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1 hogenic state investigated is infection by a human herpesvirus.
2 ous cycle of varicella-zoster virus (VZV), a human herpesvirus.
3 nse to infection with this common pathogenic human herpesvirus.
4 Human cytomegalovirus (HCMV) is a ubiquitous human herpesvirus.
5  to a small genomic region, as seen in other human herpesviruses.
6 ach of the alpha, beta, and gamma classes of human herpesviruses.
7 gG do not have orthologs in HSV-2 or 8 other human herpesviruses.
8  and gG) do not have orthologs in all 40 non-human herpesviruses.
9 d that have undetectable levels of the eight human herpesviruses.
10 he nucleoside kinase activity of coinfecting human herpesviruses.
11 of novel entry inhibitors of EBV and related human herpesviruses.
12 ey mechanism appears to be common to several human herpesviruses.
13 ciated tegument proteins compared with other human herpesviruses.
14  the circulating diversity of all classes of human herpesviruses.
15 n varicella-zoster virus (VZV; also known as human herpesvirus 3 [HHV-3]).
16 hat varicella-zoster virus (VZV; also called human herpesvirus 3 [HHV3]), the human alphaherpesvirus
17 s Prevotella spp., Streptococcus mutans, and Human herpesvirus 4 (Epstein-Barr virus [EBV]) were more
18 coma herpesvirus (KSHV), formally designated human herpesvirus 4 (HHV-4) and 8 (HHV-8), respectively,
19 n gammaherpesvirus Epstein-Barr virus (EBV) (human herpesvirus 4 [HHV4]) infects most adults and is a
20              Human cytomegalovirus (HCMV, or human herpesvirus 5 [HHV-5]) is a large DNA-containing v
21 n distinct strains of human cytomegalovirus (human herpesvirus 5) and show that gene drive viruses ca
22 receptor encoded by CMV (also referred to as human herpesvirus 5), a highly prevalent human virus tha
23  inherits a chromosomally integrated copy of human herpesvirus 6 (CI-HHV-6), but the consequences of
24 esence of inherited chromosomally integrated human herpesvirus 6 (ciHHV-6) in hematopoietic cell tran
25               Cytomegalovirus (CMV) (18.3%), human herpesvirus 6 (HHV-6) (34.2%), human herpesvirus 7
26  targets detected were enterovirus (n = 38), human herpesvirus 6 (HHV-6) (n = 30), and Streptococcus
27                                              Human herpesvirus 6 (HHV-6) and cytomegalovirus (CMV) ar
28                                              Human herpesvirus 6 (HHV-6) and oncogenic Marek's diseas
29                              The majority of human herpesvirus 6 (HHV-6) congenital infections (86%)
30        This review evaluates publications on human herpesvirus 6 (HHV-6) encephalitis recognizing fir
31                 Following primary infection, human herpesvirus 6 (HHV-6) establishes a persistent inf
32 mory subsets of CD4(+) and CD8(+) T cells to human herpesvirus 6 (HHV-6) have not been previously inv
33 te concerning the role of herpesviruses, and human herpesvirus 6 (HHV-6) in particular, in AD.
34                          Higher incidence of human herpesvirus 6 (HHV-6) infection has been documente
35                                   Congenital human herpesvirus 6 (HHV-6) infection results from germl
36 bella, 309 cases of dengue, and 260 cases of human herpesvirus 6 (HHV-6) infection.
37                                              Human herpesvirus 6 (HHV-6) is an important cause of men
38                                              Human herpesvirus 6 (HHV-6) is detected in the plasma of
39                                              Human herpesvirus 6 (HHV-6) is susceptible to latency an
40                                              Human herpesvirus 6 (HHV-6) may be associated with LFUE,
41                                              Human herpesvirus 6 (HHV-6) species have a unique abilit
42 megalovirus (CMV), Epstein-Barr virus (EBV), human herpesvirus 6 (HHV-6), herpes simplex virus types
43 uses such as Marek's disease virus (MDV) and human herpesvirus 6 (HHV-6), integrate their DNA into ho
44 , cytomegalovirus (CMV), BK virus (BKV), and human herpesvirus 6 (HHV-6).
