ライフサイエンスコーパス: hydrogen sulfide

1 smitters (carbon monoxide, nitric oxide, and hydrogen sulfide).

2 model in vitro in the absence or presence of hydrogen sulfide.

3 isolates also lacked the capacity to produce hydrogen sulfide.

4 e of the enzyme, without the intervention of hydrogen sulfide.

5 on monoxide and, as described more recently, hydrogen sulfide.

6 e (FEPO) for use in in vivo visualization of hydrogen sulfide.

7 nsoluble and unreactive until it reacts with hydrogen sulfide.

8 ation of l-cysteine from O-acetyl serine and hydrogen sulfide.

9 gh vasodilation mediated by nitric oxide and hydrogen sulfide.

10 injury that was prevented in the presence of hydrogen sulfide.

11 he gas phase were 8.7 x 10(-4) mug/L air for hydrogen sulfide, 1 x 10(-4) mug/L air for methyl mercap

12 concrete in sewers is known to be caused by hydrogen sulfide, although the role of wastewater in reg

13 These results suggest that hydrogen sulfide ameliorates sFlt1-induced hypertension,

14 te, ascorbic acid, acetaminophen, uric acid, hydrogen sulfide, ammonium, ammonia, and both protonated

15 Hydrogen sulfide, an endogenous signaling molecule, play

16 ganic matter, Ti with hydroxide, and Ag with hydrogen sulfide and ammonia; less than 1% of dissolved

17 st that for some bacterial metabolites, like hydrogen sulfide and butyrate, the extent of their oxida

18 Fe(III/II)-IL/DMF can remove hydrogen sulfide and can be regenerated through an elect

19 are based on the long-standing paradigm that hydrogen sulfide and chloride are the ligands responsibl

20 For these reasons, increases in hydrogen sulfide and hydrogen sulfide-producing enzymes

21 moisture in air lead to the release of toxic hydrogen sulfide and materials degradation, hindering la

22 Hydrogen sulfide and methyl mercaptan were derivatized u

23 mated analysis based on the reaction between hydrogen sulfide and N,N-dimethyl-p-phenylenediamine in

24 cted the ability of the wine to release back hydrogen sulfide and other mercaptans during AR-aging.

25 redox signalling, for instance nitric oxide, hydrogen sulfide and oxidized lipids.

26 product generated by enzymatic oxidation of hydrogen sulfide and polysulfides.

27 Among the VSCs, hydrogen sulfide and sulfur dioxide are considered most

28 be protective by intermediate conversion to hydrogen sulfide and thiosulfate.

29 rtant substances, including oxygen, nitrate, hydrogen sulfide, and ammonium, across the entire consid

30 ng and inhibit gas extraction, produce toxic hydrogen sulfide, and induce corrosion leading to downho

31 dehyde, acetone, acetic acid, hexanoic acid, hydrogen sulfide, and methanol were increased in the can

32 dehyde, acetone, acetic acid, hexanoic acid, hydrogen sulfide, and methanol) to discriminate cancer p

33 mitter H2S in its aqueous form (bisulfide or hydrogen sulfide anion) based on the alteration of Forst

34 studies suggest that the cellular effects of hydrogen sulfide are mediated in part by sulfane sulfur

35 tably carbon monoxide, cyanide, cyanate, and hydrogen sulfide, are potent inhibitors of Ni-containing

36 topril, bovine and human serum albumins, and hydrogen sulfide, are reported.

37 This study identifies hydrogen sulfide as a novel cellular mediator that can m

38 tudies are warranted to evaluate the role of hydrogen sulfide as a novel therapeutic agent for vascul

39 ned biosensor could detect concentrations of hydrogen sulfide as low as 0.5 ppm.

