ライフサイエンスコーパス: hyaline

1 brosis, ballooning degeneration, and Mallory hyaline).

2 ly wounded dogs did not form the superficial hyaline acellular lamina found in 92% of dogs with SCCED

3 A prominent superficial stromal hyaline acellular zone composed of collagen fibrils in t

4 is suboptimal due to the formation of mixed hyaline and fibrocartilage, resulting in inferior long-t

5 All engineered grafts contained a mixed hyaline and fibrous cartilage matrix.

6 This led to stiffer cartilage and decreased hyaline and increased calcified cartilage thickness.

7 flammation, ballooning degeneration, Mallory hyaline, and fibrosis.

8 fibrosis, ballooned hepatocytes, and Mallory hyaline, and two patients had cryptogenic cirrhosis thou

9 colemmal accumulations of myosin that have a hyaline appearance.

10 hypercellularity of the visceral epithelium, hyaline arteriolosclerosis, and interstitial fibrosis we

11 he structure, ultrastructure and function of hyaline articular cartilage (HAC) and subchondral bone (

12 ated by differential centrifugation of adult hyaline articular cartilage collagenase digests, mineral

13 ge matrix synthesis in vitro and regenerates hyaline articular cartilage in preclinical OA and cartil

14 Calcium crystals are universally present in hyaline articular cartilage, as well as the meniscus of

15 ; the highest specific activity was found in hyaline articular cartilage, the lowest in brain.

16 eleased from old and young human and porcine hyaline articular cartilage.

17 teral corner syndrome, and in evaluating the hyaline articular cartilage.

18 nd that they were phenotypically unique from hyaline articular chondrocytes isolated from the knee jo

19 n with focal cortical dysplasia type 2a with hyaline astrocytic inclusions, another rare and possibly

20 s), hippocampal sclerosis, lacunar infarcts, hyaline atherosclerosis, siderocalcinosis, and Lewy body

21 uch as Mallory-Denk bodies, intracytoplasmic hyaline bodies, alpha(1)-antitrypsin aggregates, as well

22 osis with or without the presence of Mallory hyaline bodies.

23 using hereditary inclusion body myopathy and hyaline body myopathy and the identification of mutation

24 Both hyaline cartilage and fibrocartilage can calcify in a va

25 -bearing unossified epiphyseal and articular hyaline cartilage and for the distal femoral and proxima

26 the long alpha1(IX) transcript expressed in hyaline cartilage and matched the predicted sequence of

27 issues found in a nasal septal biopsy, i.e., hyaline cartilage and perichondrium, for a novel tissue

28 Three-dimensional hyaline cartilage can be engineered using BMSCs from pat

29 ar (82 [25%] vs 21 [19%], P = .25); isolated hyaline cartilage defect (77 [23%] vs 20 [18%], P = .29)

30 a culture strategy was developed to engineer hyaline cartilage for engraftment into an acutely damage

31 key signaling events required for postnatal hyaline cartilage formation.

32 This Review discusses normal hyaline cartilage function and lubrication and examines

33 logy revealed trabecular bones in two cases, hyaline cartilage in another case and a focus of mineral

34 T2 spatial variation of patellar hyaline cartilage in children is similar to that of pate

35 , pigmentation was widespread throughout the hyaline cartilage in either granular composition or as b

36 The graft consisted of hyaline cartilage in four, mixed fibrohyaline cartilage

37 Regeneration of hyaline cartilage in human-sized joints remains a clinic

38 cartilaginous condylar cartilage, similar to hyaline cartilage in long bones, directly transform into

39 ints showed significantly improved repair of hyaline cartilage in osteochondral defects injected with

40 disc and cartilage is different from that of hyaline cartilage in other diarthrodial joints, and litt

41 dely expressed, but high levels are found in hyaline cartilage in the adult, bone tissue in newborn m

42 infused bioscaffolds were fully covered with hyaline cartilage in the articular surface.

43 MSC sheets' chondrogenic differentiation to hyaline cartilage in vitro via post-contraction cytoskel

44 The prevalence of hyaline cartilage lesions was particularly high (86% at

45 er at least 21 d, inducing the expression of hyaline cartilage matrix components and anabolic signali

46 maturation of articular chondrocytes in the hyaline cartilage of both developing and degenerated joi

47 ed to TMJ fibrocartilage and not seen in the hyaline cartilage of the knee.

48 intensity and in most cases attached to the hyaline cartilage of the lunate and scaphoid.

49 The trilaminar appearance depicted within hyaline cartilage on MR images obtained with this sequen

50 the hypertrophic differentiation of cultured hyaline cartilage particles.