45                           Here, we show that human herpesvirus 6 (HHV-6, A or B) RNA was detected in
46 (EBV), BK virus (BKV), adenovirus (ADV), and human herpesvirus 6 (HHV6) were evaluated to determine o
47 cytomegalovirus (CMV), Epstein-Bar virus and human herpesvirus 6 (HHV6), are known to infect neurons.
48 omes of interest were VR of adenovirus, EBV, human herpesvirus 6 (HHV6), cytomegalovirus (CMV), and B
49                       Coreactivation of both human herpesvirus 6 and cytomegalovirus in ICU patients
50                           The association of human herpesvirus 6 and cytomegalovirus reactivation wit
51 ilation and ICU type, only coreactivation of human herpesvirus 6 and cytomegalovirus was significantl
52        CMV, herpes simplex virus type 1, and human herpesvirus 6 infection were independently associa
53                                              Human herpesvirus 6 is associated with a variety of comp
54 expanded panel of HHV-6B antigens.IMPORTANCE Human herpesvirus 6 is highly prevalent and maintains ch
55 n Results: First-onset viral infections with human herpesvirus 6 or Epstein-Barr virus within 100 day
56                           Most patients with human herpesvirus 6 reactivation also reactivated cytome
57 ical ventilation, burn ICU, major infection, human herpesvirus 6 reactivation, and cytomegalovirus re
58 y and comprehensively assessed the impact of human herpesvirus 6 reactivation, and its interaction wi
59                                              Human herpesvirus 6 viremia occurred in 23% of patients
60 and the thymus, kidneys, and adrenal glands (human herpesvirus 6).
61 uding BK virus, cytomegalovirus, adenovirus, human herpesvirus 6, and Epstein-Barr virus.
62            Murine roseolovirus, a homolog of human herpesvirus 6, can also be reactivated in the lung
63                                     Viruses (human herpesvirus 6, Epstein-Barr virus, and cytomegalov
64 , enterovirus, herpes simplex virus 1 and 2, human herpesvirus 6, human parechovirus, varicella-zoste
65  positive by qPCR for Epstein-Barr virus and human herpesvirus 6, including the brain cortex (Epstein
66 9; p = 0.005) compared to patients with only human herpesvirus 6, only cytomegalovirus, or no viral r
67 30 febrile children positive for adenovirus, human herpesvirus 6, or enterovirus infection or with ac
68 galovirus, EBV, adenovirus, BK virus, and/or human herpesvirus 6.
69  new and conserved features in the genome of human herpesvirus 6.
70 d: 11 with infectious causes (2 influenza; 2 human herpesvirus 6; 2 group B Streptococcus; 2 Streptoc
71 inical relevance of chromosomally integrated human herpesvirus-6 (CIHHV-6) after transplantation is n
72  zoster virus 6% in saliva and 3% in GCF; of human herpesvirus-6 (HHV-6) 6% in saliva and 2% in GCF;
73                                              Human herpesvirus-6 (HHV-6) A and B are ubiquitous betah
74            In addition, viral copy number of human herpesvirus-6 (HHV-6) and human herpesvirus-7 (HHV
75                       To investigate whether human herpesvirus-6 (HHV-6) is a causative agent of ence
76                                              Human herpesvirus-6 (HHV-6) is a neurotropic virus that
77                                              Human herpesvirus-6 (HHV-6) is known to reactivate after
78         Previous research has suggested that human herpesvirus-6 (HHV-6) may integrate into host cell
79                     A real-time quantitative human herpesvirus-6 (HHV-6) polymerase chain reaction as
80           Inherited chromosomally integrated human herpesvirus-6 (iciHHV-6) results in the germ-line
81 ations in ATRX and CTNNB1 and integration of human herpesvirus-6 in chromosome 11p.
82  cytomegalovirus (genus Cytomegalovirus) and human herpesvirus 6A (genus Roseolovirus).
