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1 mide (C(MAX) = 28,900 ng/ml and T(1/2) = 8.0 hr)) were implemented in a hollow-fiber system.
2  was 4.2 hours (interquartile range, 2.7-8.0 hr), divided into a median prehospital delay of 0.52 hou
3 to mesenchymal stem/stromal cell infusion (0 hr), 1 hour, 4 hours, 12 hours, 24 hours, and 72 hours a
4 16.5 hr) compared with 170.4 hours (70-1,008 hr) on extracorporeal membrane oxygenation.
5 erage of one every 3.6 days to one every 2.1 hr.
6 was similar for both groups (150 mL/min: 9.1 hr [5.5-26 hr] vs 10 hr [4.2-17 hr]; p = 0.37).
7  induced initially by colistin (15 min and 1 hr) and later by doripenem (4 hr).
8          Colistin caused early (15 min and 1 hr) disruption of the bacterial outer membrane and cell
9 al prostates despite undetectable free BPA 1 hr postexposure.
10                                     Brief (1 hr) periods of postsynaptic bursting selectively depress
11 in sera of individual PND 3 pups collected 1 hr postexposure utilizing ultra-high-pressure tandem mas
12      We found that stimulation at 1 Hz for 1 hr resulted in an almost complete suppression of spontan
13 rst and then sintered at 150 degrees C for 1 hr.
14                    Among those who spent > 1 hr/day outdoors or who had lived 5 or more years at thei
15 t enrollment address and those who spent > 1 hr/day outdoors.
16 ctivity with recovery to near reference in 1 hr, followed by a slow activity decay to a stable, level
17 on-associated proteins in serum obtained: <1 hr (within 1-hour), 4-12 hr and 48-72 hr post injury.
18 ble to discriminate between TBI and HV at <1 hr.
19 L) alone, and their combination at 15 min, 1 hr and 4 hr (n = 4).
20 ontractility of individual cells for up to 1 hr after seeding, while collective forces on longer time
21 male was given access to the female within 1 hr.
22 ide (0.00012 +/- 0.00004 mumol N(2)O gDW(-1) hr(-1)) and carbon dioxide (3.556 +/- 0.471 mumol CO(2)
23 dioxide (3.556 +/- 0.471 mumol CO(2) gDW(-1) hr(-1)).
24 observers wore bright red glasses for five 1-hr periods per day, for 5 days.
25  appeared less and less reddish during the 1-hr periods of glasses wear.
26  anuran assemblages varied more within the 1-hr resolution than the 3-hr resolution.
27 groups (150 mL/min: 9.1 hr [5.5-26 hr] vs 10 hr [4.2-17 hr]; p = 0.37).
28 for covariates, women in the highest (>= ~11 hr/day) vs. the lowest (<= ~9 hr/day) quartile of sedent
29 timepoint images of embryos collected at 113 hr post-insemination, in embryo selection amongst 97 cli
30  serum obtained: <1 hr (within 1-hour), 4-12 hr and 48-72 hr post injury.
31  the evening (LNd) group by approximately 12 hr; PDF alone delays the phase of the DN3 group by appro
32 n primary respiratory epithelial cells at 12 hr p.i.
33  mortality (area under the curve, 0.77 at 12 hr up to 0.93 at 5-10 d after cardiogenic shock onset).
34 ngth significantly predicted major delay (12 hr: odds ratio, 6.98; CI, 2.33-20.89; 8 hr: odds ratio,
35 igned to one of four groups (n = 6/group; 12 hr): 1) no spontaneous breathing activity and positive e
36 and 70 patients with circulatory shock (< 12 hr duration), defined as the need for vasopressors to ma
37 ion of a whole mouse brain connectome in <12 hr.
38 s of artificial kidney failure: early (<= 12 hr), intermediate (> 12-24 hr), and late (> 24 hr) in 35
39 .5 hr/d) to an extended visitation model (12 hr/d).
40 e was 0.74, whereas for two samples taken 12 hr apart, R(2) was 0.90.
