ライフサイエンスコーパス: histochemistry

1 wisteria floribunda agglutinin (WFA) lectin histochemistry.

2 ochondrial marker Cytochrome C Oxidase (COX) histochemistry.

3 PVH was confirmed with in situ hybridization histochemistry.

4 haracteristic of IBM, including abnormal tau histochemistry.

5 brain using RT-PCR and in situ hybridisation histochemistry.

6 points, and processed for nucleic acids and histochemistry.

7 s using nonradioactive in situ hybridization histochemistry.

8 de adenine dinucleotide phosphate-diaphorase histochemistry.

9 nd NTrs was determined by immunofluorescence histochemistry.

10 NCs using quantitative in situ hybridization histochemistry.

11 igated using quantitative cytochrome oxidase histochemistry.

12 Transplanted cells were localized by histochemistry.

13 ng confocal microscopy and immunofluorescent histochemistry.

14 on cells, as indicated by cytochrome oxidase histochemistry.

15 on biochemical assays, immunoreactivity, and histochemistry.

16 identified based on cytochrome oxidase (CO) histochemistry.

17 ions processed by using tetramethylbenzidine histochemistry.

18 sittacus undulatus), was examined using iron histochemistry.

19 , they are very useful tools in quantitative histochemistry.

20 ne dinucleotide phosphate (NADPH)-diaphorase histochemistry.

21 cells, indicated by cytochrome oxidase (CO) histochemistry.

22 dinucleotide phosphate-diaphorase (NADPH-d) histochemistry.

23 t turtle retinas by using immunofluorescence histochemistry.

24 ta in the rat brain by in situ hybridization histochemistry.

25 ts was determined with in situ hybridization histochemistry.

26 adult murine brain by in situ hybridization histochemistry.

27 ype and RET-deficient mouse embryos by TUNEL histochemistry.

28 ght for Wisteria floribunda agglutinin (WFA) histochemistry.

29 fragmentation using in situ nick translation histochemistry.

30 upported by enzyme assays, Western blot, and histochemistry.

31 ma model using both a quantitative ELISA and histochemistry.

32 n the same sections by in situ hybridization histochemistry.

33 A expression, dendritic structure and immuno-histochemistry.

34 esponsive adult human retinas, and performed histochemistry.

35 itative analysis of conventional chromogenic histochemistry.

36 us (SCN) were assessed by cytochrome oxidase histochemistry.

37 (UPLC), weak anion exchange-UPLC, and lectin histochemistry.

38 land gorillas using immunohistochemistry and histochemistry.

39 ion (PCR) analysis and in situ hybridization histochemistry.

40 r complex IV activity was analysed by enzyme histochemistry.

41 e tissue processed for in situ hybridization histochemistry.

42 es as evidenced by improved ERGs and retinal histochemistry.

43 cro-positron emission tomography imaging and histochemistry.

44 n situ hybridization, and immunofluorescence histochemistry.

45 nzymatic activity was demonstrated by enzyme histochemistry.

46 outing in the rat dentate gyrus using Timm's histochemistry: (1) repeated spaced ECS; (2) daily admin

47 sion of iron-related genes, (2) nonheme iron histochemistry, (3) immunohistochemistry for proteins of

48 lei were also visualized by NADPH-diaphorase histochemistry, a marker of nNOS activity.

49 e dinucleotide phosphate diaphorase (NADPHd) histochemistry, a marker of nNOS activity.

50 T) immunoreactivity and acetylcholinesterase histochemistry (AChE).

51 rains were compared using cytochrome oxidase histochemistry, an endogenous marker of regional metabol

52 e issues, we performed in situ hybridization histochemistry analyses and found that Sirt1 mRNA is hig

53 ortex was labeled by cytochrome oxidase (CO) histochemistry analysis or [(3)H]proline autoradiography

54 t2 mRNA were measured by using hybridization histochemistry and a semiquantitative reverse transcript

55 gree of demyelination assessed by Black-Gold histochemistry and activation of glial cells assessed by

56 ith classical and contemporary techniques in histochemistry and allows unambiguous in vivo detection

57 lized to Muller glial cells by hybridization histochemistry and by immunohistochemistry.

58 ressing cell bodies by in situ hybridization histochemistry and by labeling beta-galactosidase driven

59 dinucleotide phosphate diaphorase (NADPH-d) histochemistry and conventional microelectrode technique

60 eral adult trigeminal relay nuclei by NADPHd histochemistry and demonstrate that fibers from DR conta

61 ls were investigated by acetylcholinesterase histochemistry and dual immunolocalization.