83                               The genomes of human herpesvirus 6A (HHV-6A) and HHV-6B have the capaci
84 ous human diseases have been correlated with human herpesvirus 6A (HHV-6A) and HHV-6B, the lack of an
85 ity of the human population is infected with human herpesvirus 6A (HHV-6A), a betaherpesvirus family
86                                              Human herpesvirus 6A (HHV-6A), a member of the betaherpe
87                                              Human herpesvirus 6A (HHV-6A), HHV-6B, and HHV-7 are cla
88                                              Human herpesvirus 6A (HHV-6A), HHV-6B, and HHV-7 are cla
89                     The human roseoloviruses human herpesvirus 6A (HHV-6A), HHV-6B, and HHV-7 compris
90        We show that in T cells infected with human herpesvirus 6A (HHV-6A), the E2F1 protein and its
91 megalovirus [CMV], Epstein-Barr virus [EBV], human herpesvirus 6A [HHV-6A], HHV-6B, herpes simplex vi
92                                              Human herpesvirus 6A and 6B (HHV-6A and HHV-6B) are clos
93 y 1% of people worldwide carry a copy of the human herpesvirus 6A or 6B (HHV-6A/B) in every cell of t
94                 The U24 protein expressed by human herpesvirus 6A, a ubiquitous human pathogen, has b
95 red to as inherited chromosomally integrated human herpesvirus 6A/B (iciHHV-6A/B).
96                                              Human herpesviruses 6A and 6B (HHV-6A and HHV-6B) are hu
97  most closely related to the roseoloviruses, human herpesviruses 6A, 6B, and 7.
98                                              Human herpesviruses 6A/B (HHV-6A/B) can integrate their
99 ier of an inherited-chromosomally-integrated human herpesvirus-6A (iciHHV-6A) genome in one 19q telom
100                          The roseoloviruses, human herpesvirus-6A -6B and -7 (HHV-6A, HHV-6B and HHV-
101                                              Human herpesvirus 6B (HHV-6B) belongs to the beta-herpes
102                                              Human herpesvirus 6B (HHV-6B) commonly reactivates after
103                                              Human herpesvirus 6B (HHV-6B) DNA is frequently detected
104                                              Human herpesvirus 6B (HHV-6B) DNA is frequently detected
105                                              Human herpesvirus 6B (HHV-6B) frequently reactivates aft
106 tomic analyses for HCT recipients.IMPORTANCE Human herpesvirus 6B (HHV-6B) is a DNA virus that infect
107  is positive for Epstein-Barr virus (EBV) or human herpesvirus 6B (HHV-6B), with one coinfection.
108                                              Human herpesvirus 6B was the species detected in eight s
109 o test for cytomegalovirus, BK polyomavirus, human herpesvirus 6B, HHV-6A, adenovirus, and Epstein-Ba
110 y CD4(+) T cells were enriched with DNA from human herpesvirus 6b.
111 18.3%), human herpesvirus 6 (HHV-6) (34.2%), human herpesvirus 7 (HHV-7) (20.5%) and Epstein-Barr vir
112  including the closely related human viruses human herpesvirus 7 (HHV-7) and human cytomegalovirus (H
113             In this report, we show that the human herpesvirus 7 (HHV-7) immunoevasin U21, itself a c
114                           Here, we show that human herpesvirus 7 (HHV-7) U21 binds to and downregulat
115                    The U21 gene product from human herpesvirus 7 binds to and redirects class I major
116 of HBV, parvovirus B19, cytomegalovirus, and human herpesvirus 7).
117 tial pathogens reported included bunyavirus, human herpesvirus 7, and enterovirus D-68, ultimately im
118                                          The human herpesvirus-7 (HHV-7) U21 glycoprotein binds to cl
119 py number of human herpesvirus-6 (HHV-6) and human herpesvirus-7 (HHV-7) were measured in both saliva
120              The U21 open reading frame from human herpesvirus-7 encodes a membrane protein that asso
121 ypothesized that they could be infected with human herpesvirus 8 (HHV-8) (Kaposi's sarcoma [KS]-assoc
122                          We have developed a human herpesvirus 8 (HHV-8) 50% tissue culture infective
123 oma (cKS) is an inflammatory tumor caused by human herpesvirus 8 (HHV-8) commonly observed in elderly