41 stis sp. PCC 6803 metabolism that spans a 12-hr light/12-hr dark cycle by segmenting it into 12 Time
42  6803 metabolism that spans a 12-hr light/12-hr dark cycle by segmenting it into 12 Time Point Models
43  oxygenation (< 66 hr: odds ratio, 1; 66-128 hr: odds ratio, 0.281; 95% CI, 0.101-0.777; p = 0.014; 1
44  and a high turnover frequency (483 and 1336 hr(-1) at 1-bar and 3-bar pure CH(4), respectively).
45 ian shift duration was 12 hours (range, 7-14 hr), and the median number of consecutive shifts allowed
46 utive shifts allowed was 7 hours (range 7-14 hr).
47  exposure to a natural winter 9 hr 20 min:14 hr 40 min light-dark cycle as compared to the modern ele
48 e regional and age-specific prevalence of 14 hr-HPV genotypes as well as their prevalence in abnormal
49 er spending a weekend camping in a summer 14 hr 39 min:9 hr 21 min natural light-dark cycle compared
50 y shown that exposure to a natural summer 14 hr 40 min:9 hr 20 min light-dark cycle entrains the huma
51 omplex genes was assessed in the lungs at 16 hr after the end of each exposure.
52  mL/min: 9.1 hr [5.5-26 hr] vs 10 hr [4.2-17 hr]; p = 0.37).
53 e phase of the DN3 group by approximately 17 hr.
54 ration of prone position was 14 hours (12-18 hr).
55            Here, we conducted a series of 18-hr short-term acute heat stress assays side-by-side with
56 me was 96 hours (interquartile range, 26-181 hr).
57  compared with the reference category (< 1.2 hr).
58 isoniazid C(MAX) = 1,200 ng/ml, T(MAX) = 2.2 hr and T(1/2) = 4.7 hr), and two combination therapy pro
59 al activation (GFAP and CD68) and HMGB1 at 2 hr, 6 hr, 24 hr, 3 days, and 7 days postinjury, and seru
60 oading (n = 8, isotonic saline, 1 L/hr for 2 hr) or diuretic treatment (n = 8, furosemide, 40 mg ever
61 ed for ROCK-mediated cell contraction from 2 hr post infection.
62 etected with beta-tubulin immunoreactivity 2 hr after stimulation.
63 rgeted analysis of unfractionated lysates (2 hr) accurately reproduces the quantification of fraction
64 3 sequential nuclear cleavages within only 2 hr of fertilization.
65                    The PET scan took place 2 hr after the induction of acute psychosocial stress usin
66                                     Within 2 hr of immature INP birth, downregulation of repressor ac
67 co) between burnt (432 +/- 83 mg CO(2) m(-2) hr(-1) ) and intact PSF (359 +/- 76 mg CO(2) m(-2) hr(-1
68 e-affected areas (7.8 +/- 2.2 mg CH(4) m(-2) hr(-1) ) compared to the intact PSF (4.0 +/- 2.0 mg CH(4
69 o the intact PSF (4.0 +/- 2.0 mg CH(4) m(-2) hr(-1) ) due to prolonged higher WT and more optimal met
70 ly higher CH(4) flux (0.88 +/- 0.03 mg m(-2) hr(-1) ) than spring thaw (0.48 +/- 0.04 mg m(-2) hr(-1)
71  ) and intact PSF (359 +/- 76 mg CO(2) m(-2) hr(-1) ).
72 ) ) than spring thaw (0.48 +/- 0.04 mg m(-2) hr(-1) ).
73 s , 78.17 +/- 1.04%, 11.23 +/- 1.05 mg/cm(2)/hr, and 12.78 +/- 0.32%, respectively, while these value
74 s , 76.17 +/- 0.76%, 10.17 +/- 0.21 mg/cm(2)/hr, and 14.37 +/- 0.42%, respectively.
75  by routine histology) in the OM, at 2 or 20 hr after termination of NA exposure, in humanized mice.
76 ngle doses of 800, 300, 100, 50, or 20 mg 21 hr post-CHMI.
