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1 hidium produced from superoxide oxidation of hydroethidine.
2 lated rat skeletal muscle mitochondria using hydroethidine.
3 ons, as monitored with the fluorescent probe hydroethidine.
4  micro mol/L)-enhanced chemiluminescence and hydroethidine (2 micro mol/L)-based confocal microscopy,
5 eased in aorta (measured using lucigenin and hydroethidine) after LPS, and levels of superoxide were
6      By use of the oxidative fluorescent dye hydroethidine, an in situ assay indicated markedly incre
7 deomicroscopy in mesentery microvessels with hydroethidine, an oxidant-sensitive fluoroprobe, showed
8 cent products of the membrane-permeable dyes hydroethidine and 2',7'-dichlorofluorescin diacetate, re
9 were determined on arterial segments via the hydroethidine assay and on stimulated endothelial cell c
10                      Immunocytochemistry and hydroethidine-based detection of intracellular superoxid
11                             The oxidation of hydroethidine by superoxide forms the membrane-impermeab
12  to measure the rate of ROS generation using hydroethidine, dicarboxyfluorescein diacetate, or MitoSO
13  fluorescence after intravenous injection of hydroethidine due to superoxide radicals in photorecepto
14 to form superoxide, as detected by ESI-MS, a hydroethidine fluorescence assay, and EPR spin trapping.
15 sections were determined by dihydroethidium (hydroethidine) fluorescence staining.
16 teolysis), and dihydrotetramethylrosamine or hydroethidine (fluorescent upon oxidation).
17                                              Hydroethidine (HE) and hydropropidine ([Formula: see tex
18 the product formed from the reaction between hydroethidine (HE) and superoxide (O(2)(.-)).
19                    The putative oxidation of hydroethidine (HE) has become a widely used fluorescent
20 ells via fluorescence microscopy with either hydroethidine (HE) or its mitochondrially targeted deriv
21 show that MPO activity can be assessed using hydroethidine (HE), a probe commonly employed for the de
22 ct of reaction of superoxide (O(2)(*-)) with hydroethidine (HE), namely 2-hydroxyethidium (2-OH-E(+))
23 aded with the O(2)(.-)-sensitive fluorophore hydroethidine (HE).
24 the spin trap and by histofluorescence using hydroethidine (HE, 5 micromol/L) and dichlorodihydrofluo
25 rformed by using the oxidation-sensitive dye hydroethidine (HEt) to determine whether the relatively
26 imaging microfluorimetry of the oxidation of hydroethidine (HEt) to ethidium can be used to monitor s
27                                        Using hydroethidine (HEt), a fluorescent probe for superoxide,
28                    By using multiple probes (hydroethidine, hydropropidine, Amplex Red, and coumarin
29 s released as evidenced by the conversion of hydroethidine in the extracellular environment to ethidi
30 gen species (ROS) generation, assessed using hydroethidine, in motor neurons.
31 ; flow cytometric analysis with a vital dye (hydroethidine) indicated that 1.5 mM NO lysed the erythr
32 onship between MMP-9 expression and oxidized hydroethidine, indicating reactive oxygen species (ROS)
33 confirmed by fluorescence techniques, mainly hydroethidine oxidation and horseradish peroxidase-based
34 maging of superoxide radical distribution by hydroethidine oxidation in vulnerable neurons.
35 mitochondrial source of this ROS generation, hydroethidine oxidation was inhibited by the mitochondri
36 as investigated by measuring the products of hydroethidine oxidation.
37 rogenic probes, as follows: superoxide using hydroethidine, peroxynitrite using boronate-based probes
38 y specific oxidation of a fluorescent probe, hydroethidine, reflecting decreased activity of Mn-SOD.
39 r studies using the oxidation-sensitive dye, hydroethidine, revealed Zn2+-dependent reactive oxygen s
40 lular production of O(2)(.-), as measured by hydroethidine staining, and inhibited cell growth.
41    Superoxide anion formation was assayed by hydroethidine staining.
42 mployed the superoxide-mediated oxidation of hydroethidine to ethidium to dynamically and directly as
43 wells were treated with triphenylphosphonium hydroethidine (TPP-HE), which forms the superoxide speci
44                                              Hydroethidine was used to identify superoxide radicals a
45 nd signals from the mitochondrially targeted hydroethidine, was increased in neurons with both the co
46 levels, markers of one-electron oxidation of hydroethidine, were observed at cytotoxic concentrations
47 xide formation was detected by reaction with hydroethidine within the first hour following activation