124 posi's sarcoma-associated herpesvirus (KSHV)/human herpesvirus 8 (HHV-8) displays two distinct life s
125                                              Human herpesvirus 8 (HHV-8) encodes four viral interfero
126                         Here, we report that human herpesvirus 8 (HHV-8) gene product viral interfero
127                                              Human herpesvirus 8 (HHV-8) infection is associated with
128                                              Human herpesvirus 8 (HHV-8) infection is endemic in sub-
129                                              Human herpesvirus 8 (HHV-8) infection occurs in early ch
130 plantation through reactivation of recipient human herpesvirus 8 (HHV-8) infection or through donor-d
131                                              Human herpesvirus 8 (HHV-8) interleukin-6 (vIL-6) is dis
132                                              Human herpesvirus 8 (HHV-8) interleukin-6 (vIL-6) promot
133                                              Human herpesvirus 8 (HHV-8) is an oncogenic virus causal
134       Viral interleukin-6 (vIL-6) encoded by human herpesvirus 8 (HHV-8) is believed to contribute vi
135                                              Human herpesvirus 8 (HHV-8) is endemic in Uganda and tra
136                                              Human herpesvirus 8 (HHV-8) is the causative agent of Ka
137                                          The human herpesvirus 8 (HHV-8) viral G protein-coupled rece
138                                              Human herpesvirus 8 (HHV-8) viral interleukin-6 (vIL-6)
139                         The contributions of human herpesvirus 8 (HHV-8) viral interleukin-6 (vIL-6)
140                                              Human herpesvirus 8 (HHV-8) viral interleukin-6 (vIL-6)
141                                              Human herpesvirus 8 (HHV-8) viral interleukin-6 (vIL-6),
142 -1, HSV-2), varicella-zoster virus (VZV) and human herpesvirus 8 (HHV-8) were not or rarely detected
143  epithelial cells persistently infected with human herpesvirus 8 (HHV-8), an oncogenic herpesvirus th
144  Other viruses, such as hepatitis B virus or human herpesvirus 8 (HHV-8), establish persistent infect
145 associated herpesvirus (KSHV), also known as human herpesvirus 8 (HHV-8), is a cancer-related human v
146 0-CD200R pathway during infection, including human herpesvirus 8 (HHV-8), the causative agent of Kapo
147 erent efficiencies and with the exception of human herpesvirus 8 (HHV-8), these chimeric variants res
148 evious analyses of vIL-6 activity.IMPORTANCE Human herpesvirus 8 (HHV-8)-encoded viral interleukin-6
149            Kaposi sarcoma is the most common human herpesvirus 8 (HHV-8)-related disease described af
150 Ebola virus (EBOV), Tacaribe arenavirus, and human herpesvirus 8 (HHV-8).
151 most always multicentric (MCD) and linked to human herpesvirus 8 (HHV-8).
152 aque (RM) monkeys that is closely related to human herpesvirus 8 (HHV-8)/Kaposi's Sarcoma-associated
153                                              Human herpesvirus 8 (HHV8) causes multicentric Castleman
154 central Italy and a surveillance program for human herpesvirus 8 (HHV8) infection after transplant, a
155 ly improved survival and reduced the risk of human herpesvirus 8 (HHV8)-associated lymphoma.
156 nsfection replication assay identified eight human herpesvirus 8 (HHV8)-encoded proteins required for
157 irus Kaposi's sarcoma-associated herpesvirus/human herpesvirus 8 (KSHV/HHV8), exploit microtubule (MT
158 -associated herpesvirus (KSHV; also known as human herpesvirus 8 [HHV-8]) is the etiologic agent of K
159 -associated herpesvirus (KSHV; also known as human herpesvirus 8 [HHV-8]), Epstein-Barr virus (EBV),
160 ma-associated herpesvirus (KSHV; also called human herpesvirus 8 [HHV-8]), upon being reactivated, ca
161 i's sarcoma-associated herpesvirus (KSHV; or human herpesvirus 8 [HHV8]) is implicated in the pathoge
162 ma-associated herpesvirus (KSHV; also called human herpesvirus 8 [HHV8]), the switch from latency to
163 ain at increased KS risk, likely due to high human herpesvirus 8 coinfection rates.
