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1 st-gluten challenge biopsies as quantitative histomorphometry.
2 Bone structure in the femur was assessed by histomorphometry.
3 4) was accomplished using computer-assisted histomorphometry.
4 ), were accomplished using computer-assisted histomorphometry.
5 in the optic nerve were compared by detailed histomorphometry.
6 biochemical markers of bone resorption, and histomorphometry.
7 the width of the wound by computer-assisted histomorphometry.
8 in the wound boundaries by computer-assisted histomorphometry.
9 was not significantly different from that at histomorphometry.
10 the right tibiae were removed on day 28 for histomorphometry.
11 IH volumes correlated strongly with ex vivo histomorphometry.
12 e assessed with biochemical markers and bone histomorphometry.
13 als were given fluorochrome bone markers for histomorphometry.
14 cal assays coupled with quantitative dynamic histomorphometry.
15 sorptiometry, micro-computed tomography, and histomorphometry.
16 puted tomography (uCT) and quantitative bone histomorphometry.
17 using biochemistry, noninvasive imaging, and histomorphometry.
18 ures by micro computed tomography (muCT) and histomorphometry.
19 outgrowth after 4 weeks was quantified using histomorphometry.
20 using quantitative coronary angiography and histomorphometry.
21 n optical coherence tomography (SD-OCT), and histomorphometry.
22 ound, invasive hemodynamic measurements, and histomorphometry.
23 calcified histology and cellular and dynamic histomorphometry.
24 aluated mineral and bone disorders with bone histomorphometry.
25 ation parameters were assessed using dynamic histomorphometry.
26 quantitative time-lapse imaging with dynamic histomorphometry.
27 sessed by micro-computed tomography (CT) and histomorphometry.
28 emoved and atrial thrombi were quantified by histomorphometry.
29 joints as demonstrated by histology and bone histomorphometry.
30 lar inflammatory infiltrate and quantitative histomorphometry.
31 o the cell-free zone using computer assisted histomorphometry.
32 was scored using a qualitative scale and by histomorphometry.
33 , and 9 was analyzed using computer-assisted histomorphometry.
34 d 12 mo as measured by bone densitometry and histomorphometry.
35 , bone mineral density measurement, and bone histomorphometry.
38 ced after TbetaRI-I treatment as detected by histomorphometry analysis compared with the placebo cont
40 teopenia of Notch2(tm1.1Ecan) mice, and bone histomorphometry analysis revealed decreased osteoclast
43 lent to intact bone but quantitative dynamic histomorphometry and cellular activity assays demonstrat
44 ified glycogen by Periodic-Acid Schiff (PAS) histomorphometry and colorimetric quantitative assay sho
47 days, capillary density, vascular diameter, histomorphometry and immunohistochemistry at the transec
50 maging, histology (H&E, Masson's Trichrome), histomorphometry and immunohistology (Tartrate-Resistant
52 post-surgery and analyzed through histology, histomorphometry and micro-computed tomography (muCT).
55 stingly, osteoblast activity, as measured by histomorphometry and osteocalcin expression, is strongly
56 ems pathology analysis program that includes histomorphometry and quantitative multiplex biomarker as
58 Clinical outcomes were determined, and renal histomorphometry and sequencing of Mendelian nephrotic s
60 containing M101 (1 and 2 g/L) groups through histomorphometry and TRAP (tartrate-resistant acid phosp
62 ic and SD-OCT volumes (8 microm, range 4-19, histomorphometry, and 10 microm, range 4-26, SD-OCT) and
63 examined by a combination of micro-CT, bone histomorphometry, and biomechanical testing and compared
64 al quantitative computed tomography, by bone histomorphometry, and by measurements of bone cell apopt
65 ensity using micro-computed tomography, bone histomorphometry, and characteristics of primary bone ma
66 s were examined blindly by light microscopy, histomorphometry, and color computer image analysis.
68 riodontal tissues were analyzed by micro-CT, histomorphometry, and immunohistochemistry for TRAP.
69 osteonectin in bone, we used contact x-ray, histomorphometry, and Northern blot analysis to characte
71 yzed using dual energy x-ray absorptiometry, histomorphometry, and vertebral compression testing.
74 etrics such as nerve conduction velocity and histomorphometry are necessary to improve prediction and
84 stration were in good agreement with ex vivo histomorphometry (Elastica van Gieson stain) and gadolin
89 , including proteinuria, blood pressure, and histomorphometry, fall short at capturing the complexity
90 ere we developed a framework for large-scale histomorphometry (FLASH) performing deep learning-based
92 ing of periodontal bone level (PBL) loss and histomorphometry for inflammatory cell infiltration and
94 ed, which can include descriptive histology, histomorphometry, immunostaining, 3D bone imaging, elect
98 hod is likely to eventually replace invasive histomorphometry in that it obviates the need to sacrifi
100 o-computed tomography (micro-CT), histology, histomorphometry, in situ hybridization (ISH), immunohis
104 ough sex-specific differences were observed, histomorphometry measurements revealed that both bone re
107 luated; alveolar bone loss was determined by histomorphometry, morphometry, and microcomputed tomogra
109 2(-/-) and Fgf2(+/+) mice were determined by histomorphometry, nanoindentation, and quantitative reve
113 one trabecular architecture using 3D digital histomorphometry of microcomputed tomography data from t
114 earning model for automated segmentation and histomorphometry of myelinated peripheral nerve fibers f
115 old stimuli and correlated with quantitative histomorphometry of myocardial architecture and connexin
122 ing and osteoclast surface, as determined by histomorphometry of the femur; increased urinary deoxypy
146 ostsurgery were measured using densitometry, histomorphometry, scanning electron microscopy (SEM), an
153 Compared to manual morphometry, automated histomorphometry showed superior agreement with the refe
155 Periodontal tissue response was assessed by histomorphometry, tartrate-resistant acid phosphatase hi
156 st number, and inflammation were assessed by histomorphometry, tartrate-resistant acid phosphatase hi
158 on cancellous bone structure from iliac bone histomorphometry that led to the demonstration that arch
159 roperties of the specimens, and we used bone histomorphometry to assess parameters of bone microstruc
160 ings at histologic examination, quantitative histomorphometry, transmission electron microscopy, and
161 n, proximal aortic lesion size quantified by histomorphometry was 5-fold-reduced in chow-fed ApoE+/-/
162 n, proximal aortic lesion size quantified by histomorphometry was 9-fold greater in chow-fed mice ino
165 d 7 PVOD) or surgery (10 PVOD), quantitative histomorphometry was performed in all analyzable arterie
169 as assessed by biochemical markers and bone histomorphometry, was markedly decreased at both ages.
170 By undecalcified histology and bone-specific histomorphometry we found that high circulating sgp130Fc
171 Following culture, cell viability and tissue histomorphometry were assessed with quantification of ma
174 a from qualitative analysis and computerized histomorphometry were statistically processed at a signi