77 membrane oxygenation circuit at day 1 (10-23 hr), day 3, and day 7 of extracorporeal membrane oxygena
78 281; 95% CI, 0.101-0.777; p = 0.014; 128-232 hr: odds ratio, 0.474; 95% CI, 0.191-1.174; p = 0.107; a
79 4; 95% CI, 0.191-1.174; p = 0.107; and > 232 hr: odds ratio, 1.084; 95% CI, 0.429-2.737; p = 0.864; o
80                                           24 hr urine albumin excretion correlated highly with decrea
81 ure: early (<= 12 hr), intermediate (> 12-24 hr), and late (> 24 hr) in 35%, 31%, and 34% of circuits
82 of RPB1 phosphorylation, however, took 12-24 hr.
83 diastolic pressures (p = 0.18; p = 0.14); 24 hr.
84  by time to intubation (<= 8 hr: 38.2%; 8-24 hr: 31.6%; >= 24 hr: 38.1%; p = 0.7), and there was no a
85 re compared to the starting values (after 24 hr).
86 control trials (RCT) reporting office and 24 hr.
87 rcadian clocks have evolved to anticipate 24 hr rhythms in environmental demands.
88 rcadian clock that promotes approximately 24 hr rhythms in many behavioral and physiological processe
89                 Circadian ( approximately 24 hr) clocks regulate daily rhythms in physiology, metabol
90 mulation produced lasting ( approximately 24 hr) enhancement of recollection precision, without effec
91             Its circadian ( approximately 24 hr) oscillations of gene expression and electrical activ
92   The mucosal transcriptional response at 24 hr revealed the involvement of activated T cells, which
93                       Similarly, combined 24 hr urine:total body water ratios for all analytes were s
94 d lung cells harvested for flow cytometry 24 hr later.
95 and at the end of the in vivo experiment (24 hr after peritonitis induction).
96 that exhibited of 4,500- fold expression, 24 hr following exposure to the toxic compound 5-hydroxymet
97 /FIO2 (change in PaO2/FIO2 over the first 24 hr) were associated with mortality, whereas pressure var
98 sed in memory accuracy over the following 24 hr.
99                     Light stimulation for 24 hr selectively increased dendritic spine density and AMP
100 on >/= 90%) and "hyperoxia" (FIO2 1.0 for 24 hr) groups.
101                               Fasting for 24 hr, which increased Npy expression in the Arc, failed to
102   Using a conventional dosing of 16 g/2 g/24 hr continuous infusion, obese patients were more likely
103 as inadequate in both groups and 16 g/2 g/24 hr was adequate only in nonobese patients.
104  Carlo simulations showed that 12 g/1.5 g/24 hr was inadequate in both groups and 16 g/2 g/24 hr was
105  longer different from the control groups 24 hr later.
106 ), intermediate (> 12-24 hr), and late (> 24 hr) in 35%, 31%, and 34% of circuits, respectively.
107 s, or prolonged mechanical ventilation (> 24 hr).
108 ation (<= 8 hr: 38.2%; 8-24 hr: 31.6%; >= 24 hr: 38.1%; p = 0.7), and there was no association betwee
109  (GFAP and CD68) and HMGB1 at 2 hr, 6 hr, 24 hr, 3 days, and 7 days postinjury, and serum was collect
110 gly, RNA pol II-mediated RNA synthesis in 24 hr-treated cells was upregulated, indicating that, in co
111 ontrol studies showed a modest benefit in 24 hr.
112             Serum creatinine peaked late (24 hr), when clinical recovery was imminent.
113 ected for total body water and the sum of 24 hr urine plus total body water metabolites of PA, TYR, H
114  combination with colchicine treatment or 24 hr fasting, to assess NPY/Npy expression locally in the
115     Consumption of turkey during the past 24 hr was not associated with total arsenic or DMA during t
116 itatory synapses, whereas more prolonged (24 hr) firing depressed both AMPAR and NMDAR EPSCs and elim
117  ON or OFF medication, with memory tested 24 hr later ON or OFF medication (4 conditions, within-subj
118  further experiments were run without the 24 hr delay, but they too failed to reproduce effects of do
119 nged from -29 to + 40% in relation to the 24 hr mean of Rs, especially in trenched plots.