164        Kaposi sarcoma-associated herpesvirus/human herpesvirus 8 is associated with multicentric Cast
165 6 homologue (viral interleukin-6 [vIL-6]) of human herpesvirus 8 is implicated in viral pathogenesis
166     Viral interleukin-6 (vIL-6) specified by human herpesvirus 8 is, unlike its cellular counterpart,
167                                              Human herpesvirus 8 seroprevalence was 4% (10/249) in do
168 CMV, herpes simplex virus types 1 and 2, and human herpesvirus 8 was 99.4%, 74.9%, 26.2%, 8.0%, and 1
169 s sarcoma-associated herpesvirus (also named human herpesvirus 8) is a gamma-herpesvirus that undergo
170 persistent gamma-herpesvirus infection (EBV, human herpesvirus 8) is a significant problem in AIDS pa
171 posi's sarcoma-associated herpesvirus (KSHV; human herpesvirus 8) is an oncogenic gammaherpesvirus th
172 ma-associated herpesvirus (KSHV, also called human herpesvirus 8) is linked to the development of Kap
173 s (CMV), herpes simplex virus types 1 and 2, human herpesvirus 8) using multiplex serology on blood s
174 aposi's sarcoma-associated herpesvirus (KSHV/human herpesvirus 8).
175  (cytomegalovirus, herpes simplex virus-1/2, human herpesvirus 8, hepatitis E virus, parvovirus B19).
176 associated herpesvirus (KSHV), also known as human herpesvirus 8, is the etiologic agent of Kaposi sa
177 associated herpesvirus (KSHV), also known as human herpesvirus 8, is the etiologic agent underlying K
178 The rhadinovirus 1 (RV1) lineage consists of human herpesvirus 8, Kaposi's sarcoma-associated herpesv
179                                              Human herpesvirus 8, which causes Kaposi sarcoma, expres
180                                              Human herpesvirus 8-DNA was detected in 6.8% and 2.9% of
181 in HHV-8 latent and lytic biology.IMPORTANCE Human herpesvirus 8-encoded IRF homologues were the firs
182                                      Because human herpesvirus 8-encoded K13 selectively activates th
183 Epstein-Barr virus-infected lymphocytes, and human herpesvirus 8-positive primary effusion lymphoma,
184 ted to rhesus macaque rhadinovirus (RRV) and human herpesvirus 8.
185 e of Kaposi's sarcoma-associated herpesvirus/human herpesvirus 8.
186  gammaherpesviruses, Epstein-Barr virus, and human herpesvirus 8/Kaposi's sarcoma-associated herpesvi
187 posi's sarcoma-associated herpesvirus (KSHV [human herpesvirus 8; HHV-8]) open reading frame 57 (ORF5
188 onsequence of dysregulated expression of the human herpesvirus-8 (HHV-8 or KSHV)-encoded G protein-co
189 effusion lymphomas (PEL) are associated with human herpesvirus-8 (HHV-8) and usually occur in immunoc
190 ranean Basin, despite the high prevalence of human herpesvirus-8 (HHV-8) infection in this region.
191 s led to a dramatic rise in the incidence of human herpesvirus-8 (HHV-8)-associated Kaposi's sarcoma
192 S), a multifocal vascular neoplasm linked to human herpesvirus-8 (HHV-8/KS-associated herpesvirus [KS
193                          Posttransplantation human herpesvirus-8 (HHV8)/Kaposi sarcoma herpesvirus (K
194 i's sarcoma-associated herpesvirus (KSHV, or human herpesvirus-8 [HHV-8]) has another, alternative em
195 sociation was observed with plasma levels of human herpesvirus-8 DNA.
196 of KS is KS herpesvirus (KSHV; also known as human herpesvirus-8), and viral proteins can induce KS-a
197 osi sarcoma-associated herpesvirus (KSHV; or human herpesvirus-8)-encoded protein called K-bZIP (also
198 mely Kaposi's sarcoma-associated herpesvirus/human herpesvirus-8, has been confirmed to be a direct c
199 nflammatory protein-II (vMIP-II), encoded by human herpesvirus-8, has garnered interest because of it
200                 We enrolled HIV-negative and human herpesvirus-8-seronegative patients with symptomat
201 liferative disorder caused by infection with human herpesvirus-8.
202 esvirus family including cytomegalovirus and human herpesvirus-8.
203 n shown to be associated with infection with human herpesvirus-8.
204 ) and HSV-2, two closely related neurotropic human herpesviruses, achieve neurotropism through ICP34.
205 onors to seropositive recipients for several human herpesviruses and adenoviruses.