40 of dissolved monomethylmercury (CH3Hg) with hydrogen sulfide as possible bacterial mediated or abiot

41 n steps are driven by ultraviolet light, use hydrogen sulfide as the reductant and can be accelerated

42 ncreased sulfide concentrations by oxidizing hydrogen sulfide before its diffusion to oxygenated surf

43 aggerated ISR and greater loss of endogenous hydrogen sulfide but normal inhibition of mTORC1 and mai

44 ated by changes to nuclear factor-kappa B or hydrogen sulfide but that occurred together with a modul

45 Hydrogen sulfide can signal through 3 distinct mechanism

46 Headspace gas concentrations (hydrogen sulfide, carbon dioxide, and methane), pore wat

47 prising methane, ethane, other hydrocarbons, hydrogen sulfide, carbon dioxide, nitrogen, and water.

48 Gaseous molecules including nitric oxide, hydrogen sulfide, carbon monoxide and oxygen mediate num

49 et al. (2015) show that the gasotransmitter hydrogen sulfide co-operates with growth factor TGF-beta

50 cell concentration, immobilization bed type, hydrogen sulfide concentration, and geometrical shape of

51 In the harmful organic-rich sediment, hydrogen sulfide concentrations were low initially but i

52 Hydrogen sulfide-dependent effects were mainly observed

53 more pronouncedly responding SO2 is used for hydrogen sulfide detection.

54 articularly short-chain fatty acids, but not hydrogen sulfide, directly modulate circadian clock gene

55 Treatment with a hydrogen sulfide donor, diallyl trisulfide, prevented th

56 ent with AP39, a novel mitochondria-targeted hydrogen sulfide donor, prevented ROS production, reduce

57 onstrate that AP39, a mitochondria-targeting hydrogen sulfide donor, significantly increases cardiomy

58 ts of hydrogen sulfide, we proposed that the hydrogen sulfide donor, sodium sulfide (Na2S), would att

59 xpressing animals intraperitoneally with the hydrogen sulfide-donor sodium hydrosulfide (NaHS) (50 mi

60 This vascular remodeling is reversible, and hydrogen sulfide donors are likely to improve pregnancy

61 This implies that hydrogen sulfide donors may be therapeutic.

62 Maintaining physiological levels of hydrogen sulfide during ischemia is necessary to limit i

63 orm for the in situ determination of gaseous hydrogen sulfide, employing a 470 nm light emitting diod

64 factor representation of the system based on hydrogen sulfide, Fe(II) and Fe(III).

65 Subsequently, the hydrogen sulfide formed by bacterial decomposition of co

66 visits from January 2008 to April 2009, and hydrogen sulfide ([Formula: see text]) presence in drink

67 dosymbionts of these progenitors made use of hydrogen sulfide from biogenic sources (e.g., decaying w

68 ponse is not known, we find that addition of hydrogen sulfide gas to growing cells recapitulates all

69 lanogaster and mice from lethal exposures of hydrogen sulfide gas.

70 neutrophil infiltration, cysteamine, mucin, hydrogen sulfide, ghrelin, adiponectin and the influence

71 yceophosphocholine, cystathionine, cysteine, hydrogen sulfide, glutathione disulfide, and glutathione

72 was most severe in sites with high levels of hydrogen sulfide (>100 ppm) and carbon dioxide (>1%) gas

73 Endogenous hydrogen sulfide (H(2) S) has anti-inflammatory and vaso

74 Hydrogen sulfide (H(2) S) is a gaseous molecule that has

75 Hydrogen sulfide (H(2) S) is an important signaling mole

76 antibiotic resistance, and it also produces hydrogen sulfide (H(2) S) that provides some defense aga

77 m-temperature responses to ammonia (NH(3) ), hydrogen sulfide (H(2) S), and humidity, respectively.

78 CBS is the predominant hydrogen sulfide (H(2) S)-producing enzyme in endothelia

79 ulfur species related to the gasotransmitter hydrogen sulfide (H(2) S).