120 showed that among sham controlled trials, 24 hr.
121  s) or lower dose heparin (up to 12,000 U/24 hr aiming for activated partial thromboplastin time < 45
122  was tested in a semiarid shrubland using 24 hr campaigns using two treatments: trenched plots and pl
123 orphological changes in LX-2 cells within 24 hr post-infection with HCV, HIV or HCV/HIV in MLH co-cul
124 se bats will exchange fat reserves within 24 hr, meaning that they must survive on the food of the da
125 entiation, completed by most cells within 24 hr.
126 posure-response analysis suggested that a 24-hr post-dose concentration of KAF156 of 21.5 ng/mL (90%
127 ariate method is based on collection of a 24-hr time series of plasma melatonin, a suprachiasmatic nu
128  Drosophila brain confer an approximately 24-hr rhythm onto locomotor activity are unclear, but invol
129 idase (PO) response and bacterial load at 24-hr postinfection were ascertained.
130 m was to evaluate the association between 24-hr dietary recall of poultry consumption and arsenic exp
131  for both groups (150 mL/min: 9.1 hr [5.5-26 hr] vs 10 hr [4.2-17 hr]; p = 0.37).
132 erized by recessive inheritance of a long 26 hr period phenotype, encodes an RNA-binding protein that
133 2 mRNA rhythms were delayed by 0.97 +/- 0.29 hr (p < 0.01), indicating that human molecular clocks ma
134 lucose rhythms were delayed by 5.69 +/- 1.29 hr (p < 0.001), and average glucose concentration decrea
135 erquartile range]) life at 12.9 hr [5.5-21.3 hr]) hours than access dysfunction-free circuits (18.8 h
136  was 2.0 hours (interquartile range, 1.3-3.3 hr).
137                An initial peak occurred at 3 hr post-administration, a secondary peak at 5 days, and
138 ic profile, with an initial plasma peak at 3 hr, followed by a rapid decline to a sub-therapeutic con
139 eral anesthetic, isoflurane (ISO, 1.5% for 3 hr), at three early postnatal ages (P3, P5 and P7).
140  ST2, and then exposed to ozone (2 ppm for 3 hr).
141 ved using the coarser temporal resolution (3 hr).
142  (5x more cells) and longer lasting (up to 3 hr) than rats.
143 te) using two temporal resolutions (1- and 3-hr intervals).
144 d more within the 1-hr resolution than the 3-hr resolution.
145 perature management and rewarming (median 34 hr from ICU admission).
146 d their fetuses were sacrificed from 5 to 35 hr after exposure.
147 nts' circadian rhythms were measured in a 37-hr constant routine that removes sleep and environmental
148 edian of 13 hours (interquartile range, 4-38 hr) of noninvasive ventilation.
149 an mechanical ventilation time (25.2 vs 19.4 hr; p < 0.01), cardiac surgical ICU readmissions (5.3% v
150 nged emergency department to ICU time (> 2.4 hr) is associated with increased hospital mortality afte
151 ysfunction-free circuits (18.8 hr [10.1-33.4 hr]; p < 0.0001).
152  and their combination at 15 min, 1 hr and 4 hr (n = 4).
153 ycan biosynthesis metabolites decreased at 4 hr by doripenem alone, reflecting its mechanism of actio
154  (15 min and 1 hr) and later by doripenem (4 hr).
155 teoglycan components of PNNs were enhanced 4 hr after fear conditioning but were no longer different
156 n occupationally relevant dose (10 ppm for 4 hr).
157 s)isolated from the circulating blood from 4 hr after injection and over the next week.
158 s resulted in rapid defervescence (median, 4 hr) and clinical improvement.
159 oth days of exposure (air or 0.8ppm ozone, 4 hr/day x 2-days).