62 15- to 18-month-old Gsalpha transgenic mice, histochemistry and electron microscopy illustrated the e

63 The evolution of research utilizing histochemistry and electron microscopy, along with disci

64 ectroscopic imaging, brightfield microscopy, histochemistry and fluorescence lifetime imaging, these

65 We employed histochemistry and fluorophore-assisted carbohydrate ele

66 en from the double null mice, as assessed by histochemistry and gas chromatography-mass spectrometry.

67 rcs) is demonstrated by acetylcholinesterase histochemistry and gene expression for class III beta-tu

68 Using in situ hybridization (ISH) histochemistry and gene-specific riboprobes, the current

69 Using in situ hybridization histochemistry and immunoblot analyses, we further exami

70 In situ hybridization histochemistry and immunocytochemistry revealed TIP39-co

71 lacZ, in combination with fluorescent X-gal histochemistry and immunocytochemistry to assess levels

72 In situ hybridization histochemistry and immunocytochemistry were used to exam

73 e prominent secretory granules identified by histochemistry and immunodetection for the MC-specific g

74 le from affected patients was evaluated with histochemistry and immunohistochemical stains for dystro

75 Cell phenotypes were analyzed by histochemistry and immunohistochemistry and by reverse t

76 Histochemistry and immunohistochemistry revealed that al

77 In this study, we used in situ hybridization histochemistry and immunohistochemistry to map the distr

78 Combined in situ hybridization histochemistry and immunohistochemistry with the neurona

79 Using in situ hybridization histochemistry and immunohistochemistry, we confirmed th

80 media to neointima by in situ hybridization histochemistry and immunohistochemistry.

81 ydrolysis deficiency, as observed in situ by histochemistry and in primary smooth muscle cell culture

82 ne dinucleotide phosphate (NADPH)-diaphorase histochemistry and in situ hybridization.

83 trapezoid body (MNTB) with NADPH-diaphorase histochemistry and in situ hybridization.

84 , light and electron microscopy, specialized histochemistry and indirect immunofluorescence microscop

85 ) were examined with cytochrome oxidase (CO) histochemistry and neurofilament protein (NF) immunoreac

86 dinucleotide phosphate-diaphorase (NADPH-d) histochemistry and NOS immunocytochemistry-to demonstrat

87 n's disease (PD) using in situ hybridization histochemistry and oligodeoxynucleotide (single-stranded

88 ere investigated using in situ hybridization histochemistry and oligodeoxynucleotide (single-stranded

89 oridia were identified in stool specimens by histochemistry and PCR of 30 (18.9%) of 159 HIV-infected

90 Histochemistry and polymerase chain reaction demonstrate

91 oscopy (SEM), immunofluorescence microscopy, histochemistry and quantitative RT-PCR methods.

92 We used in situ hybridization histochemistry and reverse transcriptase-PCR amplificati

93 ections were prepared for beta-galactosidase histochemistry and rhodopsin, TfR, or GFAP immunocytoche

94 ne dinucleotide phosphate-diaphorase (NADPH) histochemistry and SMI-32 immunocytochemistry to label p

95 assessments, spinal cords were collected and histochemistry and stereology were conducted to evaluate

96 Sections underwent immunocytochemistry or histochemistry and the overlap of microvascular and func

97 ter than adjacent divisions with CO and AChE histochemistry and was moderately stained with calbindin

98 Myocardial histochemistry and Western blot analysis revealed a sign

99 But, based on histochemistry and Western blot analysis, Pd is found al

100 specimens were evaluated by immunoperoxidase histochemistry and Western blot analysis.

101 mploying multiple-label immunocytochemistry, histochemistry, and backfills, we searched for photic en

102 of which was studied by means of PCR, X-gal histochemistry, and beta-galactosidase immunocytochemist

103 mechanisms were defined by optical mapping, histochemistry, and biochemistry.

104 ccinate dehydrogenase and cytochrome oxidase histochemistry, and electron microscopy correlated with

105 additional methods in rats (immunoblotting, histochemistry, and electron microscopy).

106 Liver function, morphology, histochemistry, and fibrotic parameters were examined.

107 oxidase (CO) and acetylcholinesterase (AChE) histochemistry, and immunocytochemistry for calbindin.

