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1 detailed mechanisms of action of Act from A. hydrophila.
2 d by the closely related bacterium Aeromonas hydrophila.
3  DNA of a diarrheal isolate SSU of Aeromonas hydrophila.
4 lay an important role in the virulence of A. hydrophila.
5 and phage showed significant reduction of A. hydrophila.
6 ile Aeromonas septicemia caused by Aeromonas hydrophila.
7  activities of a diarrheal isolate SSU of A. hydrophila.
8 a lethal challenge dose of wild-type (WT) A. hydrophila.
9 cB) from a clinical isolate SSU of Aeromonas hydrophila.
10 fying a predicted effector TseC in Aeromonas hydrophila.
11  from a diarrheal isolate, SSU, of Aeromonas hydrophila.
12 o cholerae, Vibrio vulnificus, and Aeromonas hydrophila.
13 ch could alter the virulence potential of A. hydrophila.
14 e IIH R-M system from the pathogen Aeromonas hydrophila.
15  from the diarrheal isolate SSU of Aeromonas hydrophila.
16 ild-type (WT) and complemented strains of A. hydrophila.
17 nses to a cytotoxic enterotoxin of Aeromonas hydrophila.
18 ositive or -negative background strain of A. hydrophila.
19 ious virulence factors produced by Aeromonas hydrophila, a type II secretion system (T2SS)-secreted c
20 one at 10 microM in overnight cultures of A. hydrophila abolishes exoprotease production in azocasein
21 increase in Mugil cephalus against Aeromonas hydrophila, according to the current research.
22 lates the most-potent virulence factor of A. hydrophila, Act.
23  Gram negative bacteria, including Aeromonas hydrophila (AhlABC).
24 ere, we demonstrate that one such, Aeromonas hydrophila AhQnr, is soluble, stable, and relieves quino
25 nsis SSU and environmental isolate Aeromonas hydrophila American Type Culture Collection_7966T.
26                            These data add A. hydrophila and A. salmonicida to the growing family of g
27 ent culture supernatants from both Aeromonas hydrophila and Aeromonas salmonicida activate a range of
28       Act is a potent virulence factor of A. hydrophila and has been shown to contribute significantl
29 secreted by the bacterial pathogen Aeromonas hydrophila and is capable of killing target cells by for
30 ibrio cholerae, Vibrio vulnificus, Aeromonas hydrophila and other Gram-negative bacteria.
31         Fish and chicken meat spiked with A. hydrophila and phage showed significant reduction of A.
32 ulence of diarrheal isolate SSU of Aeromonas hydrophila and showed that VasH, a sigma(54) activator a
33 a correlation between the TTSS and Act of A. hydrophila and the production of lactones.
34 the environmental isolate ATCC 7966(T) of A. hydrophila and the vacB gene of Shigella flexneri.
35  pathogens, Aeromonas salmonicida, Aeromonas hydrophila and Vibrio harveyi, to Atlantic salmon mucins
36 the T3SS from a diarrheal isolate, SSU of A. hydrophila, and defined the role of some regulatory gene
37 ted in 1971 from the fish pathogen Aeromonas hydrophila, and of the cryptic IncA/C plasmid pRAx (49,7
38 ion from Photorhabdus luminescens, Aeromonas hydrophila, and Vibrio parahaemolyticus are also sensiti
39  the TTSS translocon, from wild-type (WT) A. hydrophila as well as from a previously characterized cy
40 ic groups receiving an infusion of Aeromonas hydrophila at 0.2 mL/kg/hr, gradually increasing to 0.4
41             The complete genome of Aeromonas hydrophila ATCC 7966(T) was sequenced.
42 lase from diarrheal isolate SSU of Aeromonas hydrophila bound to human plasminogen and facilitated th
43 as then introduced into the chromosome of A. hydrophila by using the suicide vector pJQ200SK, allowin
44 hat only the full-length ACD of RtxA from A. hydrophila catalyzed the covalent cross-linking of the h
45  hand, the WT and complemented strains of A. hydrophila caused 80 to 90% of the mice to succumb to in
46      Inactivation of the ahyI gene on the A. hydrophila chromosome abolishes C4-HSL production.
47  2.6-kb SalI/HindIII DNA fragment from an A. hydrophila chromosome was cloned and sequenced.