160 UVEC retention within the engraftment site 4 hr post-administration was 24.5 +/- 3.2%.
161 to ectopic Erk signaling, except from 1 to 4 hr post-fertilization, when perturbing the spatial exten
162 f promoter-anchored chromatin loops within 4 hr after inducing differentiation of 3T3-L1 preadipocyte
163 ive memory (word pair associates) across a 4-hr daytime training-retest interval.
164 rdiac arrest to inclusion was 15 hours (6-44 hr).
165 tion was 15 hours (interquartile range, 2-45 hr).
166 patients than healthy patients (4.90 vs 1.48 hr) and was correlated with encephalopathy severity by G
167 g the phototransduction pathway within 24-48 hr.
168 tained from L. plantarum after 8, 24, and 48 hr incubation.
169 vity drops precipitously during the first 48 hr of differentiation, corresponding to a release of C/E
170 her the relationship between early (first 48 hr) net ultrafiltration and mortality is mediated by flu
171 ried in their ratio of protein to fat for 48 hr and then injected with P. luminescens.
172 2 patients (57%) awoke: late awakening (> 48 hr from sedation stop; median time to awakening 5 days [
173 day/5 days) and then challenged (5 mg/kg, 48 hr later) with cocaine.
174 ere, we show that resveratrol (10 microM, 48 hr) induces both a cell growth arrest and a metabolic re
175 triplicate, analyzing 180 samples in only 48 hr (the equivalent of 16 min/sample).
176                  Postshock resuscitation (48 hr) comprised retransfusion of shed blood, crystalloids
177  tshawytscha), and find that in less than 48 hr, males can upregulate sperm velocity when faced with
178     The analysis, which was completed in 1.5 hr, included loading data, annotation, principal compone
179 cardiopulmonary bypass was 4.6 hours (2-16.5 hr) compared with 170.4 hours (70-1,008 hr) on extracorp
180 anagement was implemented faster (1.9 vs 3.5 hr from 911 call; p < 0.001) and more frequently in the
181 ed KCNQ1 surface-density declined with a 3.5 hr t1/2 by impaired forward trafficking.
182 licy from a restricted visitation model (4.5 hr/d) to an extended visitation model (12 hr/d).
183 y of 3.6 hours (interquartile range, 2.1-7.5 hr).
184 me on ventilator (difference of median, 98.5 hr; p = 0.003) and length of ICU stay (difference of med
185 e assembled a novel MRI dataset containing 5 hr of RSFC data, 6 hr of task fMRI, multiple structural
186 andling was used to sleep deprive mice for 5 hr, from lights on (7 am) to 12 noon.
187 h different post-mortem time delays (up to 5 hr) to mimic the way the human brain tissue is usually p
188            We investigated the effect of a 5-hr delay in meals on markers of the human master clock a
189 s (breakfast, lunch, dinner) were given at 5-hr intervals, beginning either 0.5 (early) or 5.5 (late)
190 rds having a AHI-ODI difference exceeding >5/hr, and 6% exceeding >10 events/hr.
191 ys for 24-hour intervals (median time, 18.52 hr); 31 (26%) for 12-hour intervals (median, 10.58 hr);
192 113 (72%) for 6-hour intervals (median, 9.55 hr).
193 1 (26%) for 12-hour intervals (median, 10.58 hr); 117 (47%) for 8-hour intervals (median, 9.60 hr); a
194 ; 8 hr: odds ratio, 23.70; CI, 8.13-69.11; 6 hr: odds ratio, 71.95; CI, 25.13-206.0).
195 anges in inundation duration between 2 and 6 hr/day did not affect SOM decay, the treatment without i
196 ng of the inundation duration to 0, 2, and 6 hr/day), and their interaction.
197  MRI dataset containing 5 hr of RSFC data, 6 hr of task fMRI, multiple structural MRIs, and neuropsyc
198 se in circulating WBCs, augment the early (6 hr postcecal ligation and puncture) recruitment of infla
199 d hydrocortisone at a 50 mg IV bolus every 6 hr and fludrocortisone as a 50 mug tablet once daily).