80 oth hydrogenotrophic SO(4) (2-) reduction to hydrogen sulfide (H(2)S) and carbon dioxide (CO(2)) redu

81 Dysregulation of hydrogen sulfide (H(2)S) by inhibition of cystathionine

82 We also examined whether hydrogen sulfide (H(2)S) could dehomocysteinylate eNOS t

83 types of ancient bacteria and archea rely on hydrogen sulfide (H(2)S) for their energy production, eu

84 The ubiquitous gasotransmitter hydrogen sulfide (H(2)S) has been recognized to play a c

85 Hydrogen sulfide (H(2)S) has emerged as an important sig

86 Hydrogen sulfide (H(2)S) is a biologically relevant mole

87 Hydrogen sulfide (H(2)S) is a gaseous signaling molecule

88 Hydrogen sulfide (H(2)S) is an important signaling molec

89 Hydrogen sulfide (H(2)S) is dichotomous in nature as it

90 Hydrogen sulfide (H(2)S) is emerging as an important pla

91 Hydrogen sulfide (H(2)S) is involved in numerous pathoph

92 Hydrogen sulfide (H(2)S) is known to play a physiologica

93 The biological mediator hydrogen sulfide (H(2)S) is produced by bacteria and has

94 Hydrogen sulfide (H(2)S) participates in prokaryotic met

95 Hydrogen sulfide (H(2)S) production in the intestinal mi

96 ablished that exposure of mammalian cells to hydrogen sulfide (H(2)S) suppresses mitochondrial functi

97 is the pressure-driven disproportionation of hydrogen sulfide (H(2)S) to H(3)S, with a confirmed tran

98 This study examined the role of hydrogen sulfide (H(2)S), a novel gasotransmitter, in th

99 Hydrogen sulfide (H(2)S), a signaling molecule implicate

100 CO) emissions (~60-70%), and dominate NH(3), hydrogen sulfide (H(2)S), and volatile organic compounds

101 ert dietary sources of sulfur into genotoxic hydrogen sulfide (H(2)S), have been associated with deve

102 rotein increases gut bacterial production of hydrogen sulfide (H(2)S), indole, and indoxyl sulfate.

103 erlying convergent evolution of tolerance to hydrogen sulfide (H(2)S)-a toxicant that impairs mitocho

104 fides (RSSH), presumed signaling products of hydrogen sulfide (H(2)S)-mediated thiol (RSH) modificati

105 lyase (CSE; also cystathionase), a principal hydrogen sulfide (H(2)S)-synthesizing enzyme in the live

106 proposed to mediate the signaling effects of hydrogen sulfide (H(2)S).

107 at C77 due to the induction of intracellular hydrogen sulfide (H(2)S).

108 nt overproduction of the gaseous transmitter hydrogen sulfide (H(2)S).

109 e endogenously generate the gaseous molecule hydrogen sulfide (H(2)S).

110 xide (CO(2)), water vapor (H(2)O vapor), and hydrogen sulfide (H(2)S).

111 (4) Hydrogen sulfide (H(2)S/HS(-)) inhibits CO oxidation but

112 Hydrogen sulfide (H2 S) and hydrogen polysulfides (H2 Sn

113 Hydrogen sulfide (H2 S) exhibits promising protective ef

114 Hydrogen sulfide (H2 S) has emerged as a crucial biomole

115 Although hydrogen sulfide (H2 S) is perhaps best known as a toxic

116 Here we propose that hydrogen sulfide (H2 S), as a glial-released inflammator

117 The endogenously produced gasotransmitter, hydrogen sulfide (H2 S), is a novel molecule that mitiga

118 a the reaction of the gaseous biotransmitter hydrogen sulfide (H2S) and disulfides (RSSR) and/or sulf

119 evidence suggests that the gaseous molecules hydrogen sulfide (H2S) and nitric oxide (NO) enhances pl

120 thionine-beta-synthase (CBS) and its product hydrogen sulfide (H2S) are aberrantly upregulated in col

121 Gaseous signaling molecules such as hydrogen sulfide (H2S) are produced endogenously and med

122 We identified the gaseous molecule hydrogen sulfide (H2S) as the major effector molecule dr

123 to a significant down-regulation of cellular hydrogen sulfide (H2S) by 50% and of glutathione (GSH) b

