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1 viduals with LTB with or without concomitant hookworm infection.
2 but not IL-4-driven Type 2 responses during hookworm infection.
3 SP-2-specific IgE likely induced by previous hookworm infection.
4 ency anemia that is the clinical hallmark of hookworm infection.
5 on the risk of an individual harboring heavy hookworm infection.
6 anti-ASP-2 IgE levels and the risk of heavy hookworm infection.
7 important new tool for the control of human hookworm infection.
8 -1 is a possible drug and vaccine target for hookworm infection.
9 are efficacious vaccines in animal models of hookworm infection.
10 of Australia who were suffering from endemic hookworm infection.
11 weight loss and anemia caused by a secondary hookworm infection.
12 27 to control the early lung larval stage of hookworm infection.
13 immunity and restricts lung injury following hookworm infection.
14 species for examining host immunity to human hookworm infections.
15 ide showed activity against T. trichiura and hookworm infections.
16 afety of emodepside against T. trichiura and hookworm infections.
17 s expressing IFNgamma were reduced following hookworm infection (23.9%-11.5%; P = .04), with correspo
19 e association between Plasmodium-species and hookworm infection among preschool-aged children (odds r
20 s highlight the importance of neutrophils in hookworm infection and a potential conserved mechanism o
21 We examined the association between maternal hookworm infection and birth outcomes in a cohort of wom
27 s observed among children without detectable hookworm infection, but no association was observed amon
28 risk of wheeze was independently reduced by hookworm infection by an odds ratio of 0.48 (95% CI 0.24
29 erences between the immune responses against hookworm infection by patients from epidemic versus none
31 er and can deliver higher protection against hookworm infection compared with the clinical vaccine ca
33 ngly suggest nutritional impacts of maternal hookworm infection during pregnancy may be divergent bas
35 g human immunity to both schistosomiasis and hookworm infection has been associated with IgE response
39 t loss and anemia) of Ancylostoma ceylanicum hookworm infection in Syrian golden hamsters of the outb
41 pathogenesis by allowing the study of adult hookworm infections in a species with well-characterized
42 ty of ascending tribendimidine doses against hookworm infections in African school-aged children, key
44 ficant role for host genetics in determining hookworm infection intensity has also been shown, but th
52 escence-activated cell sorting revealed that hookworm infection is associated with reduced percentage
55 nd 73% of severe anemia were attributable to hookworm infection; < 10% of anemia was attributable to
58 hamsters (i.e., those with neither previous hookworm infections nor treatment) were also vaccinated.
60 sed to determine the impact of A. ceylanicum hookworm infection on host cytokine responses and lympho
62 so we examined the influence of experimental hookworm infection on the predicted outcomes of escalati
63 he effect of concurrent (active) and treated hookworm infections on the immunogenicity of vaccination
64 ng shoes was associated with reduced odds of hookworm infection (OR 0.29, 95% CI 0.18-0.47) and infec
66 5% confidence interval [CI], 1.03-2.14), and hookworm infection (OR, 1.77; 95% CI, 1.22-2.55) were th
69 found to protect BALB/c mice against rodent hookworm infections, resulting in worm burden reductions
70 lopment of NTD vaccines, including those for hookworm infection, schistosomiasis, leishmaniasis, and
71 demonstrate that the presence of coincident hookworm infection significantly diminished both spontan
72 rticipants who were cured of T. trichiura or hookworm infection (the cure rate) with emodepside 14 to
74 ring chronic helminth infection; at least in hookworm infection, this suppression may protect against
76 iron restriction mediates susceptibility to hookworm infection using the hamster model of Ancylostom
78 1.34; 95% CI, 1.05-1.71; 20 studies), while hookworm infection was associated with a significantly s
81 h P. falciparum infection, but the effect of hookworm infection was seen only in the absence of M. pe