108 rse transcription-PCR, in situ hybridization histochemistry, and immunocytochemistry.

109 ells, as shown by single-cell RT-PCR, enzyme histochemistry, and immunofluorescence.

110 Our proteomics, histochemistry, and immunohistochemistry data also revea

111 real-time polymerase chain reaction, (immuno)histochemistry, and light and electron microscopy.

112 had mild exercise intolerance, normal muscle histochemistry, and normal respiratory chain activity in

113 d using immunofluorescence or lectin binding histochemistry, and percentages of single and double-lab

114 Developmental analysis by histology, histochemistry, and SEM revealed a significant reduction

115 Histopathology and histochemistry are providing a better understanding of t

116 nsitometry, microphotometry, and video-based histochemistry, are effective in quantitative and detail

117 amide adenine diphosphate (NADPH)-diaphorase histochemistry as an indicator of nitric oxide synthase

118 ntrols using quantitative cytochrome oxidase histochemistry as an intracellular measure of oxidative

119 tivity using quantitative cytochrome oxidase histochemistry as in our previous study using congenital

120 Lectin histochemistry as well as tissue binding patterns of TCo

121 re identified by acetylcholinesterase (AChE) histochemistry, as well as immunostaining for calbindin-

122 The senescence-associated beta-galactosidase histochemistry assay, adapted for use in the primate pos

123 sured retrospectively using a novel affinity histochemistry assay.

124 r, and were alkaline phosphatase positive by histochemistry but showed no other evidence of bone line

125 sera corroborated the results of the NADPH-d histochemistry by staining the same two cells in the cer

126 ional roles in the retina using fluorescence histochemistry, confocal microscopy, immuno-electron mic

127 romatography (HPLC), cytochrome oxidase (CO) histochemistry, cresyl violet, or demonstration of TCAs

128 Dihydroxyphenylalanine (DOPA) histochemistry demonstrated an increase in melanization

129 tibialis anterior (TA) muscle morphology and histochemistry demonstrated an increase in the cross sec

130 beta-Galactosidase histochemistry demonstrated gene expression throughout t

131 Affinity histochemistry demonstrated that hyaluronan accumulates

132 Electron microscopic histochemistry disclosed a complex array of corneal PGs

133 Brain histochemistry disclosed abnormal aggregates of ferritin

134 Histochemistry documented successful implantation of myo

135 s for these mutations was analyzed by enzyme histochemistry during embryogenesis, postnatal developme

136 of H5N1 virus receptors, by virus and lectin histochemistry, during highly pathogenic avian influenza

137 pathological tests with regard to studies of histochemistry examination.

138 lateral symmetry of cytochrome oxidase (COX) histochemistry following unilateral eye enucleation was

139 hicle for the adjuvant by using fluorescence histochemistry for catecholamines, with morphometric ana

140 e healing pattern was assessed by histology, histochemistry for collagen deposition, and immunohistoc

141 ere analyzed utilizing in situ hybridization histochemistry for enkephalin (ENK) mRNA in the ventral

142 on microscopy, we performed immunoperoxidase histochemistry for ICAM-5 in mouse visual cortex at post

143 transcriptase (RT)-PCR, immunostaining, and histochemistry for key proteins underlying ATP secretion

144 ial cells) and GFAP (astrocytes), and enzyme histochemistry for menadione-dependent a-glycerophosphat

145 such injury, we performed immunofluorescence histochemistry for metabotropic glutamate receptor 1alph

146 Myeloperoxidase histochemistry for neutrophils was performed in jejunal

147 Myeloperoxidase histochemistry for neutrophils was performed in jejunal

148 studied with myosin immunohistochemistry and histochemistry for the mitochondrial enzyme, nicotinamid

149 ation was confirmed by in situ hybridization histochemistry for two strongly down-regulated genes, my

150 By quantitative histochemistry, hepatic fibrosis in treated animals was

151 Liver tissues were analyzed by histochemistry, hydroxyproline determination, reverse-tr

152 In situ hybridization histochemistry identified strong mRNA labeling for BMP-4

153 We analyzed structural changes in muscle by histochemistry, immunocytochemistry, and electron micros

154 enucleated, and retinal sections studied by histochemistry, immunofluorescence labeling, and confoca

155 Histochemistry, immunohistochemistry, and electron micro

156 Using histochemistry, immunohistochemistry, and image analysis

157 uction profiles were evaluated by histology, histochemistry, immunohistochemistry, and quantitative a

158 MS cases together with complex IV/complex II histochemistry, immunohistochemistry, laser dissection m

159 hology were evaluated using biochemistry and histochemistry in 62 subjects with a premortem diagnosis

160 f NADPHd activity was demonstrated by NADPHd histochemistry in both central and peripheral nervous sy

161 ha regulation, we used in situ hybridization histochemistry in C57Bl/6J mice and Sprague-Dawley rats.