124 acrophage inducible NOS was inhibited by the hydrogen sulfide (H2S) donor Na2S with IC50 values of ap

125 Hydrogen sulfide (H2S) elicits pleiotropic physiological

126 Hydrogen sulfide (H2S) emissions were determined from an

127 Hydrogen sulfide (H2S) exhibits beneficial effects in th

128 a new biological process for the removal of hydrogen sulfide (H2S) from low pressure biogas, the nee

129 A new sensor for the detection of hydrogen sulfide (H2S) gas has been developed to replace

130 provide strong evidence for the role of the hydrogen sulfide (H2S) gas in the protective effects of

131 Hydrogen sulfide (H2S) has been increasingly recognized

132 Hydrogen sulfide (H2S) has been recognized as a signalli

133 Hydrogen sulfide (H2S) has emerged as the most important

134 Hydrogen sulfide (H2S) has long been known as a toxic ga

135 rapeutic manipulation of the gasotransmitter hydrogen sulfide (H2S) has recently been proposed as a n

136 Hydrogen sulfide (H2S) has recently been recognized as a

137 Hydrogen sulfide (H2S) impairs mitochondrial respiration

138 (CSE) is one of the major enzymes producing hydrogen sulfide (H2S) in lungs, participating in the re

139 We sought to investigate the role of hydrogen sulfide (H2S) in regulating vasomotor tone in t

140 Cystathionine gamma-lyase (CSE) produces hydrogen sulfide (H2S) in the cardiovascular system.

141 is study was designed to examine the role of hydrogen sulfide (H2S) in the generation of oxidized low

142 The physiological functions of hydrogen sulfide (H2S) include vasorelaxation, stimulati

143 Because hydrogen sulfide (H2S) inhibits high glucose-induced mat

144 We have previously shown that hydrogen sulfide (H2S) inhibits high glucose-induced pro

145 Hydrogen sulfide (H2S) is a biologically important small

146 Hydrogen sulfide (H2S) is a critical gaseous signaling m

147 Hydrogen sulfide (H2S) is a gaseous mediator that promot

148 Hydrogen sulfide (H2S) is a hazardous gas, which not onl

149 Hydrogen sulfide (H2S) is a reactive small molecule gene

150 Hydrogen sulfide (H2S) is a signaling molecule that is t

151 Hydrogen sulfide (H2S) is an attractive agent for myocar

152 Hydrogen sulfide (H2S) is an endogenous gaseous mediator

153 Hydrogen sulfide (H2S) is an endogenous gasotransmitter

154 Hydrogen sulfide (H2S) is an endogenously produced gaseo

155 Hydrogen sulfide (H2S) is an essential biological signal

156 Hydrogen sulfide (H2S) is an important biological mediat

157 Hydrogen sulfide (H2S) is an important biological signal

158 Hydrogen sulfide (H2S) is an important biological signal

159 The gasotransmitter hydrogen sulfide (H2S) is an important tuner of the card

160 Hydrogen sulfide (H2S) is an integral signaling molecule

161 Hydrogen sulfide (H2S) is increasingly recognized to mod

162 Hydrogen sulfide (H2S) is now recognized as a physiologi

163 Hydrogen sulfide (H2S) is now recognized as an important

164 Hydrogen sulfide (H2S) is produced by yeast during winem

165 Hydrogen sulfide (H2S) is produced endogenously in vivo

166 cent experimental studies have revealed that hydrogen sulfide (H2S) is produced enzymatically in all

167 The gasotransmitter, hydrogen sulfide (H2S) is recognized as an important med

168 Hydrogen sulfide (H2S) is the most abundant gas-phase sp

169 Hydrogen sulfide (H2S) is the most recently accepted end

170 Hydrogen sulfide (H2S) is the third biological gasotrans

171 Hydrogen sulfide (H2S) plays important roles in the ente

172 enocarcinoma tissue expresses high levels of hydrogen sulfide (H2S) producing enzymes, namely, cystat

