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1 hogenic state investigated is infection by a human herpesvirus.
2 ous cycle of varicella-zoster virus (VZV), a human herpesvirus.
3 nse to infection with this common pathogenic human herpesvirus.
4 Human cytomegalovirus (HCMV) is a ubiquitous human herpesvirus.
5 to a small genomic region, as seen in other human herpesviruses.
6 ach of the alpha, beta, and gamma classes of human herpesviruses.
7 gG do not have orthologs in HSV-2 or 8 other human herpesviruses.
8 and gG) do not have orthologs in all 40 non-human herpesviruses.
9 d that have undetectable levels of the eight human herpesviruses.
10 he nucleoside kinase activity of coinfecting human herpesviruses.
11 of novel entry inhibitors of EBV and related human herpesviruses.
12 ey mechanism appears to be common to several human herpesviruses.
13 ciated tegument proteins compared with other human herpesviruses.
14 the circulating diversity of all classes of human herpesviruses.
16 hat varicella-zoster virus (VZV; also called human herpesvirus 3 [HHV3]), the human alphaherpesvirus
17 s Prevotella spp., Streptococcus mutans, and Human herpesvirus 4 (Epstein-Barr virus [EBV]) were more
18 coma herpesvirus (KSHV), formally designated human herpesvirus 4 (HHV-4) and 8 (HHV-8), respectively,
19 n gammaherpesvirus Epstein-Barr virus (EBV) (human herpesvirus 4 [HHV4]) infects most adults and is a
21 n distinct strains of human cytomegalovirus (human herpesvirus 5) and show that gene drive viruses ca
22 receptor encoded by CMV (also referred to as human herpesvirus 5), a highly prevalent human virus tha
23 inherits a chromosomally integrated copy of human herpesvirus 6 (CI-HHV-6), but the consequences of
24 esence of inherited chromosomally integrated human herpesvirus 6 (ciHHV-6) in hematopoietic cell tran
26 targets detected were enterovirus (n = 38), human herpesvirus 6 (HHV-6) (n = 30), and Streptococcus
32 mory subsets of CD4(+) and CD8(+) T cells to human herpesvirus 6 (HHV-6) have not been previously inv
42 megalovirus (CMV), Epstein-Barr virus (EBV), human herpesvirus 6 (HHV-6), herpes simplex virus types
43 uses such as Marek's disease virus (MDV) and human herpesvirus 6 (HHV-6), integrate their DNA into ho
46 (EBV), BK virus (BKV), adenovirus (ADV), and human herpesvirus 6 (HHV6) were evaluated to determine o
47 cytomegalovirus (CMV), Epstein-Bar virus and human herpesvirus 6 (HHV6), are known to infect neurons.
48 omes of interest were VR of adenovirus, EBV, human herpesvirus 6 (HHV6), cytomegalovirus (CMV), and B
51 ilation and ICU type, only coreactivation of human herpesvirus 6 and cytomegalovirus was significantl
54 expanded panel of HHV-6B antigens.IMPORTANCE Human herpesvirus 6 is highly prevalent and maintains ch
55 n Results: First-onset viral infections with human herpesvirus 6 or Epstein-Barr virus within 100 day
57 ical ventilation, burn ICU, major infection, human herpesvirus 6 reactivation, and cytomegalovirus re
58 y and comprehensively assessed the impact of human herpesvirus 6 reactivation, and its interaction wi
64 , enterovirus, herpes simplex virus 1 and 2, human herpesvirus 6, human parechovirus, varicella-zoste
65 positive by qPCR for Epstein-Barr virus and human herpesvirus 6, including the brain cortex (Epstein
66 9; p = 0.005) compared to patients with only human herpesvirus 6, only cytomegalovirus, or no viral r
67 30 febrile children positive for adenovirus, human herpesvirus 6, or enterovirus infection or with ac
70 d: 11 with infectious causes (2 influenza; 2 human herpesvirus 6; 2 group B Streptococcus; 2 Streptoc
71 inical relevance of chromosomally integrated human herpesvirus-6 (CIHHV-6) after transplantation is n
72 zoster virus 6% in saliva and 3% in GCF; of human herpesvirus-6 (HHV-6) 6% in saliva and 2% in GCF;
84 ous human diseases have been correlated with human herpesvirus 6A (HHV-6A) and HHV-6B, the lack of an
85 ity of the human population is infected with human herpesvirus 6A (HHV-6A), a betaherpesvirus family
91 megalovirus [CMV], Epstein-Barr virus [EBV], human herpesvirus 6A [HHV-6A], HHV-6B, herpes simplex vi
93 y 1% of people worldwide carry a copy of the human herpesvirus 6A or 6B (HHV-6A/B) in every cell of t
99 ier of an inherited-chromosomally-integrated human herpesvirus-6A (iciHHV-6A) genome in one 19q telom
106 tomic analyses for HCT recipients.IMPORTANCE Human herpesvirus 6B (HHV-6B) is a DNA virus that infect
107 is positive for Epstein-Barr virus (EBV) or human herpesvirus 6B (HHV-6B), with one coinfection.