162 Dual-labeling histochemistry in caudoputamen demonstrated that densely

163 We used cytochrome oxidase (CO) histochemistry in conjunction with other histological me

164 s by beta-galactosidase expression and X-gal histochemistry in gastrointestinal epithelia.

165 ed by semiquantitative in situ hybridization histochemistry in melanized neurons of human substantia

166 studied by using NADPH-diaphorase (NADPH-d) histochemistry in the CNS and peripheral organs.

167 disease were processed using cholinesterase histochemistry in the presence or absence of rivastigmin

168 cells, as reflected by GAD67 mRNA expression histochemistry, in the rat substantia nigra pars reticul

169 Using histochemistry, in vivo Ca(2+) imaging, and behavioral a

170 leukocytic phytohemagglutinin (LPHA) lectin-histochemistry] in 119 archival specimens of human melan

171 rther characterized by in situ hybridization histochemistry, including pituitary adenylate cyclase ac

172 Hybridization histochemistry indicated robust KiSS-1 and GPR54 mRNA ex

173 ne serum and characterized using morphology, histochemistry, indirect immunofluorescence microscopy,

174 cultures together with in situ hybridization histochemistry (ISHH) in sections of adult rat tissue.

175 the present study used in situ hybridization histochemistry (ISHH) to map the temporal and sexually d

176 spinal cord, by using in situ hybridization histochemistry (ISHH) with a novel cRNA probe.

177 by using radioisotopic in situ hybridization histochemistry (ISHH) with a novel sensitive cRNA probe.

178 he CNS, as assessed by in situ hybridization histochemistry (ISHH), has been described previously in

179 In rat and monkey amygdala, WFA histochemistry labeled perineuronal nets, but not glial

180 ption polymerase chain reaction, immunoblot, histochemistry, laser-capture microscopy, and terminal d

181 Subcellular studies and histochemistry localized UGD protein to the perinuclear

182 (R(2)>0.80, p<1E-20) between MRI and immuno-histochemistry measurements with 95% lower bound of the

183 tagged" and imaged using conventional immuno-histochemistry methods.

184 Using NADPH-diaphorase histochemistry, neuronal nitric oxide synthase (nNOS) an

185 g overall normal tissue morphology and brain histochemistry, normal blood and urine chemistries, norm

186 vity were validated by in situ hybridization histochemistry of COX-2 mRNA and Western blot analysis.

187 llular changes in dendritic mitochondria and histochemistry of cytochrome c oxidase (CO) activity wer

188 and D(2) receptors and in situ hybridisation histochemistry of D(1) and D(2) mRNA were performed.

189 Immune histochemistry of murine and human poststroke autoptic b

190 Horseradish peroxidase (HRP) histochemistry of OB following a 10-day recovery period

191 es used RT-PCR and beta-galactosidase (LacZ) histochemistry of retinas from transgenic mice heterozyg

192 Enzyme histochemistry of the adult rat brain for ectonucleotida

193 ol or injured liver and also by quantitative histochemistry of tissue sections.

194 Histochemistry of tissues from CNC patients is indicativ

195 ther, the integration of OR-PAM with (immuno)histochemistry offers a simple and versatile technique w

196 antitative confocal analysis of isolectin B4 histochemistry on days 7 and 14.

197 ing microtubule-associated protein-2 (MAP-2) histochemistry on P28 while the animals were hypoxic (n=

198 Their axons were visualized by X-gal histochemistry or anti-beta-galactosidase immunofluoresc

199 In normal rats, synapses labeled by Timm histochemistry or dynorphin immunohistochemistry were ra

200 ned for CTb immunocytochemistry or for CytOx histochemistry or for Nissl.

201 e dinucleotide phosphate diaphorase (NADPHd) histochemistry or immunocytochemistry using an antibody

202 in production at a cellular level by in situ histochemistry or immunocytochemistry.