48                     Members of the Aeromonas hydrophila complex (A. hydrophila, HG2, and A. salmonici
49               The exoprotease activity of A. hydrophila consists of both serine protease and metallop
50 environmental isolate ATCC 7966 of Aeromonas hydrophila consists of six genes (rtxACHBDE) organized i
51 d 100% of the animals inoculated with the A. hydrophila control strain.
52  regulation in the fur isogenic mutant of A. hydrophila could be restored by complementation.
53 se in gidA and act gene expression in the A. hydrophila Dam-overproducing strain, and these data matc
54        Three enterotoxins from the Aeromonas hydrophila diarrheal isolate SSU have been molecularly c
55 n, we showed that animals challenged with A. hydrophila die because of kidney and liver damage, hypog
56 ps), Pseudomonas aeruginosa (xcp), Aeromonas hydrophila (exe), and Vibrio cholerae (eps).
57 splayed 87% sequence similarity to Aeromonas hydrophila ExeE, a member of the PulE (GspE) family of p
58                          The tagA gene of A. hydrophila exhibited 60% identity with that of a recentl
59           In contrast, the wild-type (WT) A. hydrophila exhibited significant growth at this low temp
60 ction with ciprofloxacin-resistant Aeromonas hydrophila following leech therapy.
61 ntation experiments demonstrated that the A. hydrophila fur gene could restore iron regulation in an
62 is more similar to the X. campestris than A. hydrophila genes.
63                                 Thus, the A. hydrophila genome sequence provides valuable insights in
64                                        An A. hydrophila genomic library was transferred into a P. aer
65 bers of the Aeromonas hydrophila complex (A. hydrophila, HG2, and A. salmonicida), a group that has p
66 le aspects of the metabolic repertoire of A. hydrophila include dissimilatory sulfate reduction and r
67 ablished a role for three enterotoxins in A. hydrophila-induced gastroenteritis in a mouse model with
68 tropenic animals were more susceptible to A. hydrophila infection than normal mice.
69 vive of the Mugil cephalus against Aeromonas hydrophila infection.
70 e and enhances their survivability during A. hydrophila infection.
71  mice with the above AHLs prior to lethal A. hydrophila infection.
72 predominant immune cells inflicted during A. hydrophila infections, such as murine macrophages, when
73 (n = 5), which received continuous Aeromonas hydrophila infusion plus antiprostacyclin antibody infus
74  from a diarrheal isolate, SSU, of Aeromonas hydrophila is aerolysin related and crucial to the patho
75                                    Aeromonas hydrophila is both a human and animal pathogen, and the
76 sport in the fresh water bacterium Aeromonas hydrophila is found to occur by means of an indiscrimina
77 ion of the S-layer on the cell surface in A. hydrophila is more similar to the X. campestris than A.
78 , 1 Serratia marcescens isolate, 1 Aeromonas hydrophila isolate, 1 Aeromonas veronii isolate, 2 Chrys
79                           In contrast, WT A. hydrophila killed 100% of the mice within 48 h.
80 ansit through turtles colonized by Aeromonas hydrophila, leading to the hypothesis that SdiA is used
81                                    Aeromonas hydrophila leads to both intestinal and extraintestinal
82 f the effector domains of V. cholerae and A. hydrophila MARTX toxins to elucidate the mechanism of th
83       The biological activity of selected A. hydrophila mutants was restored after complementation.
84 ure supernatants from deletion mutants of A. hydrophila, namely, a Delta act mutant (a T2SS-associate
85 f T6SS and ExoA in pathogenesis caused by A. hydrophila NF strains in both mouse peritonitis and NF m
86 ndividual with multiple strains of Aeromonas hydrophila (NF1-NF4), the latter three constituted a clo
87  rosenbergii nodovirus), bacteria (Aeromonas hydrophila or Vibrio harveyi) or heavy metals (cadmium o
88 ages (44RR2.8t, 25 and 31) and one Aeromonas hydrophila phage (Aeh1).
89                                       The A. hydrophila pilD homologue, tapD, was identified by its a
90 nstrated for the first time that VgrG1 of A. hydrophila possessed actin ADPRT activity associated wit
91     Our study is the first to report that A. hydrophila possesses a functional RtxA having an ACD tha
92 ted cytotoxic enterotoxin (Act) of Aeromonas hydrophila possesses multiple biological activities, whi
93   A cytotoxic enterotoxin (Act) of Aeromonas hydrophila possesses several biological activities, and
94   A cytotoxic enterotoxin (Act) of Aeromonas hydrophila possesses several biological activities, indu
95 totoxic, actin-targeting mART from Aeromonas hydrophila PPD134/91.