200 IV acetaminophen treatment (15 mg/kg every 6 hr).
201 icting severe oliguria (< 0.3 mL/kg/hr for 6 hr) occurring within the next 12 hours were tested in th
202 12 wk at a concentration of 15 mg/m(3) for 6 hr/d for 5 consecutive d/wk.
203 ivation (GFAP and CD68) and HMGB1 at 2 hr, 6 hr, 24 hr, 3 days, and 7 days postinjury, and serum was
204 117 (47%) for 8-hour intervals (median, 9.60 hr); and 113 (72%) for 6-hour intervals (median, 9.55 hr
205 f 0.52 hours (interquartile range, 0.40-0.66 hr) and a median emergency department delay of 3.6 hours
206 of extracorporeal membrane oxygenation (< 66 hr: odds ratio, 1; 66-128 hr: odds ratio, 0.281; 95% CI,
207 odds ratio, 1.29; 95% CI, 1.02-1.64 (2.4-3.7 hr) and odds ratio, 1.54; 95% CI, 1.11-2.14 (> 3.7 hr),
208 d odds ratio, 1.54; 95% CI, 1.11-2.14 (> 3.7 hr), both compared with the reference category (< 1.2 hr
209 ,200 ng/ml, T(MAX) = 2.2 hr and T(1/2) = 4.7 hr), and two combination therapy protocols including dru
210 predefined time points (3, 6, 12, 24, 48, 72 hr), animals (>/= 6 per group) underwent echocardiograph
211 ed: <1 hr (within 1-hour), 4-12 hr and 48-72 hr post injury.
212 e quantification of fractionated lysates (72 hr analysis) while obviating the need for peptide detect
213 ter (PM2.5) and carbon monoxide (CO) over 72 hr among a cohort of Tanzanian pregnant women.
214 ave setup for the first time a long-term (72 hr) respiratory distress syndrome model in spontaneously
215 50,000 simulated exomes and it required 1.75 hr for the analysis of a quantitative trait using severa
216 ane administration was 3.33 hours (2.33-5.75 hr), range 1-19 hours with a mean consumption of 7.89 mL
217  than access dysfunction-free circuits (18.8 hr [10.1-33.4 hr]; p < 0.0001).
218  (12 hr: odds ratio, 6.98; CI, 2.33-20.89; 8 hr: odds ratio, 23.70; CI, 8.13-69.11; 6 hr: odds ratio,
219 y did not differ by time to intubation (<= 8 hr: 38.2%; 8-24 hr: 31.6%; >= 24 hr: 38.1%; p = 0.7), an
220 n duration of monitoring was 47 hours (15-88 hr), and median duration from cardiac arrest to inclusio
221  (median [interquartile range]) life at 12.9 hr [5.5-21.3 hr]) hours than access dysfunction-free cir
222 ighest (>= ~11 hr/day) vs. the lowest (<= ~9 hr/day) quartile of sedentary time had higher risk for C
223  exposure to a natural summer 14 hr 40 min:9 hr 20 min light-dark cycle entrains the human circadian
224 a weekend camping in a summer 14 hr 39 min:9 hr 21 min natural light-dark cycle compared to a typical
225 tored for spontaneous, convulsive seizures 9 hr/day every day for 24-36 days.
226 fter a week's exposure to a natural winter 9 hr 20 min:14 hr 40 min light-dark cycle as compared to t
227 ained and applied on a large dataset (28,939 hr of data) using a stratified 20-fold cross-validation.
228 t-drop and were euthanized 8, 24, 48, 72, 96 hr, 7, 14 and 30 days later.
229 irrored in cellular responses observed at 96 hr in isolated mucosal T cells.
230 ology to examine the transcriptome of the 96 hr developmental program.