109 o test for cytomegalovirus, BK polyomavirus, human herpesvirus 6B, HHV-6A, adenovirus, and Epstein-Ba
111 18.3%), human herpesvirus 6 (HHV-6) (34.2%), human herpesvirus 7 (HHV-7) (20.5%) and Epstein-Barr vir
112 including the closely related human viruses human herpesvirus 7 (HHV-7) and human cytomegalovirus (H
117 tial pathogens reported included bunyavirus, human herpesvirus 7, and enterovirus D-68, ultimately im
119 py number of human herpesvirus-6 (HHV-6) and human herpesvirus-7 (HHV-7) were measured in both saliva
121 ypothesized that they could be infected with human herpesvirus 8 (HHV-8) (Kaposi's sarcoma [KS]-assoc
123 oma (cKS) is an inflammatory tumor caused by human herpesvirus 8 (HHV-8) commonly observed in elderly
124 posi's sarcoma-associated herpesvirus (KSHV)/human herpesvirus 8 (HHV-8) displays two distinct life s
130 plantation through reactivation of recipient human herpesvirus 8 (HHV-8) infection or through donor-d
142 -1, HSV-2), varicella-zoster virus (VZV) and human herpesvirus 8 (HHV-8) were not or rarely detected
143 epithelial cells persistently infected with human herpesvirus 8 (HHV-8), an oncogenic herpesvirus th
144 Other viruses, such as hepatitis B virus or human herpesvirus 8 (HHV-8), establish persistent infect
145 associated herpesvirus (KSHV), also known as human herpesvirus 8 (HHV-8), is a cancer-related human v
146 0-CD200R pathway during infection, including human herpesvirus 8 (HHV-8), the causative agent of Kapo
147 erent efficiencies and with the exception of human herpesvirus 8 (HHV-8), these chimeric variants res
148 evious analyses of vIL-6 activity.IMPORTANCE Human herpesvirus 8 (HHV-8)-encoded viral interleukin-6
152 aque (RM) monkeys that is closely related to human herpesvirus 8 (HHV-8)/Kaposi's Sarcoma-associated
154 central Italy and a surveillance program for human herpesvirus 8 (HHV8) infection after transplant, a
156 nsfection replication assay identified eight human herpesvirus 8 (HHV8)-encoded proteins required for
157 irus Kaposi's sarcoma-associated herpesvirus/human herpesvirus 8 (KSHV/HHV8), exploit microtubule (MT
158 -associated herpesvirus (KSHV; also known as human herpesvirus 8 [HHV-8]) is the etiologic agent of K
159 -associated herpesvirus (KSHV; also known as human herpesvirus 8 [HHV-8]), Epstein-Barr virus (EBV),
160 ma-associated herpesvirus (KSHV; also called human herpesvirus 8 [HHV-8]), upon being reactivated, ca
161 i's sarcoma-associated herpesvirus (KSHV; or human herpesvirus 8 [HHV8]) is implicated in the pathoge
162 ma-associated herpesvirus (KSHV; also called human herpesvirus 8 [HHV8]), the switch from latency to
165 6 homologue (viral interleukin-6 [vIL-6]) of human herpesvirus 8 is implicated in viral pathogenesis
166 Viral interleukin-6 (vIL-6) specified by human herpesvirus 8 is, unlike its cellular counterpart,
168 CMV, herpes simplex virus types 1 and 2, and human herpesvirus 8 was 99.4%, 74.9%, 26.2%, 8.0%, and 1
169 s sarcoma-associated herpesvirus (also named human herpesvirus 8) is a gamma-herpesvirus that undergo
170 persistent gamma-herpesvirus infection (EBV, human herpesvirus 8) is a significant problem in AIDS pa
171 posi's sarcoma-associated herpesvirus (KSHV; human herpesvirus 8) is an oncogenic gammaherpesvirus th
172 ma-associated herpesvirus (KSHV, also called human herpesvirus 8) is linked to the development of Kap
173 s (CMV), herpes simplex virus types 1 and 2, human herpesvirus 8) using multiplex serology on blood s
175 (cytomegalovirus, herpes simplex virus-1/2, human herpesvirus 8, hepatitis E virus, parvovirus B19).