173 ne gamma-lyase (CGL), resulting in increased hydrogen sulfide (H2S) production and protection from he

174 we tested the hypothesis that restoration of hydrogen sulfide (H2S) production in diabetic BMCs impro

175 Previous studies have demonstrated that hydrogen sulfide (H2S) protects against multiple cardiov

176 Hydrogen sulfide (H2S) regulates various physiological p

177 Endogenous hydrogen sulfide (H2S) renders bacteria highly resistant

178 O2 sensing by carbon monoxide (CO)-sensitive hydrogen sulfide (H2S) signaling contribute to reflex va

179 ed memory impairment in brain is mediated by hydrogen sulfide (H2S) synthesized by cystathionine beta

180 Carbon monoxide (CO) and hydrogen sulfide (H2S) used to be thought of simply as l

181 Hydrogen sulfide (H2S) was discovered as a third gasotra

182 Here we found that hydrogen sulfide (H2S) was required for Foxp3(+) Treg ce

183 ido-aza-BODIPY 3a selectively interacts with hydrogen sulfide (H2S) when compared to other molecules.

184 e hydrolisis of sodium thiophosphate (TP) to hydrogen sulfide (H2S) which in the presence Cd(2+) ions

185 Hydrogen sulfide (H2S), a cardioprotective gas, is endog

186 Hydrogen sulfide (H2S), a gaseous messenger produced mai

187 Hydrogen sulfide (H2S), a gaseous species produced by bo

188 Hydrogen sulfide (H2S), a novel gasotransmitter, is endo

189 Hydrogen sulfide (H2S), a small signaling gas molecule i

190 ns emit odorant chemicals including ammonia, hydrogen sulfide (H2S), and volatile organic compounds.

191 fur network generate the signaling molecule, hydrogen sulfide (H2S), from the amino acids cysteine an

192 chemical approach to the direct detection of hydrogen sulfide (H2S), in aqueous solutions, covering a

193 Precursors to hydrogen sulfide (H2S), methanethiol (MeSH), ethanethiol

194 with lower wine pH associated with decreased hydrogen sulfide (H2S), methanethiol, dimethyl sulfide,

195 The relationship between hydrogen sulfide (H2S), microRNAs (miRs), matrix metallo

196 fering gas species, including ammonia (NH3), hydrogen sulfide (H2S), nitric oxide (NO), carbon mono-o

197 rine acidification dynamics based on oxygen, hydrogen sulfide (H2S), pH, dissolved inorganic carbon a

198 l-containing compounds, including thiols and hydrogen sulfide (H2S), play important but differential

199 t signaling molecules, nitric oxide (NO) and hydrogen sulfide (H2S), proceeds via elusive mechanism(s

200 Additionally, we reveal that hydrogen sulfide (H2S), rather than sulfide, was likely

201 Buildup of hydrogen sulfide (H2S), which functions as a signaling m

202 a higher number of sulfane sulfur atoms than hydrogen sulfide (H2S), which has various physiological

203 ftenberg isolates, including 17 atypical non-hydrogen sulfide (H2S)-producing isolates, were detected

204 nitric oxide (NO), carbon monoxide (CO), and hydrogen sulfide (H2S).

205 the actual signaling molecules derived from hydrogen sulfide (H2S).

206 Hydrogen sulfide has been implicated in a large number o

207 We prepared microbubbles carrying hydrogen sulfide (hs-MB) with different H2S/C3F8 ratios

208 erapeutic effect and mechanisms of action of hydrogen sulfide in an animal model of sFlt1-induced hyp

209 l for tracking the variation of bisulfide or hydrogen sulfide in extracellular fluids.

210 e in our understanding of the role played by hydrogen sulfide in life processes.

211 c heme group and catalyzes the production of hydrogen sulfide in mammalian cells.

212 previous findings, demonstrates the role of hydrogen sulfide in regulating autophagy.