203 NADPH-diaphorase (NADPHd) histochemistry or NOS-immunostaining was combined with s

204 status of mutant mice, indicated by a lectin histochemistry pattern similar to that of wild-type mice

205 odologies: cytoarchitecture (cresyl violet), histochemistry (peanut agglutinin), immunocytochemistry

206 evelopment by means of in situ hybridization histochemistry, quantitative RT-PCR, and immunocytochemi

207 e specific lectin, and subsequent GNA lectin histochemistry refined the localization of N-glyans cont

208 mitochondria using immunohistochemistry and histochemistry, respectively, in chronic active and inac

209 In human amygdala, WFA lectin histochemistry resulted in labeling of perineuronal nets

210 aging, magnetic resonance imaging and immuno/histochemistry results show that the engineered ASCs act

211 Traditional microscopies in combination with histochemistry reveal glycogen accumulation within glia

212 In situ hybridization histochemistry revealed expression of Kir6.1/SUR2B mRNAs

213 In the Melibe brain, NADPH-d histochemistry revealed only a single pair of bilaterall

214 nses were improved (P<0.05) with Tempol, and histochemistry revealed oxidative stress in KW animals,

215 Quantitative in situ hybridization histochemistry revealed that baclofen (2.5 mg/kg, i.p.)

216 Histochemistry revealed that LNA-92a increased capillary

217 Immuno histochemistry revealed that the inclusions were immunor

218 Double-labeling immunofluorescence histochemistry revealed that the nNOS-like immunoreactiv

219 Cytochrome oxidase histochemistry reveals large-scale cortical modules in a

220 Furthermore, histochemistry reveals markedly increased NEDD4-1 immuno

221 d the absence of NTPDase2, and ATPase enzyme histochemistry reveals no reaction product in taste buds

222 es followed by their localization with DPPIV histochemistry showed 3- to 5-fold increases in the numb

223 weeks after infection, in situ hybridization histochemistry showed a pattern of chronic overexpressio

224 Oxidative histochemistry showed cytochrome c oxidase-deficient fib

225 Muscle histochemistry showed mitochondrial proliferation, and b

226 Immunocytochemistry/histochemistry showed that a TN-C-rich ECM surrounds Prx

227 Protein histochemistry showed that the tissue binding specificit

228 erminal sequences, and in situ hybridization histochemistry showed that these glypican-1 ligands are

229 In situ hybridization histochemistry shows that melanopsin expression is restr

230 In situ hybridization histochemistry shows that NAALADase-related mRNAs have a

231 We examined normal vessels and plaques by histochemistry, Southern blotting, and fluorescence in s

232 Immunofluorescence and enzymatic histochemistry staining were performed to assess puriner

233 Using enzymatic histochemistry staining, we also demonstrated functional

234 Histochemistry study and real-time PCR further confirmed

235 s from typical confounding factors common to histochemistry, such as variation in reagent penetration

236 the site of transection combined with TUNEL histochemistry suggested that neuronal death, including

237 s early lineage progenitors, undetectable by histochemistry, that leave the bone marrow to enter the

238 tional protein immunolocalization and lignin histochemistry, these results suggest that the distinct

239 Here we used cytochrome oxidase (CO) histochemistry to demonstrate that GAP-43 heterozygous (

240 resent report, we used in situ hybridization histochemistry to demonstrate that the 5-HT(3B) subunit

241 t's visual cortex using alkaline phosphatase histochemistry to demonstrate the capillary endothelial

242 In this study, we used in situ hybridization histochemistry to determine the change in the levels of

243 as analyzed using Western blotting and Golgi histochemistry to examine the hypothesized outcomes.

244 We used in situ hybridization histochemistry to examine the patterns of expression of

245 S-1 cortex by using postembedding immunogold histochemistry to examine the subcellular distribution o

246 ne dinucleotide phosphate (NADPH)-diaphorase histochemistry to identify populations of neurons contai

247 dinucleotide phosphate diaphorase (NADPH-d) histochemistry to identify the source of nitrergic inner

248 , we used double label in situ hybridization histochemistry to investigate the potential direct actio

249 Using serotonin transporter (5HT-T) histochemistry to label thalamocortical afferents (TCAs)

250 Here, we use X-gal histochemistry to map CRE-mediated gene transcription in

251 We used histochemistry to map differentially expressed proteins

252 boprobes, we performed in situ hybridization histochemistry to map the distribution of orexin recepto