176 associated herpesvirus (KSHV), also known as human herpesvirus 8, is the etiologic agent of Kaposi sa
177 associated herpesvirus (KSHV), also known as human herpesvirus 8, is the etiologic agent underlying K
178 The rhadinovirus 1 (RV1) lineage consists of human herpesvirus 8, Kaposi's sarcoma-associated herpesv
181 in HHV-8 latent and lytic biology.IMPORTANCE Human herpesvirus 8-encoded IRF homologues were the firs
183 Epstein-Barr virus-infected lymphocytes, and human herpesvirus 8-positive primary effusion lymphoma,
186 gammaherpesviruses, Epstein-Barr virus, and human herpesvirus 8/Kaposi's sarcoma-associated herpesvi
187 posi's sarcoma-associated herpesvirus (KSHV [human herpesvirus 8; HHV-8]) open reading frame 57 (ORF5
188 onsequence of dysregulated expression of the human herpesvirus-8 (HHV-8 or KSHV)-encoded G protein-co
189 effusion lymphomas (PEL) are associated with human herpesvirus-8 (HHV-8) and usually occur in immunoc
190 ranean Basin, despite the high prevalence of human herpesvirus-8 (HHV-8) infection in this region.
191 s led to a dramatic rise in the incidence of human herpesvirus-8 (HHV-8)-associated Kaposi's sarcoma
192 S), a multifocal vascular neoplasm linked to human herpesvirus-8 (HHV-8/KS-associated herpesvirus [KS
194 i's sarcoma-associated herpesvirus (KSHV, or human herpesvirus-8 [HHV-8]) has another, alternative em
196 of KS is KS herpesvirus (KSHV; also known as human herpesvirus-8), and viral proteins can induce KS-a
197 osi sarcoma-associated herpesvirus (KSHV; or human herpesvirus-8)-encoded protein called K-bZIP (also
198 mely Kaposi's sarcoma-associated herpesvirus/human herpesvirus-8, has been confirmed to be a direct c
199 nflammatory protein-II (vMIP-II), encoded by human herpesvirus-8, has garnered interest because of it
204 ) and HSV-2, two closely related neurotropic human herpesviruses, achieve neurotropism through ICP34.
