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1 TOH7 is dynamically expressed during retinal histogenesis.
2 k period before the onset of cerebral cortex histogenesis.
3 ily are essential for animal development and histogenesis.
4 ential for cell survival and normal cortical histogenesis.
5 is and support the conversion theory for OCS histogenesis.
6 dysregulation of genes involved in forebrain histogenesis.
7 ggesting that they play key roles in retinal histogenesis.
8 ity cues to the MC and for postnatal retinal histogenesis.
9 icular zone and is required for normal brain histogenesis.
10 st cell type to differentiate during retinal histogenesis.
11  CMZ during late stages of embryonic retinal histogenesis.
12 hose interactions direct cell sorting during histogenesis.
13 ending to their gene expression patterns and histogenesis.
14  assumed to underlie all defects in cortical histogenesis.
15  act as regulators of dynein during cortical histogenesis.
16 rturbed PTPmu dramatically disrupted retinal histogenesis.
17 ious genetic interaction between them in PVN histogenesis.
18 on of the pial basement membrane in cortical histogenesis.
19 ntrol of progenitor cell differentiation and histogenesis.
20 enitor cell proliferation throughout retinal histogenesis.
21 h embryos for mutations that disrupt retinal histogenesis.
22 the role of these mutant SCs in neurofibroma histogenesis.
23 ticularly during the later stages of retinal histogenesis.
24 st a unique role of pia in cerebellar cortex histogenesis.
25 for certain stages of central nervous system histogenesis.
26 bited, leading to a disruption of the tectal histogenesis.
27 nct phases of rod development during retinal histogenesis.
28 chanisms that play important roles in tissue histogenesis.
29  and neurite outgrowth during nervous tissue histogenesis.
30 ll-matrix interactions during nervous tissue histogenesis.
31 attern formation, neural differentiation and histogenesis.
32                 None of the abnormalities in histogenesis and axonal pathways were observed when the
33  heterogeneous group of diseases with varied histogenesis and biological behavior.
34                                              Histogenesis and classification are therefore uncertain.
35           Here, we study the relationship of histogenesis and clonal diversification in GCTs by analy
36  likely to reflect underlying differences in histogenesis and disease progression rather than growth
37 l forms of human liver disease; however, the histogenesis and function of DRs in liver injury are not
38 malignant stages of human cutaneous melanoma histogenesis and investigated its potential causative ro
39 s transiently expressed during early retinal histogenesis and is necessary for retinal ganglion cell
40  the cadherin family have been implicated in histogenesis and maintenance of cellular structure and f
41 ns enable chondrogenic mesenchyme to undergo histogenesis and morphogenesis, but precise molecular an
42 kewise, little information exists on the ear histogenesis and neurogenesis in many mutants.
43 death may have important implications in the histogenesis and pathology of the nervous system.
44 nt for creating descriptive models of tissue histogenesis and renewal in embryonic development and ad
45   Foxl2 disruption thus provides a model for histogenesis and reproductive competence of the ovary.
46  each sensory system, developmental times of histogenesis and the earliest ages of innervation and fu
47 clones, in a series of 54 gliomas of varying histogenesis and tumor grade.
48 hese findings challenge traditional views of histogenesis and tumor origin.
49 opathology and ultrastructure, controversial histogenesis, and enigmatic clinical behavior.
50 BMP receptor gene mutation alone, cerebellar histogenesis appears normal, thereby demonstrating funct
51 tal pathways underpinning GCT initiation and histogenesis are incompletely understood.
52  coordinate the control of cell division and histogenesis are poorly understood.
53      Vertebrate central nervous system (CNS) histogenesis depends on glia-guided migration of postmit
54        Among brain regions, human cerebellar histogenesis differs in complexity compared with nonhuma
55 helix transcription factor essential for GNP histogenesis, does not induce medulloblastomas when expr
56 on is disrupted, reveals abnormal cerebellar histogenesis due to the disturbance of multiple cellular
57  required in vivo during early stages of ENS histogenesis for the survival of undifferentiated enteri
58 nsitive first-line information about ovarian histogenesis for which no in vitro cell models are curre
59 ogy emerged, the cellular events of cortical histogenesis have been intensively scrutinized.
60 onship between PAX3-FKHR expression and ARMS histogenesis in primary tumors and cell culture systems.