253 henotype, we have used in situ hybridization histochemistry to map the IKAP mRNA in sections of whole

254 By using Fluoro-jade histochemistry to mark neurodegeneration and dual immuno

255 ogical techniques with in situ hybridization histochemistry to produce both 2D and 3D images and to v

256 odies) and fluorescent in situ hybridization histochemistry to search for Y chromosome-positive cells

257 We used established lectin histochemistry to show that PNNs undergo drastic reconst

258 In this study we used in situ hybridization histochemistry to show that TCF7L2 has a unique expressi

259 We used Wisteria Floribunda agglutinin histochemistry to visualize PNNs to investigate whether

260 in conjunction with cytochrome oxidase (CO) histochemistry, to investigate the distribution of thala

261 gment was performed after beta-galactosidase histochemistry using 0.1% to 1% potassium permanganate i

262 vid-19 patients was processed for hyaluronan histochemistry using a direct staining method and compar

263 The complementary technique of Southwestern histochemistry using a labeled Smad-binding element demo

264 In situ hybridization histochemistry using an antisense probe to this novel re

265 culture was studied by in situ hybridization histochemistry using an intron-specific VP heteronuclear

266 Immunofluorescence histochemistry using antibodies to three domains of the

267 s ('shape modules') were located by electron histochemistry using Cupromeronic blue methodology.

268 alamus and amygdala by in situ hybridization histochemistry using monkey-specific cRNA probes.

269 pecimens were assessed by immunofluorescence histochemistry using polyclonal antibodies specific for

270 lla and rat vestibular brainstem; diaphorase histochemistry was done in the chinchilla periphery.

271 Immunofluorescent histochemistry was employed to detect changes in peptidi

272 In situ hybridization histochemistry was performed for such "claustrum-enriche

273 dase (COX) and succinate dehydrogenase (SDH) histochemistry was performed on 46 EOM samples to determ

274 Double-label fluorescent histochemistry was used for IT and AVT (by using antibod

275 ative and double-label in situ hybridization histochemistry was used selectively to confirm a number

276 Histochemistry was used to assess hyalinosis in Klotho(-

277 First, isotopic in situ hybridization histochemistry was used to examine mRNA expression of pa

278 r immunohistochemistry or cytochrome oxydase histochemistry was used to reveal thalamic afferent patt

279 Using histochemistry, we demonstrated accumulation of mucus an

280 Using enzymatic histochemistry, we detected ecto-AMPase activity in dent

281 Finally, using Timm histochemistry, we detected progressive sprouting of mos

282 Using Fos histochemistry, we found changes in the activation of se

283 By using double in situ hybridization histochemistry, we found co-expression of the functional

284 d whole-mount placental alkaline phosphatase histochemistry, we found that itch-sensing skin arbors e

285 In conclusion, using in situ hybridization histochemistry, we have shown that mRNA for both the exc

286 in situ hybridization and immunofluorescence histochemistry, we show that hippocampal D4R mRNA and pr

287 l c-fos mRNA probe for in situ hybridization histochemistry, we systematically analyzed and identifie

288 dinucleotide phosphate diaphorase (NADPH-d) histochemistry were used to explore the existence of sex

289 Culture, 16S ribosomal DNA sequencing, and histochemistry were used to guide subsequent FISH.

290 n Pde6b(H620Q) homozygotes was documented by histochemistry, whereas PDE6beta expression and activity

291 g Holtzman rat pups using cytochrome oxidase histochemistry, which reflects long-term changes in brai

292 istochemistry and NADPH-diaphorase (NADPH-d) histochemistry, which yielded almost identical results e

293 s against NOS and NADPH-diaphorase (NADPH-d) histochemistry, which, with the exception of the primary

294 cts were processed for in situ hybridization histochemistry with (35)S-oligonucleotide probes for GAT

295 ion were determined by in situ hybridization histochemistry with a digoxigenin (DIG)-labeled antisens

296 We have shown by Western blotting and immuno-histochemistry with a polyclonal antibody to a specific

297 uleus were measured by in situ hybridization histochemistry with autoradiographic analysis.

298 Using double labeling fluorescent histochemistry with confocal microscopy we found TRalpha

299 In situ hybridization histochemistry with SPACRCAN riboprobes indicates that h

300 Conventional histochemistry yields rich morphological data from tissu