51 tilage survived engraftment and maintained a hyaline cartilage phenotype, but did not form mature art

52 Cell-based therapy for articular hyaline cartilage regeneration predominantly involves th

53 over expression of type X collagen which, in hyaline cartilage structure is not characteristic of the

54 Transplanted hyaline cartilage survived engraftment and maintained a

55 robust method to generate 3D, scaffold-free, hyaline cartilage tissue constructs from hESCs that are

56 Given that chondrocytes in healthy hyaline cartilage typically display a spherical shape, b

57 Once hyaline cartilage was extensively pigmented, there was a

58 by lubrication is fundamental for decreasing hyaline cartilage wear and for maintaining the function

59 cruciate), elastic cartilage, meniscus, and hyaline cartilage were analyzed for NTTPHase activity (t

60 jority of vertebrate species have a layer of hyaline cartilage within the fibrous sclera giving an ex

61 -treated cells results in differentiation of hyaline cartilage, and single cell RNAseq analysis ident

62 al type II, IX, and XI collagen phenotype of hyaline cartilage, but the fibrils appear abnormally thi

63 otential and impaired capacity in generating hyaline cartilage, compared to cells isolated from young

64 vitro under hypoxia (2.5% O2) produced more hyaline cartilage, which expressed typical articular car

65 howed that both MC-Chs and NCC-Chs expressed hyaline cartilage-associated markers and were capable of

66 iated markers and were capable of generating hyaline cartilage-like tissue ectopically and at joint d

67 enting the low intrinsic repair potential of hyaline cartilage.

68 nt enabled the maintenance of functional and hyaline cartilage.

69 liably restore the structure and function of hyaline cartilage.

70 promote osteogenic progression of engrafted hyaline cartilage.

71 ble of alleviating necrosis at the centre of hyaline cartilage.

72 gical development of these distinct types of hyaline cartilage.

73 rogenesis into either permanent or transient hyaline cartilage.

74 has a similar collagen phenotype to that of hyaline cartilage.

75 scaffold and exercise treatment experienced hyaline-cartilage regeneration and completely healed car

76 creased subchondral bone formation, improved hyaline-cartilage structure, and good mechanical propert

77 tion of type II collagen is observed in both hyaline cartilages and is secondary to proteoglycan loss

78 a constitutively active human MMP-13 to the hyaline cartilages and joints of transgenic mice.

79 The collagen framework of hyaline cartilages, including articular cartilage, consi

80 kidney pathology, evidenced by proteinuria, hyaline casts in kidneys and increased renal mRNA expres

81 edge of the BP to ramify extensively in the hyaline cell area.

82 longitudinal network in the border cell and hyaline cell region.

83 e how colonization of Sphagnum angustifolium hyaline cells by Nostoc muscorum modifies S abundance an

84 like anomaly phenotype), and accumulation of hyaline cells in vitreous.

85 eria associations with Sphagnum's living and hyaline cells make this a rich area for future research,

86 At the scale of individual hyaline cells, colonized cells exhibited localized enric

87 types of SCs, homogene cells, border cells, hyaline cells, ganglion cells, and connective tissue cel

88 ic margin, while others were granular with a hyaline core.

89 by its more severe phenotype, which includes hyaline deposits in multiple organs, recurrent infection

90 Muscle fibers with hyaline desmin-containing cytoplasmic inclusions in comb

91 bind to all three major types of cartilage (hyaline, elastic, and fibrocartilage) and perform equall

92 Juvenile hyaline fibromatosis (JHF) and infantile systemic hyalin

93 Juvenile hyaline fibromatosis (JHF) is an autosomal recessive con

94 ns linked to the genetic disorders, juvenile hyaline fibromatosis, and infantile systemic hyalinosis.

95 The identification of hyaline fibrous tissue, with numerous crystalline basoph

96 Of the 80 dematiaceous and 154 hyaline fungi sequenced, 65 and 51.2%, respectively, gav

97 ophilic protein isolated from characteristic hyaline gastric lesions was identified as Ym2, a member

98 logically, there is widespread deposition of hyaline (glycoprotein) material and disruption/reduplica

99 Histologic analysis revealed invasive hyaline hyphae and some darkly pigmented structures that

100 ctures of the joints, and an accumulation of hyaline in the dermis.

101 hological diagnosis of neuronal intranuclear hyaline inclusion disease may also have polyglutamine re

102 Rosenthal fibers (RF), intra-astrocytic hyaline inclusions, accumulate in various pathological c

103 ibit distinct gene expression signatures and hyaline inclusions, supporting a novel genetic associati

104 rimentally wounded dogs is distinct from the hyaline lamina observed in dogs with SCCED.

105 brane involved the thickening of the central hyaline layer and a reduction in the epithelial cells on

106 s contributes to the ordered assembly of the hyaline layer and elevation of the fertilization envelop

107 y mAb183 to show that cell attachment to the hyaline layer is necessary for bottle cell formation and

108 The major constituent of the hyaline layer is the protein hyalin, a fibrillar glycopr

109 The hyaline layer of echinoderm embryos is an extraembryonic

110 or a fibronectin-like substrate, and for the hyaline layer.