206 the structure of region IX is present in all human herpesviruses and thus represents a potential stru
207             Human cytomegalovirus (CMV) is a human herpesvirus, and infection is widespread in the hu
208                                              Human herpesviruses are among the most prevalent pathoge
209 e against Epstein-Barr virus (EBV) and other human herpesviruses are limited to those targeting viral
210                                              Human herpesviruses are responsible for a range of debil
211                              Infections with human herpesviruses are ubiquitous and a public health c
212 logues of pUL7 and pUL51 is conserved across human herpesviruses, as is their association with trans-
213           Epstein-Barr virus is a ubiquitous human herpesvirus associated with epithelial and lymphoi
214           Because KSBcl-2 and BHRF1 are from human herpesviruses associated with malignancies, we scr
215                                              Human herpesviruses-associated vasculitis can cause vasc
216 de analog, is thought to be specific for the human herpesviruses because it requires a virally encode
217 ched the genome-wide level for CMV and other human herpesviruses, but study of HHV-6 is at an earlier
218     This study examined the association of 4 human herpesviruses (CMV, herpes simplex virus type 1, h
219 olution imaging of individual virions of the human herpesvirus cytomegalovirus (CMV) showed that viri
220 -regulating master circuits in HIV-1 and the human herpesvirus cytomegalovirus along with potential c
221 more differentiated T cells specific for the human herpesviruses cytomegalovirus and Epstein-Barr vir
222               By simulating infection of the human herpesvirus, cytomegalovirus, we hypothesize that
223                                       Of the human herpesviruses, cytomegalovirus (HCMV) and Epstein-
224          The ORF54-related proteins of other human herpesviruses do not possess this activity, sugges
225 Currently, there are approximately 150 known human herpesvirus-encoded miRNAs, although an miRNA(s) e
226             Our results show VZV, like other human herpesviruses, encodes several sncRNAs and miRNAs,
227 genic latency gene expression program of the human herpesvirus Epstein-Barr virus (EBV).
228                                              Human herpesviruses establish life-long latency in the h
229 sized that HHV-6A, like other members of the human herpesvirus family, encodes miRNAs, which function
230 l mechanism by which HHV-6A, a member of the human herpesvirus family, may contribute to inadequate m
231 g 3 HSV-1 genomes, one HSV-2 genome, 8 other human herpesvirus genomes, and 40 non-human herpesvirus
232  other human herpesvirus genomes, and 40 non-human herpesvirus genomes.
233                                    All eight human herpesviruses have a conserved herpesvirus protein
234                              Since all eight human herpesviruses have UL25 orthologs, this discovery
235                                              Human herpesvirus (HHV) 6 causes substantial morbidity a
236 nd that certain infectious pathogens such as human herpesvirus (HHV) 6, HHV-7, and adenovirus, which
237 immunological tools available to detect both human herpesvirus (HHV) and immune control of replicatio
238 d men have at least one actively replicating human herpesvirus (HHV) in their mucosal secretions at a
239                                              Human herpesvirus (HHV) infections are common during inf
240  Both human immunodeficiency virus (HIV) and human herpesvirus (HHV) infections persist lifelong, and
241                              Reactivation of human herpesvirus (HHV)-6 and HHV-7 has been linked to v
242 ced hypersensitivity syndrome (DIHS), latent human herpesvirus (HHV)-6 is frequently reactivated in a
243 identification of suitable animal models for human herpesvirus (HHV)-6A and HHV-6B infections has bee
244 for a single virus: Epstein-Barr virus (23), human herpesvirus (HHV)-6B (10), human parainfluenza vir
245 KS) is a rare vascular tumor associated with human herpesvirus (HHV)-8 infection.
246 nt studies have found evidence of occasional human herpesvirus (HHV)-8 transmission via blood transfu
247                                              Human herpesviruses (HHV) 6 and 7 are ubiquitous infecti
248                  Infections due to the eight human herpesviruses (HHV) are exacerbated by immunosuppr
249                                              Human herpesviruses (HHV) establish lifelong latent infe
250                  Asymptomatic replication of human herpesviruses (HHV) is frequent in HIV-infected me
251 were as follows: cytomegalovirus (CMV), 44%; human herpesvirus [HHV] 6, 18%; HHV8, 6%; Epstein-Barr v
252                             Reactivations of human herpesviruses (HHVs) contribute to the development
253               Seminal HIV RNA and DNA from 7 human herpesviruses (HHVs) were measured by real-time po
254 , acts as a DNA chain terminator for several human herpesviruses (HHVs), including HHV-2 (HSV-2), a c
255 lly occurring genetic variants of persistent human herpesviruses imprints on the evolution of the ant
256 us (EBV), known as an oncovirus, is the only human herpesvirus in the genus Lymphocryptovirus (LCV).