231 ventilation (weighted mean difference, -2.98 hr [95% CI, -5.08 to -0.89]; I = 34%; n = 334, five tria
232 perate at a sorting speed of up to 125 beads/hr (~3.5 times faster than a trained operator) providing
233       Times of symptom-onset-to-baseline-CT (hr) (median [interquartile range]: 3 [4] vs 5 [5]; p = 0
234  performed an associative memory task during hr-fMRI in which they encoded and later retrieved face-n
235 atients with OSA on placebo (AHI > 10 events/hr), AHI was lowered by 74% (62-88%) (P < 0.001) and all
236 exceeding >5/hr, and 6% exceeding >10 events/hr.
237 sleep apnea (apnea-hypopnea index > 5 events/hr) were administered an antioxidant or placebo cocktail
238 , 8.1 +/- 2.2 years, AHI:11.1 +/- 5.9 events/hr), and 200 control subjects (84 male, 8.2 +/- 2.0 year
239  recessive ppd mutants on a spring-flowering hr mutant background show early, photoperiod-insensitive
240 les with a CV < 5% at a rate as high as 60 g/hr (>1 trillion particles/hour).
241 acquired class A infections were detected in hr-MSM only.
242 = 0.015), time from admission to intubation (hr) (unit odds ratio, 1.01; 1.01-1.03; p = 0.017), and u
243                 Approximately 6.2 +/- 1.1 kg hr(-1) was emitted from off-road diesel activities withi
244 nds facilities, and an additional 116-186 kg hr(-1) formed from the photochemical oxidation of diesel
245 icate that AP infusion at the dose of 25U/kg/hr corrects serum and tissue AP deficiency and may preve
246 ses of bovine intestinal AP (1, 5, or 25U/kg/hr).
247 inimum urine output between 0.3 and 0.5mL/kg/hr), or 3) severe acute kidney injury (serum creatinine
248 , 0.15 mg/kg/hr (3.7 mg/kg/d) and 0.11 mg/kg/hr (2.8 mg/kg/d), increased by 36% and 58%, respectively
249  doses on the day of cannulation, 0.15 mg/kg/hr (3.7 mg/kg/d) and 0.11 mg/kg/hr (2.8 mg/kg/d), increa
250 g/kg/hr opioids (9.7 mg/kg/d) and 0.39 mg/kg/hr benzodiazepines (9.4 mg/kg/d) at decannulation, an in
251 ischarge, patients were receiving 0.40 mg/kg/hr opioids (9.7 mg/kg/d) and 0.39 mg/kg/hr benzodiazepin
252 d (propofol 2.6 +/- 1.2 vs 2.6 +/- 1.2 mg/kg/hr; p = 0.23 and remifentanil 1.27 +/- 0.17 vs 1.27 +/-
253 onists ELA or Apelin-13 (39 and 15 microg/kg/hr, respectively) versus normal saline.
254 ol: 3 mL/kg during 1 hour before and 1 mL/kg/hr during 6 hours after contrast medium exposure.
255  for predicting severe oliguria (< 0.3 mL/kg/hr for 6 hr) occurring within the next 12 hours were tes
256 ultrafiltration rate greater than 1.75 mL/kg/hr was associated with significantly greater mortality (
257  net ultrafiltration greater than 1.75 mL/kg/hr was independently associated with increased hospital
258 ne < 132 umol/L or urine output >= 0.5 mL/kg/hr), 2) mild-moderate acute kidney injury (serum creatin
259 sure adsorption), 2) low clearance (20 mL/kg/hr), and 3) high clearance (40 mL/kg/hr).
260  therapy or minimum urine output < 0.3 mL/kg/hr).
261 0 mL/kg/hr), and 3) high clearance (40 mL/kg/hr).
262 h a net ultrafiltration less than 1.01 mL/kg/hr, a net ultrafiltration rate greater than 1.75 mL/kg/h
263 py clearance rates: 1) no clearance (0 mL/kg/hr, to measure adsorption), 2) low clearance (20 mL/kg/h
264     Reduction in morphine use of 0.25 mug/kg/hr (95% CI, -0.68 to 1.18 mug/kg/hr) was not statistical
265 0.25 mug/kg/hr (95% CI, -0.68 to 1.18 mug/kg/hr) was not statistically significant.
