures in specialized hind-leg pockets(3) using volatiles acquired from multip

51 njection of 0.75% bupivacaine into the right hind leg prior to CIP was used for peripheral nerve bloc

52 ard oil (100%) to the lateral surface of the hind leg produced a facilitation of the tail-flick refle

53 e and found that F-PRT of the C57BL/6 murine hind leg produced fewer severe toxicities leading to dea

54 Brief electrical stimulation of a single hind leg proprioceptor, the lump receptor (LR), led to p

55 y and labial palps, and unmodified femora of hind legs, providing key information for the evolutionar

56 ergy and its quick release to accelerate the hind legs rapidly.

57 dition, immunohistochemistry of mouse embryo hind legs showed that Sox9 phosphorylated at serine 211

58 ting to jump from smooth glass, the insects' hind legs slipped, resulting in weak, uncontrolled jumps

59 es that exhibit more moderate enlargement of hind legs, such as mantids and cockroaches.

60 ic dystrophic mice a progressive lameness of hind legs, suggestive of a nerve defect.

61 n of a well-studied model system: the locust hind leg tibial extensor muscle.

62 raptorial front legs, and the two propulsive hind legs to produce a controlled jump with a precise la

63 Dytiscus marginalis simultaneously uses its hind legs to propel itself through the water.

64 ) and femoral blood flow (80%) and decreased hind leg vascular resistance (31%).

65 but increased femoral blood flow and lowered hind leg vascular resistance to a similar extent as insu

66 od pressure, heart rate, femoral blood flow, hind leg vascular resistance, and glucose uptake were me

67 If one hind leg was cocked then the spikes only occurred in mot

68 sults suggest that diversification of insect hind legs was influenced by changes in both the spatial

69 Its rodent analog-unsupported rearing on hind legs-was a classic model in deciphering neural sign

70 roduced normally, they developed progressive hind leg weakness and decline in motor coordination at 1

71 mice with subcutaneous PC3 xenografts in the hind leg were treated with 2ME2 (75 mg/kg) p.o. for 5 da

72 ther than the middle legs, and also that the hind legs were able to generate a larger angular velocit

73 Second, we concluded that the hind legs were able to propel the beetle farther than th

74 First, both hind legs were moved into a cocked position by high-freq

75 joint angles, the motion trajectories of the hind legs were obtained by using MATLAB.

76 Mice bearing the sarcoma FSA in the hind legs were treated daily for 10 days with SC-'236 (6

77 as shown that spinal rats given shock to the hind leg when it is in an extended position (contingent

78 nditioned orienting behavior (rearing on the hind legs) when a visual stimulus was paired with food.

79 The hind leg, which bore most of the weight, has a half-upri

80 d the motion data and kinematic model of the hind legs will be helpful in the design of bionic underw

81 Forty-two BDIX rats were inoculated in each hind leg with a DHD/K12/TRb tumor cell line.

82 ssive mouse mutation, exhibits ataxia of the hind legs with a slight side-to-side wobble while walkin

83 individuals regularly touching others on the hind legs within populations that have become concentrat