213 his work will detail the known mechanisms of hydrogen sulfide in the mitochondria and the implication

214 sess the direct anti-inflammatory effects of hydrogen sulfide in vivo, we used a peritonitis model by

215 Hydrogen sulfide is a cardioprotective signaling molecul

216 Hydrogen sulfide is a critical signaling molecule, but h

217 Hydrogen sulfide is a highly reactive molecule that is c

218 Hydrogen sulfide is a highly toxic gas-second only to ca

219 Hydrogen sulfide is a signaling molecule that regulates

220 Hydrogen sulfide is a vasorelaxant and proangiogenic gas

221 Hydrogen sulfide is an essential catabolite that interve

222 Hydrogen sulfide is an important biological signaling mo

223 Hydrogen sulfide is an important endogenous mediator tha

224 Hydrogen sulfide is detected by UV-assisted quantitative

225 Hydrogen sulfide is found in many environments including

226 c acid decarboxylase produces taurine, while hydrogen sulfide is recycled into cysteine by cystathion

227 -phase reaction of methylidyne radicals with hydrogen sulfide, leading to thioformaldehyde (H(2)CS) a

228 The subsequent generation of hydrogen sulfide led to the formation of iron-sulfur pre

229 ching between low steady-state intracellular hydrogen sulfide levels and the higher concentrations wh

230 Hydrogen sulfide maintains differentiated smooth muscle

231 e have examined the underlying mechanisms of hydrogen sulfide-mediated lung protection and determined

232 activated receptor gamma inhibitor abolished hydrogen sulfide-mediated protection in ventilated anima

233 Hydrogen sulfide-mediated protection was associated with

234 els of sulfur-containing compounds including hydrogen sulfide, mercaptans, and sulfur dioxide.

235 man (patho)physiology with a central role in hydrogen sulfide metabolism.

236 th anaerobic biostimulation processes (e.g., hydrogen sulfide, methane production) during full-scale

237 dehyde, acetone, acetic acid, hexanoic acid, hydrogen sulfide, methanol, and phenol-were found to be

238 A novel hydrogen sulfide microbial biosensor was developed based

239 In plants, hydrogen sulfide mitigates stress and regulates importan

240 Hydrogen sulfide monitoring has become essential in the

241 ith LEVC, the potency and efficacy of sodium hydrogen sulfide (NaHS) (10-1000 muM) on K(ATP) currents

242 re able to detect H(2)S produced from sodium hydrogen sulfide (NaHS) added at concentrations as low a

243 ing HHcy were treated without or with sodium hydrogen sulfide (NaHS), a H(2)S donor (30 uM), in drink

244 In Arabidopsis (Arabidopsis thaliana), hydrogen sulfide negatively regulates autophagy independ

245 between sulfur oxides, SOn (n = 1, 2, 3) and hydrogen sulfide (nH2S), resulting in the efficient form

246 s at mid-depths where acids are produced via hydrogen sulfide oxidation in waters mixed upward from a

247 Reactive sulfur species, such as hydrogen sulfide, persulfides, and polysulfides, have re

248 directly deliver a single species, including hydrogen sulfide, perthiol, and COS, and will be very us

249 ynthesis, TgCBS can also efficiently produce hydrogen sulfide, preferentially via condensation of cys

250 ic bottom-water conditions, often with toxic hydrogen sulfide present, were interrupted by brief oxyg

251 Hydrogen sulfide produced by sulfate-reducing microorgan

252 e reasons, increases in hydrogen sulfide and hydrogen sulfide-producing enzymes have been implicated

253 first summarize our current understanding of hydrogen sulfide production and metabolism, as well as i

254 ls independently of Atf4 expression, whereas hydrogen sulfide production is promoted via GCN2-ATF4 pa

255 sfunction reflected by a failure to maintain hydrogen sulfide production or apolipoprotein B100 (ApoB

256 other oral surfaces, and stool; hydrogen and hydrogen sulfide production were also differentially dis

257 hereas those encoding for methanogenesis and hydrogen sulfide production were higher in native Africa

258 duction of placental CSE activity, decreased hydrogen sulfide production, and smooth muscle cell dedi

259 ccumulation of CBS in mitochondria increased hydrogen sulfide production, which prevented Ca(2+)-medi

260 Below toxic levels, hydrogen sulfide promotes mitochondrial biogenesis and f

261 Hydrogen sulfide radicals in the ground state, SH(X), an

262 Hydrogen sulfide reduces ventilator-induced lung injury

263 from post-translational processes involving hydrogen sulfide-related chemistry.