257 ass A infections, the high seroprevalence of human herpesviruses in lr-MSM warrants further investiga
258 of this study was to analyse the presence of human herpesviruses in saliva and gingival crevicular fl
259       Nineteen proteins are conserved in all human herpesviruses, including capsid scaffold protein U
260  we found that HDAC inhibiting proteins from human herpesviruses induce human NKG2D ligand ULBP-1.
261 t as additional therapeutic options to treat human herpesvirus infections.
262 vide additional therapeutic options to treat human herpesvirus infections.
263 s sarcoma-associated herpesvirus (KSHV) is a human herpesvirus involved in the causation of several h
264                 The most recently identified human herpesvirus is Kaposi's sarcoma-associated herpesv
265    The epidemiology and essential biology of human herpesvirus is reviewed.
266                                       EBV, a human herpesvirus, is commonly acquired during childhood
267 nding recruiting p53 to Zp.IMPORTANCE EBV, a human herpesvirus, is latently present in most nasophary
268 llular protein and has homologs in all other human herpesviruses, it has potential importance as a th
269  the data suggest that HHV-6 is unique among human herpesviruses: it specifically and efficiently int
270  Kaposi sarcoma (KS) to infection by a novel human herpesvirus (Kaposi sarcoma-associated herpesvirus
271                                              Human herpesviruses may cause severe complications after
272 is to investigate the effect of any of eight human herpesviruses on development of dementia or mild c
273                           All members of the human herpesvirus protease (HHV Pr) family are active as
274 yzed the incidence of infection of the eight human herpesviruses simultaneously in 1 045 peripheral b
275         pUL47 may have a similar function in human herpesviruses such as varicella-zoster virus or he
276 Human cytomegalovirus (HCMV) is a ubiquitous human herpesvirus that can cause life-threatening diseas
277                Epstein-Barr virus (EBV) is a human herpesvirus that can cause lymphomas, epithelial c
278 -zoster virus (VZV) is a medically important human herpesvirus that causes chickenpox and shingles, b
279     Epstein-Barr virus (EBV) is an oncogenic human herpesvirus that dramatically reorganizes host gen
280                Epstein-Barr Virus (EBV) is a human herpesvirus that efficiently establishes latent in
281                          EBV is an oncogenic human herpesvirus that has the ability to infect and tra
282                          EBV is a ubiquitous human herpesvirus that is associated with various lympho
283     Epstein-Barr virus (EBV) is a ubiquitous human herpesvirus that is causally implicated in the dev
284 , the replication of herpes simplex virus, a human herpesvirus that is closely related to HCMV, was n
285 pstein-Barr virus, one of the most prevalent human herpesviruses that also causes cancer, we have dis
286 ibody response.IMPORTANCE HHV-6A and -6B are human herpesviruses that have the unique property of bei
287      Epstein-Barr virus (EBV) is one of nine human herpesviruses that persist latently to establish p
288 genomes, combined with the inability of most human herpesviruses to replicate in animals, has until r
289                              Enterovirus and human herpesvirus type 6 had agreements of 95.7% and 85.
290 esviruses (CMV, herpes simplex virus type 1, human herpesvirus type 6, and Epstein-Barr virus) with c
291 afamilial dynamics of endemic infection with human herpesvirus type 8 (HHV-8) in Amerindian populatio
292 quently Epstein-Barr virus (EBV) positive or human herpesvirus type-8 (HHV-8) positive.
293                           Strikingly, as all human herpesviruses use conserved mRNA processing pathwa
294 that the hexon-capping SCP-the largest among human herpesviruses-uses its N-terminal half to bridge h
295 is substantially smaller than those of other human herpesviruses, VZV has a similarly sized capsid, c
296                          Levels of HIV and 7 human herpesviruses were measured by real-time polymeras
297                    Compared to blood donors, human herpesviruses were more prevalent in all MSM group
298  we address this issue for EBV, a widespread human herpesvirus with oncogenic potential.
299                  Epstein-Barr virus (EBV), a human herpesvirus with potent B cell growth transforming
300                                  We focus on human herpesviruses, with key insights drawn from veteri

 
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