206 the structure of region IX is present in all human herpesviruses and thus represents a potential stru
209 e against Epstein-Barr virus (EBV) and other human herpesviruses are limited to those targeting viral
212 logues of pUL7 and pUL51 is conserved across human herpesviruses, as is their association with trans-
216 de analog, is thought to be specific for the human herpesviruses because it requires a virally encode
217 ched the genome-wide level for CMV and other human herpesviruses, but study of HHV-6 is at an earlier
218 This study examined the association of 4 human herpesviruses (CMV, herpes simplex virus type 1, h
219 olution imaging of individual virions of the human herpesvirus cytomegalovirus (CMV) showed that viri
220 -regulating master circuits in HIV-1 and the human herpesvirus cytomegalovirus along with potential c
221 more differentiated T cells specific for the human herpesviruses cytomegalovirus and Epstein-Barr vir
225 Currently, there are approximately 150 known human herpesvirus-encoded miRNAs, although an miRNA(s) e
229 sized that HHV-6A, like other members of the human herpesvirus family, encodes miRNAs, which function
230 l mechanism by which HHV-6A, a member of the human herpesvirus family, may contribute to inadequate m
231 g 3 HSV-1 genomes, one HSV-2 genome, 8 other human herpesvirus genomes, and 40 non-human herpesvirus
236 nd that certain infectious pathogens such as human herpesvirus (HHV) 6, HHV-7, and adenovirus, which
237 immunological tools available to detect both human herpesvirus (HHV) and immune control of replicatio
238 d men have at least one actively replicating human herpesvirus (HHV) in their mucosal secretions at a
240 Both human immunodeficiency virus (HIV) and human herpesvirus (HHV) infections persist lifelong, and
242 ced hypersensitivity syndrome (DIHS), latent human herpesvirus (HHV)-6 is frequently reactivated in a
243 identification of suitable animal models for human herpesvirus (HHV)-6A and HHV-6B infections has bee
244 for a single virus: Epstein-Barr virus (23), human herpesvirus (HHV)-6B (10), human parainfluenza vir
246 nt studies have found evidence of occasional human herpesvirus (HHV)-8 transmission via blood transfu
251 were as follows: cytomegalovirus (CMV), 44%; human herpesvirus [HHV] 6, 18%; HHV8, 6%; Epstein-Barr v
254 , acts as a DNA chain terminator for several human herpesviruses (HHVs), including HHV-2 (HSV-2), a c
255 lly occurring genetic variants of persistent human herpesviruses imprints on the evolution of the ant
256 us (EBV), known as an oncovirus, is the only human herpesvirus in the genus Lymphocryptovirus (LCV).
257 ass A infections, the high seroprevalence of human herpesviruses in lr-MSM warrants further investiga
258 of this study was to analyse the presence of human herpesviruses in saliva and gingival crevicular fl
260 we found that HDAC inhibiting proteins from human herpesviruses induce human NKG2D ligand ULBP-1.
263 s sarcoma-associated herpesvirus (KSHV) is a human herpesvirus involved in the causation of several h
267 nding recruiting p53 to Zp.IMPORTANCE EBV, a human herpesvirus, is latently present in most nasophary
268 llular protein and has homologs in all other human herpesviruses, it has potential importance as a th
269 the data suggest that HHV-6 is unique among human herpesviruses: it specifically and efficiently int
270 Kaposi sarcoma (KS) to infection by a novel human herpesvirus (Kaposi sarcoma-associated herpesvirus
272 is to investigate the effect of any of eight human herpesviruses on development of dementia or mild c
274 yzed the incidence of infection of the eight human herpesviruses simultaneously in 1 045 peripheral b
276 Human cytomegalovirus (HCMV) is a ubiquitous human herpesvirus that can cause life-threatening diseas
278 -zoster virus (VZV) is a medically important human herpesvirus that causes chickenpox and shingles, b
279 Epstein-Barr virus (EBV) is an oncogenic human herpesvirus that dramatically reorganizes host gen
283 Epstein-Barr virus (EBV) is a ubiquitous human herpesvirus that is causally implicated in the dev
284 , the replication of herpes simplex virus, a human herpesvirus that is closely related to HCMV, was n
285 pstein-Barr virus, one of the most prevalent human herpesviruses that also causes cancer, we have dis
286 ibody response.IMPORTANCE HHV-6A and -6B are human herpesviruses that have the unique property of bei
287 Epstein-Barr virus (EBV) is one of nine human herpesviruses that persist latently to establish p
288 genomes, combined with the inability of most human herpesviruses to replicate in animals, has until r
290 esviruses (CMV, herpes simplex virus type 1, human herpesvirus type 6, and Epstein-Barr virus) with c
291 afamilial dynamics of endemic infection with human herpesvirus type 8 (HHV-8) in Amerindian populatio
294 that the hexon-capping SCP-the largest among human herpesviruses-uses its N-terminal half to bridge h
295 is substantially smaller than those of other human herpesviruses, VZV has a similarly sized capsid, c