96 activity against both vAh strains and the A. hydrophila reference strain ATCC 35654.
97 e pathogens, including a strain of Aeromonas hydrophila resistant to amikacin, tobramycin, and multip
98          Overproduction of mutated Dam in A. hydrophila resulted in bacterial motility, hemolytic and
99                                       The A. hydrophila RNase R-lacking strain was found to be less v
100 er, we showed that the full-length ACD of A. hydrophila RtxA disrupted the actin cytoskeleton of HeLa
101                                    Aeromonas hydrophila secretes several extracellular proteins that
102 the DNA adenine methyltransferase gene of A. hydrophila SSU (dam(AhSSU)) in a T7 promoter-based vecto
103 ntially be important for the viability of A. hydrophila SSU as we could delete the chromosomal copy o
104 cid residues ((252)FYDAEKKEY(260)) in the A. hydrophila SSU enolase involved in plasminogen binding.
105 -expressed enolase in the pathogenesis of A. hydrophila SSU infections and of any gram-negative bacte
106 shed by 55% compared to that of a control A. hydrophila SSU strain harboring the pBAD vector alone.
107 ctively, compared to those of the control A. hydrophila SSU strain.
108 gene to be essential for the viability of A. hydrophila SSU, and, therefore, to better understand the
109 e bacterium, and overproduction of Dam in A. hydrophila SSU, using an arabinose-inducible, P(BAD) pro
110 e the expression of act and gidA genes in A. hydrophila SSU.
111 e act/aopB mutant, compared to that of WT A. hydrophila SSU.
112 erated a fur isogenic mutant of wild-type A. hydrophila SSU.
113 343, 394, 420, 427, and 430 of enolase in A. hydrophila SSU; the mutated forms of enolase were hypere
114 nd lateral flagellin proteins from Aeromonas hydrophila strain AH-3 (serotype O34) were found to be g
115 ) caused by mixed infection with 4 Aeromonas hydrophila strains (NF1-NF4).
116 ral flagellum, that are reported in other A. hydrophila strains are not identified in the sequenced i
117  was used, whereby either single or mixed A. hydrophila strains were injected intramuscularly.
118                               We purified A. hydrophila TagA as a histidine-tagged fusion protein (rT
119 il gene cluster that resembles the Aeromonas hydrophila tap gene cluster and other type IV-A pilus as
120 Pseudomonas aeruginosa (PilA), and Aeromonas hydrophila (TapA).
121 ns of a diarrheal isolate, SSU, of Aeromonas hydrophila that exhibited a 50 to 53% reduction in the h
122  was noted in the gidA isogenic mutant of A. hydrophila that was generated by marker exchange mutagen
123                                 In Aeromonas hydrophila, the ahyI gene encodes a protein responsible
124  against Edwardsiella ictaluri and Aeromonas hydrophila, the causative agents of enteric septicemia o
125 d secretion compared to that of wild-type A. hydrophila; the triple-knockout mutant failed to induce
126 ic enterotoxin gene (act)-minus strain of A. hydrophila, thus generating aopB and act/aopB isogenic m
127 the cytotoxic enterotoxin (Act) of Aeromonas hydrophila to examine global cellular transcriptional re
128 EpsL and its homologue, ExeL, from Aeromonas hydrophila together with either EpsE or its Aeromonas ho
129               The tagA isogenic mutant of A. hydrophila, unlike its corresponding wild-type (WT) or t
130                                       The A. hydrophila VacB protein contained 798 amino acid residue
131 teriophage infecting hypervirulent Aeromonas hydrophila (vAh) was isolated and characterized.
132 ogether, our data indicated alteration of A. hydrophila virulence by overproduction of Dam.
133 f RNase R in modulating the expression of A. hydrophila virulence.
134 m was essential for attenuation of Aeromonas hydrophila virulence.
135                                 An Aeromonas hydrophila VolA homolog complements a V. cholerae VolA m
136                            The RNase R of A. hydrophila was a cold-shock protein and was required for
137 e cytotoxic enterotoxin (Act) from Aeromonas hydrophila was hyperexpressed with the pET, pTRX, and pG
138 ol group (n = 6), in which 1010/mL Aeromonas hydrophila was infused intravenously at 0.2 mL.kg-1.hr-1
139  well as from the clinical isolate SSU of A. hydrophila, was exclusively expressed and produced durin
140  viability of Escherichia coli and Aeromonas hydrophila were compared to spherical nanostructures (an

 
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