111 the mechanisms of cell interactions with the hyaline layer.

112 n that is deposited to the inner side of the hyaline layer.

113 ote then becomes covered by a newly secreted hyaline layer.

114 moted the intrinsic regeneration of GAG-rich hyaline-like cartilage and improved tissue integration.

115 present study aims to develop transplantable hyaline-like cartilage constructs by stimulating MSC cho

116 et technology for fabricating scaffold-free, hyaline-like cartilage constructs from MSCs for future t

117 Hyaline-like cartilage matrix production was observed in

118 myofibrillar disarray and degeneration with hyaline-like inclusions.

119 be suitable contenders for the generation of hyaline-like repair tissue.

120 tivity in micromass culture, and generated a hyaline-like translucent cartilage particle in serum-fre

121 Ultrastructurally, the hyaline masses are composed of intermediate-density amor

122 The hyaline masses are intensely congophilic; react strongly

123 all vacuoles, and nemaline rods flanking the hyaline masses or congregated under the sarcolemma.

124 g in aggregation of the degraded residues in hyaline masses, and causes replication of Z disks, resul

125 titin, alpha-actinin, and slow myosin in the hyaline masses, signal nonlysosomal protein degradation.

126 e, irregularly polygonal, and mostly central hyaline masses, small vacuoles, and nemaline rods flanki

127 The hyaline material is positive with periodic acid-Schiff a

128 subpopulation of smaller cells that surround hyaline material.

129 f a residual mass, which revealed a shrunken hyaline matrix with preserved collagenous architecture.

130 HFOV had less pulmonary inflammation in the hyaline membrane disease (HMD) recovery phase.

131 use alveolar damage with pulmonary edema and hyaline membrane formation associated with accumulation

132 e clinical disease, diffuse alveolar damage, hyaline membrane formation, alveolitis, and death were n

133 iffuse alveolar disease manifested as edema, hyaline membrane formation, and interstitial cellular in

134 tory cells, interstitial and alveolar edema, hyaline membrane formation, and ultimately fibrosis.

135 use alveolar damage (DAD) with its prominent hyaline membrane formation, by mapping the 3D distributi

136 n pulmonary edema and acute lung injury with hyaline membrane formation, leading to decreased oxygena

137 aracterized by acute alveolar cell death and hyaline membrane formation, sustained up-regulation of h

138 characterized by diffuse alveolar damage and hyaline membrane formation.

139 The presence of hyaline membranes (HM) and airway fibrin (AF) was signif

140 turation, fibrosis, peripheral inflammation, hyaline membranes, and foamy alveolar macrophages, a phe

141 fuse alveolar damage (DAD), including edema, hyaline membranes, and proliferation of pneumocytes and

142 e in preterm infants, including atelectasis, hyaline membranes, and the lack of pulmonary surfactant

143 respiratory distress such as atelectasis and hyaline membranes.

144 aphs corresponded to intraalveolar edema and hyaline membranes.

145 results were categorized into those showing hyaline, mixed fibrohyaline cartilage, fibrocartilage, a

146 ies show ballooning degeneration and Mallory hyaline or fibrosis.

147 presence of hepatocyte ballooning, Mallory's hyaline, or fibrosis, which are important features in th

148 ation of extracellular matrix' attributed to hyaline rotaliid shells, or 'mineralization within intra

149 Microscopically, hyphae were hyaline, septate, and branched and remained totally devo

150 of biopsy tissue from lungs and brain showed hyaline, septate, branched hyphae with clamp connections

151 's methenamine silver stains showed numerous hyaline, septate, fungal hyphae of various lengths, many

152 The hyaline structures are strongly congophilic.

153 ghly electron-dense material, resembling the hyaline structures of myofibrillar myopathy.

154 arbouring amorphous, granular or pleomorphic hyaline structures, and vacuoles containing membranous m

155 ncluding lipid and proteinaceous substances (hyaline substances).

156 pathological signature with numerous round, hyaline, TAR DNA-binding protein 43 (TDP-43)-positive in

157 l fibrosis (CI), tubular atrophy, arteriolar hyaline thickening, fibrous intimal thickening (CV), and

158 hritis, highlighted by widespread glomerular hyaline thrombi, being common among NZM.Baff(-/-) mice b

159 The fundamental pathologic lesion is a hyaline thrombus composed of platelets and some fibrin a

160 foci in the bone marrow and soft tissues and hyaline tubular casts in the kidneys.

161 tric vs multicentric), histopathologic type (hyaline vascular [HV] vs plasma cell [PC]), anatomical l

162 Three histologic variants (hyaline vascular, plasma-cell, and mixed) and two clinic

163 her subdivided according to whether they had hyaline vascular, plasma-cell, or mixed disease.

164 Castleman disease (n = 24) typically had the hyaline-vascular type (n = 23), were asymptomatic (n = 1