264 e cystathionine gamma lyase (CSE), generates hydrogen sulfide-related sulfane sulfur compounds (H(2)S

265 f Fe(II) in Fe(III)-IL is beneficial for the hydrogen sulfide removal and the electrochemical regener

266 To improve the hydrogen sulfide removal efficiency with the application

267 Hydrogen sulfide removal is a long-standing economic and

268 bacterial gasotransmitters, nitric oxide and hydrogen sulfide represent.

269 ne to form cystathionine and either water or hydrogen sulfide, respectively.

270 s cysteine and sulfite into cysteic acid and hydrogen sulfide, respectively.

271 Buzzing with activity: A hydrogen sulfide selective fluorogenic probe, 7-azido-4-

272 Hydrogen sulfide signaling in mitochondria and disease.

273 al blood flow via oxidative stress, reducing hydrogen sulfide signaling.

274 ons and volatile organic compounds, ammonia, hydrogen sulfide, sulfur dioxide and hydrogen are then d

275 granulocyte infiltration negatively affects hydrogen sulfide synthesis.

276 two distinct methods of measuring leukocyte hydrogen sulfide synthesis; methylene blue formation fol

277 designed to examine roles of the endogenous hydrogen sulfide synthesizing enzyme cystathionine beta-

278 e that physiologic modification of parkin by hydrogen sulfide, termed sulfhydration, enhances its cat

279 en conducted on the physiological effects of hydrogen sulfide, the underlying mechanisms are poorly u

280 ght the biologic effects of nitric oxide and hydrogen sulfide, their seemingly indistinguishable effe

281 loits only prebiotically plausible molecules-hydrogen sulfide, thioacetate(12,14) and ferricyanide(12

282 in oil reservoirs produces toxic, corrosive hydrogen sulfide through microbial sulfate reduction, of

283 ced the relatively lower-affinity binding of hydrogen sulfide to IDO1, inspiring the use of the small

284 zable sulfur while mitigating the effects of hydrogen sulfide toxicity is poorly understood.

285 One hydrogen sulfide-triggered mechanism in the protection a

286 coupons were exposed to different levels of hydrogen sulfide under well-controlled conditions in lab

287 Prodrugs that release hydrogen sulfide upon esterase-mediated cleavage of an e

288 Hydrogen sulfide upregulates renal AQP-2 protein express

289 Hydrogen sulfide was found to be strongly inhibitory to

290 Because of the anti-inflammatory effects of hydrogen sulfide, we proposed that the hydrogen sulfide

291 uch as thioglycosides, thioacids, and sodium hydrogen sulfide were also alkynylated successfully to l

292 The effects of hydrogen sulfide were analyzed in a mouse ventilator-ind

293 rs (SMBs) nitric oxide, carbon monoxide, and hydrogen sulfide were initially viewed as environmental

294 d, likely due to the microbial production of hydrogen sulfide which favors stabilization of metals in

295 (MGL) catalyzes the decomposition of Hcy to hydrogen sulfide which forms fluorescent CdS nanoparticl

296 ulfate reducing microorganisms (SRM) produce hydrogen sulfide which is toxic, explosive, and corrosiv

297 cean, often accompanied by the production of hydrogen sulfide, which is toxic to higher organisms.

298 erial to parts-per-billion concentrations of hydrogen sulfide with potential applications in industri

299 gaseous molecules, such as nitric oxide and hydrogen sulfide, within the human body began a new conc

300 Such prodrugs directly release hydrogen sulfide without the involvement of perthiol spe