266 exmedetomidine dosage was 0.7 +/- 0.2 mug/kg/hr.
267 ntanil 1.27 +/- 0.17 vs 1.27 +/- 0.16 mug/kg/hr; p = 0.52, respectively).
268 owed by continuous administration of 1 ng/kg/hr during 3 hours.
269  units/kg/hr] vs 15.1 international units/kg/hr [10.7-18.3 international units/kg/hr] in the treatmen
270 se heparin dose (13.5 international units/kg/hr [9.6-17.9 international units/kg/hr] vs 15.1 internat
271 nits/kg/hr [10.7-18.3 international units/kg/hr] in the treatment and control group, respectively; p
272 units/kg/hr [9.6-17.9 international units/kg/hr] vs 15.1 international units/kg/hr [10.7-18.3 interna
273  weeks, at three levels of exposure (0.1 kGy/hr/wk [low], 1 kGy/hr/wk [medium] and 3 kGy/hr/wk [high]
274 vels of exposure (0.1 kGy/hr/wk [low], 1 kGy/hr/wk [medium] and 3 kGy/hr/wk [high]).
275 /hr/wk [low], 1 kGy/hr/wk [medium] and 3 kGy/hr/wk [high]).
276 r fluid loading (n = 8, isotonic saline, 1 L/hr for 2 hr) or diuretic treatment (n = 8, furosemide, 4
277 ared to those without (+0.2 vs -0.006 mmol/L/hr; p < 0.001).
278 , beginning either 0.5 (early) or 5.5 (late) hr after wake.
279               CART identifed an AUC >=435 mg*hr/L for TCS.
280 boli (adjusted risk ratio, 1.03/microembolus/hr; 95% CI, 1.01-1.05) and with persistently positive tr
281 at an infarct core growth rate (IGR) <4.1 ml/hr and initial infarct core volumes (ICV) <19.9 ml had a
282  all LVO stroke patients have an IGR <4.1 ml/hr.
283 -19 hours with a mean consumption of 7.89 mL/hr (SD, 2.99).
284 efore angiotensin II initiation was 40 ng/mL/hr (normal, 0.6-3.0 ng/mL/hr) with resultant drop to 22
285 L/hr) with resultant drop to 22 and 12 ng/mL/hr at 2 and 6 hours after angiotensin II initiation, res
286 ation was 40 ng/mL/hr (normal, 0.6-3.0 ng/mL/hr) with resultant drop to 22 and 12 ng/mL/hr at 2 and 6
287 ed to infuse sustained low rates (0.15 mug / hr) of (R)-DOI.HCl to mice fed a high-fat "Western" diet
288 iometric growth of SrTiO(3) exceeding 600 nm hr(-1).
289 st NO(3) production was approximately 1 ppbv hr(-1), while NO(3) and N(2)O(5) were at or below 3 pptv
290 edian IP than qualified lr-MSM and qualified hr-MSM (2 [interquartile range {IQR}, 1-2] vs 3 [IQR, 2-
291 SM but was significantly higher in qualified hr-MSM and unqualified MSM.
292 9% of qualified lr-MSM, and 27% of qualified hr-MSM.
293 ied as low risk (lr) or medium-to-high risk (hr) based on self-reported sexual behavior and as qualif
294 utic vaccines have focused on two high-risk (hr) HPV genotypes, 16 and 18, and two viral targets, E6
295        Subsequently, all cases of high-risk (hr) HPV-associated high-grade lesions and carcinomas in
296 rom December 1, 2016, to August 2, 2018, the hr-HPV prevalence among 60,135 women was 24.78%, the mos
297 ants received 800 mg KAF156 or placebo three hr before CHMI with Pf-infected mosquitoes.
298 g AI annotation, surgeons reviewed 50 videos/hr.
299 h pentobarbital (Pento, 40 mg/kg body weight/hr) or propofol (Prop, 12 mg/kg body weight/hr), respect
300 /hr) or propofol (Prop, 12 mg/kg body weight/hr), respectively.

 
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