61 and probably irreversible aberrations in the histogenesis in the developing central nervous system.
62 results suggest that progenitor behavior and histogenesis in the mammalian neocortex conform to a rem
63 d activation in vitro and disrupted cortical histogenesis in vivo in both mouse and ferret models, in
64             These results suggest a model of histogenesis in which the activity of factors that promo
65 have been reported in the literature and the histogenesis is controversial as, at the time of diagnos
66 mally in the mi/mi mutant, but later retinal histogenesis is dependent on the presence of a different
67                       In Drosophila, retinal histogenesis is precisely coordinated and is associated
68  an in vitro system in which much of retinal histogenesis is recapitulated.
69                                          Its histogenesis is still controversial.
70 tics have led to better understanding of the histogenesis, natural history, and molecular events that
71 e processes of size regulation in the brain, histogenesis, neuronal differentiation, and the maintena
72                        In vertebrate retina, histogenesis occurs over an extended period.
73 ondyle demonstrated two stratified layers of histogenesis of cartilaginous and osseous phenotypes.
74 ng of the biliary compartment to analyze the histogenesis of DRs and their potential contribution to
75  to Ewing's sarcoma and provide clues to the histogenesis of Ewing's sarcoma in bone.
76         The results provide insight into the histogenesis of GIST and the clinical behavior of this t
77 film and its derangements as well as for the histogenesis of mucus-producing carcinomas.
78                                          The histogenesis of PanIN and PDAC in both mice and men rema
79                             Furthermore, the histogenesis of photoreceptors and Muller glia is extend
80                                          The histogenesis of prostatic neuroendocrine cells is contro
81 genously expressed Wnt-13 could modulate the histogenesis of teratocarcinomas by mediating interactio
82  developing cerebellum and their role in the histogenesis of the cerebellar cortex.
83 vel cell adhesion molecule essential for the histogenesis of the embryonic ectoderm in Xenopus, and d
84  c-Ret function during renal development and histogenesis of the enteric nervous system.
85 n pathogenesis, most do not recapitulate the histogenesis of the human disease.
86                               We studied the histogenesis of the lizard visual system (E30 to adultho
87     Programmed cell death contributes to the histogenesis of the nervous system, and is believed to b
88 a regenerates, but little is known about the histogenesis of the new tissue, including the structure
89 sion mediates patterning, morphogenesis, and histogenesis of tissues in which they are expressed.
90 ession of ancient and conserved genes during histogenesis, organization of embryonic axes, and initia
91 profiles associated with drug resistance and histogenesis, patient immunity, and HIV dynamics and mut
92  division, with progressively later onset as histogenesis proceeds.
93                                              Histogenesis relies on cues that promote the chondrogeni
94 ational stages preceding and during cortical histogenesis results in the early onset of hypertrophic
95  a phase 2 study to assess the role of tumor histogenesis (subtype), fluorodeoxyglucose positron emis
96 basal lamina had dramatic effects on retinal histogenesis, such as an irreversible retraction of the
97 ght into the elusive molecular mechanisms of histogenesis, the conserved temporal order by which neur
98 mily of tumors (ESFT) shares a common neural histogenesis, tumor genetics, and a fascinating biology,
99 f DNMT1 in the regulation of postnatal liver histogenesis under homeostasis and stress conditions.
100 esis in a set of 50 human gliomas of various histogenesis, using cDNA microarrays, inferential and de
101            Finally, the role of primary-cell histogenesis was assessed by infecting primary cultures
102                              Also, forebrain histogenesis was preserved in mice with neuron-specific
103                                        Tumor histogenesis was the only characteristic associated with
104        To better understand hemangioblastoma histogenesis, we analyzed postmortem CNS tissues from fo
105 l migration and the consequences for retinal histogenesis when migration is impaired.
106                        The period of retinal histogenesis when SPACRCAN is detected first is coincide
107 ntral nervous system (CNS) tumors of unknown histogenesis, which can occur sporadically or in von Hip
108 e a dynamically changing process of abnormal histogenesis, which drives more changes; ii) the faulty
109                                 The first is histogenesis, which entails the production of cartilage
110 ish a causal relationship of proper cortical histogenesis with the presence of an intact pial basemen

 
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